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1 mber, secretory capacity, and sensitivity to lactogenic hormones.
2 the milk protein beta-casein in response to lactogenic hormones.
3 iates in response to a basement membrane and lactogenic hormones.
4 lveolar PCD even in the presence of systemic lactogenic hormones.
5 ultivation with the combination of the three lactogenic hormones.
6 dered receptor binding for each of the three lactogenic hormones.
8 ctivation of signal transduction pathways by lactogenic hormones and cell-substratum interactions act
10 nstrated that AFAP1 responds to prolactin, a lactogenic hormone, by forming a complex with cSrc and b
11 kling, the consequent decline in circulating lactogenic hormone concentrations initiates remodeling o
12 ot functionally differentiate in response to lactogenic hormones despite their organization into thre
13 However, the exact mechanisms by which the lactogenic hormones drive beta cell expansion remain unc
15 se was disrupted in the presence of systemic lactogenic hormones: (i) sealing of the teats, (ii) mamm
19 GADD153 expression was upregulated by the lactogenic hormones insulin and progesterone and associa
20 mouse mammary GPT gene is stimulated by the lactogenic hormones, insulin, glucocorticoid, and prolac
21 duction, lactation, and immune function, the lactogenic hormones likely play roles in tissue differen
22 n increased cell proliferation and inhibited lactogenic hormone-mediated differentiation as revealed
26 val, and function and mediated mainly by the lactogenic hormones prolactin (PRL) and placental lactog
27 old and 12.5-fold, respectively, whereas the lactogenic hormone, prolactin (PRL) stimulated a 3.5-fol
29 ng of ordered binding to include three human lactogenic hormones: prolactin, growth hormone, and plac
32 a transgenic mouse model engineered so that lactogenic hormones stimulate a sustained increase in eI
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