ライフサイエンスコーパス: lacrimal gland

1 ptors P2X(1-4) and P2X(6) are present in the lacrimal gland.

2 olycystin-2L2) were expressed in adult mouse lacrimal gland.

3 that P2X(7)receptors were functional in the lacrimal gland.

4 ion of interleukin (IL)-1 into the exorbital lacrimal gland.

5 es (Hs2st, Hs6st1, and Hs6st2) in developing lacrimal gland.

6 esenchymal transition (EMT) in repair of the lacrimal gland.

7 thelium, but not in the CN epithelium or the lacrimal gland.

8 gher uptake in kidneys, urinary bladder, and lacrimal gland.

9 f Tregs and CD4(+) IFN-gamma(+) cells in the lacrimal gland.

10 in AL2 mRNA was detected only in male rabbit lacrimal gland.

11 2, BL, CL, and CL2 were detected only in the lacrimal gland.

12 veral lipophilins were expressed only in the lacrimal gland.

13 of unipotent KRT5(+) epithelial cells in the lacrimal gland.

14 ntry into the acinar epithelial cells of the lacrimal gland.

15 e coregulation of the stress response in the lacrimal gland.

16 cell and stimulate protein secretion in rat lacrimal gland.

17 elated differences in gene expression of the lacrimal gland.

18 eNOS and nNOS were both present in lacrimal gland.

19 en clearance, was investigated in the rabbit lacrimal gland.

20 nd suppress BMP-induced proliferation in the lacrimal gland.

21 expression of hundreds of genes in the mouse lacrimal gland.

22 n two arborized structures, the lung and the lacrimal gland.

23 ression and activity of CYP3A6 in the rabbit lacrimal gland.

24 s (mU) of botulinum toxin B (BTX-B) into the lacrimal gland.

25 and specifically CYP3A6 in the naive rabbit lacrimal gland.

26 drenergic-induced protein secretion from the lacrimal gland.

27 mediated assembly to form a depot inside the lacrimal gland.

28 e formation of secretory acinar lobes in the lacrimal gland.

29 on were determined at the ocular surface and lacrimal gland.

30 nic inflammation in the underlying stroma or lacrimal gland.

31 alpha1, collagen type IIIalpha1 and NF-kB in lacrimal glands.

32 ltration of inflammatory/immune cells in the lacrimal glands.

33 utoimmune disease affecting the salivary and lacrimal glands.

34 from IL-1-injected, but not saline-injected, lacrimal glands.

35 autoimmune diseases of ovary, prostate, and lacrimal glands.

36 surface epithelia and on fluid secretion by lacrimal glands.

37 so known as lipophilins, are found in rabbit lacrimal glands.

38 njunctiva, cornea, and tears produced by the lacrimal glands.

39 lymphocyte infiltrations in the extraorbital lacrimal glands.

40 e exocrine glands including the salivary and lacrimal glands.

41 340 is abundant in secreted tears and in the lacrimal glands.

42 er number of CD8+ T cells and B cells in the lacrimal glands.

43 results from involvement of the salivary and lacrimal glands.

44 y excessive inflammation and fibrosis in the lacrimal glands.

45 immune disease mainly affecting salivary and lacrimal glands.

46 uclear cell infiltration in the salivary and lacrimal glands.

47 V levels were detectable in the salivary and lacrimal glands 14-28 days after i.p. infection and corr

48 The lacrimal gland (28%), conjunctiva (28%), and orbit (28%)

49 g from the orbit (19), conjunctivae (18) and lacrimal gland (6).

50 ses growth and proliferation of the lung and lacrimal gland, a result that was confirmed in vivo usin

51 Lacrimal gland abscess or dacryoadenitis was the present

52 Fourteen patients with primary lacrimal gland ACC were included.

53 Lacrimal gland ACCs are frequently positive for the MYB-

54 Our findings show that lacrimal gland ACCs are genetically and clinically simil

55 The results show that lacrimal gland acinar cells are lost through programmed

56 Dysfunction of lacrimal gland acinar cells can ultimately result in ocu

57 hexosaminidase secretion from primary rabbit lacrimal gland acinar cells.

58 Rat lacrimal gland acini were incubated with H89, an inhibit

59 Rat lacrimal gland acini were isolated by collagenase digest

60 Rat lacrimal gland acini were isolated by collagenase digest

61 Lacrimal gland acini were isolated by collagenase digest

62 Rat lacrimal gland acini were isolated by collagenase digest

63 signaling components of this pathway in rat lacrimal gland acini.

64 Idiopathic inflammatory tumor of the lacrimal gland, also called idiopathic dacryoadenitis, g

65 t (LFU), comprising the cornea, conjunctiva, lacrimal gland and interconnecting innervation.

66 (typically the lateral) and, less often, the lacrimal gland and is often mild when it arises during o

67 but not Pax6(PE/PE) mice, developed stunted lacrimal gland and lens hypoplasia which was significant

68 on the regulation of gene expression in the lacrimal gland and may provide genetic evidence for a co

69 Lacrt has prosecretory activity in the lacrimal gland and mitogenic activity at the corneal epi

70 regulator (aire)-deficient mice, we assessed lacrimal gland and ocular surface immunopathology by qua

71 Lymphocytic infiltration of the lacrimal gland and ocular surface in autoimmune diseases

72 TLR2 and 5 were upregulated in lacrimal gland and overall, there was a corresponding ch

73 an essential transcription factor for lens, lacrimal gland and pancreas development.

74 whether these cells can be isolated from the lacrimal gland and propagated in vitro.

75 ESP22 is secreted from the lacrimal gland and released into tears of 2- to 3-week-o

76 gle injection of interleukin-1alpha into the lacrimal gland and that this effect was reversible.

77 y localized gp340 to the acinar cells of the lacrimal gland and the deeper layers of the conjunctival

78 vated by adenosine triphosphate (ATP) in rat lacrimal gland and to determine their role in protein se

79 X(7) purinergic receptors are present in rat lacrimal gland and when stimulated increase [Ca(2+)](i),

80 lands of Wolfring is similar to that of main lacrimal glands and are consistent with secretion electr

81 l infiltration of the stroma or acini of the lacrimal glands and conjunctivae of both saline-injected

82 oimmune disease starting in the salivary and lacrimal glands and continuing to involve the lungs and

83 y complete removal of the main and accessory lacrimal glands and nictitating membranes.

84 ation of primarily CD4(+) T cells within the lacrimal glands and with increased expression of IL-4.

85 re equally noted to involve the conjunctiva, lacrimal gland, and orbit.

86 erved in bone marrow, colon, lung, pancreas, lacrimal gland, and salivary gland, but not in heart, th

87 s, and 3) autoimmune disease of the thyroid, lacrimal gland, and salivary gland.

88 ially therapeutic cyclosporine levels to the lacrimal gland, and showed efficacy in a clinically rele

89 cells in the inflammatory infiltrates of the lacrimal gland, and the presence of anti-Sjogren's syndr

90 tissue PMN population in the corneal limbus, lacrimal glands, and cervical lymph nodes of healthy mal

91 low, occurred mainly around the salivary and lacrimal glands, and could easily be corrected.

92 MECs can be isolated from rat lacrimal glands, and they express P2X(7), P2Y(1), P2Y(11

93 ch as marked lymphocytic infiltration of the lacrimal glands, antinuclear antibodies in the serum, an

94 The accessory lacrimal glands are assumed to contribute to the product

95 The results of our study indicate that lacrimal glands are capable of tissue repair after duct

96 ne disease mainly involving the salivary and lacrimal glands, are poorly understood.

97 matory illness that affects the salivary and lacrimal glands as well as other organ systems.

98 ounds localize to the parotid, salivary, and lacrimal glands as well as to the kidney, leading to dos

99 idosis (7 in adipose tissue; 5 affecting the lacrimal gland) as well as comparable tissue from 6 heal

100 inflammatory CD4(+) T cells detected in the lacrimal gland, as well as those in the periphery of old

101 to mononuclear infiltration of salivary and lacrimal glands, as well as to expansion of bronchial ly

102 increased apoptosis and deterioration in the lacrimal gland, associated dysfunction, and development

103 ealkylation was measured in the naive rabbit lacrimal gland at a rate of 14 +/- 7 picomoles/mg protei

104 pproach, however, we have identified a novel lacrimal gland autoantigen, odorant binding protein 1a,

105 to investigate the pathogenic mechanisms in lacrimal gland autoimmunity and associated ocular surfac

106 expression profiles of C57BL/6.NOD-Aec1Aec2 lacrimal glands before, or concomitant with, the first a

107 es were reviewed and microscopic sections of lacrimal gland biopsy samples were critically re-evaluat

108 mp7-null mice have distinctive reductions in lacrimal gland branch number, and that inhibition of Bmp

109 , downstream of FGF signaling, in regulating lacrimal gland branching and differentiation.

110 In the lacrimal gland, branching morphogenesis depends on the i

111 ran sulfates by Ndst genes at the tip of the lacrimal gland bud.

112 Consistent with this, Fgf10-induced ectopic lacrimal gland budding in explant cultures is dependent

113 , and epithelial deletion of Fgfr2 abolishes lacrimal gland budding, its specific modification of hep

114 on, we demonstrate that Shp2 is required for lacrimal gland budding, lens cell proliferation, surviva

115 st1, Fgfr2 or Shp2 disrupts ERK signaling in lacrimal gland budding.

116 g robustly rescue the lens proliferation and lacrimal-gland-budding defects in the Shp2 mutants.

117 P2X(7) receptors were present in the lacrimal gland by RT-PCR and Western blot analysis.

118 ) and P2X(6)receptors were identified in the lacrimal gland by RT-PCR, Western blot, and immunofluore

119 ession in sarcoidosis involving the orbit or lacrimal gland can be distinguished from gene expression

120 Lacrimal gland carcinoma can form a triangle of tissue b

121 It is significantly more common in lacrimal gland carcinoma compared with dacryoadenitis an

122 aging for 116 patients was reviewed: 39 with lacrimal gland carcinoma, 37 with lymphoma, and 40 with

123 The "wedge sign" is most common in lacrimal gland carcinoma, but can occur in patients with

124 mposed of rabbit conjunctival epithelium and lacrimal gland cell spheroids, and recapitulates the aqu

125 In lacrimal gland cells, the activation of M3AChRs stimulat

126 Mice were killed at various times, and lacrimal gland cellularity was analyzed by flow cytometr

127 ioma (n = 4; 10%), melanocytoma (n = 3; 8%), lacrimal gland choristoma (n = 2; 5%), gliosis (n = 1; 3

128 eased infiltration of mononuclear cells into lacrimal glands compared with 4-1BB intact lpr mice.

129 Murine lacrimal glands contain mesenchymal stem cells that seem

130 The lacrimal gland contains stem/progenitor cells capable of

131 auxiliary eye structures such as the eyelid, lacrimal gland, cornea and conjunctiva.

132 Human fibroblasts were isolated from the lacrimal gland, cornea, and Tenon's capsule and treated

133 l, T-cell infiltration into the salivary and lacrimal glands could be successfully visualized.

134 genetic rescue experiments in which the Ugdh lacrimal gland defect is ameliorated by constitutive Ras

135 tive FGF receptor only partially rescued the lacrimal gland defects in Sox9 heterozygotes, suggesting

136 To address if lacrimal gland development and FGF signaling depends on

137 Therefore, Fgf10-Fgfr2b signaling during lacrimal gland development is sensitive to the content o

138 Here, we show that lacrimal gland development requires specific modificatio

139 We further show that lacrimal gland development requires the mesenchymal expr

140 ogether, our data reveal crucial features of lacrimal gland development that have broad implications

141 nly for modulating Ras signaling in lens and lacrimal gland development, but also for RTK signaling i

142 st;Hs6st double mutants completely abolished lacrimal gland development, suggesting that both 2-O and

143 e main downstream target of Shp2 in lens and lacrimal gland development.

144 nd epithelial lineage dynamics that underlie lacrimal gland development.

145 ellular matrix components during early stage lacrimal gland development.

146 hy in NOD/LtJ mice an in-depth evaluation of lacrimal gland disease in the NOD/LtJ mouse has remained

147 The cause of bilateral lacrimal gland disease most commonly was inflammatory, f

148 Klf5CN lacrimal glands displayed increased vasculature and larg

149 The cyclosporine implant produced lacrimal gland drug levels 1 to 2 log units higher than

150 provides an excellent approach for studying lacrimal gland duct cells about which relatively little

151 d expression of transporters and channels on lacrimal gland duct membranes is consistent with the hyp

152 Lacrimal gland ductal cysts (dacryops) are uncommon, occ

153 infective debris (1 case) from the affected lacrimal gland ductule--typically the most inferolateral

154 Infective lacrimal gland ductulitis, commonly from Actinomyces inf

155 Immunohistochemistry studies of lacrimal gland dysfunction and conjunctival inflammation

156 disease manifested primarily by salivary and lacrimal gland dysfunction resulting in dry mouth/dry ey

157 disease, (2) salivary gland dysfunction and lacrimal gland dysfunction, and (3) limited mouth-openin

158 in ATD and Sjogren syndrome, conditions with lacrimal gland dysfunction.

159 gland involvement, orbital pseudotumor, and lacrimal gland enlargement.

160 ecific requirement of Ndst1 and Ndst2 in the lacrimal gland epithelial, but not mesenchymal, cells, a

161 the co-receptors for Fgf10 signaling in the lacrimal gland epithelium, but their function in the Fgf

162 or budding morphogenesis in the presumptive lacrimal gland epithelium.

163 beta-catenin conditionally expressed in the lacrimal gland epithelium.

164 c scopolamine, or by performing extraorbital lacrimal gland excision.

165 otype but led to significant improvements in lacrimal gland exocrinopathy and tear secretion.

166 Cultured MSCs isolated from injured lacrimal glands expressed Snai1 and vimentin alongside n

167 multifactorial chronic disorder in which the lacrimal glands fail to produce enough tears to maintain

168 may provide genetic evidence for a cornea-to-lacrimal gland feedback mechanism in dry eye.

169 portance of heparan sulfate 6-O sulfation in lacrimal gland FGF signaling.

170 fibroblasts were elevated in cGVHD-affected lacrimal gland fibroblasts and (2) that they could be re

171 absence of recent upper respiratory illness, lacrimal gland focus, multiple orbital abscesses, and la

172 2283 genes) were significantly lower in the lacrimal gland for patients with sarcoidosis.

173 l types and lineage relationships that drive lacrimal gland formation are unclear.

174 other orbital inflammatory conditions in the lacrimal gland fossa.

175 We show that the lacrimal gland from its earliest developmental stages is

176 Frozen sections of lacrimal glands from five rats were subjected to LCM to

177 Exorbital lacrimal glands from male Sprague-Dawley rats were divid

178 Exorbital lacrimal glands from male Sprague-Dawley rats were divid

179 e new clues for a molecular understanding of lacrimal gland function and mechanisms of coordinated ti

180 deficient in 4-1BB were generated, and their lacrimal gland function was studied.

181 molecular signalling processes that control lacrimal gland function will give insight into correctiv

182 Lacrimal glands function to produce an aqueous layer, or

183 se in reducing the morbidity associated with lacrimal gland graft-versus-host disease after allogenei

184 The Hs6st mutants exhibited significant lacrimal gland hypoplasia and a strong genetic interacti

185 IL-1beta activated ERK in the lacrimal gland in vitro and in vivo, and this effect was

186 uctal inflammation in both submandibular and lacrimal glands in contrast to the much milder infiltrat

187 te complex on the cell surface and prevented lacrimal gland induction by Fgf10 in explant cultures.

188 ts demonstrate that mesenchymal GAG controls lacrimal gland induction by restricting the diffusion of

189 Lacrimal gland inflammation was induced by injection of

190 se occurs in the setting of conjunctival and lacrimal gland inflammation, potentially mediated by the

191 FN-gamma and substantial IL-4 at the site of lacrimal gland inflammation.

192 IL-1beta injection into the lacrimal gland inhibited aqueous tear production by 52%

193 Injection of LPS into the lacrimal gland inhibited neurally and adrenergic agonist

194 was to determine the mechanisms involved in lacrimal gland injury and repair.

195 R(-/-)) had the same submandibular gland and lacrimal gland injury as did the IL14alphaTG mice, but t

196 Lacrimal gland injury was induced by injection of interl

197 It was recently reported that repair of the lacrimal gland involved the mobilization of mesenchymal

198 Lacrimal gland involvement in granulomatosis with polyan

199 inical and imaging features of patients with lacrimal gland involvement secondary to GPA and to compa

200 nd to have orbital inflammatory disease with lacrimal gland involvement, of whom 7 had a final diagno

201 estrogen's and progesterone's action on the lacrimal gland involves the regulation of numerous genes

202 The tear-producing lacrimal gland is a tubular organ that protects and lubr

203 Ongoing studies demonstrate that the murine lacrimal gland is capable of repair after experimentally

204 Previously, it was reported that the murine lacrimal gland is capable of repair after experimentally

205 e effect of estrogen and progesterone on the lacrimal gland is mediated through specific receptors an

206 The lacrimal gland is primarily responsible for the aqueous

207 , and secretion from the acinar cells of the lacrimal gland is regulated by both cholinergic and adre

208 disease affecting primarily the salivary and lacrimal glands leading to xerostomia (dry mouth) and xe

209 Deletion of 4-1BB in lpr mice accelerates lacrimal gland lesions through increased CD4(+) T-cell i

210 s; it is associated with inflammation of the lacrimal gland (LG) and in some cases involves T cell in

211 The lacrimal gland (LG) delivers defensive and metabolic fac

212 The lacrimal gland (LG) develops through branching morphogen

213 Bilateral lacrimal gland (LG) disease is a unique presentation tha

214 d corneal injury (CI) trigger alterations of lacrimal gland (LG) growth factor expression and redistr

215 To describe the involvement of the lacrimal gland (LG) in blepharophimosis-ptosis-epicanthu

216 Lacrimal gland (LG) morphogenesis and repair are regulat

217 us, and evaluate its effects on the inflamed lacrimal gland (LG) of non-obese diabetic mouse (NOD), a

218 in the parasympathetic neural stimulation of lacrimal gland (LG) secretion.

219 ia receptor-mediated transcytosis across the lacrimal gland (LG), which produces the bulk of human te

220 progenitor cells in the uninjured, adult rat lacrimal gland (LG).

221 -adrenergic receptors in the mouse exorbital lacrimal gland (LG).

222 me-related immunopathological changes in the lacrimal glands (LGs) of CD25KO mice, we examined LGs of

223 Lacrimal glands (LGs) of male NOD mice, a model of Sjogr

224 Paraffin-embedded lacrimal glands (LGs) were stained with hematoxylin and

225 xual dimorphic expression patterns of rabbit lacrimal gland lipophilins AL, AL2, BL, CL, and CL2 were

226 N) preferentially suppress AOD and Treg from lacrimal gland LN preferentially suppress dacryoadenitis

227 re, the systemic illness (skin, salivary and lacrimal glands, lungs, lymphadenopathy, and splenomegal

228 ents of alpha-fodrin were found in tears and lacrimal gland lysates, respectively, of lpr/4-1BB(-/-)

229 egenerative potential in a rabbit model with lacrimal gland main excretory duct ligation-induced inju

230 erwent debulking surgery of the inflammatory lacrimal gland mass for diagnostic and therapeutic reaso

231 t therapy, and bioartificial devices such as lacrimal gland microdevices and keratoprostheses, or tis

232 the ocular surface, with the conjunctiva and lacrimal gland most commonly affected.

233 the eyelids (n = 53 [82%]), followed by the lacrimal gland (n = 5), conjunctiva (n = 4), and eyebrow

234 ha, 1 microg in 2 microL) into the exorbital lacrimal glands of anesthetized female BALB/c mice.

235 gene expression profiles were generated for lacrimal glands of C57BL/6.NOD-Aec1Aec2 mice 4 to 20 wee

236 s infiltrate the corneal stroma, limbus, and lacrimal glands of diseased mice.

237 amma than IL-13 mRNA relative transcripts in lacrimal glands of MRL/lpr/IL-4(tm) mice (mean differenc

238 tial biomarkers of impending autoimmunity in lacrimal glands of SjS-prone mice.

239 ot attenuate lymphocytic infiltration of the lacrimal gland or eye, it significantly reduced ocular s

240 eyelid, conjunctiva, choroid, ciliary body, lacrimal gland, or orbit (OA-uveal lymphoma) were includ

241 A total of 36 tumors from 32 patients with lacrimal gland PA or Ca-ex-PA were included in the study

242 tance for disease progression in a subset of lacrimal gland PAs.

243 oma and generally indicates life-threatening lacrimal gland pathology that requires urgent biopsy.

244 w of images from patients with biopsy-proven lacrimal gland pathology.

245 Carbachol increased ATP release from lacrimal gland pieces but not from acini.

246 Biophysical properties of lacrimal gland polycystin-2 channels were similar to tho

247 The lacrimal gland possesses many features that make it an e

248 Lacrimal gland progenitor cells isolated from ligated ti

249 Isolated lacrimal gland progenitor cells were tested and characte

250 L-1beta activates the ERK pathway to inhibit lacrimal gland protein secretion and aqueous tear produc

251 Lacrimal gland protein secretion was measured using a sp

252 t of IL-1beta on aqueous tear production and lacrimal gland protein secretion.

253 However, reactivities to other salivary and lacrimal gland proteins were readily detected.

254 An enlarged salivary gland or lacrimal gland raises a wide differential diagnosis that

255 ioration of the autonomic innervation of the lacrimal glands rather than an impaired corneal innervat

256 Lacrimal glands regulate the production and secretion of

257 ment of one of more salivary gland(s) and/or lacrimal gland(s).

258 Lacrimal glands secrete proteins, electrolytes and water

259 Lacritin protein is highly expressed in the lacrimal gland, secreted into tear fluid, and detected o

260 LPS inhibits lacrimal gland secretion by activating caspase 1, and IL

261 (ERK) in inflammation-induced inhibition of lacrimal gland secretion.

262 1 alleviated the inhibitory effect of LPS on lacrimal gland secretion.

263 tear protein, it promotes basal tearing and lacrimal gland secretion.

264 ants (e.g., urinary pheromones, extraorbital lacrimal gland secretions, major histocompatibility comp

265 ferential display analysis, and a new rabbit lacrimal gland secretoglobin, lipophilin AL2, was identi

266 ut ((-/-)) mice have impaired ocular surface-lacrimal gland signaling, rendering them susceptible to

267 e a critical role for Orai1-mediated SOCE in lacrimal gland signalling and function.

268 The results provide further insights into lacrimal gland stem/progenitor cell physiology and their

269 The existence of lacrimal gland stem/progenitor cells was proposed in sev

270 Secretory function also increased in the lacrimal gland, suggesting this local therapy could trea

271 rkers between the developing mouse and human lacrimal gland, supporting the use of mice to understand

272 The ligation-injured lacrimal glands temporarily decreased in weight and had

273 iple pustules/abscesses in the region of the lacrimal gland that were expressing purulent fluid into

274 owed that gp340 transcripts were abundant in lacrimal gland tissue and were also present in the corne

275 ryops) are uncommon, occurring anywhere that lacrimal gland tissue is present.

276 Studies were performed in adult lacrimal gland tissue of Swiss-Webster mice.

277 fforts, the molecular and cellular events in lacrimal gland tissues initiating inflammatory responses

278 P3A expression and activity was performed on lacrimal gland tissues obtained from naive untreated and

279 und that EMT is induced during repair of the lacrimal gland to generate MSCs to initiate repair, and

280 ied the expression of MYB-NFIB in 19 non-ACC lacrimal gland tumors.

281 cterize the role of Orai1 in the function of lacrimal glands using a mouse model in which the gene fo

282 : 1) initial injury to the submandibular and lacrimal glands via an environmental insult and LTalpha;

283 notype, in which the exorbital branch of the lacrimal gland was absent in most cases.

284 The major lacrimal gland was involved in 13 lesions; 2 lesions aro

285 The total RNA of the lacrimal gland was then extracted, at eight time points-

286 Cytokine expression of lacrimal glands was assessed by flow cytometry and RT-PC

287 usly, in single lacrimal cells isolated from lacrimal glands, we demonstrated that muscarinic recepto

288 e were noted to have significantly increased lacrimal gland weight, with enlarged, carbohydrate-rich,

289 Acinar cells from monkey lacrimal gland were cultured and characterized.

290 Acini from rat lacrimal gland were isolated by collagenase digestion.

291 Caspase 1 and ERK activities in the lacrimal gland were measured by immunohistochemistry, We

292 Lacrimal glands were collected from young adult, ovariec

293 RNAs from male and female rabbit lacrimal glands were compared in a differential display

294 Acinar cells from monkey lacrimal glands were cultured with or without tumor necr

295 ted acinar epithelial monolayers from rabbit lacrimal glands were exposed to varying concentrations o

296 Two and half days after injection, the lacrimal glands were removed and used to prepare explant

297 raries generated from normal human and mouse lacrimal glands were sequenced and analyzed by PHRED, Re

298 Rat lacrimal glands were subjected to collagenase digestion,

299 Exorbital lacrimal glands were then removed and processed for meas

300 voked macrophage infiltration to the eye and lacrimal gland, where they played a functional role in d