ライフサイエンスコーパス: lactobacilli

1 ng the rectum may be a reservoir for vaginal lactobacilli.

2 he identification of 97 strains of commensal lactobacilli.

3 edictive of an absence of H(2)O(2)-producing lactobacilli.

4 strains as well as other species of vaginal lactobacilli.

5 unique DNA fingerprint compared to all other lactobacilli.

6 colonized with commensal bacteria, primarily lactobacilli.

7 ilus, are the most common species of vaginal lactobacilli.

8 ift involves organisms other than the MS and lactobacilli.

9 thesis about the natural history and role of lactobacilli.

10 (+) T lymphocytes and vaginal H2O2-producing lactobacilli.

11 pecific traits in the genomes of the vaginal lactobacilli.

12 genes (ISGs) being the most affected by both lactobacilli.

13 teria and levels of Streptococcus mutans and Lactobacilli.

14 or total bacteria, Streptococcus mutans, and Lactobacilli.

15 toxicity, and does not inhibit the growth of lactobacilli.

16 is also highly conserved in streptococci and lactobacilli.

17 the increasing use of probiotics containing lactobacilli.

18 onii is a member of the acidophilus group of lactobacilli.

19 Lactobacilli, a component of the normal vaginal flora, c

20 n a pathogen-free facility were colonized by Lactobacilli, a component of the oropharyngeal flora.

21 91; 20.2%) and those with H(2)O(2)-producing lactobacilli absent (n=756; 79.8%).

22 Lactobacilli acidify CVM to pH approximately 4 by contin

23 Engineered lactobacilli also caused a modest, but statistically sig

24 Lactobacilli also increased tumor necrosis factor-alphaa

25 Lactobacilli also inhibited gonococcal invasion of epith

26 The viability of the microcapsulated lactobacilli also remained high (>7 log cfu g(-1)) for 2

27 ted sustained colonization by H2O2-producing lactobacilli among women already colonized (relative ris

28 factors for an absence of H(2)O(2)-producing lactobacilli among women with bacterial vaginosis.

29 s: a nucleoside deoxyribosyltransferase from lactobacilli and a 5'-monophosphate-2'-deoxyribonucleosi

30 ant aciduric bacteria from root lesions were lactobacilli and A. israelii, while from sound root surf

31 l women have a relative depletion of vaginal lactobacilli and an increase in vaginal E. coli compared

32 This correlates with a loss of lactobacilli and an overgrowth of diverse bacteria.

33 to examine the effect of N-9 use on vaginal lactobacilli and bacterial vaginosis.

34 The growth of health-promoting lactobacilli and bifidobacteria is supported by FOS, giv

35 o evidence that a multistrain preparation of lactobacilli and bifidobacteria was effective in prevent

36 ia, while a slight increase on the growth of lactobacilli and bifidobacteria was observed after expos

37 receive either a multistrain preparation of lactobacilli and bifidobacteria, with a total of 6 x 10(

38 nal flora characterized by reduced levels of lactobacilli and concomitant overgrowth of anaerobic bac

39 obial community was found to be dominated by Lactobacilli and Enterobacteria, both typically facultat

40 We conclude that the association between lactobacilli and gonococci is complex and may be subject

41 0) had significantly lower concentrations of lactobacilli and higher concentrations of Gardnerella va

42 g thus allows high efficiency mutagenesis in lactobacilli and lactococci, and may be used to further

43 Quantitative bacterial counts for lactobacilli and M. hominis are better correlates of CVL

44 aginal bacterial flora with disappearance of lactobacilli and overgrowth of Gardnerella vaginalis and

45 aracterized by the replacement of beneficial lactobacilli and the augmentation of anaerobic bacteria.

46 ptide production, which allows overgrowth of Lactobacilli and triggers T regulatory cell expansion in

47 he inverse association between H2O2-positive lactobacilli and vaginal E. coli colonization remained i

48 le to displace pre-coated vaginal protective lactobacilli and we hypothesize this to be a trigger for

49 ensal gut bacteria (e.g., bifidobacteria and lactobacilli) and increase the abundance of enterobacter

50 nderstood, and although mutans streptococci, lactobacilli, and A. naeslundii have been implicated in

51 onserved in streptococci, staphylococci, and lactobacilli, and are required for bacterial biofilm for

52 totoxicity, lack of activity against vaginal lactobacilli, and effectiveness against both HSV-2 and H

53 s, salivary log10 mutans streptococci, log10 lactobacilli, and fluoride level, did not represent stat

54 olved in fatty acid biosynthesis, amounts of lactobacilli, and saturated LCFA were measured in fecal

55 vels of Bifidobacteria, mutans streptococci, lactobacilli, and yeasts were correlated with each other

56 genous microbes that stably colonize a host, lactobacilli appear to be planktonic, opportunistic sett

57 Lactobacilli are a diverse group of species that occupy

58 h an associated decrease in caries, if these lactobacilli are fed to rats in an established caries mo

59 Lactobacilli are found in a wide variety of habitats.

60 Lactobacilli are low-virulence, commensal organisms of t

61 en who are colonized with H(2)O(2)-producing lactobacilli are more likely to maintain a normal vagina

62 Lactobacilli are non-spore forming, lactic acid producin

63 Lactobacilli are part of the normal bacterial flora of t

64 Bifidobacteria and lactobacilli are purportedly beneficial to human health

65 sufficient data exist to support the use of lactobacilli as candidates for the development of new or

66 by churning the cream that was fermented by lactobacilli at 37 degrees C for 24 h.

67 bserved in levels of mutans streptococci and lactobacilli at all time-points.

68 In multivariate analysis, only lactobacilli bacterial counts (P=.006; inverse associati

69 lationship between vaginal colonization with lactobacilli, bacterial vaginosis (BV), and acquisition

70 Combinations of Lactobacilli, Bifidobacteria, and Streptococcus salivari

71 ffect vaginal colonization by H2O2-producing lactobacilli but that such use may promote loss of non-H

72 udies have shown that oral administration of lactobacilli can be an efficient approach to treat lacta

73 Lactobacilli can lead to bacteremia and liver abscesses

74 MDCs activated with lactobacilli clearly skewed CD4(+) and CD8(+) T cells to

75 Lactobacilli colonizing the rectum may be a reservoir fo

76 0%), and L. gasseri (10%) were the prevalent lactobacilli colonizing the rectums of 290 females.

77 ophilus CRL 1014 on microbial metabolism and lactobacilli community composition for improving human h

78 The DGGE obtained showed changes in the lactobacilli community from the colon compartments of th

79 nificantly with any of the preparations, but lactobacilli concentrations decreased transiently.

80 adic reported cases of infections related to lactobacilli containing probiotics.

81 L. monocytogenes infection by treatment with lactobacilli correlates with a decrease in host gene exp

82 obacilli in the vagina, suggest that vaginal lactobacilli could reacidify the vagina at the rate obse

83 ein concentrations were linked to changes in lactobacilli counts (P < 0.05, R(2) = -0.33 for the mode

84 12 months, the proportion with H2O2-positive lactobacilli decreased (n = 32; 53% vs 27%; P = .03).

85 However, lactobacilli decreased by > or = 10(2) cfu/mL in 9 (56%)

86 lenged IL-10-deficient murine colitis model, lactobacilli demonstrated probiotic effects by direct mo

87 The proportion positive for H2O2+ or H2O2- lactobacilli did not change significantly with any of th

88 Absence of lactobacilli did not increase acquisition of VVC.

89 rmal Nugent score at all visits had a stable lactobacilli dominated microbiota with prevailing Lactob

90 with BV compared with women with a "normal" lactobacilli-dominated microbiota.

91 pported by the observation of persistence of lactobacilli during and after treatment of CDI.

92 Proteinase K treatment of methanol-fixed lactobacilli eliminated the inhibitory effect, suggestin

93 Diabetic rats were fed daily with human lactobacilli engineered to secrete GLP-1(1-37).

94 PCRs to detect bifidobacteria, bacteroides, lactobacilli, Escherichia coli, Clostridium difficile, a

95 have shown nonoxynol-9 (N-9) to be toxic to lactobacilli, especially to strains that produce H2O2.

96 ilm community, and successfully compete with lactobacilli for dominance in the vaginal environment.

97 ial efficacy of an anti-ICAM Ab delivered by lactobacilli for use as an anti-HIV-1 microbicide.

98 the assertion from other investigators that lactobacilli found in the GI tract originate in the oral

99 The absence of lactobacilli from the vaginal flora was associated with

100 Lactobacilli, Gardnerella vaginalis, and Mycoplasma homi

101 HIV RNA, lactobacilli, Gardnerella vaginalis, and Mycoplasma homi

102 Pectic oligosaccharides also enhanced lactobacilli growth during mixed batch faecal fermentati

103 Exploiting lactobacilli has been complicated by metabolic diversity

104 ural explanation for the mechanisms in which lactobacilli have adapted to their host niche by maximiz

105 Lactobacilli have been associated with dental caries for

106 uated the hypothesis that women colonized by lactobacilli have decreased acquisition of vaginal infec

107 The effect that vaginal lactobacilli have on trichomoniasis is not known.

108 Thus, vaginal lactobacilli have the ability to inhibit gonococci at tw

109 sociated with lack of vaginal H2O2-producing lactobacilli (hazard ratio [HR] = 4.0, P < .001) or pres

110 .001) or presence of only non-H2O2-producing lactobacilli (HR = 2.2, P = .02).

111 otal levels of LCFA correlated with those of lactobacilli in fecal samples from patients with active

112 are necessary for sustained colonization of lactobacilli in humans: 1) a stagnant, retentive niche t

113 s emphasize a potentially important role for lactobacilli in modulating immunological functions of DC

114 tobacillus after using probiotics containing lactobacilli in the course of her treatment of Clostridi

115 foundation for further exploring the role of lactobacilli in the ecological dynamics of vaginal micro

116 and Lactobacillus jensenii, the most common lactobacilli in the female genital tract, inhibit gonoco

117 ed how glycogen could be utilized by vaginal lactobacilli in the genital tract.

118 ially fermented by mainly bifidobacteria and lactobacilli in the human gut.

119 s pronounced differences were observed among lactobacilli in the induction of IL-6 and IL-10.

120 to reflect on the health implications of the lactobacilli in the mouth and downstream GI and to ponde

121 iginate in the oral cavity by proposing that lactobacilli in the oral cavity arise from caries lesion

122 ined with an estimate of the total number of lactobacilli in the vagina, suggest that vaginal lactoba

123 High numbers of lactobacilli in the vaginal tract have been correlated w

124 terial counts (G. vaginalis, M. hominis, and lactobacilli) in our model improved the sensitivity and

125 ict failure to become colonized by probiotic lactobacilli include exposure to semen, vaginal intercou

126 heir selective effects on bifidobacteria and lactobacilli, influence many aspects of bowel function t

127 m scores were recorded at baseline and after lactobacilli ingestion.

128 capacity for biotransformations catalysed by lactobacilli is an untapped biotechnology resource.

129 differentiation of group B streptococci from lactobacilli is not possible, but lactobacillus cells al

130 cterial microbiome of the stomach, including lactobacilli, is vital in promoting colonization resista

131 Vaginal lactobacilli isolated from 215 sexually active women wer

132 Vaginal lactobacilli may be associated with trichomoniasis in wo

133 Thus, absence of H2O2-positive lactobacilli may be important in the pathogenesis of rec

134 Absence of H2O2-producing lactobacilli may contribute to the development of cervic

135 We hypothesize that commensal lactobacilli may enhance growth of gonococci in vivo by

136 een suggested that vaginal colonization with lactobacilli may reduce the risk of vulvovaginal candidi

137 at predict sustained colonization by vaginal lactobacilli, microbiologic, behavioral, and demographic

138 ed Atopobium vaginae, Gardnerella vaginalis, lactobacilli, Mycoplasma hominis, and the human albumin

139 en, vaginal intercourse, and the presence of lactobacilli of the same species at enrollment.

140 During follow-up, absence of vaginal lactobacilli on culture was associated with an increased

141 to measure the effects of H(2)O(2)-producing lactobacilli on gonococcal infection.

142 ection was adapted to examine the effects of lactobacilli on gonococcal interactions with endometrial

143 onectin and casein, as did CbpG expressed on lactobacilli or as a purified full-length or truncated r

144 P < .001) and with absence of H2O2-positive lactobacilli (OR, 2.9; P = .04).

145 ngs the colonization rates at age 5 weeks of lactobacilli (P < .001) and bacteroides (P = .02) increa

146 emales colonized by other H(2)O(2)-producing lactobacilli (P < .001).

147 increased the bifidobacterial (P < 0.01) and lactobacilli (P < 0.001) populations but significantly d

148 loss of colonization with H(2)O(2)-producing lactobacilli (P=.018), as was antibiotic use (P< or =.00

149 In particular, elevated levels of lactobacilli physically displaced and attenuated C. rode

150 tide-1 content as well as Bifidobacteria and Lactobacilli populations in the caecum.

151 into 2 groups: those with H(2)O(2)-producing lactobacilli present (n=191; 20.2%) and those with H(2)O

152 Lactobacilli present in caries lesions represent both a

153 ined the ability of G. vaginalis to displace lactobacilli previously adhered to HeLa cells.

154 guished CTV-05 from other endogenous vaginal lactobacilli prior to and after vaginal capsule use.

155 ver, until now, most research has focused on lactobacilli probiotics.

156 ever, coliform colonization occurred whether lactobacilli produced H2O2 or not.

157 t the hypothesis that H2O2-producing vaginal lactobacilli protect against acquisition of BV but do no

158 iological studies suggest H(2)O(2)-producing lactobacilli protect women against gonorrhea.

159 estinal colonization by TNF-alpha-inhibitory lactobacilli reduced intestinal inflammation in H. hepat

160 Priming with live lactobacilli resulted in diminished granulocyte recruitm

161 itially colonized only by non-H2O2-producing lactobacilli resulted in loss of vaginal lactobacilli (R

162 OS promoted the growth of bifidobacteria and lactobacilli, resulting in high levels of short chain fa

163 ing lactobacilli resulted in loss of vaginal lactobacilli (RR, 2.5; 95% CI, 1.2-5.3).

164 V and promotion of vaginal colonization with lactobacilli should be evaluated as potential interventi

165 Lactobacilli species are considered non-pathogenic organ

166 vable microbes, including oral streptococci, lactobacilli, Streptococcus mutans, and Candida, in sali

167 We also demonstrate that commensal lactobacilli support the growth of N. gonorrhoeae in vit

168 udies indicate that probiotics, particularly lactobacilli, suppressCandidagrowth and biofilm developm

169 detection of H(2)O(2) by a broader range of lactobacilli than a published, widely used agar formulat

170 P < .001) and in women without H2O2-positive lactobacilli than in women with (odds ratio [OR], 4.0; P

171 ntain a normal vaginal flora than women with lactobacilli that do not produce H(2)O(2).

172 We identified new shuffled lactobacilli that grow at substantially lower pH than do

173 esence of novel CRISPR-Cas immune systems in lactobacilli that may be exploited for genome editing.

174 Commensal lactobacilli that produce hydrogen peroxide (H(2)O(2)) i

175 Since strains of lactobacilli that produce hydrogen peroxide inhibit the

176 The effect of adding the cells of four lactobacilli to a squid pen powder (SPP)-containing medi

177 longitudinal loss or acquisition of vaginal lactobacilli used as probiotics.

178 Heavy growth of lactobacilli was associated with a lower frequency of va

179 the vagina and rectum by H(2)O(2)-producing lactobacilli was associated with the lowest prevalence o

180 s can naturally or therapeutically encounter lactobacilli, we investigated the effects of several wel

181 Furthermore, lactobacilli were able to displace adherent gonococci fr

182 females, vaginal and rectal colonization by lactobacilli were assessed by culture methods.

183 Low levels of vaginal lactobacilli were associated with trichomoniasis (odds r

184 Lactobacilli were detected in 64% of subjects, and 42% o

185 No lactobacilli were detected in the lungs of wild-type (WT

186 Salivary levels of mutans streptococci and lactobacilli were enumerated at baseline, 3, 6, and 12 m

187 ariable fragment Abs secreted by transformed lactobacilli were evaluated for their protective efficac

188 The longevity-promoting benefits of lactobacilli were hypothesized as early as 1907.

189 Lactobacilli were identified on the basis of DNA homolog

190 A total of 272 isolates of lactobacilli were identified to the species level by use

191 Vaginal and rectal lactobacilli were identified to the species level by usi

192 Women with reduced lactobacilli were less likely to regain normal flora tha

193 Levels of S. mutans were positively and Lactobacilli were negatively associated with caries.

194 Lactobacilli were observed to be a microbiome component

195 Vaginal lactobacilli were present in 62% of the women and were s

196 Murine-derived probiotic lactobacilli were selected in vitro for their ability to

197 alis and M. hominis and decreasing levels of lactobacilli were significantly associated with BV by Nu

198 The predominant lactobacilli were the species Lactobacillus crispatus (3

199 to regain normal flora than were those whose lactobacilli were unaffected.

200 Lactobacilli, when targeted to the respiratory epitheliu

201 biotics, or by endogenous bifidobacteria and lactobacilli, whose metabolic activity and growth may al

202 ; and (2) that toxicological assays of these lactobacilli will show them to be benign.

203 which is indicative of DC maturation, those lactobacilli with greatest capacity to induce IL-12 were

204 tobacillus acidophilus BG2FO4 is a strain of lactobacilli with properties of marked intestinal adhere

205 t juice appeared to support the viability of lactobacilli, with higher microorganism numbers observed

206 ontained mainly streptococci, staphylococci, lactobacilli, yeasts, and enterobacteria.