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1 rone, placental growth factor, and placental lactogen.
2 es: prolactin, growth hormone, and placental lactogen.
3 genic hormones prolactin (PRL) and placental lactogens.
4 GH genes include placental GHs and placental lactogens.
6 t transgenic mice expressing mouse placental lactogen-1 (mPL1) in beta cells under the rat insulin II
7 pment with decreased expression of Placental lactogen-1 and -2 (Pl1 and Pl2) and increased expression
8 in, estradiol, progesterone, human placental lactogen, and prolactin) were measured in conjunction wi
9 in (PRL), growth hormone (GH), and placental lactogen are expressed by endothelia and have angiogenic
10 owing fetus, whereas prolactin and placental lactogen counterbalance this resistance and prevent mate
15 ion of the placental hormone human placental lactogen (hPL), experiments were performed to determine
16 lacentas lacking GATA-3 accumulate placental lactogen I and proliferin mRNAs to a level 50% below tha
17 ATA-2 gene had a similar effect on placental lactogen I expression, but led to a markedly greater red
18 ulate transcription from the mouse placental lactogen I gene promoter in a transfected trophoblast ce
19 tors regulate transcription of the placental lactogen I gene, as well as the related proliferin gene,
20 P-C, decidual/trophoblast PRP, and placental lactogen I variant, only which are expressed in the spon
22 ot directly involved in cAMP metabolism, the lactogen-induced increase in cAMP was most likely due to
24 Bcl-XL-specific siRNA significantly inhibits lactogen-mediated protection against DEX-induced beta ce
25 ve this is the first direct demonstration of lactogens mediating their protective effect through the
28 s (hepatocyte growth factor [HGF], placental lactogen, or parathyroid hormone-related protein) have b
32 rder to explore the role of murine placental lactogen (PL)-I (mPL-I) in islet mass regulation in vivo
35 her, we identify the mechanism through which lactogens protect beta cells against DEX-induced death.
39 re we show that serotonin acts downstream of lactogen signaling to stimulate beta cell proliferation.
42 ed glucose-stimulated insulin secretion from lactogen-treated islets could be accounted for by increa
43 in the upregulation of islet function after lactogen treatment, we examined the relationship between
44 e-related peptide (PTHrP) or mouse placental lactogen type 1 (mPL1) in pancreatic beta-cells, using t
45 sely related to growth hormone and placental lactogen with properties and functions resembling both a
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