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1 stitutional symptoms, and elevated values of lactic dehydrogenase.
3 flow, reduced release of the cardiac enzyme lactic dehydrogenase, and reduced myocardial concentrati
6 age >/= 60 years, stages III to IV disease, lactic dehydrogenase (LDH) > normal, extranodal sites (E
7 5%] of eight, P: <.005) and higher levels of lactic dehydrogenase (LDH) (339 U/L +/- 65 [SD] vs 258 U
8 inase (AST), alanine transaminase (ALT), and lactic dehydrogenase (LDH) (but not alkaline phosphatase
9 oorer failure-free survival (FFS) included a lactic dehydrogenase (LDH) level greater than normal (P
10 horter survival included age >/=60, elevated lactic dehydrogenase (LDH) or beta-2-microglobulin (beta
12 dependent increase in cell death measured by lactic dehydrogenase (LDH) release and vital dye exclusi
15 Combining elevated beta2 microglobulin and lactic dehydrogenase (neither elevated v one or both ele
16 ded sex, stage, Karnofsky performance score, lactic dehydrogenase, number of extranodal sites, B symp
17 (glyceraldehyde-3-phosphate dehydrogenase or lactic dehydrogenase) or even 3 mM magnesium ions can al
18 id not injure T84 monolayers, as assessed by lactic dehydrogenase release and protein synthesis assay
20 ubular segment injury (up to 92, 71, and 30% lactic dehydrogenase release, respectively) and massive
21 etastases and the presence of elevated serum lactic dehydrogenase to be used in the M category; (4) a
22 arameters of the labile enzyme rabbit muscle lactic dehydrogenase were determined as a function of th
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