ライフサイエンスコーパス: large-cell

1 heterogeneous phenotype with the presence of large cells.

2 of Ccr4-Not complexes results in abnormally large cells.

3 generate action potentials (APs) than medium-large cells.

4 ze increases in small cells and decreases in large cells.

5 etworks even prevent granule condensation in large cells.

6 Of these large cells, 20 to 37% were apoptotic, according to both

7 , 19.7% squamous cell, 6.1% undifferentiated large cell, 7.2% non-small cell not otherwise specified,

8 imilar staining patterns for cytokeratins in large cell acanthoma and normal conjunctiva, except for

9 a low Ki-67 proliferation index (PI) in the large cell acanthoma compared with high indices in dyspl

10 Conjunctival large cell acanthoma is a morphologically distinctive cl

11 The large cell acanthoma recurred 4 times but retained its b

12 One large cell acanthoma, 7 epidermoid dysplasias, and 4 squ

13 Evidence for large cell aggregation was only found in N. Pacific samp

14 independently of SHH and displays classic or large cell anaplastic (LCA) pathology and poor prognosis

15 and morphologic changes that resemble human large cell anaplastic medulloblastoma.

16 or desmoplastic tumours, and 57% (33-80) for large-cell anaplastic tumours.

17 ne amplification has been identified in many large cell/anaplastic medulloblastoma, but no causative

18 -induced PNETs are histologically similar to large cell/anaplastic medulloblastomas and can occur in

19 t that sPNET with varied differentiation and large cell/anaplastic medulloblastomas may be two tumor

20 Medulloblastomas that display a large cell/anaplastic morphology and overexpress the cel

21 Metastatic disease and large-cell/anaplastic (LC/A) phenotype were the clinicop

22 % CI, 0 to 55) and 40% (95% CI, 0 to 83) for large-cell/anaplastic (n = 5) medulloblastoma ( P < .001

23 ural progenitors induced tumors exhibiting a large-cell/anaplastic histopathology adjacent to the fou

24 0.19 for desmoplastic/nodular and 45.97 for large-cell/anaplastic medulloblastoma) and nonresponse t

25 lps identify preneoplastic lesions including large cell and small cell change.

26 uited to convey morphogenetic information in large cells and tissues and provides a versatile model s

27 ell lines, including mantle cell, anaplastic large cell, and Hodgkin lymphoma cell lines.

28 Captured melanoma CTCs were large cells, and consisted of 2 subpopulations, based on

29 pe illumination optical path that produces a large cell- and beam-angle invariant stray excitation th

30 otherwise specified; 2 (13%) had anaplastic large cell; and 1 each had extranodal natural killer/T c

31 ls (decay time, approximately 25 ms) than in large cells ( approximately 2 ms), and repetitive stimul

32 compared with the distribution of all cells, large cells are more dense in the vicinity of veins and

33 Large cells are produced by increasing DNA content or pl

34 Other large cells arise more peripherally by initiating endocy

35 Diffuse large cell (B) lymphoma was the most common histologic d

36 nd R. minor Grell, another with a relatively large cell body forming lacunae, and another that has si

37 ffect is more pronounced for bacteria with a large cell body.

38 tant in maintaining arousal, we first placed large cell-body-specific lesions in these targets.

39 ding rates of its slowest proteins, and--for large cells--by the rates of its protein diffusion.

40 tological, and functional characteristics of large-cell calcifying Sertoli cell tumors of the testes

41 est that all primary producers, not just the large cells, can contribute to export from the surface l

42 ge cell carcinoma (H460) and drug resistance large cell carcinoma (COR-L23/5010) observing a concomit

43 alveolar epithelial cancer cell line (A549); large cell carcinoma (H460) and drug resistance large ce

44 ) and irregular margin (n = 3, 60%); and for large cell carcinoma (n = 1), solid attenuation and spic

45 adenocarcinoma, squamous cell carcinoma, and large cell carcinoma) caused up-regulation of CCR2/CCL2

46 histological spectrum (adeno-, squamous and large-cell carcinoma) and more frequent metastasis compa

47 olar carcinoma, squamous cell carcinoma, and large-cell carcinoma.

48 adenocarcinoma, squamous-cell carcinoma, and large-cell carcinoma.

49 rcinoma, squamous cell, and undifferentiated large cell carcinomas comparing the highest with the low

50 squamous cell carcinomas, 38.5% (5 of 13) of large-cell carcinomas, and 60% (3 of 5) of small-cell lu

51 Quantitative Nissl studies indicated that large cells (cell body area >2 standard deviations (SD)

52 of hepatic neoplasia, liver cell dysplasia (large cell change), long thought to be a premalignant le

53 In contrast, the wild type formed large cell clumps of dead and live cells, indicating the

54 pective of the age of the donor hepatocytes, large cell clusters appeared in juvenile, but only small

55 proliferate within distinct islands, forming large cell clusters that eventually fuse during metamorp

56 Deletion of the enzyme results in large cell clusters with disordered division patterns, i

57 oil, chromoplasts, small cell clusters, and large cell clusters) were isolated from different types

58 w strategy for the mechanical stimulation of large cell clusters, taking advantage of dielectrophores

59 f small groups of cells and the expansion of large cell colonies.

60 lopmental branching points between these two large cell compartments are still controversial, and lit

61 y functional theory (DFT) calculation with a large cell containing 1024 atoms (614 Ni and 410 Nb).

62 ham-1 mutant embryos contain many unusually large cell-death corpses.

63 Large cell deposits first appeared at 1 month in 40% of

64 so inhibition potentially prevents damage of large cell envelope components, but not host digestion.

65 d 16 females, 50 with squamous cell, 16 with large cell, five with adenosquamous, and 35 with adenoca

66 ps and for subsets including advanced-stage, large-cell FL and the combined subset of small cleaved-

67 To accommodate the large cells following zygote formation, early blastomere

68 Common to all succulents is the presence of large cells for water storage.

69 Large cell formation is functionally important because w

70 We propose that large cells generated by polyploidization or cell fusion

71 ve disease (86% v 60%; P < .001), anaplastic large-cell histology (53% v 40%; P = .04), and two or fe

72 PreBII large cells in adults, but not in children, showed highl

73 cting to VPM; and 7) approximately 30-50% of large cells in PrV send collaterals to ZI and VPM.

74 l cells were 10-100 times more abundant than large cells in the N. Pacific, whereas in the S. Pacific

75 , replication-competent virus measurement by large cell input quantitative viral outgrowth assay, and

76 macrophages had similar numbers of small and large cells; interstitial macrophages were mainly small.

77 ctivity for neurofilament was predominant in large cells led us to directly examine the relationship

78 In large cells like neurons, how is microtubule polymerizat

79 th senescence should be useful for screening large cell line panels to identify genomic biomarkers of

80 n therapy monitoring, 2 murine tumor models (large cell lung cancer cell line H460 and prostate cance

81 g adenocarcinomas, squamous cell carcinomas, large cell lung cancer, adenoid cystic carcinomas and ce

82 ose with pancreatic tumours or small-cell or large-cell lung cancer, as well as those without complet

83 One patient with cutaneous anaplastic large cell lymphoma (8 mg/kg) achieved a complete respon

84 (8 out of 34) activated B cell-like diffuse large cell lymphoma (ABC-DLBCL), but not in GC B cell-li

85 ose expression is up-regulated on anaplastic large cell lymphoma (ALCL) and Hodgkin lymphoma (HL) cel

86 contributes to the oncogenesis of anaplastic large cell lymphoma (ALCL) are not completely understood

87 We used various ALK-positive anaplastic large cell lymphoma (ALCL) cell lines to evaluate two in

88 malignant lymphocytes, including anaplastic large cell lymphoma (ALCL) cells.

89 ic lymphoma kinase (ALK)-positive anaplastic large cell lymphoma (ALCL) constitutes an ideal model di

90 ic lymphoma kinase (ALK)-positive anaplastic large cell lymphoma (ALCL) frequently carries the t(2;5)

91 dies in Hodgkin lymphoma (HL) and anaplastic large cell lymphoma (ALCL) has had profound clinical suc

92 ic lymphoma kinase (ALK)-negative anaplastic large cell lymphoma (ALCL) is a CD30-positive T-cell non

93 Anaplastic large cell lymphoma (ALCL) is a distinct entity of T-cel

94 Anaplastic large cell lymphoma (ALCL) is a mature T-cell lymphoma t

95 Anaplastic large cell lymphoma (ALCL) is a peripheral T-cell lympho

96 Systemic anaplastic large cell lymphoma (ALCL) is an aggressive CD30(+) non-

97 Anaplastic large cell lymphoma (ALCL) is the most common type of pe

98 a subset of T-cell lymphomas with anaplastic large cell lymphoma (ALCL) morphology (ALK+ ALCL), the v

99 lymphoma kinase-positive (ALK(+)) anaplastic large cell lymphoma (ALCL) patients.

100 In ALK-rearranged anaplastic large cell lymphoma (ALCL), a specific subtype of T-cell

101 sed Hodgkin lymphoma and systemic anaplastic large cell lymphoma (ALCL), the single agent response ra

102 LK) are detected in 50% to 70% of anaplastic large cell lymphoma (ALCL), which is a T/null cell non-H

103 in (NPM)-ALK fusion protein in an anaplastic large cell lymphoma (ALCL)-derived cell line carrying th

104 sed Hodgkin lymphoma and systemic anaplastic large cell lymphoma (ALCL).

105 y efficacious in animal models of anaplastic large cell lymphoma (ALCL).

106 ic lymphoma kinase (ALK)-positive anaplastic large cell lymphoma (ALCL).

107 ncluding Hodgkin disease (HD) and anaplastic large cell lymphoma (ALCL).

108 psed or refractory (R/R) systemic anaplastic large cell lymphoma (ALCL).

109 ic lymphoma kinase (ALK)-negative anaplastic large cell lymphoma (ALK-negative), despite their unique

110 Breast implant-associated anaplastic large cell lymphoma (BIA-ALCL), a rare peripheral T-cell

111 of aberrant somatic hypermutation in diffuse large cell lymphoma (DLCL), the most common form of non-

112 as robust enough to differentiate anaplastic large cell lymphoma (n = 32) from other PTCLs.

113 a in patients with secondary CL were diffuse large cell lymphoma (n = 5, 28%), chronic lymphocytic le

114 phoma kinase (NPM-ALK) expressing anaplastic large cell lymphoma are not completely understood.

115 breaks and translocations in the anaplastic large cell lymphoma breakpoint regions of NPM1 and ALK.

116 ase (ALK)-positive and -negative, anaplastic large cell lymphoma cell lines and primary patient tumou

117 plastic lymphoma kinase-positive, anaplastic large cell lymphoma cell lines and that ectopically expr

118 Breast implant-associated anaplastic large cell lymphoma is a rare cancer in patients with br

119 interfering RNA transfection of T-anaplastic large cell lymphoma Karpas 299.

120 target different receptors on the anaplastic large cell lymphoma line L-82, but delivered the same cy

121 ation detected in the majority of anaplastic large cell lymphoma patients, and has recently been impl

122 s fungoides (T-MF), and cutaneous anaplastic large cell lymphoma were studied in parallel with corres

123 morphologically more high grade (50% diffuse large cell lymphoma) compared with primary CL (37% low-g

124 16 relapsed CLL and 9 RT (all proven diffuse large cell lymphoma) patients were enrolled, and 60% rec

125 aplastic lymphoma kinase-positive anaplastic large cell lymphoma), upfront auto-SCT was associated wi

126 ALK)-positive and 48 ALK-negative anaplastic large cell lymphoma, 14 adult T-cell leukemia/lymphoma a

127 to 3% of patients per year into aggressive, large cell lymphoma, a critical event in the course of t

128 angioimmunoblastic, ALK-negative anaplastic large cell lymphoma, and enteropathy-associated T-cell l

129 rge B-cell lymphoma, ALK-positive anaplastic large cell lymphoma, chronic myelogenous leukemia, and a

130 pressed on Hodgkin's lymphoma and anaplastic large cell lymphoma, it is a promising target for immuno

131 actory Hodgkin lymphoma, systemic anaplastic large cell lymphoma, relapsed or refractory B-cell precu

132 rhabdomyosarcoma, neuroblastoma, anaplastic large cell lymphoma, renal cell carcinoma, and inflammat

133 ymphoma and, in a subset of cases of diffuse large cell lymphoma, the mechanism of Bcl-6 overexpressi

134 ry syndrome, or primary cutaneous anaplastic large cell lymphoma, with disease progression after >/=

135 NS relapse or dissemination in patients with large cell lymphoma.

136 hronic lymphocytic leukemia (CLL) to diffuse large cell lymphoma.

137 als in CD30+ Hodgkin lymphoma and anaplastic large cell lymphoma.

138 ng classical Hodgkin lymphoma and anaplastic large cell lymphoma.

139 of MALT lymphoma to activated B-cell diffuse large-cell lymphoma (ABC-DLBCL).

140 -derived neoplasias, most notably anaplastic large-cell lymphoma (ALCL) and Hodgkin's lymphoma.

141 herapy of certain cancers such as anaplastic large-cell lymphoma (ALCL) and inflammatory myofibroblas

142 ents in several tumors, including anaplastic large-cell lymphoma (ALCL) and non-small cell lung carci

143 Most of the anaplastic large-cell lymphoma (ALCL) cases carry the t(2;5; p23;q3

144 Anaplastic large-cell lymphoma (ALCL) frequently carries the t(2;5)

145 c proteins in the pathogenesis of anaplastic large-cell lymphoma (ALCL) have been well defined; never

146 Anaplastic large-cell lymphoma (ALCL) is a clinical and biological

147 Breast implant-associated anaplastic large-cell lymphoma (ALCL) is a recently described clini

148 Systemic anaplastic large-cell lymphoma (ALCL) is a T-cell lymphoma, whose a

149 Systemic anaplastic large-cell lymphoma (ALCL) is an aggressive subtype of T

150 c proteins in the pathogenesis of anaplastic large-cell lymphoma (ALCL) is well established.

151 Anaplastic large-cell lymphoma (ALCL) represents a heterogeneous gr

152 regulated in T-lymphomas, such as anaplastic large-cell lymphoma (ALCL) tumors.

153 c lymphoma kinase (ALK) -positive anaplastic large-cell lymphoma (ALCL) was excluded.

154 Anaplastic large-cell lymphoma (ALCL) was initially recognized on t

155 lymphoma kinase-positive (ALK(+)) anaplastic large-cell lymphoma (ALCL), and adult T-cell leukemia/ly

156 dgkin's lymphoma (HL), three with anaplastic large-cell lymphoma (ALCL), and two with CD30+ T-cell ly

157 ts with Hodgkin lymphoma (HL) and anaplastic large-cell lymphoma (ALCL), the study by Jacobsen and co

158 ic lymphoma kinase (ALK)-positive anaplastic large-cell lymphoma (ALCL).

159 LK) in patients with ALK-positive anaplastic large-cell lymphoma (ALCL).

160 portant pathogenetic role in ALK+ anaplastic large-cell lymphoma (ALCL).

161 aplastic lymphoma kinase-positive anaplastic large-cell lymphoma (ALK(+) ALCL) is a unique type of T-

162 ree of 10 primary cutaneous CD30+ anaplastic large-cell lymphoma (C-ALCL).

163 In patients with diffuse large-cell lymphoma (DLBCL), we observed higher levels o

164 NHL and of two predominant subtypes, diffuse large-cell lymphoma (DLCL) (n = 233) and follicular lymp

165 ligible pediatric patients with stage III/IV large-cell lymphoma (LCL); 90 patients were randomly ass

166 T-cell lymphoma/leukemia (n = 4), anaplastic large-cell lymphoma (n = 2), and extranodal natural kill

167 astic T-cell lymphoma [AITL], and anaplastic large-cell lymphoma [ALCL]) is difficult, with the morph

168 All seven patients without anaplastic large-cell lymphoma achieved CR.

169 ALK translocations, particularly anaplastic large-cell lymphoma and inflammatory myofibroblastic tum

170 y expressed in a subset of T-cell anaplastic large-cell lymphoma and non-small-cell lung cancer, resp

171 The BCL-6 gene is involved in diffuse large-cell lymphoma and overexpressed in other types of

172 Hodgkin's lymphoma and anaplastic large-cell lymphoma are the two most common tumors expre

173 of relapsed Hodgkin lymphoma and anaplastic large-cell lymphoma by the Food and Drug Administration.

174 patients, particularly those with anaplastic large-cell lymphoma histology.

175 In T-cell lymphomas, anaplastic large-cell lymphoma is defined by mutually exclusive rea

176 lymphoma and treated patients with advanced large-cell lymphoma on a separate protocol (doxorubicin,

177 is fungoides or primary cutaneous anaplastic large-cell lymphoma who had been previously treated.

178 pression of CD30 (the hallmark of anaplastic large-cell lymphoma) and of immunosuppressive cytokine I

179 's transformation (CLL that has evolved into large-cell lymphoma) and only one case of CLL progressio

180 Outcomes of 241 patients (112 anaplastic large-cell lymphoma, 102 peripheral T-cell lymphoma not

181 iated with an improved outcome in anaplastic large-cell lymphoma, ALK positive.

182 , respectively, for patients with anaplastic large-cell lymphoma, and 63.8% (SE, 10.3%) and 70.3% (SE

183 t otherwise specified (PTCL-NOS), anaplastic large-cell lymphoma, and adult T-cell leukemia/lymphoma

184 amine T-cell lymphomas, including anaplastic large-cell lymphoma, angioimmunoblastic T-cell lymphoma,

185 to Hodgkin lymphoma and systemic anaplastic large-cell lymphoma, CD30 expression of malignant lympho

186 , or oncogenic mutations, such as anaplastic large-cell lymphoma, inflammatory myofibroblastic tumour

187 K aberrations (eight of nine with anaplastic large-cell lymphoma, one of 11 with neuroblastoma, three

188 valuable solid or CNS tumours, or anaplastic large-cell lymphoma, refractory to therapy and for whom

189 with refractory solid tumours and anaplastic large-cell lymphoma.

190 nd blood samples of patients with anaplastic large-cell lymphoma.

191 primarily Hodgkin's lymphoma and anaplastic large-cell lymphoma.

192 atients with Hodgkin lymphoma and anaplastic large-cell lymphoma.

193 and ATLL and a lower incidence of anaplastic large-cell lymphoma; Hispanics had a higher incidence of

194 netic lesion underlying pediatric anaplastic large cell lymphomas (ALCL) and inflammatory myofibrobla

195 nucleophosmin-ALK oncoprotein in anaplastic large cell lymphomas (ALCL).

196 (NSCLC) and approximately 70% of anaplastic large cell lymphomas (ALCL).

197 ccurring in approximately half of anaplastic large cell lymphomas (ALCLs).

198 event in the pathogenesis of most anaplastic large cell lymphomas (ALCLs).

199 aplastic lymphoma kinase (ALK)(-) anaplastic large cell lymphomas (ALCLs).

200 ubset included lines derived from anaplastic large cell lymphomas, non-small-cell lung cancers, and n

201 of relapsed Hodgkin and systemic anaplastic large cell lymphomas--both characterized by high express

202 ng Classical Hodgkin and systemic anaplastic large cell lymphomas.

203 Anaplastic large-cell lymphomas (ALCLs) are a group of clinically a

204 Anaplastic large-cell lymphomas (ALCLs) bearing the t(2;5) transloc

205 Anaplastic large-cell lymphomas (ALCLs) encompass at least 2 system

206 the survival and proliferation of anaplastic large-cell lymphomas (ALCLs).

207 ts in mice, including Karpas-299 (anaplastic large-cell lymphomas [ALCL]) and H3122 (NSCLC).

208 In addition to 100% anaplastic large-cell lymphomas, 57% of other PTCL entities were CD

209 indistinguishable from patients' anaplastic large-cell lymphomas.

210 characteristic of patient-derived anaplastic large-cell lymphomas.

211 induced inward current densities than medium-large cells, most of which did not respond to capsaicin.

212 logic appearance of totally undifferentiated large-cell NBs (LCNs), the most aggressive and deadly fo

213 arcinoid, small cell lung cancer (SCLC), and large cell NE cancer.

214 10, 71%); for small cell or mixed small and large cell neuroendocrine carcinoma (n = 7), solid atten

215 s with small-cell lung cancer and eight with large-cell neuroendocrine carcinoma, all of whom receive

216 using platinum plus etoposide for those with large-cell neuroendocrine carcinoma.

217 In contrast to small-cell lung cancer and large-cell neuroendocrine lung tumours, TP53 and RB1 mut

218 ogically confirmed small-cell lung cancer or large-cell neuroendocrine tumours with progressive measu

219 transfer represents a promising therapy, the large cell number required to achieve clinical efficacy

220 unit-fibroblasts (CFU-Fs) on plastic and the large cell numbers required for transplantation.

221 (23 classic, eight desmoplastic-nodular, two large cell, one anaplastic), 17 ependymomas (13 World He

222 But this approach is limited where large cells or searches over millions of structure types

223 nvironment of bird macrophages, exhibiting a large cell phenotype that rescues growth suppression.

224 nced suppression of mat3 and even to cause a large-cell phenotype that is comparable to that caused b

225 core component of both the crumbs and discs large cell polarity complexes.

226 uranoside hydrochloride (Ara-C) expanded the large cell population but the residual proliferative cap

227 e in the patient's hematopoietic system of a large cell population with a mutation in the X-linked PI

228 This matrix is averaged over a large cell population, revealing diagonal blocks called

229 revents the application of these assays to a large cell population, which produces information with s

230 dual cells in an organ-scale simulation of a large cell population.

231 ipal neurons, the Kenyon cells (KCs), form a large cell population.

232 deformation of a macroscopic substrate by a large cell population.

233 d to measuring average radiotracer uptake in large cell populations and, as a result, lack the abilit

234 fast and quantitative mechanical sampling of large cell populations becomes feasible.

235 ntermediate-sized clusters to segregation of large cell populations by formation of smooth boundaries

236 matic study of mechanical characteristics of large cell populations complementing conventional tools

237 lication of single-cell genome sequencing to large cell populations has been hindered by technical ch

238 ugh the overall levels of gene expression in large cell populations seem to be precisely controlled,

239 These measurements, performed over large cell populations, show quite generally that sequen

240 ng improved optical and analytical tools and large cell populations, we show that P1 plasmid foci are

241 cannot be obtained by techniques relying on large cell populations.

242 studying heterogeneity in gene expression in large cell populations.

243 Large cells received short-latency excitatory inputs and

244 ndividual cells from a particular cell line, large cells should be easier to electroporate than small

245 Large cell size is not restricted to a particular bacter

246 ftsZ gene, which may be responsible for the large cell size of members of this species, as well as g

247 With the evolution of extreme polyploidy and large cell size, Epulopiscium has acquired some of the a

248 lastic leukemia-like cells, characterized by large cell size, multiploid nuclei, and expression of CD

249 id, increasing their DNA content to attain a large cell size.

250 We uncover that large cell-size increase is accompanied by downregulatio

251 econdary dendrites of neurons with medium or large cell somas.

252 with cell length in small cells, but in very large cells spindle length approaches an upper limit of

253 Mucin-4 (Muc4) is a large cell surface glycoprotein implicated in the protec

254 CD13 is a large cell surface peptidase expressed on the monocytes

255 xamined how AP-2 mediates internalization of large cell surface receptors, such as the eight-chain TC

256 Polysialic acid (PSA), a large cell-surface carbohydrate that regulates cell inte

257 erologous cells, PSD-93 induces formation of large cell-surface clusters.

258 GspB is a large cell-surface glycoprotein expressed by Streptococc

259 nin-like sequence) gene family encodes eight large cell-surface glycoproteins (Als1-Als7 and Als9) th

260 he cytoplasmic portion of plexins, which are large cell-surface semaphorin receptors, and to mediate

261 cterized histologically by the appearance of large cells that are either aneuploid or polyploid.

262 mal rate, approximately 70 spikes/s), unlike large cells that project to premotor areas (maximal rate

263 In addition to forming large cells, the two-molecule-long bendy walls give this

264 Gunflint populations that display relatively large cells (thick-walled spheres, filament-forming rods

265 To achieve sensitivity comparable to that of large cells, those that report upon a small region of vi

266 sion and acid transport, helping to overcome large cell-to-capillary distances that are characteristi

267 Due to large cell-to-cell variability and the lack of direct me

268 Interferon-beta (IFNB1) mRNA shows very large cell-to-cell variability in primary human dendriti

269 This corresponded to large cell-to-cell variability in the decay (tau(w) = 3-

270 Pom1p gradients exhibit large cell-to-cell variability, as well as dynamic fluct

271 propensity for calcium oscillations, despite large cell-to-cell variability.

272 present in a mix of oligomeric states with a large cell-to-cell variation.

273 Surprisingly, there were large cell-to-cell variations for all supplied lutropin

274 Despite large cell-to-cell variations in the concentrations of i

275 cells marked by emergence of slow kinetics, large cell-to-cell variations of copy numbers, temporall

276 Large cell transformation (LCT) in mycosis fungoides (MF

277 ith advanced-stage disease (ASD, n = 92) and large cell transformation (LCT, n = 22).

278 Age at diagnosis, large cell transformation and secondary bacterial infect

279 Large cell transformation often hallmarks cases with a m

280 the CTCL lymph node lesions with or without large cell transformation.

281 f positive cells present in the cases with a large cell transformation.

282 ated with intermediate-grade (P = 0.002) and large-cell transformation histology (P = 0.04).

283 phocytic leukemia for 29 and 7 months before large-cell transformation in the eye.

284 bles tested, four (stage IV, age > 60 years, large-cell transformation, and increased lactate dehydro

285 onstrated tumor-stage MF,with no evidence of large-cell transformation.

286 ary cutaneous anaplastic T-cell lymphoma and large-cell transformed cutaneous T-cell lymphoma.

287 pharmacologic inhibition of EZH2 activity in large-cell transformed cutaneous T-cell lymphoma.

288 had squamous-cell carcinoma, and 7 (2%) had large-cell undifferentiated carcinoma.

289 e applied for the microfluidic enrichment of large cells, up to the 40 microm diameter level examined

290 by the transition from small-cell variant to large-cell variant.

291 mall-cell variants (SCVs) are generated from large-cell variants (LCVs) is considered fundamental to

292 discriminative power of chemometric tools on large cell wall-chromatographic data sets to discover no

293 nection between protein localization and the large cell-wall curvature at the poles.

294 Large cells, which are found in all subdivisions of the

295 Large cells, which were targeted under two-photon guidan

296 These attributes contrast with large cells, whose brief IPSCs and rapid firing rates ca

297 nges as very young thread cells develop into large cells with fully mature coiled threads.

298 iple consecutive abnormal mitoses, producing large cells with giant nuclei and high ploidy that event

299 It includes calretinin immunoreactive large cells with oval or polygonal cell bodies.

300 ndant anti-phospho-ribosomal S6 labelling of large cells within the frontal cortex, basal ganglia, hi