1 ized particles permeate extensively into the lamina densa.

2 bril insertion or are present throughout the lamina densa.

3 rofibrils appear to insert directly into the lamina densa.

4 lacking either LN or LG domains and formed a lamina densa.

5 ed that morpholino-injected embryos lacked a lamina densa and lamina lucida at 24 hpf, and BM defects

6   The tissue separation in TBDN is below the lamina densa, and electron microscopy has revealed abnor

7  heritable skin disease manifesting with sub-lamina densa blistering, erosions, and chronic ulcers.

8     Anti-vWFA2 autoantibodies located at the lamina densa bound to the dermal side of salt-split skin

9 kin and localized to the lower lamina lucida/lamina densa by direct and indirect immunoelectron micro

10 brillin microfibrils might be present in the lamina densa, epithelial cell cultures (WISH, HaCaT, and

11 nt membrane appeared to be thickened and the lamina densa had an irregular appearance after treatment

12 ement membrane, as seen by a nearly complete lamina densa, hemidesmosomes, and the polarized, linear

13 d the presence of fibrillin-1 throughout the lamina densa in the dermal-- epidermal junction.

14 ha3 IV), the GBM is irregularly swollen, the lamina densa is absent, and permeation is increased.

15 n of in utero epidermal blisters beneath the lamina densa of the basement membrane and also in renal

16 cate that size-dependent permeation into the lamina densa of the GBM and the podocyte glycocalyx, tog

17  nanoparticles show that permeation into the lamina densa of the GBM is size-sensitive.

18                     Particles traversing the lamina densa tend to accumulate upstream of the podocyte

19                       TEM confirmed that the lamina densa was partly or completely absent along the a

20 ents and anchoring fibril connections to the lamina densa were more numerous compared with the compos

21 that both basement membrane proteins and the lamina densa were retained at the BMZ throughout healing