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1 natural immunity conferred by carriage of N. lactamica.
2 the challenge group became colonized with N. lactamica.
3 of 17 strains examined but was present in N. lactamica.
4 rizontal gene transfer with the commensal N. lactamica.
5  and Y, Neisseria gonorrhoeae, and Neisseria lactamica.
6 restriction endonuclease NlaIII in Neisseria lactamica.
7 methyltransferases, including M.NlaIII in N. lactamica.
8            Two weeks after inoculation of N. lactamica, 33.6% (48/143) of the challenge group became
9 lted in the acquisition of 26 consecutive N. lactamica alleles.
10  between colonization by commensal Neisseria lactamica and meningococcal disease, we investigated whe
11 in four individuals cocolonized by Neisseria lactamica and Neisseria meningitidis One HGT event resul
12 h all participants were reinoculated with N. lactamica and resampled 2 weeks later.
13 gitidis, Neisseria gonorrheae, and Neisseria lactamica are often regarded as highly recombining bacte
14 e other commensals, whole bacteria of two N. lactamica commensal strains had low inflammatory potenti
15 nce of adaptive cell-mediated immunity to N. lactamica during the peak age of carriage.
16 us of N. meningitidis, N. gonorrheae, and N. lactamica in the light of the PCE model.
17                                    Neisseria lactamica is a commensal bacteria that colonizes the hum
18 e inhibition of meningococcal carriage by N. lactamica is even more potent than after glycoconjugate
19 urified lipooligosaccharide nor PorB from N. lactamica is likely to be responsible for this activity.
20                                    Neisseria lactamica is not associated with disease, but N. meningi
21 loid, while the commensal organism Neisseria lactamica maintains chromosomes in single copy.
22 ead, outer membrane vesicles derived from N. lactamica mediate a B cell-dependent proliferative respo
23 dered to be nonpathogenic, such as Neisseria lactamica, N. subflava, and N. sicca.
24  six commensal Neisseria isolates, Neisseria lactamica, Neisseria subflava, Neisseria flava, Neisseri
25                                    Neisseria lactamica or its components could be a novel bacterial m
26 ulated with 10(4) colony-forming units of N. lactamica or were sham-inoculated, and carriage was moni
27                 However, commensal Neisseria lactamica organisms and purified N. lactamica PorB (publ
28     These effects are reproduced by whole N. lactamica organisms.
29 s abrogated the proliferative response to N. lactamica outer membrane vesicles, suggesting that this
30 sseria meningitidis (P < 0.05) and Neisseria lactamica (P < 0.002) (2-sided Fisher's exact test) was
31 eisseria lactamica organisms and purified N. lactamica PorB (published elsewhere as Nlac PorB) induce
32  loop residues have been switched between N. lactamica PorB and N. meningitidis PorB, we identified r
33 himeric N. meningitidis strain expressing N. lactamica PorB induces lower levels of interleukin 8 (IL
34                              Furthermore, N. lactamica PorB is a poor inducer of proinflammatory medi
35                     Our studies show that N. lactamica PorB is a TLR2 ligand but its binding specific
36 rolled infection of human volunteers with N. lactamica prevents colonization by Neisseria meningitidi
37 I restriction enzyme isolated from Neisseria lactamica recognizes the sequence 5'-CATG-3', cleaving a
38    Crossover inoculation of controls with N. lactamica replicated the result.
39                    Analysis of the LPS of N. lactamica strain NL4 revealed two distinct and interconv
40 ghly conserved property and is present in N. lactamica strains derived from diverse clonal complexes.
41 cB were also found in N. meningitidis and N. lactamica strains, and an insertion in the meningococcal
42                                 In Neisseria lactamica, this promoter involves another dcw-associated
43 of N. polysaccharea (Np), four strains of N. lactamica, three strains of N. subflava and nine strains
44 carriage was only observed in carriers of N. lactamica, was due both to displacement of existing meni
45  of N. meningitidis, the commensal Neisseria lactamica, which chiefly colonizes infants not adults.
46 gitidis, Neisseria gonorrhoeae and Neisseria lactamica, which exhibit very different phenotypes with
47 nd signaling, commensal organisms such as N. lactamica would benefit from expressing a product that i

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