ライフサイエンスコーパス: lateral wall

1 e of sheet orientation in the late-activated lateral wall.

2 e and retained the geometry of the infarcted lateral wall.

3 ns and actually twisted clockwise at the AVV lateral wall.

4 mammalian hearing and originates within its lateral wall.

5 eter of the PM was 22% of that of the intact lateral wall.

6 for electromotile force coupling in the OHC lateral wall.

7 esses in the cochlea, including the cochlear lateral wall.

8 in part because it is softer than the mature lateral wall.

9 s the second reduced counts primarily in the lateral wall.

10 l ganglion neurons, or cells of the cochlear lateral wall.

11 ade peptidoglycan in the division septum and lateral wall.

12 neurons, and, finally, the fibrocytes in the lateral wall.

13 regions, whereas 84% of LV leads were in the lateral wall.

14 ficantly less at the flow divider versus the lateral wall.

15 tiating fibrocytes and marginal cells in the lateral wall.

16 the RV free wall, ventricular septum, and LV lateral wall.

17 rominence and central region in the cochlear lateral wall.

18 for interactions between the bundle and the lateral wall.

19 on of new peptidoglycan into portions of the lateral wall.

20 ased motor mechanism that resides in the OHC lateral wall.

21 ebral ventricle walls, mainly located in the lateral wall.

22 membrane protein highly expressed in the OHC lateral wall.

23 all sheet extension was >2 times that in the lateral wall (0.22+/-0.12 versus 0.09+/-0.06).

24 g (IVS, 0.009 +/- 0.002 mL.min-1.g-1.beat-1; lateral wall, 0.005 +/- 0.001 mL.min-1.g-1.beat-1; P = .

25 basal and midcavity septum compared with the lateral wall (11.0+/-1.4 versus 10.1+/-1.3 mm; P<0.001).

26 ); at 21 to 28 days, the difference was 50% (lateral wall, 14 +/- 6%; IVS, 28 +/- 6%; P = .0001).

27 H levels were significantly increased in the lateral wall 2 and 4 h post-exposure and returned to nor

28 followed by the tongue (57%), oropharyngeal lateral wall (49%), and epiglottis (26%).

29 was required for tether formations from OHC lateral wall (499 +/- 152 pN) than from OHC basal end (1

30 r each patient, located at the extreme basal lateral wall (8 of 11 patients) and other regions (9 of

31 Lateral wall ablation was performed either by creating a

32 ia characteristics, resulting in a defective lateral wall, abnormal neuroblast migration and differen

33 The OHC lateral wall above the nuclear level is composed of the

34 found that deletion of Panx1 in the cochlear lateral wall almost abolished ATP release under physiolo

35 nt tether length, was 3.71 pN/microm for OHC lateral wall and 4.57 pN/microm for OHC basal end.

36 ween the mitral valve annulus pACC at the LV lateral wall and mean LAP.

37 of Corti and modiolar core, by L-NAME in the lateral wall and modiolar core, and by NAC in all three

38 ted nonlinear capacitance resided in the OHC lateral wall and was not detected at the apical cuticula

39 erenol stimulation in DHF (both anterior and lateral walls), and CRT substantially improved both.

40 A is present in the neurosensory epithelium, lateral wall, and spiral limbus of the cochlea, with hig

41 lume of the tongue, parapharyngeal fat pads, lateral walls, and soft palate) was similar between subj

42 The lateral walls are affected most by strain, and the septa

43 d C(peak) increase with the logarithm of the lateral wall area (A(LW)) and determine from the functio

44 omputer simulation model showed that the hot lateral wall artifact found in SPECT myocardial perfusio

45 d, reconstructed SPECT images showed the hot lateral wall artifact with a mean septal-to-lateral wall

46 hing caused an additional 9% decrease in the lateral wall as a result of the adjacent lung-lateral wa

47 tially linked to beta(1-3)glucan, and in the lateral wall, attached in part to beta(1-6)glucan.

48 ural bead columns were inserted into the mid-lateral wall between the PMs.

49 ateral wall as a result of the adjacent lung-lateral wall boundary.

50 the septal ring and scattered foci along the lateral wall, but mutants have no phenotypic change.

51 eart and around the circumference toward the lateral wall, but not the interventricular septum.

52 s is markedly upregulated selectively in the lateral wall by noise exposure, presumably in response t

53 IVS and LV lateral wall circumferential strain (CS) were both reduc

54 We selectively labeled and imaged the lateral wall components in the isolated guinea pig OHC u

55 ional LV ischemia involving the inferior and lateral walls confers increased likelihood of RV dysfunc

56 cal vent insertion (49%), in the inferior or lateral wall consistent with infarction (5%), or in othe

57 With RBBB, the lateral wall contracts early so that biventricular (BiV)

58 ions in the mechanical properties of the OHC lateral wall could play a role in the modulation of OHC

59 id not show hot lateral wall (mean septal-to-lateral wall count ratio = 1.04) and the ratios changed

60 the filling of the left lung (mean septal-to-lateral wall count ratio = 1.05).

61 id not show hot lateral wall (mean septal-to-lateral wall count ratio = 1.07).

62 lateral wall artifact with a mean septal-to-lateral wall count ratio of 0.86.

63 ups persisted for IVS CS (P<0.01) but not LV lateral wall CS (P=0.09).

64 Although IVS CS and LV lateral wall CS correlated with pulmonary artery systoli

65 Subsequently, attachments to lateral walls disappear and attachments to microtubule e

66 provide durability and may contact with the lateral wall either to give additional anchoring of the

67 ere approximately 2 times higher than in the lateral wall (epicardium, 0.14+/-0.07 versus 0.05+/-0.03

68 ope and is determined by the balance between lateral wall extension and synthesis of peptidoglycan at

69 PBP3, PBP5 and PBP4a, and possibly PBP4, in lateral wall growth.

70 nvolved the subepicardial layer inferior and lateral wall in 154 patients (41%; IL group), the midwal

71 T is achieved over a fairly broad area of LV lateral wall in both nonfailing and failing hearts, with

72 uld have subsequently expanded to the entire lateral wall in outer hair cells.

73 y less at the flow divider compared with the lateral wall in the DES group (0.07 mm [interquartile ra

74 reater at the flow divider compared with the lateral wall in the DES group (uncovered: 40% [IQR 16% t

75 (P < 0.0032) and ventral displacement of the lateral walls in the mid- and caudal regions (P < 0.0001

76 beling in tanycytes of the ventral floor and lateral walls in the tuberal and mammillary recess porti

77 PBP3 and PBP4a localized specifically to the lateral wall, in distinct foci, whereas PBP1 and PBP2b l

78 of the molecular and ionic substrates of the lateral wall, including those necessary for electromotil

79 The outer hair cell (OHC) lateral wall is a unique trilaminate structure consistin

80 The OHC lateral wall is a unique trilaminate structure consistin

81 vide on the mechanical properties of the OHC lateral wall is important for understanding the mechanis

82 Electromechanical coupling in the cell's lateral wall is modeled in terms of linear constitutive

83 ration of contraction of both the septum and lateral wall is shorter in obstructed patients with the

84 stable for generations while material in the lateral walls is diluted by growth and turnover.

85 Regional inferior and lateral wall ischemia was greater among patients with RV

86 e cytoskeletal framework associated with the lateral wall known as the cortical lattice.

87 Recurrent conduction through the lateral wall lesions was associated with intraprocedural

88 RV lateral wall longitudinal strain (LS) and interventricul

89 RV lateral wall LS and IVS LS, but not LV lateral wall LS,

90 2+/-3.9%, P=0.002, respectively), whereas LV lateral wall LS was preserved.

91 RV lateral wall LS and IVS LS, but not LV lateral wall LS, correlated with pulmonary artery systol

92 thout left lung wrap-around did not show hot lateral wall (mean septal-to-lateral wall count ratio =

93 ients, reconstructed images did not show hot lateral wall (mean septal-to-lateral wall count ratio =

94 Participants received lateral wall, medial wall, 2.5 wall, or 3 wall decompres

95 duces an electromechanical response in their lateral wall membrane.

96 tality was increased in patients with severe lateral wall motion abnormalities (p = 0.05), ST-segment

97 F at presentation, length of stay, prior MI, lateral wall motion abnormality at presentation, and pea

98 m -20.4 +/- 9.6% to -30.5 +/- 14.0%, whereas lateral wall motion occurred earlier with no net magnitu

99 decrease in systolic wall thickening in the lateral wall occurred compared with the interventricular

100 duction of a localized cell expansion in the lateral wall of a root epidermal cell.

101 cells, whereas it was detected in the entire lateral wall of cochlear outer hair cells and had an int

102 ior alveolar artery (PSAA) is located on the lateral wall of maxillary sinus and may become injured d

103 cified pseudoaneurysm arising from the right lateral wall of the abdominal aorta with the neck of the

104 The lateral wall of the avian braincase, which is indicative

105 The lateral wall of the braincase lies along a boundary in t

106 pulations of mesoderm, will give rise to the lateral wall of the braincase.

107 r may determine the final composition of the lateral wall of the braincase.

108 eactivity is present in the spiral ganglion, lateral wall of the cochlea, and organ of Corti, all tar

109 e mammalian subventricular zone (SVZ) of the lateral wall of the forebrain ventricle retains a popula

110 ventricular zone (SVZ), lies adjacent to the lateral wall of the lateral ventricle and is responsible

111 ration of neuronal precursors throughout the lateral wall of the lateral ventricle in the adult mamma

112 The lateral wall of the lateral ventricle in the human brain

113 e present in the brains, particularly in the lateral wall of the lateral ventricle of juvenile (3-mon

114 Whole mounts of the lateral wall of the lateral ventricle stained for the ne

115 the subventricular zone (SVZ) throughout the lateral wall of the lateral ventricle.

116 orm regenerative units (pinwheels) along the lateral wall of the lateral ventricle.

117 We show here that the SVZ of the lateral wall of the lateral ventricles in adult mice is

118 ependymal cells during the formation of the lateral wall of the lateral ventricles in the brain.

119 It is the subependymal zone of the lateral wall of the lateral ventricles.

120 cell niche that lies along the length of the lateral wall of the lateral ventricles.

121 art failure induced by rapid pacing from the lateral wall of the left ventricle (220 +/- 9 bpm for 26

122 his study is to compare the thickness of the lateral wall of the maxillary sinus (TLWMS) and the thic

123 collected an additional five swabs from the lateral wall of the mid-vagina.

124 vely flat central ulcer on the right postero-lateral wall of the midesophagus below the level of the

125 taining cells fated to form a portion of the lateral wall of the neural tube at essentially all rostr

126 n kinase type IV-were immunolocalized to the lateral wall of the OHC.

127 The time to peak velocity in the lateral wall of the patients, as compared with that in t

128 Lesions were present in the lateral wall of the RA (six of seven maps) and in the LA

129 in six cases, three involving lesions in the lateral wall of the RA and three involving the LA septum

130 y, temporary pacing leads were placed in the lateral wall of the right atrium and at the roof of the

131 ateral ventricle on images that depicted the lateral wall of the ventricle.

132 se that forms U-shaped patterns facing outer lateral walls of epidermis and cortex cells.

133 Human chromosomes attach initially to lateral walls of microtubules.

134 aining nucleation complexes localized to the lateral walls of pre-existing CMTs.

135 ns, localizes peptidoglycan synthesis in the lateral walls of rod-shaped cells.

136 of the CD1b heavy chain in the superior and lateral walls of the A' pocket.

137 ained and some IcsA is distributed along the lateral walls of the bacillus.

138 n irreplaceable role in the extension of the lateral walls of the cell.

139 ntraoperative tissue (n = 43) samples of the lateral walls of the human lateral ventricles.

140 s located in the adhesions of the medial and lateral walls of the rostral LVs upregulated parvalbumin

141 CT caused a major artifactual defect in the lateral wall on the PET image.

142 etimes with irregular distribution along the lateral wall or a preference for the septum.

143 The incidence of oscillopsia after lateral wall orbital decompression was 35% when patients

144 Prior tumor resection with lateral wall osteotomy, delay in IACC implementation or

145 m stretch was significantly increased in the lateral walls (P < 0.023) in a ventral-lateral direction

146 ltivariable analysis, inducible inferior and lateral wall perfusion defects increased the likelihood

147 mutant (or depletion of all three isoforms) lateral wall PG synthesis was impaired and the cells bec

148 We conclude that the lateral wall PM is more compliant than the CL/SSC comple

149 ided septal flash with simultaneous RV basal lateral wall prestretch/late activation.

150 al activation (right-sided septal flash), RV lateral wall prestretch/late contraction, postsystolic s

151 g" strains were significantly greater at the lateral wall, regardless of short-axis level, whereas E(

152 loaded chamber (containing the electromotile lateral wall) relative to the lower portion (containing

153 ventricular septum, and the left ventricular lateral wall, reproducing the pattern of fibrosis found

154 (2) All lateral wall right atrial activation can be uniform duri

155 icular, we sought an explanation for the hot lateral wall seen in SPECT images from normal female and

156 In nonstented coronary bifurcations, the lateral wall showed significantly greater intima as well

157 Optimal CRT was achieved from LV lateral wall sites, slightly more anterior than posterio

158 d to affect both the motor protein and other lateral wall stiffness components.

159 cells by stereocilia displacement and/or by lateral wall stretching result in a frequency-dependent

160 e of the different functional regions (e.g., lateral wall, synaptic basal pole) of the polarized cell

161 dibular symphysis area, and thickness of the lateral wall (T-LW) of the maxillary sinus.

162 The maxillary sinus lateral wall tends to increase in thickness from the sec

163 rotease activity was higher in the infarcted lateral wall than in the remote, uninjured septum on ex

164 includes taste buds, papilla epithelium and lateral walls that extend into underlying connective tis

165 d flow per beat between the left ventricular lateral wall (the site of stimulation) and the IVS was f

166 As compared with the intensity at the lateral wall, the intensities of prestin labeling at the

167 of the present study is to examine the sinus lateral wall thickness (LWT) of atrophic posterior maxil

168 also indicate that lipid peroxidation in the lateral wall tissues does not influence permanent thresh

169 formed in the organ of Corti, but not in the lateral wall tissues.

170 LVLWS/LVSWS was more robust than the LV lateral wall to LV septal wall ratio of early diastolic

171 ablation procedure, conduction block in the lateral wall was verified in 10 of 14 patients using 1 o

172 ession analysis the volume of the tongue and lateral walls was shown to independently increase the ri

173 ling was found at the outer layer of the OHC lateral wall, which was further evidenced by use of a hy

174 to shift and rotate in the plane of the OHC lateral wall with a dynamics tightly regulated by membra