ライフサイエンスコーパス: lamina cribrosa

1 tinal layers, optic nerve head, choroid, and lamina cribrosa.

2 ost were located immediately anterior to the lamina cribrosa.

3 umatically induced optic nerve damage at the lamina cribrosa.

4 issues, such as the peripapillary sclera and lamina cribrosa.

5 reactivity, and increased thickening of the lamina cribrosa.

6 nt visualization of the choroid, sclera, and lamina cribrosa.

7 st posterior to the retina, analogous to the lamina cribrosa.

8 ts contribution to increased rigidity of the lamina cribrosa.

9 nally passing through the full extent of the lamina cribrosa.

10 erfusion of the neurons as they traverse the lamina cribrosa.

11 anent) and hypercompliant deformation of the lamina cribrosa and anterior scleral canal wall are pres

12 The anterior lamina cribrosa and its continuity with the prelaminar g

13 e-dependent effects cause bowing back of the lamina cribrosa and optic disc cupping.

14 thinning, stretching, and deformation of the lamina cribrosa and peripapillary sclera that are minima

15 d the anterior and posterior surfaces of the lamina cribrosa and peripapillary sclera were delineated

16 e scleral foramen (the mouse does not have a lamina cribrosa), and exited the inferior retrobulbar op

17 abled improved visualization of the choroid, lamina cribrosa, and sclera.

18 In the lamina cribrosa, ANGPTL7 expression was associated with

19 rmanent posterior deformation of the central lamina cribrosa, as well as expansion of the anterior an

20 authors aimed to study the potential role of lamina cribrosa astrocytes as a component of activated i

21 Although activation of lamina cribrosa astrocytes has been identified in glauco

22 by IL-10 was approximately 6 times higher in lamina cribrosa astrocytes incubated under simulated isc

23 gamma induced HLA-DR expression in brain and lamina cribrosa astrocytes, up to 25-fold, (P < 0.001) e

24 rogresses along the optic nerve to reach the lamina cribrosa by P34, coincident with the time of eye

25 ng the retinal schisis cavity and gap in the lamina cribrosa corresponding to the optic pit.

26 intracranial pressure (ICP) at the level of lamina cribrosa could have important implications for un

27 e anomalies is the presence of a scleral (or lamina cribrosa) defect permitting anomalous communicati

28 In glaucoma patients, lamina cribrosa depth changes are detected with similar

29 To determine whether: (1) change in lamina cribrosa depth occurs more frequently than change

30 Lamina cribrosa depth should be measured from an anterio

31 The authors predicted the lamina cribrosa displacement (LCD), scleral canal expans

32 To estimate the pressures at the lamina cribrosa, geometrical distances were estimated fr

33 he translaminar pressure gradient across the lamina cribrosa, has been reported in glaucoma patients.

34 he major lysyl oxidase isoform in the normal lamina cribrosa in association with a complex elastic fi

35 disc drusen and their relationship with the lamina cribrosa in vivo.

36 ifically within the peripapillary sclera and lamina cribrosa) in response to intraocular pressure (re

37 Posterior migration of the lamina cribrosa is a component of early cupping in monke

38 ss, strain, and geometric deformation of the lamina cribrosa (LC) and peripapillary sclera.

39 ogeneous material properties defined for the lamina cribrosa (LC) based on local connective tissue vo

40 e associated alterations in the phenotype of lamina cribrosa (LC) cells are implicated in changes occ

41 human optic nerve head (ONH) astrocytes and lamina cribrosa (LC) cells express BMP and BMP receptor

42 g the potential clinical importance of focal lamina cribrosa (LC) defects as a characteristic structu

43 The properties of the lamina cribrosa (LC) greatly influenced its response to

44 gions comparable to the human prelaminar and lamina cribrosa (LC) in the optic nerve head and the ret

45 essel shift inside the ONH occurs within the lamina cribrosa (LC) or the prelaminar tissue.

46 We assessed in vivo lamina cribrosa (LC) position within the optic nerve hea

47 an increased pressure difference across the lamina cribrosa (LC) related to a low intracranial press

48 Deep ONC structures, including the lamina cribrosa (LC), short posterior ciliary artery (SP

49 y segmented (prelamina, choroid, sclera, and lamina cribrosa [LC]).

50 visualization of the porous structure of the lamina cribrosa, nerve fiber layer, choroid, photorecept

51 ared with normal contralateral controls, the lamina cribrosa of eyes with elevated IOP exhibited mark

52 border tissue of Elschnig, Bruch's membrane, lamina cribrosa, optic nerve septa, pial sheath, and vas

53 The ability to consistently resolve lamina cribrosa pores in vivo has applications in the st

54 the ONH tissues in all 17 monkeys, anterior lamina cribrosa position, laminar thickness, and scleral

55 y between retinal schisis and the gap in the lamina cribrosa present in the optic disc pit, supportin

56 Trans-lamina cribrosa pressure difference (TLCPD) was calculat

57 The trans-lamina cribrosa pressure difference (TLCPD) was calculat

58 ressure, leading to an abnormally high trans-lamina cribrosa pressure difference, could result in bar

59 iven that the biomechanics of the sclera and lamina cribrosa probably influence retinal ganglion cell

60 udoexfoliation-specific elastinopathy of the lamina cribrosa resulting from a primary disturbance in

61 y the anterior and posterior surfaces of the lamina cribrosa, scleral flange, and peripapillary scler

62 E), neural canal opening (NCO), and anterior lamina cribrosa surface (ALCS) by using custom software.

63 um, neural canal opening (NCO), and anterior lamina cribrosa surface (ALCS) were delineated using cus

64 and the ONH surface depth (ONHSD), anterior lamina cribrosa surface depth (ALCSD), and prelaminar ti

65 iridocorneal angle, posterior bowing of the lamina cribrosa, swelling and loss of large retinal gang

66 laminar deformation, neural canal expansion, lamina cribrosa thickening, and posterior (outward) bowi

67 Compared with normal and POAG specimens, lamina cribrosa tissues obtained from early and late sta

68 The anterior lamina cribrosa was consistently imaged in vivo in norma

69 Compared with the regional PID maximums, the lamina cribrosa was posteriorly deformed centrally, infe

70 s of the eight monkeys with IOP 0 mm Hg, the lamina cribrosa was posteriorly displaced and thicker an

71 Within the high-IOP early-glaucoma eyes, the lamina cribrosa was posteriorly displaced and thicker an

72 Within the high-IOP normal eyes, the lamina cribrosa was posteriorly displaced compared with

73 Reflectance images (840 nm) of the anterior lamina cribrosa were acquired using the AOSLO in four or

74 eripapillary sclera, scleral canal wall, and lamina cribrosa were present among the three normal eyes

75 iliary muscle, corneoscleral fibroblast, and lamina cribrosa) were cultured in 24-well plates in the

76 eyes, elastin appeared as fine fibers in the lamina cribrosa, without elastotic aggregates, and witho