ライフサイエンスコーパス: large bowel

1 c expansion of Enterobacteriaceae within the large bowel.

2 erial antigens, and homogenates of small and large bowel.

3 patients the lesions involved both small and large bowel.

4 s tumorigenesis in juvenile polyposis of the large bowel.

5 ll intestine, but rarely was observed in the large bowel.

6 e for faecal pellet propulsion in the murine large bowel.

7 rical activity and/or contractions along the large bowel.

8 per colonoscopic examination of the proximal large bowel.

9 ), on recurrence of neoplastic polyps of the large bowel.

10 ide pouch that extends from the cecum of the large bowel.

11 te chemopreventive effect on adenomas in the large bowel.

12 n the pathogenesis of sporadic tumors of the large bowel.

13 in CD if the inflammation is limited to the large bowel.

14 aspirin has an antineoplastic effect in the large bowel.

15 the recurrence of adenomatous polyps in the large bowel.

16 uired for colonization of the suckling mouse large bowel.

17 of the OA(-) strains were maintained in the large bowel.

18 cells were also recovered from the cecum and large bowel.

19 ow that NSAIDs can lead to the regression of large bowel adenomas.

20 Participants with large-bowel adenomatous polyps diagnosed in the past 6 m

21 El Tor TCP(-) strain colonized the cecum and large bowel almost as well as the wild-type strain.

22 nts and scintigraphy in most segments of the large bowel and a negative correlation with the small bo

23 effect of dietary cellulose in the mammalian large bowel and highlight the potential role of dietary

24 ntly less damage in thymus, small bowel, and large bowel, but not in liver or skin tissues from recip

25 lian gut, (b) on biofilm distribution in the large bowel, (c) the association of lymphoid tissue with

26 foods was inversely related to incidence of large bowel cancer (adjusted relative risk 0.75 [95% CI

27 etween intakes of different PUFAs and distal large bowel cancer in a population-based case-control st

28 ed the association between NSAIDs and distal large bowel cancer in African Americans and whites, usin

29 tudy of patients who underwent resection for large bowel cancer in Maryland.

30 with those from the same dietary factors for large bowel cancer in this cohort.

31 s was associated with reduced risk of distal large bowel cancer in whites (multivariable odds ratios

32 AID use was inversely associated with distal large bowel cancer in whites (odds ratio (OR) = 0.60, 95

33 was associated with increased risk of distal large bowel cancer in whites, but not among African Amer

34 tios and 95% confidence intervals for distal large bowel cancer risk in relation to quartiles of PUFA

35 aenoic acids was inversely related to distal large bowel cancer risk, whereas the ratio of omega-6 to

36 e patients with excluded bowel tumor died of large bowel cancer within 2.4 years; by contrast, the ac

37 Of these, 23 subsequently died (disseminated large bowel cancer, 12; unrelated causes, 9; related cau

38 anti-inflammatory drugs have reduced risk of large bowel cancer.

39 The activity of oral UFT in large-bowel cancer when administered with oral LV (appro

40 Most large bowel cancers are moderately to well-differentiate

41 ectal adenomas, which are precursors of most large-bowel cancers.

42 Rag2-/- mice, rapidly developed colitis and large bowel carcinoma.

43 0.02) and a greater propensity for small and large bowel complications (overall: 9.0 vs. 2.6%; P< 0.0

44 ily history of colorectal cancer, history of large bowel conditions and symptoms, and previous colono

45 s between symptoms and changes in small- and large-bowel contents after oral challenge.

46 in ileocolonic inflammation and favored for large bowel disease.

47 r trigger host responses that cause small or large bowel diseases (such as enteroaggregative or enter

48 immunity are integral in the progression of large bowel diseases.

49 ny, detailed history and results of previous large-bowel endoscopies were obtained by interview and f

50 compared with participants without previous large-bowel endoscopy was assessed according to time sin

51 reviewed included distribution (small bowel, large bowel), extent (mild, moderate, extensive), and mo

52 beverages or different anatomic sites in the large bowel have been inconsistent.

53 istologically verified adenoma in the distal large bowel (ie, descending colon, sigmoid colon, or rec

54 ominantly expressed in the epithelium of the large bowel in individuals with HMPS.

55 e risk of recurrent neoplastic polyps of the large bowel in patients with a history of colorectal ade

56 The major motor functions of the large bowel include storage, propulsion and defecation.

57 ms and characteristic mucosal lesions of the large bowel (including pseudomembranous colitis) are des

58 e immunological target in the development of large bowel inflammation in IL-10(-/-) mice and argue th

59 aditionally, fecal leukocyte testing detects large bowel inflammation or disruption, conditions that

60 usly develop microbiota-driven, TNF-mediated large bowel inflammation that resembles human ulcerative

61 on should be strongly considered for limited large bowel inflammation.

62 f the prostate (IRR 3.46, 95% CI 1.25-9.59), large bowel (IRR 2.35, 95% CI 0.96-5.77), and lung (IRR

63 For most practical purposes, however, the large bowel is inaccessible for routine investigation, a

64 y into the host and adapts for growth in the large bowel is limited.

65 uced by fermentation of dietary fiber in the large bowel, it may be an important regulator of apoptos

66 Crps from mouse large bowel lumen were bactericidal in the low micromola

67 n persist in a functional state in the mouse large bowel lumen.

68 esis in multiple organ systems including the large bowel, lung, breast, and prostate.

69 dian, 131 v 90 mm; P < .0001), the length of large bowel (median, 314 v 206 mm; P < .0001), and ileum

70 Diaphragm disease and large bowel mesentery implants were the only CT predicto

71 downstream functional effects in the healthy large-bowel mucosa remain to be investigated.

72 tcomes after surgical treatment of malignant large bowel obstruction (MBO) and to identify risk facto

73 Subsequent symptoms of large bowel obstruction necessitated a left hemicolectom

74 e in the nonsurgical management of malignant large bowel obstruction.

75 rvival in patients presenting with malignant large bowel obstruction.

76 g was introduced for palliation of malignant large-bowel obstruction (MLBO) more than 20 years ago bu

77 , the imaging findings in multiple causes of large-bowel obstruction are illustrated and compared wit

78 Large-bowel obstruction is an abdominal emergency with h

79 dy performed in patients suspected of having large-bowel obstruction, it may not be sufficient to dis

80 gent enema may be used to confirm or exclude large-bowel obstruction.

81 it can establish the diagnosis and cause of large-bowel obstruction.

82 In the large bowel, p21WAF-1/CIP1 and p53 expression were obser

83 neration of migrating motor complexes in the large bowel, particularly in the mouse colon.

84 rmined the effects of wheat and oat brans on large-bowel physiology were fractionated by using a phys

85 independent predictors of ECF/EAF/IAS were a large bowel resection (adjusted odds ratio [AOR], 3.56 [

86 Large bowel resection, large-volume fluid resuscitation,

87 -hysterectomy vaginal cuff, and the small or large bowel, resulting in protrusion of the vagina, uter

88 onsumed constant diets to determine selected large-bowel, serum cholesterol and triacylglycerol, and

89 xpressed and is most abundant in the thymus, large bowel, small bowel, stomach, and prostate.

90 oxically low levels of HIV expression in the large bowel suggest that different processes drive HIV p

91 pose patients to anastomotic leak (AL) after large bowel surgery.

92 of specialist units, improved results after large-bowel surgery, and the demise of outmoded techniqu

93 stinal epithelial cells lining the small and large bowel, thus identifying apoptosis as the driving f

94 in suppressing urgency, prolonging small and large bowel transit and relieving symptoms in IBS-D.

95 CA formation and absorption, prolongation of large bowel transit is a pathogenic factor in the format

96 s were related to mouth-to-caecum (MCTT) and large bowel transit times (LBTTs) in 4 groups of 8 indiv

97 Prolonged large bowel transit, and an increase in the proportion o

98 d and solid gastric emptying, and small- and large-bowel transit, using (111)In-diethylenetriaminepen

99 l-bowel transit, and at 24, 48, and 72 h for large-bowel transit.

100 (GVHD), and infection after total (small and large) bowel transplantation in pigs.

101 imultaneous DSBMI and total (i.e., small and large) bowel transplantation.

102 al content transferred with total small plus large bowel transplants (TBTx) might aggravate the alloi

103 the erythrocyte sedimentation rate (ESR) and large bowel uptake of (99m)Tc-WBC (P < 0.05) and a negat

104 ars to predispose to carcinoma in the liver, large bowel, urinary bladder, and gastric mucosa.

105 The large bowel was involved in 36 (82%), the small intestin

106 istologically verified adenoma of the distal large bowel with 29,413 control subjects.

107 of CMMCs were made from the isolated murine large bowel, with or without a fecal pellet.