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2 t expression raises the possibility that the lack of effect of a missing alpha i2 on AC inhibition in
3 eased p21Cip1 expression, as demonstrated by lack of effect of AA-Akt on cell proliferation in p21-/-
5 BRET(2) analyses in intact cells indicate a lack of effect of agonist on the propensity of the hLHR
7 reactive intermediates, as evidenced by the lack of effects of antioxidants N-acetyl-L-cysteine and
8 by the N-terminal domain accounting for the lack of effect of apo(a) size polymorphism on the bindin
15 trinsic binding specificity and the complete lack of effect of disrupting individual RRM4 contact poi
18 leled iNOS protein and NO while indicating a lack of effect of either of the kinase inhibitors on TNF
19 he high rate of stunting at baseline and the lack of effect of either the meat or multiple micronutri
21 high density of mu-opioid receptors, but the lack of effects of estrogen on [35S]GTPgammaS binding, w
26 creases in 3H2-DOG uptake occurred despite a lack of effect of IGF-I on GLUT1 protein levels (P > 0.5
27 ificity of this finding was confirmed by the lack of effect of IgPKD antibody on cell adhesion in a P
28 Anatomical specificity was confirmed by the lack of effect of inhibition of mGlu5 receptors (10-30 m
29 k voltage dependence of inactivation and the lack of effect of intracellular Ca2+ buffers, the Ca2+-b
33 d at understanding the mechanisms behind the lack of effect of MMN supplementation on morbidity measu
37 N-linked (Asn-Xaa-Ser/Thr) glycosylation, a lack of effect of N-glycosidase F, the presence of 70 an
40 cement by hypertonic solutions, the relative lack of effect of osmolarity on evoked release, and the
41 synthetic dipalmitoyl PI-5-P to PI-4,5-P(2), lack of effect of phosphatidic acid, inhibition by hepar
44 gradation of the protein as evidenced by the lack of effect of proteasomal and lysosomal inhibitors.
46 iscounted for a number of reasons, including lack of effect of radical traps, no apparent Si-Cl bond
49 he increase in SR Ca(2+) load by RR, and the lack of effects of RR on single RyR open time and conduc
55 p38 MAPK both increased AP-1 transcription, lack of effect of TGF-beta on lipopolysaccharide-stimula
56 ted endocytosis of leptin contrasts with the lack of effect of the caveolae inhibitors nystatin and f
57 stant for evoked and spontaneous IPSCs and a lack of effect of the GABA transport-inhibitor 1-[2([(di
62 nactivation of PKC was also indicated by the lack of effect of the tripeptides Tyr-Gly-Gly and Gly-Al
66 healthy status of this cohort may explain a lack of effect of vitamin K supplementation on cytokine
67 tic tissues to vitamin K antagonists and the lack of effects of vitamin K antagonists on the function
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