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1 abrogating postreplicative intermediate and late gene transcription.
2 holoenzyme reflects its role in stimulating late gene transcription.
3 eraction in regulation of terminase or other late gene transcription.
4 y DNA-binding protein, ICP8, also stimulates late gene transcription.
5 iral DNA and bound proteins in activation of late gene transcription.
6 A synthesis and more pronouncedly inhibiting late gene transcription.
7 l DNA replication and as the activator of T4 late gene transcription.
8 nuclei as sites of viral DNA replication and late gene transcription.
9 f the gp45 sliding clamp in activation of T4 late-gene transcription.
10 for TBP in vaccinia virus intermediate- and late-gene transcription.
11 and it blocks both viral DNA replication and late gene transcription, although to different degrees.
12 utations, that E74B is a potent repressor of late gene transcription and E74A is sufficient to premat
13 ive role in the regulation of vaccinia virus late gene transcription and suggest that poxviruses have
14 included a defect in viral DNA replication, late gene transcription, and virus production as well as
15 Cs), domains in which viral DNA replication, late-gene transcription, and encapsidation take place, i
16 ssential for either viral DNA replication or late gene transcription, but the absence of lef-6 result
17 that the BR-C early gene directly activates late gene transcription by interacting with late gene ci
18 ial cells increased the induced level of JCV late gene transcription by the viral early protein, T-an
19 activator of herpes simplex virus early and late gene transcription during infection and in vitro ca
21 cting plasmids containing T7 promoter-driven late gene transcription factors and a late promoter repo
22 s viral trans factors crucial for activating late gene transcription following viral DNA replication
23 discovery of the viral mutants that uncouple late gene transcription from DNA replication lays an imp
24 ICP4 is required for all delayed-early and late gene transcription, ICP0 stimulates transcription o
25 , given that these mutations do not increase late gene transcription in the absence of genome replica
26 eletion of lef-6 affected DNA replication or late gene transcription in the context of an infection.
27 xpression of gC, and that the high levels of late-gene transcription may be largely due to the induct
28 y, it was thought that only intermediate and late gene transcription produced double-stranded (ds) RN
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