ライフサイエンスコーパス: latrophilin

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1 teins as endogenous postsynaptic ligands for latrophilins.

2 as interactors for the lectin-like domain of latrophilins.

3 rs: a G-protein-coupled receptor called CIRL/latrophilin 1 (CL1) and a cell-surface protein called ne

4 e probably by binding to two receptors, CIRL/latrophilin 1 (CL1) and neurexin Ialpha.

5 r alpha-latrotoxin have been described: CIRL/latrophilin 1 (CL1) and neurexin-1alpha.

6 The G-protein-coupled receptor CIRL1/latrophilin-1 (CL1) and the type-1 membrane proteins neu

7 ons of soluble teneurin-binding fragments of latrophilin-1 decreased synapse density, suggesting that

8 urin binding but has no effect on binding of latrophilin-1 to another ligand, FLRT3.

9 Latrophilin-1, -2, and -3 are adhesion-type G protein-co

10 ternative splicing at an N-terminal site; in latrophilin-1, this alternative splicing modulates teneu

11 ch includes olfactomedin, TIGR, Noelin-2 and latrophilin-1.

12 vation between the adhesion-GPCRs Celsr1 and Latrophilin-1.

13 deaminase, lipocalin 2, synaptotagmin 4, and latrophilin 2, whose time-dependent induction following

14 nd knockout mice, we show in this study that latrophilin-2 (Lphn2), a cell-adhesion G protein-coupled

15 EMT markers including slug, runx2, RhoA, and latrophilin-2.

16 s examined (megalin, thrombospondin-4, KR18, latrophilin-3, and phosphatidylinositol-3-OH kinase P101

17 hich shows approximately 65% identity to rat latrophilin, a G-coupled, seven span transmembrane prote

18 Using latrophilin, a well-studied member of the LNB family, we

19 Latrophilin adhesion-GPCRs (Lphn1-3 or ADGRL1-3) and Unc

20 LTX(N4C) binds to both LTX receptors (latrophilin and neurexin) and greatly enhances the frequ

21 Like the latrophilins and other members of the secretin family of

22 ndependent receptors for latrotoxin (CIRL or latrophilin) and neurexin 1 alpha receptors were found t

23 erve terminals, neurexins and CLs (CIRLs and latrophilins), and then executes a critical, second step

24 All latrophilins are subject to alternative splicing at an N

25 dhesion molecules, in addition to binding to latrophilins as heterophilic cell-adhesion molecules.

26 (2) a Ca(2+)-independent mechanism with CIRL/latrophilins as receptors, in which alpha-latrotoxin dir

27 1 decreased synapse density, suggesting that latrophilin binding to teneurin may directly or indirect

28 hesion, different from heterophilic teneurin-latrophilin binding.

29 Here we show that the Drosophila aGPCR Latrophilin/dCIRL acts in mechanosensory neurons by modu

30 yperactivity disorder, suggesting a role for latrophilins in human cognitive function.

31 onsistent with a role of teneurin binding to latrophilins in trans-synaptic interactions.

32 Latrophilins (LPHNs) are a small family of G protein-cou

33 phan receptor lat-1, a homolog of vertebrate latrophilins, plays an essential role in the establishme

34 neuroligins, cerebellin/GluD complexes, and latrophilins, thereby shaping the input/output relations

35 l-surface receptors, neurexins and CLs (Cirl/latrophilins), which probably have a physiological funct

36 a, together with binding studies, prove that latrophilin, which is linked to G proteins and inositol

37 We show that teneurin binds to latrophilins with nanomolar affinity and that this bindi

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