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1 its expression from a second cell type, the lactotrope.
2 itary dwarfism and a complete loss of mature lactotropes.
3 n of PRL synthesis and cell proliferation in lactotropes.
4 on PRL production and cell proliferation in lactotropes.
5 erm repression of the growth hormone gene in lactotropes.
6 , which induce PRL gene expression in mature lactotropes.
7 of PRL transcription in fully differentiated lactotropes.
8 ctive extinction of hGH expression in mature lactotropes.
9 the precursors of prolactin (PRL)-producing lactotropes.
10 eneral lack of thyrotropes, somatotropes and lactotropes.
11 oduction and cell proliferation of pituitary lactotropes.
12 thanol's inhibitory action on Gi3 protein in lactotropes.
13 nction leads to the opposite result, loss of lactotrope and thyrotrope cell specification, and an inc
15 proteins and cell proliferation in enriched lactotropes and lactotrope-derived PR1 cells containing
16 DAPT treatment leads to increased numbers of lactotropes and loss of corticotropes in the anterior pa
17 n factor Pit-1 is expressed in somatotropes, lactotropes, and thyrotropes of the anterior pituitary.
19 inally differentiated dorsal somatotrope and lactotrope cell types and a marked increase in the expre
20 e, thyrotrope, somatotrope, corticotrope and lactotrope cells in the anterior lobe and the intermedia
22 pes-gonadotropes, thyrotropes, somatotropes, lactotropes, corticotropes, and melanotropes-appear to b
23 ll proliferation in enriched lactotropes and lactotrope-derived PR1 cells containing various D2 recep
24 F-2 could play a key role both in initiating lactotrope differentiation and maintaining PRL expressio
29 ors required for thyrotrope, somatotrope and lactotrope ontogeny, but their relative roles are differ
31 ive Lhx3 genes are deficient in gonadotrope, lactotrope, somatotrope, and thyrotrope pituitary cells.
34 ts indicate that hst overexpression mediates lactotrope tumor growth and potently stimulates PRL synt
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