1  goblet cells independent of CsA's effect on lacrimation.

2  be activated by ocular stimuli important to lacrimation.

3 onomic feature during exacerbations: 73% had lacrimation, 51% nasal congestion, 46% conjunctival inje

4                       Of the cohort, 87% had lacrimation, 68% had conjunctival injection, 58% rhinorr

5   At high concentrations, menthol can induce lacrimation and nocifensive behaviors in a TRPM8 indepen

6          Moreover, treatment restored normal lacrimation and resistance to eye and airway infections

7 y sparse hair, inability to sweat, decreased lacrimation, frequent pulmonary infections, and missing

8 ed transaminases in one patient, and grade 3 lacrimation in the other patient.

9                              The increase in lacrimation is likely due to an increase in cool cell ac

10 mic symptoms such as conjunctival injection, lacrimation, nasal congestion, rhinorrhoea, ptosis or ey

11 o 90 seconds, and accompanied by ipsilateral lacrimation of the eye, rhinorrhea, and conjunctival inj

12 parasympathetic) autonomic symptoms, such as lacrimation or conjunctival injection.

13   Exposures to industrial isocyanates induce lacrimation, pain, airway irritation, and edema.

14 sfunction of these corneal afferents and the lacrimation reflex pathway they activate lead to some fo

15  ipsilateral ptosis, conjunctival injection, lacrimation, rhinorrhoea and facial flushing.

16 lacrimal gland injection of BTX-B suppressed lacrimation, thereby establishing a dry eye state.

17 ocular surface has been reported to increase lacrimation via activation of corneal cool primary affer

18 nthol induces corneal cool cell activity and lacrimation via the transient receptor potential melasta

19 t low concentrations of menthol can increase lacrimation via TRPM8 channels without evoking nocifensi

20        Symptomatic onycholysis and excessive lacrimation were observed after several months or more o