ライフサイエンスコーパス: lack the ability to

1 might be because the mature mammalian retina lacks the ability to accept and incorporate stem cells o

2 on using molecular dynamics simulations that lacked the ability to account for charge transfer and di

3 importantly, every theory (and every model) lacks the ability to account for some key autoimmune dis

4 Deltazrc1Deltacot1 lacks the ability to accumulate Zn in the vacuole and ha

5 Unlike CXCL12, these truncates either lack the ability to act as a chemoattractant for CD34(+)

6 beta-Catenin-deficient mice, which lack the ability to activate canonical Wnt signaling, ex

7 ike NeuroD, the c-Myc transactivation domain lacked the ability to activate insulin gene expression.

8 Importantly, this mutant lacks the ability to activate p53-dependent apoptotic ge

9 ther, a compound that binds to PPARgamma but lacks the ability to activate transcription, also inhibi

10 lymeric IgR knockout (pIgR(-/-)) mice, which lack the ability to actively secrete IgA into the mucosa

11 nt transport, such as cystic fibrosis, might lack the ability to adapt to the infection and present w

12 fmt) mutant strains of L. monocytogenes that lacked the ability to add formyl groups to nascent polyp

13 health-related impacts of climate change but lacking the ability to address these impacts.

14 The glnD null mutant completely lacked the ability to adenylylate GlnK.

15 se tia deletion strains were noninvasive and lacked the ability to adhere to human ileocecal cells.

16 cause these methods have a limited scale and lack the ability to afford particles of varied shapes.

17 pocket and surrounding hydrophobic ridge, it lacks the ability to agglutinate guinea pig erythrocytes

18 in K14E6(WT)), the mice expressing E6(I128T) lacked the ability to alter the radiation-induced block

19 r PCB 126, which is inactive toward Ry1R and lacks the ability to alter the Ca2+ leak pathway.

20 a mixed population of viruses containing or lacking the ability to antagonize BST-2.

21 ct into JH-deleted embryonic stem cells that lack the ability to assemble HC variable region genes an

22 f mu1, unable to release mu1N or form pores, lack the ability to associate with membranes.

23 variants with interface point substitutions lack the ability to be fully activated by Vps75, and one

24 LIP-1 was attenuated in VILIP-1 mutants that lacked the ability to be myristoylated or to bind calciu

25 al calcium binding domains as well as HA-CaM lacking the ability to be phosphorylated at residues Tyr

26 ns 2 and 3, and this poly(A)-binding protein lacks the ability to be regulated by oxidation or reduct

27 logous domain termed Zbeta, which appears to lack the ability to bind left-handed nucleic acids.

28 ce, the three follistatin domains inherently lack the ability to bind or neutralize activin.

29 18BPd isoforms lack a complete Ig domain and lack the ability to bind or neutralize IL-18.

30 tion kinetics via binding to Na(V)1.x site 3 lack the ability to bind phospholipids, whereas site 4 t

31 fied using genomic approaches alone, as they lack the ability to bind specific DNA sequences.

32 the propagation of targeted Ad vectors that lack the ability to bind the native fiber receptor.

33 ll-size antibodies, these antibody fragments lack the ability to bind the neonatal Fc receptor (FcRn)

34 by the cotransfection of mutants of E1A that lack the ability to bind to some or all members of the p

35 ifying viral envelope proteins, so that they lack the ability to bind to their cognate receptor but s

36 Expression of Ng mutants that lack the ability to bind to, or dissociate from, calmodu

37 Mutants of Bro1 that lack the ability to bind Ub were dramatically impaired i

38 pressed by primates; known rodent homologues lack the ability to bind virus due to amino acid differe

39 Similarly, the single wild-type isolate that lacked the ability to bind hemoglobin also appeared to l

40 GPIHBP1-W109S lacked the ability to bind LPL but had a reduced propens

41 A p21 mutant that lacked the ability to bind proliferating cell nuclear an

42 Recently we have shown that mutants of Kv4.2 lacking the ability to bind an intersubunit Zn(2+) betwe

43 7 with wild type CaM or CaM1,2,3,4, a mutant lacking the ability to bind Ca2+, rescued surface expres

44 ransgenes encoding mutant versions of CBP60a lacking the ability to bind calmodulin failed to complem

45 s adjuvanticity, we examined how a PA mutant lacking the ability to bind EF (PA-U7) or another mutant

46 ting moiety of P947 with a scrambled peptide lacking the ability to bind MMPs.

47 e expression of beta(1)Pix SH3m(W43K), which lacks the ability to bind Pak, and beta(1)PixDHm(L238R/L

48 ation knock-in mouse (EPCR(R84A/R84A)) which lacks the ability to bind PC/APC.

49 However, GABA(B(1e)) lacks the ability to bind the radiolabeled antagonist [(

50 The p53 mutant that lacks the ability to bind to mortalin remains at centros

51 w here that GFI136N, in contrast to GFI136S, lacks the ability to bind to the Gfi1 target gene that e

52 Mutant NKCC1 that lacks the ability to bind to this kinase showed K+ trans

53 However, they lack the ability to bond along specific directions as at

54 ine receptor mutation resulted in a receptor lacking the ability to catalyze the binding of guanosine

55 ave diminished CAAX proteolytic activity and lack the ability to cleave the amino terminus of the a-f

56 of a palmitoylation-deficient PSD-95 mutant lacking the ability to cluster and to interact with NMDA

57 tive medicine, but standard assays generally lack the ability to combine different cell stimulations

58 y lipoprotein receptor-deficient mice, which lack the ability to control lipid levels in the blood, i

59 at maintain tumor antigen responsiveness but lack the ability to control melanoma outgrowth.

60 Synthetic polymer approaches generally lack the ability to control the primary sequence, with s

61 se conventional approaches, although useful, lack the ability to control the spatial and temporal asp

62 growth inhibition was not observed in cells lacking the ability to correctly upregulate this protein

63 Finally, we also showed that HSV strains lacking the ability to counter autophagy and the PKR-dri

64 A splice variant of MyD88 (MyD88(S)), which lacks the ability to couple IRAK-1 to NF-kappaB, has bee

65 e when dealing with thousands of samples and lacks the ability to create scores that could be used as

66 Expression of the IEG form of Homer, which lacks the ability to cross-link, modulates mGluR-induced

67 hat, unlike typical circulating HIV strains, lack the ability to degrade IRF3.

68 Filaments of nhx1 nhx2 did not elongate and lacked the ability to dehisce and release pollen, result

69 er, individual solid-state plasmonic devices lack the ability to deliver multiple functionalities.

70 Mannose receptor knockout (MR(-/-)) mice lack the ability to detect trimannose.

71 struction methodologies for cDNA microarrays lack the ability to determine array integrity prior to h

72 niform sampling of the genome and inherently lack the ability to differentiate highly similar paralog

73 Gfi-null progenitors accumulate because they lack the ability to differentiate.

74 inding of NADP(+) but resulted in the enzyme lacking the ability to differentiate between the oxidize

75 ndocytosis motif in K5 resulted in a protein lacking the ability to direct an increased rate of MHC-I

76 transcription or translation, they generally lack the ability to directly sense cellular signals.

77 In addition, many studies lack the ability to distinguish between lifetime and rec

78 ic cleavage, while a guide-specific approach lacked the ability to distinguish between the wild-type

79 nal cellulosic aerogel processing approaches lack the ability to easily fabricate complete aerogel st

80 Furthermore, the Ku70(-/-) cells lack the ability to effectively rejoin signal and coding

81 The mammalian CNS lacks the ability to effectively compensate for injury b

82 re that primary fibroblasts from CS patients lack the ability to efficiently repair these particular

83 These mutants uniformly lacked the ability to either grow photoautotrophically o

84 vealed that Rce1-deficient embryos and cells lacked the ability to endoproteolytically process Ras pr

85 are isosteric to natural DNA bases but which lack the ability to engage in Watson-Crick (W-C) hydroge

86 ic mouse-human DII-FL MAb (E28) variant that lacks the ability to engage FcgammaR and C1q also did no

87 HuR-deficient PDAC cells lacked the ability to engraft successfully in immunocomp

88 ation and the C-terminal deletion completely lacked the ability to enhance phagocytosis.

89 Further analysis showed that Tok07 PB1 alone lacked the ability to enhance the pathogenicity of the r

90 Hdm2NLS lacks the ability to enter the nucleus, whereas Hdm2NES

91 n through activation of RIG-I signaling, but lacks the ability to evade the human IFN response.

92 ons between wild-type and deficient mice and lack the ability to examine reversal/inhibition of injur

93 was virtually nonexistent because all OPMRs lacked the ability to exchange data electronically with

94 Males appear to lack the ability to exhibit an LH surge, but it is uncle

95 16-Delta strains of Saccharomyces cerevisiae lacking the ability to export all RNAs, including poly(A

96 ed human aortic smooth muscle cells (HASMCs) lack the ability to express E-selectin.

97 strategy, a GPCMV mutant virus was used that lacked the ability to express an essential capsid gene (

98 l(+/+) CTLs because these remained immature, lacking the ability to express costimulatory molecules C

99 nother population of similar cells but which lacked the ability to fire phasically.

100 produce bone within the injected bone, they lack the ability to form mineralized bone nodules when e

101 l lines expressing the ANG-ALS variants also lack the ability to form stress granules.

102 l lines expressing the ANG-ALS variants also lack the ability to form stress granules.

103 CD133KD cells also lacked the ability to form CD144(+) VM-like channels in

104 on ancestor of tetrapods and ray-finned fish lacked the ability to form ectomesenchyme and its deriva

105 DM action, we prepared peptide variants that lacked the ability to form one or more of the hydrogen b

106 f 24,444 and an isoelectric point of 7.7 and lacked the ability to form the cystine loop structure ch

107 A Cys-to-serine mutant of wheat eIF4E, which lacked the ability to form the disulfide, crystallized w

108 tified that did not elicit cell rounding and lacked the ability to form typical plaques.

109 ntiospecificity was observed for 2-pyridones lacking the ability to form H-bonds.

110 3,5-benzenetricarboxyester (BTE) derivative, lacking the ability to form the H-bonding network, demon

111 , a SipW mutant protein was constructed that lacks the ability to form a solid-surface biofilm but st

112 an the others, divides at a slower rate, and lacks the ability to form an eye.

113 trical function and syntaxin 1A binding, but lacks the ability to form clusters, does not enhance gra

114 tion when 3-deazaguanine, a base analog that lacks the ability to form minor-groove hydrogen bonds wi

115 ic variant of SIAE was catalytically active, lacked the ability to function in a dominant negative ma

116 tibodies against conformational epitopes but lacked the ability to function in cell-cell fusion assay

117 This gRNA lacks the ability to function in trans.

118 Here, we show that CD73(-/-) mice, which lack the ability to generate extracellular adenosine, ar

119 Mice that lack the ability to generate NE or epinephrine show incr

120 An ackA mutant, lacking the ability to generate ATP from acetyl phosphat

121 nstrate that stem cell transplant recipients lacking the ability to generate or signal IL-17 develop

122 Prosthetic valves currently used in children lack the ability to grow with the patient and often requ

123 ltahtx strain was mutagenized and one mutant lacking the ability to grow on methylphosphonate as the

124 otype similar to that of a crp mutant, which lacks the ability to grow anaerobically with DMSO, fumar

125 h thymine replaced by difluorotoluene, which lacks the ability to hydrogen bond.

126 We demonstrate that human Tdp1 lacks the ability to hydrolyze a phosphodiester linked 5

127 plex departures from the null hypothesis but lack the ability to identify the specific alternative hy

128 Permeabilized cells of a dltD::erm mutant lacked the ability to incorporate D-alanine into LTA.

129 Mutants also lack the ability to increase mEJP rate in response to sp

130 Consequently, they lack the ability to increase the physiological salience

131 ability to organize axis formation, but they lack the ability to induce neuroectodermal tissues, a ch

132 that lack the NF-kappaB essential modulator lack the ability to induce the IFN genes following DNA d

133 Two antagonists that lacked the ability to induce any detectable CTL effector

134 A Fab fragment of the antibody, however, lacked the ability to induce cell death.

135 macrophages prepared from the same monocytes lacked the ability to induce IL-12 or IFN-gamma response

136 icroscopy demonstrated that the tssE mutants lacked the ability to induce multinucleated giant cell f

137 a cytokine-dependent murine T-cell line but lacked the ability to induce proliferation in response t

138 t the N-terminal-truncated CXCL12/SDF-1alpha lacks the ability to induce the migration of CD34(+) cor

139 A mutant Salmonella strain which lacks the ability to induce TNF-alpha was isolated from

140 r support for our hypothesis that drugs that lack the ability to inhibit transport by all three monoa

141 Axin mutants with C-terminal truncations lacked the ability to inhibit Lef-1 reporter activity, e

142 d 3) all TRPV1 mutations insensitive to ACEA lacked the ability to inhibit MO-evoked calcium accumula

143 EC strain that has a disruption in this gene lacks the ability to inhibit lymphokine production and l

144 ion of transport to the plasma membrane, and lacks the ability to inhibit the formation of CopII coat

145 he LIM domains is sufficient to bind DAT but lacks the ability to inhibit transporter activity.

146 They lack the ability to initiate pheromone-induced G1 cell c

147 Some model eukaryotic organisms lack the ability to insert selenocysteine, and prokaryot

148 The mutant proteins also lack the ability to interact with the large subunit of R

149 structure-based BCAR1 and BCAR3 mutants that lack the ability to interact, we show that BCAR3-induced

150 of Scs2p (scs2Delta) and a mutant, OPI1FFAT, lacking the ability to interact with Scs2p were utilized

151 nulogenic truncation mutant of SgIII (1-210) lacks the ability to interact with CgA.

152 an IgG1 antibody with a constant region that lacks the ability to interact with Fcgamma receptors.

153 cts with another bZIP protein TaFDL13, which lacks the ability to interact with the VRN1 promoter, su

154 The toolbox of rat genetics currently lacks the ability to introduce site-directed, heritable

155 responsive tissues, we have used female mice lacking the ability to localize ERalpha to the membrane

156 ond to patterning and hydrodynamic cues, but lack the ability to maintain their precise orientation.

157 We have studied Sl/Sl(d) mice, which lack the ability to make membrane-bound stem cell factor

158 Mice that lacked the ability to make both CD8(+) and CD4(+) iTregs

159 protein receptor Rag 1 double-knockout mice (lacking the ability to make immunoglobulins due to loss

160 Seven M. truncatula mutants, lacking the ability to make nodules, were tested for Nod

161 the same dose of AMPH is not blunted in mice lacking the ability to make norepinephrine and epinephri

162 Whereas wild-type C. bescii lacks the ability to make ethanol, 70% of the fermentati

163 ility of a C10S/V30M TTR double mutant which lacks the ability to make mixed disulfides, but retains

164 tant of Streptococcus pyogenes Manfredo that lacks the ability to make streptococcal acid glycoprotei

165 ory activity; however, the monomeric species lacked the ability to manifest anti-proliferative activi

166 However, this approach lacks the ability to measure and evaluate important meta

167 s cultured with glucose as the carbon source lacked the ability to metabolize acetone at the onset of

168 lidated bioprocessing of lignin, and it also lacks the ability to metabolize sugars or organic acids;

169 Lysates from Icmt-/- embryos lacked the ability to methylate either recombinant K-Ras

170 Lysates from Icmt-/- cells lacked the ability to methylate farnesyl-K-Ras4B or smal

171 In addition, Icmt-/- cells lacked the ability to methylate Rab proteins.

172 d this hypothesis using Stat1-/- mice, which lack the ability to mount Th1 immune responses.

173 eatly reduced when donor and recipient cells lack the ability to move because of mutations in any of

174 ously shown to induce an oncogenic phenotype lack the ability to move from caveolae in response to EG

175 10,000 clones were screened for mutants that lacked the ability to move away from the edge of a colon

176 re many ways to detect kinase activity, most lack the ability to "multiplex" the analysis (i.e., to d

177 cterized bevacizumab as species specific and lacking the ability to neutralize murine (m) VEGF-A.

178 sferase, hepatocarcinoma cells were found to lack the abilities to oxidize choline to betaine and to

179 e determined that RDH10(trex) mutant protein lacks the ability to oxidize retinol to retinal, resulti

180 on reaction leading to lariat formation, but lacks the ability to participate in the second reaction.

181 Bax toxicity was reduced in strains lacking the ability to perform oxidative phosphorylation

182 EKK1, four of the five altered MKK4 proteins lacked the ability to phosphorylate stress-activated pro

183 yeast (Saccharomyces cerevisiae) line, which lacks the ability to phosphorylate glucose and fructose

184 -7 assemble motile, full-length flagella but lack the ability to phototax.

185 actin machinery was intact, Cdc42 null cells lacked the ability to polarize their Golgi and coordinat

186 MxiE-regulated genes yielded no mutants that lacked the ability to prevent apoptosis.

187 Furthermore, most techniques lack the ability to process multiple samples simultaneou

188 beta converting enzyme (ICE)-deficient mice, lacking the ability to process mature IL-18 and IL-1beta

189 the absence of anti-Gal B cells, these mice lack the ability to produce anti-Gal.

190 Most importantly, they lack the ability to produce interferon-gamma in response

191 possess any antimicrobial activity and hence lack the ability to produce microbial resistance, but wh

192 a-hydroxylase knock-out (Dbh-/-) mice, which lack the ability to produce the SNS transmitters, norepi

193 ), and the respective isogenic mutants which lacked the ability to produce alginate, for their suscep

194 is cell line to generate a viral mutant that lacked the ability to produce B2.

195 nd had a ferric citrate transport system but lacked the ability to produce or use aerobactin.

196 Mice lacking the ability to produce BMP2 in their limb bones

197 Transcription of rrn operons in strains lacking the ability to produce either NusA or NusB was e

198 Mice lacking the ability to produce eosinophils should prove

199 220 or its isogenic mutant strain (Deltafmt) lacking the ability to produce formylated peptides.

200 tratracheal transfer of airway CD8 TRM cells lacking the ability to produce IFN-gamma were less effec

201 NK cells with a normal mature phenotype, but lacking the ability to produce IFN-gamma, whereas cocult

202 Soybean (Glycine max L. Merr.) mutants lacking the ability to produce the lectin normally found

203 the presence of PCA, a P. aeruginosa mutant lacking the ability to produce the siderophores pyoverdi

204 lly equivalent to the inner cell mass, which lacks the ability to produce all extraembryonic tissues.

205 cell line that stably expresses human TP and lacks the ability to produce TXA(2) was created by gene

206 Moreover, a Bcr mutant that lacks the ability to promote hydrolysis of Rac-GTP bound

207 They also lacked the ability to propel latex spheres and were resi

208 tionally specialized for killing, while they lack the ability to provide B cell help.

209 However, these methods lack the ability to provide spatial information for thes

210 in large cell populations and, as a result, lack the ability to quantify cell-to-cell variations.

211 imated strength of physical interactions and lack the ability to rank interactants as a function of t

212 This was because it lacked the ability to rearrange itself upon binding.

213 g and short isoforms, with the short isoform lacking the ability to recruit HDACs.

214 to hyperphosphorylated RNA polymerase II but lacks the ability to recruit SR proteins because of repl

215 l domain, whereas the TLS-ERG fusion protein lacks the ability to recruit SR proteins due to replacem

216 t the discretion of the attending physician, lack the ability to redefine the standard of care and mi

217 xicity, whereas obinutuzumab, a type II mAb, lacks the ability to redistribute into lipid rafts and i

218 P450 reconstituted system, Mn-cyt b5, which lacks the ability to reduce oxyferrous cyt P450 2B4, was

219 porary bladder tissue engineering strategies lack the ability to reform bladder smooth muscle, vascul

220 Mammals, in contrast to zebrafish, lack the ability to regenerate hair cells.

221 the developed world, partly because mammals lack the ability to regenerate heart tissue.

222 mutations of the COOH-terminal half of DPC4 lacked the ability to regulate transcription while retai

223 XDbf4 mutant that inhibits Wnt signaling but lacks the ability to regulate Cdc7.

224 were tightly adherent to surfaces but which lacked the ability to release cells into the medium and

225 , dilute bleach, and mechanical cleaning all lack the ability to remove cellular debris completely, w

226 ApoA-I lacked the ability to remove alpha-TOH from Tangier dise

227 Mitochondria lack the ability to repair certain helix-distorting lesi

228 lacking exon 3) that produces a protein that lacks the ability to repress transcription by NGFI-A and

229 In general, nonlegumes were assumed to lack the ability to respond to the rhizobial lipo-chitin

230 HCs are formed before birth, and mammals lack the ability to restore the sensory deficits associa

231 xpression patterns of nonimprinted genes but lack the ability to restore those of imprinted genes.

232 d a sedimentation coefficient of 5 to 7S and lacked the ability to restore translation in extracts of

233 s a harsh microenvironment for islets and it lacks the ability to retrieve the graft.

234 nference software offerings that use ABC-SMC lack the ability to scale in parallel computing environm

235 imeras in which only either B-1 or B-2 cells lack the ability to secrete IgM show mortality rates sim

236 ted conditional Wntless (Wls) KO mice, which lacked the ability to secrete Wnts from either liver epi

237 assessed the functional capacity of T(regs) lacking the ability to secrete both IL-10 and IL-35, whi

238 that mice deficient in the IL-17 receptor or lacking the ability to secrete IL-17 are highly suscepti

239 We show that this peptide lacks the ability to seed aggregation of tau244-372 in c

240 typical bovine insulin fibrils, although it lacks the ability to seed the intact protein.

241 However, multipotent progenitors lack the ability to self-renew, therefore their mitotic

242 However, NF180 lacked the ability to self-assemble, suggesting that lik

243 es possessing the same functional groups but lacking the ability to self-assemble do not catalyze sub

244 Api g 1, the Bet v 1 homolog in celery, lacks the ability to sensitize and is devoid of major T-

245 ic inhibitor but not with an HBx mutant that lacked the ability to sequester Crm1 in the cytoplasm.

246 Furthermore, knockout mice lacking the ability to signal through the type I interfe

247 In addition, mice lacking the ability to signal through TLR4 had significa

248 CXC families with nanomolar affinity, yet it lacks the ability to signal upon ligand binding.

249 The majority of existing vaccine adjuvants lack the ability to significantly stimulate the cellular

250 MM11453 lacked the ability to significantly activate RARs and re

251 a popular standard coalescent simulator, it lacks the ability to simulate sequences with recombinati

252 suggesting that some S. aureus isolates may lack the ability to site-specifically integrate SCCmec i

253 ABCA1-dependent cholesterol efflux, and they lacked the ability to stabilize ABCA1.

254 and thyroxine preferential binding sites and lacked the ability to stabilize TTR.

255 ommitment at a first encounter, primary IPCs lacked the ability to stimulate naive T cells.

256 3 cell lines, while the PF-deficient mutants lacked the ability to stimulate, and the complemented PF

257 binding and activation of the HGF receptor, lacks the ability to super-activate the Ras-MAP kinase p

258 ate that under low Ci conditions, the mutant lacks the ability to supply Rubisco with adequate CO(2)

259 c imaging revealed that beta1-depleted cells lacked the ability to sustain protrusions into the suben

260 igoadenylate synthetase-like proteins, which lack the ability to synthesize 2'-5' A, have been recent

261 In contrast to most animals, humans lack the ability to synthesize ascorbic acid as a result

262 provement in the development of fetuses that lack the ability to synthesize cholesterol, such as thos

263 DD mice lack the ability to synthesize dopamine (DA) specificall

264 se knockout (Dbh -/-) mice that specifically lack the ability to synthesize NE.

265 r bacteria comprising the order Chlamydiales lack the ability to synthesize nucleotides de novo and m

266 Mutants which lack the ability to synthesize PAI-1, PAI-2, or both aut

267 Cilia lack the ability to synthesize proteins and thus require

268 All parasitic protozoa lack the ability to synthesize purine nucleotides de nov

269 parasites, they are purine auxotrophs (i.e. lack the ability to synthesize purines de novo) and ther

270 completely devoid of selenoprotein genes and lack the ability to synthesize Sec.

271 xpression profile in a luxS-deficient strain lacking the ability to synthesize AI-2.

272 Transcriptome analysis of mutants lacking the ability to synthesize cAMP or the cAMP bindi

273 But children lacking the ability to synthesize cortisol (because of v

274 In addition, mice lacking the ability to synthesize GalC or sulfatide form

275 missive to HMPV infection, mutant cell lines lacking the ability to synthesize glycosaminoglycans (GA

276 Members of the Nematoda are heme auxotrophs, lacking the ability to synthesize heme; however, the hum

277 xpression were still observed in backgrounds lacking the ability to synthesize JA or the key transcri

278 5) Lacking the ability to synthesize new receptors and lack

279 depletion of available carbon, swarmer cells lacking the ability to synthesize ppGpp or polyP imprope

280 ultisensory neurons are strikingly immature, lacking the ability to synthesize the cross-modal inform

281 ltalro1Deltaare1Deltaare2Delta strain, which lacks the ability to synthesize both TAG and SE, is not

282 istolytica, is a nucleo-base auxotroph (i.e. lacks the ability to synthesize purines or pyrimidines d

283 mophilum is an aerobic photoheterotroph that lacks the ability to synthesize several essential nutrie

284 The results argue that people with autism lack the ability to take into consideration what others

285 only the CL20i4-EF1alpha-hgamma(c)OPT vector lacked the ability to transactivate LMO2 protein express

286 We establish that the human Fat homolog FAT4 lacks the ability to transduce Hippo signaling in Drosop

287 ein (MTP), Chinese hamster ovary (CHO) cells lack the ability to translocate apoB into the lumen of t

288 hat the newly isolated H5N2 and H5N8 viruses lacked the ability to transmit between ferrets and exhib

289 Moreover, these influenza H5Nx viruses lacked the ability to transmit between ferrets in a dire

290 xpected, the addition of AI-2 to cells which lack the ability to transport AI-2 (lsr null mutant) fai

291 The defective GPIHBP1 proteins also lacked the ability to transport LPL from the basolateral

292 ar-native levels of secondary structure, but lack the ability to undergo a cooperative thermal transi

293 Consequently, these cells lack the ability to undergo activation-induced cell deat

294 This mutant receptor has been shown to lack the ability to undergo agonist-induced internalizat

295 round cells under all growth conditions and lack the ability to undergo morphological differentiatio

296 Exosc3-deficient B cells lack the ability to undergo normal levels of class switc

297 tigations showed that IDO2-deficient B cells lacked the ability to upregulate the costimulatory marke

298 The dipeptide system mutants lacked the ability to use certain dipeptides, hexapeptid

299 C. dubliniensis has been reported to lack the ability to utilize xylose (XYL) and alpha-methy

300 es such as urease and xylose utilization and lacking the ability to utilize nitrate and nitrite.