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1 through the protrusion of the actin-enriched lamella.
2 esions at localized regions of the advancing lamella.
3 ransmission electron microscopy image of the lamella.
4 the sensory epithelium within each olfactory lamella.
5 ic organization more typical of a fibroblast lamella.
6 ll periphery and at dynamic spots within the lamella.
7 ars to cause MT buckling and breaking in the lamella.
8 o depolymerize into the field of view at the lamella.
9 .4 mircon/min rate of retrograde flow in the lamella.
10 olocalizes with actinin4, is present at each lamella.
11 chains of pectin in the cell wall and middle lamella.
12 lls and is especially abundant in the middle lamella.
13 tion of FA growth at the leading edge of the lamella.
14 myosin activity and absence of a contractile lamella.
15 be generated about focal adhesions in a thin lamella.
16 d style, most likely by weakening the middle lamella.
17 s in the cell body but bind along MTs in the lamella.
18 assembly of the primary cell wall and middle lamella.
19  myosin phosphorylation within the spreading lamella.
20 F-actin) networks, the lamellipodium and the lamella.
21 layer is constrained to within an individual lamella.
22 ited higher affinity for disk rims than disk lamella.
23 ngate shape with narrow protrusions and wide lamellas.
24 09 hours (range, 96-630 hours) for posterior lamellas.
25  graft consisted of a large anterior stromal lamella (9.0 mm in diameter and +/- 250 mum in thickness
26 ce of the cell, indicating continuity of the lamella across the outer wall.
27                                       In the lamella, all marked and observed actin filaments remain
28                 On the dorsal surface of the lamella, an optical gradient trap measured rearward forc
29 lly in the control of both the angle between lamella and backbone axes and the angle between surface
30 imate boundary between the lamellipodium and lamella and continued to grow as they were swept back to
31 sults show MLCK is a myosin regulator in the lamella and contractile ring, and pinpoints sites where
32 t structures besides lamellipodia, including lamella and filopodia, may have unappreciated roles in c
33 cells were more motile and had more abundant lamella and filopodia.
34 lear region generates rearward forces in the lamella and forward forces in the cell rear.
35 ing edge ruffling and retrograde flow in the lamella and lamellipodia.
36 tire microtubule lattice in the leading edge lamella and lamellipodium.
37 ow that distribution of most proteins in the lamella and PM domains is preserved even in the absence
38 from different regions of a single olfactory lamella and simultaneously from widely separated lamella
39 istinct accumulations of LFA-1-ICAM-1 in the lamella and TCR-MHC in the uropod, consistent with a mot
40 n the transition zone between the peripheral lamella and the cell body, a subset of MTs remains stati
41 esponsible for the degradation of the middle lamella and the loosening of the primary cell wall surro
42 in mechanical stiffening of both the leading lamella and the perinuclear region of motile cells.
43 ced in the root and stem cortex and the leaf lamella and trichomes in response to heavy metal stress.
44 08 hours (range, 108-678 hours) for anterior lamellas and 339 +/- 109 hours (range, 96-630 hours) for
45 a function resulted in significantly smaller lamellas and decreased process number, length, and branc
46 their growth cones had significantly smaller lamellas and reduced levels of F-actin in vitro.
47  periphery as extended by smaller individual lamella, and a newly discovered whole-interface actin-dr
48  in the outer segment: plasma membrane, disk lamella, and disk rim.
49 , our results suggest that the unique folded lamella architecture of the cone OS may maximize density
50 and that approximately 80% of the MTs in the lamella are not centrosome bound.
51 re more concentrated in the cell wall-middle lamella area of the parenchyma cells.
52 ted motoneurones had RER with a more ordered lamella arrangement than controls.
53 or MLCK-mediated myosin contractility in the lamella as a driving force for migration.
54 rence of the microtubule cytoskeleton in the lamella as compared with the cell body and provide the f
55 ain its localization just behind the leading lamella as PMNs migrate, indicating that membrane recycl
56 reate the proximal surface of an evaginating lamella, as well as membrane protrusions that extend bet
57 e the acquisition of cryo-ET data within FIB-lamellas at specific locations, unambiguously identified
58 s to produce enzymes that degrade the middle lamella between cells in the AZ.
59 ction is limited by mass transfer across the lamella boundaries.
60 lusters at the mucosal tip of each olfactory lamella but scattered in the neuroepithelial region.
61 rils oriented in a common direction within a lamella but varying by ~30 to 90 degrees between adjacen
62 nt from the highly lignified compound middle lamella, but xylan occurred throughout the cell walls of
63                                       In the lamella, cell body, and tail there are two observable ty
64 lidated by adhesion of a cotton fiber middle lamella (CFML).
65 s were small and rearward directed under the lamella, changed direction in front of the nucleus, and
66  the volume represented within a typical FIB lamella constitutes a small fraction of the biological s
67 ) transplantation of a 9-mm anterior corneal lamella cut by microkeratome-assisted dissection (400-mu
68                                  The leading lamella develops no detectable propulsive traction, even
69               Microfibril direction within a lamella did not change gradually or abruptly across the
70 bition of Paks abolished F-actin flow in the lamella, displaced myosin IIA from the cell edge, and de
71 scence in Kanzi apples was not due to middle lamella dissolution, as tissue failure still occurred by
72            MTs are excluded from the leading lamella during polarization and motility, but treatment
73 its function in modulating lamellipodium and lamella dynamics, and the implications of these dynamics
74 tions of Arp2/3 and ADF/cofilin, whereas the lamella exhibits spatially random punctae of F-actin ass
75              We propose that the coupling of lamella extensions to fluctuating rearward tractions in
76 y and reversibly blocked protrusion-mediated lamella formation and chemotaxis.
77 f proteins involved in actin dynamics during lamella formation in Drosophila S2 cells.
78  90 proteins implicated in actin function on lamella formation in Drosophila S2 cells.
79 ent properties, beta-actin localization, and lamella formation in motile cells.
80                 Actin arginylation regulates lamella formation in motile fibroblasts, but the underly
81                              We propose that lamella formation may reduce tension building at cell-ce
82 r the rho kinase inhibitor, Y-27632, blocked lamella formation, myosin phosphorylation within the pro
83 d filopodial formation and stabilization and lamella formation.
84                Top coats were applied to the lamella-forming block copolymers poly(styrene-block-trim
85              This method produces magic size lamella, having a well-spaced discrete melting point, Tm
86 on that paved the way for an adhesive middle lamella in multicellular land plants.
87 spatial interaction of the lamellipodium and lamella in response to upstream signals.
88 blasts in the body correlates with epidermal lamella invasion and subsequent adult skin differentiati
89 of living cells, whereas ER removal from the lamella is powered by actomyosin-based retrograde flow o
90 very thin amorphous layer on the crystalline lamella, just sufficient to accommodate a loop, but like
91 FA-1 that includes talin and encompasses the lamella/lamellipodial interface as well as further back
92 sed by oligodendrocytes and localizes to the lamella leading edge where actin polymerization is activ
93 of wound healing, with reduced appearance of lamella-like membrane protrusions at the cell leading ed
94                           MTs in the leading lamella move rearward relative to the substrate, as has
95                             GSK3beta-induced lamella MT destabilization was partially rescued by expr
96  constitutively active GSK3beta destabilizes lamella MTs by disrupting lateral MT interactions with t
97                             The plus ends of lamella MTs persist in growth perpendicular to the leadi
98 both plus end tracking and association along lamella MTs, we show that partial phosphorylation of the
99 sed spatial overlap of the lamellipodium and lamella networks and reduced cell-edge protrusion effici
100 al surface),7.0 mm in diameter, with a donor lamella obtained by microkeratome-assisted dissection, p
101 anism by which caspase 8 is recruited to the lamella of a migrating cell, promoting cell migration in
102 icate that ColXVII-actinin4 complexes at the lamella of a moving keratinocyte regulate actin dynamics
103 lls, MT turnover is increased twofold in the lamella of HGF-treated cells but unchanged in the retrac
104 ase (MRCK) has been shown to localize to the lamella of mammalian cells through its interaction with
105                    This contrasts MTs in the lamella of migrating cells at the noncontacted leading e
106                 MLCK is highly active in the lamella of migrating cells, but not at the retracting ta
107  flow rearward as occurs in the leading edge lamella of migrating cells.
108 olic distribution, caspase 8 is recruited to lamella of migrating cells.
109 radial (dorsal) stress fibers at the leading lamella of migrating renal epithelia.
110  apparent apoptosis in and under the cornoid lamella of PMVK-deficient lesional tissues, with incompl
111 ort a systematic study of melting of layered lamella of silver alkanethiolates (AgSCn).
112 ting the nucleus by envelopment at the inner lamella of the nuclear membrane.
113  lamella of the principal olive, the ventral lamella of the principal olive, and the rostral half of
114 ents from parts of, respectively, the dorsal lamella of the principal olive, the ventral lamella of t
115 termined scintigraphically ex vivo in a 1-cm lamella of the resected tumorous kidney.
116 omposite has been developed, wherein stacked lamellas of 1D vanadium pentoxide nanofibres, intercalat
117 ropulsive forces generated by the keratocyte lamella on both the ventral and the dorsal surfaces.
118  integrates the trajectories and dynamics of lamella open margin lattice components.
119 in the bulk of the film, consistent with the lamella orientation observed by GIWAXS, a more "edge-on"
120 aulic response through size change of middle-lamella pectins.
121 ted endothelial keratoplasty using posterior lamella prepared with a 300-mum head microkeratome (Mori
122  retrograde fluxes at focal adhesions in the lamella region.
123 2) is critical for the formation of the disc/lamella rim in photoreceptor outer segments (OSs), but p
124  spectroscopic analysis that is performed on lamella samples.
125 iate in fruiting bodies illuminated from the lamella side, in sliced fruiting bodies, and in the stip
126 llagen XVII (ColXVII) is found in actin-rich lamella, situated behind the lamellipodium.
127 lls showed altered localization of a leading lamella-specific marker, talin, and a uropod-specific ma
128 eserved even in the absence of RDS, rim, and lamella structures.
129  the notochord and lie beneath the epidermal lamella (subepidermal fibroblasts).
130 in bundle retrograde movement at the site of lamella, such that actin bundle movement is enhanced mor
131 he dorsal (0.4 nN/microm(2)) surfaces of the lamella, suggesting that dorsal matrix contacts are as e
132  which are regularly connected by pillars as lamella support.
133 mbly at adhesions but retained a contractile lamella that generated large tension on adhesions.
134 ary cell wall and are abundant in the middle lamella that holds plant cells together.
135 ely fluorescently labeled lipid droplets, in lamellas that are 300 nm thick.
136 ion of the corresponding "twin" granule cell lamella, thereby lateralizing and amplifying the influen
137 can move through the crystal as evidenced by lamella thickening without disturbing the crystalline or
138 metry shows stepwise increases in Tm, as the lamella thickness increases by integer increments of cha
139                              Here, we show a lamella-to-toroid transition, captured through the disso
140 ide an excellent thermal stability for these lamellas up to 350 degrees C.
141                       Anterior and posterior lamella was obtained using 60 kHz and 150 kHz FS laser.
142 n in the apical cortex is transmitted to the lamella where force-sensitive FAs start to grow.
143 ead increased in the thickened region of the lamella where myosin condensation has been observed.
144 h stimulation both within and outside of the lamella where the mossy cell ablation took place.
145 ction of the second colocalized network, the lamella, where actomyosin contraction was integrated wit
146 ium lying along the proximal portion of each lamella, where it attaches to the midline raphe.
147  proteins affect the integrity of the middle lamella, which controls cell-to-cell adhesion and thus i
148 and elaborate architecture of equally spaced lamellas, which are regularly connected by pillars as la
149 ed by an elongate cell body with an anterior lamella whose cell edge is divided into protrusion-formi
150 reorganizes into a concentric lamellipod and lamella with retrograde actin flow that helps regulate t

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