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1 bortus 2308, live Escherichia coli HB101, or latex beads).
2 y their internalisation of 1 mum fluorescent latex beads.
3 icroruby dextran amine, or rhodamine labeled latex beads.
4 er systems such as colloidal gold or colored latex beads.
5 d standards such as proteins and polystyrene latex beads.
6 the binding of neutrophils to albumin-coated latex beads.
7 y decreasing (85%) binding of ROS but not of latex beads.
8  was lower than for heat-killed organisms or latex beads.
9 es but also phagocytosable chitosan and even latex beads.
10 ulation with chitin but not with chitosan or latex beads.
11 ion was similar to the rate for phagocytosed latex beads.
12 reatment did not affect uptake of IgG-coated latex beads.
13 ed by motors driving tethered cells or 1-mum latex beads.
14 served following ingestion of oxidized OS or latex beads.
15 agocytosis of apoptotic neutrophils, but not latex beads.
16 ch as viruses, pathogenic bacteria, and even latex beads.
17 cules with monoclonal antibodies adsorbed to latex beads.
18 hagocytosis of M. tuberculosis or TDM-coated latex beads.
19 dvanced glycation end product-BSA-conjugated latex beads.
20 -8) and increasing phagocytosis of opsonized latex beads.
21 of particulate substances such as zymosan or latex beads.
22 mosan, and mannan (a mannose polymer)-coated latex beads (1 microm) at 1, 10, or 100 microg/ml.
23 crom beta-glucan particles, laminarin-coated latex beads, 1 microm latex beads, 50 to 100 microm chit
24 labeled with a fluorescent antibody [2] or a latex bead [5], are seen to move longitudinally down the
25 cles, laminarin-coated latex beads, 1 microm latex beads, 50 to 100 microm chitin particles, or 50 to
26 inding of unopsonized particles (e.g., TiO2, latex beads; 63 +/- 5 and 67 +/- 4% inhibition, respecti
27 g of unopsonized particles (TiO2, Fe2O3, and latex beads; 66 +/- 5, 77 +/- 2, and 85 +/- 2% inhibitio
28 oad), while the second motor rotated a small latex bead (a light load).
29 o the same degree that phagosomes containing latex beads acquired these markers after 1.5 h of infect
30 or bR did not occur around neutrally charged latex beads acting as cell surrogates, demonstrating tha
31 urified SOF N-terminal peptide could promote latex bead adherence to HEp-2 cells and inhibit GAS inva
32 e, with the microcantilever technique, where latex beads affixed on silicon cantilevers were used as
33 an immunochromatographic assay (ICGA), and a latex bead agglutination assay (LBAA).
34 osable dichlorodihydrofluorescein-conjugated latex beads and cyclic hydroxylamines with differing mem
35 nthesized alpha-1,2-trimannose cap sugars on latex beads and demonstrated that C57BL/6 mice coinocula
36     We illustrate this by using two sizes of latex beads and demonstrating the simultaneous detection
37 agocytic uptake of serum-coated or -uncoated latex beads and E. coli However, consistent with previou
38 een fibrinogen molecules covalently bound to latex beads and either wild-type alphaIIbbeta3 molecules
39 slowed the motility of phagosomes containing latex beads and endogenous pigment granules.
40                              Phagocytosis of latex beads and Escherichia coli bacteria was also not a
41 up, agglutination of Gal(alpha1-4)Gal-coated latex beads and human or sheep erythrocytes, and hemolys
42 nts, the Tfp of immobilized bacteria bind to latex beads and retract, pulling beads from the tweezers
43 uction; and in vitro capacity to phagocytose latex beads and to migrate toward the chemokine (C-C mot
44 polymerized from the surface of mDia1-coated latex beads, and deformed by manipulating both ends thro
45 th increased uptake of Ig-opsonized targets, latex beads, and fluid phase markers, and it was accompa
46 ntibacterial activity during phagocytosis of latex beads, and this too was dependent in large part on
47 the ingestion process, cells were exposed to latex beads at 15-20 degrees C, which allows engulfment
48 ts, up-converting phosphor technologies, and latex beads, between others) in LFBs.
49 n (90-226 emm1::km) was greatly reduced, and latex beads bound to M1 protein were readily internalize
50                                              Latex beads bound to the cell surface move in a myosin I
51 re stably attached to 1 mum (bacteria-sized) latex beads, but not directly linked to each other, in c
52 uMC generate ROS upon exposure to IgG-coated latex beads by 5-LO and COX; and ROS appear to have no s
53                       Preferential uptake of latex beads by follicle-associated epithelium indicates
54  observation that soon after phagocytosis of latex beads by M phi, cell surface expression of HSA rap
55 uptake of nonpathogenic Escherichia coli and latex beads by nonphagocytic mammalian cells.
56 hly scattering metal-coated polystyrene (PS) latex beads by using solvent-controlled heterocoagulatio
57 n molecule 1 (CD54) of EC was measured using latex beads coated with antibody to CD54 as a model for
58                  Flow cytometry testing with latex beads coated with human leukocyte antigen (HLA) cl
59          By analyzing the internalization of latex beads coated with invasin derivatives, an addition
60 tent with a direct role of the minor pilins, latex beads coated with SpaB or SpaC protein bind specif
61 g Jurkat T cells as effectors and cell-sized latex beads coated with various antibodies as artificial
62  of adenovirus and transferrin-coated 100-nm latex beads compared to MH-S, a GM(+/+) AM cell line.
63 rganisms and by 280% in phagocytosis of FITC-latex beads compared to those that received the antisens
64  and a 65% reduction in phagocytosis of FITC-latex beads compared to those treated with the sense oli
65                                        These latex bead compartments (LBCs) are encased in membrane a
66  phagosomes but not in phagosomes containing latex beads conjugated with another protein.
67          Murine macrophages were pulsed with latex beads conjugated with OVA.
68       BFA inhibited macrophage processing of latex bead-conjugated Ag for presentation to T cells, su
69 eomic analysis of the membrane fraction from latex bead-containing (LBC) phagosomes isolated from mac
70       After 120 minutes of in vivo exposure, latex beads could be found in cervical lymph nodes.
71 igen and antibody immobilized on polystyrene latex beads (diameter, 0.109 +/- 0.0025 micron).
72                                   IgG-coated latex beads did stimulate ROS production in huMC, and in
73                        MSU crystals, but not latex beads, directly bound recombinant soluble (s) CD14
74 es of the murine eye that ingest fluorescent latex beads do not migrate to regional lymph nodes.
75 ents, in comparison to phagosomes containing latex beads, do not readily interact with incoming endoc
76 at keratinocytes phagocytose melanosomes and latex beads equally well and that this phagocytic proces
77  during uptake of diverse targets, including latex beads, Escherichia coli, Salmonella typhimurium, a
78                                              Latex bead-exposed PEC were not more potent APC, and the
79  endocytic pathway do not permit endocytosed latex beads from reaching terminal lysosomes in an anter
80                              Phagocytosis of latex beads had no effect on the expression of thymosin
81 eas medium conditioned by Mphi phagocytizing latex beads had no proapoptotic effects upon neutrophils
82               In response to phagocytosis of latex beads, human embryonic kidney 293 cells synthesize
83 n not expressed by Pn, but not with 1-microm latex beads in adjuvant.
84  phoP(+) or phoP mutant Y. pestis strains or latex beads in J774A.1 macrophages.
85 ize in the vicinity of phagosomes containing latex beads in stimulated macrophages.
86 in vitro and opsonized particles (IgG-coated latex beads) in vitro and in vivo in intact mice.
87                               OVA-conjugated latex beads, in addition to stimulating brisk proliferat
88 fluorescein isothiocyanate (FITC)-conjugated latex beads, indicating that alveolar macrophages from P
89 taken to examine the mechanisms that mediate latex bead-induced resistance to Acanthamoeba keratitis.
90                      In contrast, IgG-coated latex bead-induced ROS production in human polymorphonuc
91  used to evaluate the role of neutrophils in latex-bead-induced protection against Acanthamoeba kerat
92 and determine the role of macrophages in the latex-bead-induced resistance.
93 ults indicate that intracorneal injection of latex beads induces a remarkable resistance to Acanthamo
94           Instillation of sterile 1.0 microM latex beads into the central corneal epithelium renders
95 ts of a retrograde tracer (usually rhodamine latex beads) into the superficial layers of the superior
96 icles (WGA-Au-HRP) or fluorescein-conjugated latex beads, into the LC labeled numerous (approximately
97                                              Latex bead labeling of peripheral blood Gr1(lo) monocyte
98 somes with phagosomes containing scintillant latex beads led to light emission in a reaction requirin
99 the giant vacuoles acquired large numbers of latex beads, M. tuberculosis, and avirulent L. pneumophi
100 o microscopy by tracking 10 mum, polystyrene latex beads mixed into the solution.
101  and LRG-47 localized to vacuoles containing latex beads, neither protein localized to vacuoles conta
102 was also stimulated by immune complex-coated latex beads or cross-linking of Abs specific for Fc gamm
103                        Phagosomes containing latex beads or heat-killed M. tuberculosis, on the other
104           In contrast, phagosomes containing latex beads or heat-killed organisms appeared to be proc
105 s between micrometer sized particles, either latex beads or red blood cells (RBCs), create aggregates
106 CD103(+) DCs were able to ingest and traffic latex beads or soluble antigen.
107 the specific nature of phagosomes containing latex beads or wild-type S. typhimurium (viable or heat-
108  alpha-MSH, were conjugated to microspheres (latex beads) or macrospheres (polyamide beads) through a
109  albumin or ovalbumin), amino-functionalized latex beads, or dextran polymer and arrayed at the surfa
110 e optical labels such as gold nanoparticles, latex beads, or fluorescent nanoparticles to visualize t
111 administered i.p. 200 mg/kg GaAs crystals or latex beads, or vehicle.
112        Also analyzed were phagocytized black latex beads, phagocytized melanosomes pretreated to simu
113  during intracellular transport, we examined latex beads phagocytosed into living mammalian macrophag
114 ll line internalized fluid phase markers and latex beads (phagocytosis) at one-third the rate of cont
115 ding protein profiles of these vesicles with latex bead phagosomal compartments (LBC).
116  diminished EEA1 recruitment to newly formed latex bead phagosomes and blocked phagosomal acquisition
117  of translocation of SK1G82D and SK1S225A to latex bead phagosomes were indistinguishable from those
118 osis H37Rv, inhibited recruitment of EEA1 to latex bead phagosomes, and diminished their maturation.
119 ced acquisition of late endocytic markers by latex bead phagosomes, demonstrating an essential role o
120 a decrease in EEA1 colocalization with model latex bead phagosomes, which normally acquire EEA1 and s
121 cytochalasin D prevented iNOS recruitment to latex bead phagosomes.
122 xin 4 and transferrin receptor on PIM-coated latex bead phagosomes.
123 er demonstrated by converse experiments with latex bead phagosomes.
124                                              Latex beads preferentially bound to a subpopulation of c
125                            ILC3s can take up latex beads, process protein antigen, and consequently p
126 actor (prfA) mutants of L. monocytogenes, or latex beads produced mss1 expression levels similar to t
127 e the opsonophagocytic uptake of ClfA-coated latex beads, protect against an intravenous challenge in
128    Neutrophil depletion did not abrogate the latex beads' protective effect.
129 crophagicidal drug clodronate eliminated the latex beads' protective effect.
130            Plasma membrane tethers formed by latex beads pull back on the beads with a force that was
131                                The uptake of latex beads showed that M-cell function was similar in a
132 is factor alpha/fibrinogen, immunoglobulin G latex beads, Staphylococcus aureus, formyl-methionyl-leu
133  Engulfment of the apoptotic bodies, but not latex beads, stimulated Kupffer cell generation of death
134 es ingest less apoptotic thymocytes (but not latex beads) than wild-type macrophages, and this defect
135 ling forces to human neutrophils adhering to latex beads that were coated with antibodies to CD62L (L
136 osomes were specifically labeled with 800-nm latex beads that were conjugated with streptavidin and A
137 , having been tethered to glass or marked by latex beads, the rotation of the internal components has
138     In fibroblasts plated on collagen-coated latex beads there are large increases of [Ca(2+)]i, time
139 nce was examined by assessing the binding of latex beads to neutrophils.
140  or beta1 integrin antibodies immobilized on latex beads to promote integrin aggregation.
141 ic binding of synthetic Le(x), conjugated to latex beads, to gastric tissue.
142                                              Latex bead treatment did not affect adherence of trophoz
143                                              Latex bead treatment induced a significant infiltration
144  In vitro experiments examined the effect of latex bead treatment on the capacity of A. castellanii t
145                   Model phagosomes harboring latex beads undergo a coordinated Rab5-Rab7 exchange, wh
146                   Phagocytosis of 4- micro m latex beads was also impaired in mAM cells as determined
147     The colocalization of iNOS with ingested latex beads was an actin-dependent process, since treatm
148  killed M.tb, live Staphylococcus aureus, or latex beads was associated with translocation of cytosol
149 mycobacterial antigens covalently coupled to latex beads was chloroquine insensitive.
150 ated epithelium, transcytosis of fluorescent latex beads was evaluated with confocal microscopy.
151 ocytosis of opsonized bacteria particles and latex beads was observed upon incubation of murine macro
152 of intimin, when purified and immobilized on latex beads, was sufficient to trigger A/E lesions on pr
153 , biocytin, fluorogold, and rhodamine-linked latex beads), we have shown that, as in other avian grou
154                                              Latex beads were coated with a recombinant fragment cont
155                                  Fluorescent latex beads were coated with fibronectin (FN), collagen
156           Fluorescent 0.2-microm polystyrene latex beads were either instilled into the conjunctival
157       In contrast, heat-killed organisms and latex beads were in acidic vacuoles which contained the
158 flammatory agents were also stimulatory, but latex beads were ineffective, indicating that microbial
159    Within 20 minutes of an in vivo exposure, latex beads were internalized by the follicle-associated
160  that infection and LPS were stimulatory but latex beads were not.
161    The rates of phagocytosis of bacteria and latex beads were significantly higher in Rap1 WT(+) and
162                                   The 100-nm latex beads were surface-modified with the temperature-s
163  acids was inefficient at promoting entry of latex beads, whereas dimerization of this derivative by
164 tion arrest, and model phagosomes containing latex beads, which follow the normal pathway of maturati
165 tics and the ability to bind and translocate latex beads, which make it indistinguishable from antige
166       Unlike the model phagosomes containing latex beads, which transiently recruited EEA1, mycobacte
167 athogen mimics (namely glycodendrimer-coated latex beads with acid-labile linkers) were synthesized.
168                                   Coating of latex beads with the major mycobacterial cell envelope g
169                     We have used fluorescent latex beads, with or without covalently conjugated OVA,

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