ライフサイエンスコーパス: lavage

1 d with maximal levels at 4 h (2.2 +/- 0.4 pg/lavage).

2 mphocytes and neutrophils in bronchoalveolar lavage.

3 All patients underwent bronchoalveolar lavage.

4 ell-free DNA (cfDNA) were isolated from each lavage.

5 sly reported cfDNA fraction from the uterine lavage.

6 entilated patients underwent bronchoalveolar lavage.

7 ry, chest x-ray study, and a bronchoalveolar lavage.

8 mbers of cells by whole-lung bronchoalveolar lavage.

9 patients of whom 159 underwent laparoscopic lavage.

10 and E. coli colony counts in bronchoalveolar lavage.

11 us and total fungus in their bronchoalveolar lavage.

12 Following lung injury induced by polysorbate lavage, a higher PEEP (20-24 cm H2O) with LTVV resulted

13 Laparoscopic lavage alone has been suggested as definitive treatment.

14 zed, out of whom 39 patients in laparoscopic lavage and 36 patients in the Hartmann procedure groups

15 ive procedures such as diagnostic peritoneal lavage and exploratory laparotomy were commonly utilized

16 -producing CD4(+) T cells in bronchoalveolar lavage and lung were significantly decreased in mice tha

17 f 12, 9, 6, 3, and 0 cm H2O before and after lavage and mechanical ventilation induced injury.

18 Uterine lavage and paired blood samples were collected and analy

19 ent T cell populations in the lower FGT from lavage and patient-matched peripheral blood to investiga

20 operational taxonomic units, as well as with lavage and peripheral blood neutrophilia.

21 reduces sEphrin-B2 levels in bronchoalveolar lavage and prevents lung fibrosis in mice.

22 Double-stranded RNA in bronchoalveolar lavage and serum samples following lung contusion was me

23 proinflammatory cytokines in both peritoneal lavage and serum.

24 To compare the outcomes of laparoscopic lavage and sigmoid resection in perforated diverticuliti

25 ients were randomly assigned to laparoscopic lavage and the Hartmann procedure with a median (first,

26 Randomization between laparoscopic lavage and the Hartmann procedure.

27 Bronchoalveolar lavage and tissues were sampled for mycobacterial persis

28 nimals through evaluation of bronchoalveolar lavage and tissues.

29 stress syndrome was induced by repeated lung lavages and injurious mechanical ventilation.

30 eritonitis" AND "therapeutic irrigation" or "lavage" AND randomized controlled trial and any derivati

31 lammatory cytokine levels in bronchoalveolar lavage, and alveolar capillary leakage.

32 putum, endobronchial biopsy, bronchoalveolar lavage, and blood samples.

33 hils were examined in blood, bronchoalveolar lavage, and endobronchial biopsies 48 hours after challe

34 hlebotomy, sputum induction, bronchoalveolar lavage, and endobronchial biopsy.

35 CR was done on urine, biopsy, cervicovaginal lavage, and genital mucosal surface specimens.

36 means of forced oscillation, bronchoalveolar lavage, and histologic analysis.

37 induced SPP1 in lung tissue, bronchoalveolar lavage, and serum increased more in male than in female

38 in the lungs, blood, liver, bronchoalveolar lavage, and spleens of mice at 24 h postinfection.

39 y antibodies present in sera, cervicovaginal lavages, and breast milk from HIV-1-infected individuals

40 In this trial, laparoscopic lavage as treatment for patients with perforated diverti

41 Cells recovered by bronchoalveolar lavage (BAL) after imaging were stained immunohistochemi

42 f human small airway tissue, bronchoalveolar lavage (BAL) and an experimental murine model of COPD.

43 ed numbers of eosinophils in bronchoalveolar lavage (BAL) and increased collagen content in the lung

44 test, airway inflammation in bronchoalveolar lavage (BAL) and lung tissue, and total free IgE in seru

45 activity were determined in bronchoalveolar lavage (BAL) and lungs of human donors and patients with

46 methods to identify HBEC in bronchoalveolar lavage (BAL) and wash (BW) have throughput limitations i

47 ived from mRNA isolated from bronchoalveolar lavage (BAL) cells and leukocytes of sarcoidosis patient

48 te the expression of iNOS in bronchoalveolar lavage (BAL) cells and tissue from central and periphera

49 of allergic asthma, matched bronchoalveolar lavage (BAL) fluid and plasma were collected from three

50 rmance has been validated on bronchoalveolar lavage (BAL) fluid and serum specimens, but recent evide

51 of inflammatory cytokines in bronchoalveolar lavage (BAL) fluid and the lungs.

52 ryngeal aspirates (NPAs) and bronchoalveolar lavage (BAL) fluid before HCT.

53 tor (VEGF) levels in hamster bronchoalveolar lavage (BAL) fluid early after intranasal challenge.

54 g was performed on acellular bronchoalveolar lavage (BAL) fluid from 30 subjects infected with HIV wi

55 Unlike blood ILC2s, bronchoalveolar lavage (BAL) fluid ILC2s from asthmatic patients were re

56 We examined bronchoalveolar lavage (BAL) fluid leukocytes, cytokines, mediators, and

57 eomycin injury increases the bronchoalveolar lavage (BAL) fluid levels of ATX protein 17-fold.

58 h nodes (MLNs), jejunum, and bronchoalveolar lavage (BAL) fluid of healthy and SIV-infected rhesus ma

59 f viruses collected from the bronchoalveolar lavage (BAL) fluid of the animals.

60 oteome analysis of acellular bronchoalveolar lavage (BAL) fluid samples on an observational prospecti

61 ous body sites, including bronchial alveolar lavage (BAL) fluid; sputum and lung tissue samples; and

62 ulosis-specific responses in bronchoalveolar lavage (BAL) from persons with latent M. tuberculosis in

63 ured mtDNA concentrations in bronchoalveolar lavage (BAL) from subjects with and without IPF, as well

64 s quantified by weight loss, bronchoalveolar lavage (BAL) inflammatory cell number, cytokine concentr

65 terminal airways-alveoli by bronchoalveolar lavage (BAL) of human adult lungs.

66 in digested lung tissue and bronchoalveolar lavage (BAL) simultaneously, 6 h after each ovalbumin (O

67 y and a multiplex assay with bronchoalveolar lavage (BAL) specimens (n = 68) from 52 highly character

68 CMV DNA threshold exists in bronchoalveolar lavage (BAL) to differentiate pneumonia from pulmonary s

69 responsiveness was assessed, bronchoalveolar lavage (BAL) was performed, and lung cells harvested for

70 d B cells in the spleens and bronchoalveolar lavage (BAL) were also observed.

71 nderwent SAC, and cells from bronchoalveolar lavage (BAL) were collected after 24 hours.

72 n anaerobic supernatants and bronchoalveolar lavage (BAL) were determined by gas chromatography.

73 ils and macrophages into the bronchoalveolar lavage (BAL), and human CD45(+) cells in the lungs.

74 Subjects underwent bronchoalveolar lavage (BAL), and peripheral whole blood was collected i

75 l immune responses in blood, bronchoalveolar lavage (BAL), and tracheobronchial lymph nodes.

76 ted fiberoptic bronchoscopy, bronchoalveolar lavage (BAL), endobronchial brushings, and biopsy.

77 ion of CD4(+) T cells in the bronchoalveolar lavage (BAL).

78 raphy, and bronchoscopy with bronchoalveolar lavage (BAL).

79 aeruginosa) were analyzed in bronchoalveolar lavage (BAL); and alveolar SGLT1 was analyzed by immunoh

80 ptom scores, spirometry, and bronchoalveolar lavage before and after rhinovirus-induced asthma exacer

81 Given that a uterine lavage can be easily and quickly performed even outside

82 Following silica treatment, bronchoalveolar lavage cell infiltrates decreased in female Spp1-/- mice

83 Bronchoalveolar lavage cell mRNA levels of iNOS or iNOS expression in ce

84 two independent study sites, bronchoalveolar lavage cells from donors with latent tuberculosis infect

85 lambda in bronchial epithelial and bronchial lavage cells in atopic asthmatics.

86 No differences in Th17 or Th1 lavage cells were found compared with controls.

87 osomal RNA gene sequencing of cervicovaginal lavage clustered each participant visit into a unique mi

88 y-four couples were enrolled; cervicovaginal lavage (CVL) and tissue were collected 2 hours after sex

89 ting study, 17 women provided cervicovaginal lavage (CVL) specimens at baseline (all had abstained fr

90 HIV-1 assay using plasma and cervicovaginal lavage (CVL) specimens.

91 lization for pneumonia with bronchioalveolar lavage demonstrating Pneumocystis jiroveci on confirmato

92 Morbidity and mortality after laparoscopic lavage did not differ when compared with the Hartmann pr

93 protein concentration in the bronchoalveolar lavage diminished, showing the impact of CXCR7 on stabil

94 allele fraction were present in the uterine lavage DNA samples.

95 ation phenotype indicated by bronchoalveolar lavage eosinophil surface markers, as well as the releas

96 as diagnosed on the basis of bronchoalveolar lavage eosinophilia and blood findings.

97 in circulating eosinophils, bronchoalveolar lavage eosinophilia, and eosinophil peroxidase depositio

98 efore mepolizumab treatment, bronchoalveolar lavage eosinophils had more surface IL-3 and granulocyte

99 s correlated with FEV1 and the proportion of lavage eosinophils.

100 bronchoscopic airway survey, bronchoalveolar lavage, esophageal pH monitoring, and a swallowing study

101 cles (EVs) are detectable in bronchoalveolar lavage fluid (BALF) and culture medium of lung epithelia

102 was to analyze cytokines in bronchoalveolar lavage fluid (BALF) and explore predicting factors of se

103 treptococcus pneumoniae, and bronchoalveolar lavage fluid (BALF) and lung CFU values were determined.

104 ics of GT and bmGT in serum, bronchoalveolar lavage fluid (BALF) and lungs of A. fumigatus-infected c

105 metabolites between matched bronchoalveolar lavage fluid (BALF) and plasma, identifies the degree of

106 BORT reduced bronchoalveolar lavage fluid (BALF) and tissue eosinophils and inflammat

107 s were assessed in lungs and bronchoalveolar lavage fluid (BALF) by multiparametric flow cytometry.

108 characterized by increased broncho-alveolar lavage fluid (BALF) cells and cytokines (IL-6 and TNF-al

109 the proinflammatory role of bronchoalveolar lavage fluid (BALF) exosomes in patients with sarcoidosi

110 We studied bronchoalveolar lavage fluid (BALF) from 36 patients with ARDS (20 survi

111 ng of serum, lung tissue and bronchoalveolar lavage fluid (BALF) from a non-lethal mouse model with i

112 of two biomarkers present in bronchoalveolar lavage fluid (BALF) from chlorine gas exposed mice.

113 when stimulated with LPS or bronchoalveolar lavage fluid (BALF) from patients with ARDS.

114 (HCMV) DNA detection in the bronchoalveolar lavage fluid (BALF) indicates HCMV replication in the pu

115 ed controls, ozone increased bronchoalveolar lavage fluid (BALF) protein, a marker of lung permeabili

116 Neutrophils in bronchoalveolar lavage fluid (BALF) served as markers of inflammation.

117 arallel, adenosine levels in bronchoalveolar lavage fluid (BALF) were increased by approximately 3-fo

118 g function measurements, and bronchoalveolar lavage fluid (BALF), serum, and lungs were collected on

119 gnificantly increased in the bronchoalveolar lavage fluid 48 hours after segmental allergen challenge

120 We found that relative bronchioalveolar lavage fluid adenosine levels are progressively elevated

121 neutrophil numbers and KC in bronchoalveolar lavage fluid and caused slight peribronchiolar inflammat

122 le reproductive tract tissue, cervicovaginal lavage fluid and its intracellular metabolite (TFV dipho

123 numbers of leukocytes in the bronchoalveolar lavage fluid and lower expression of inflammatory chemok

124 n ILC2s and TH2 cells in the bronchoalveolar lavage fluid and lung tissue were assessed.

125 biochemical measurements in bronchoalveolar lavage fluid and lung tissue.

126 monocytes/macrophages, mouse bronchoalveolar lavage fluid and lung tissues, and AHR in mice.

127 e presence of PRELP in human bronchoalveolar lavage fluid and showed that PRELP can be found in alveo

128 No changes in bronchoalveolar lavage fluid angiotensin-converting enzyme 2 activity we

129 spiratory distress syndrome broncho-alveolar lavage fluid by 60%, whereas serum amyloid P replenishme

130 munosuppressive functions of bronchoalveolar lavage fluid cells, inhibited bone marrow cell transendo

131 endobronchial brushings and bronchoalveolar lavage fluid collected from 39 asthmatic patients and 19

132 Analysis of bronchoalveolar lavage fluid collected from human patients with P. aerug

133 nophil and T cell numbers in bronchoalveolar lavage fluid compared with those in diluent-treated or c

134 eumonia demonstrated cytotoxic activity, and lavage fluid contained amyloid molecules, including olig

135 use, human CD49d(+) PMNs isolated from nasal lavage fluid during a viral respiratory tract infection

136 d dramatically increased in broncho-alveolar lavage fluid during acute respiratory distress syndrome.

137 frequency was significantly higher in nasal lavage fluid during acute respiratory symptoms in all su

138 n important mediator in resolving tissue and lavage fluid eosinophilia in allergic mouse models.

139 mucosal IgA was detected in bronchioalveolar lavage fluid for up to 6 weeks.

140 Mutations originally only detected in lavage fluid fractions were later confirmed to be presen

141 Bronchoalveolar lavage fluid from 23 lean, 12 overweight, and 20 obese s

142 counts, and total protein in bronchoalveolar lavage fluid from acute respiratory distress syndrome 1.

143 In bronchoalveolar lavage fluid from humans with ARDS, gut-specific bacteri

144 inhibited by treatment with bronchoalveolar lavage fluid from inhibitory kappaB kinase beta transact

145 H complexes were detected in bronchoalveolar lavage fluid from patients with acute respiratory distre

146 ssion patterns in plasma and bronchoalveolar lavage fluid from patients with ARDS.

147 were quantified in serum and bronchoalveolar lavage fluid from patients with CF, asthmatic patients,

148 ophilia were measured in the bronchoalveolar lavage fluid from patients with mild asthma 48 hours aft

149 s elevated in both serum and bronchoalveolar lavage fluid from Rasgrp1-deficient mice; 4) GM-CSF-spec

150 as non-ventilated controls' broncho-alveolar lavage fluid had no effect on fibrocyte differentiation.

151 eosinophilia, and increased bronchoalveolar lavage fluid IL-4 and IL-5, whereas adoptive transfer of

152 onverting enzyme activity in bronchoalveolar lavage fluid increased 3.2-fold in elderly when compared

153 spiratory distress syndrome broncho-alveolar lavage fluid inhibited by 71% (55-94) fibrocyte differen

154 elial cell proliferation and bronchoalveolar lavage fluid levels of keratinocyte growth factor were o

155 Bronchoalveolar lavage fluid LT levels were increased in neonatal and ad

156 Similarly, bronchoalveolar lavage fluid obtained from human volunteers exposed to O

157 ng could detect somatic mutations in uterine lavage fluid obtained from women undergoing hysteroscopy

158 nodes, peripheral blood, and bronchoalveolar lavage fluid of AGMs and rhesus macaques (in which CD4 d

159 tion driver mutations were identified in the lavage fluid of approximately half of the women without

160 OCS3 protein was elevated in bronchoalveolar lavage fluid of both virus- and bacteria-infected mice,

161 ines and chemokines into the bronchoalveolar lavage fluid of Lipa(-/-) mice.

162 e frequently detected in the bronchoalveolar lavage fluid of lung transplant patients diagnosed with

163 MUC1-ED were elevated in the bronchoalveolar lavage fluid of mechanically ventilated patients with P.

164 mined in protein, serum, and bronchoalveolar lavage fluid of mice and lungs and serum of human donors

165 he levels of chemerin in the bronchoalveolar lavage fluid of WT mice.

166 from eyelid skin and peritoneal and uterine lavage fluid provide unprecedented opportunities to diss

167 spiratory distress syndrome broncho-alveolar lavage fluid restored their full inhibitory effect.

168 virulence, while analysis of bronchoalveolar lavage fluid revealed that tumor necrosis factor alpha (

169 1, we used paired serum and bronchoalveolar lavage fluid samples obtained within 48 hours of acute r

170 Plasma and broncho-alveolar lavage fluid serum amyloid P contents were determined by

171 y attenuated the increase in bronchoalveolar lavage fluid SOCS3 noted in lungs of mice challenged wit

172 A3 and intracellular IL-4 in bronchoalveolar lavage fluid T cells, but expression of the TH17 transcr

173 Ventilated controls' broncho-alveolar lavage fluid was a less potent inhibitor (51% [23-66%] o

174 alyzed for immune cell subsets, and alveolar lavage fluid was analyzed for ILC2-derived cytokines.

175 Bronchoalveolar lavage fluid was collected from 23 steroid-free nonsmoki

176 Bronchoalveolar lavage fluid was collected from patients with severe ast

177 onor; however, pretransplant bronchoalveolar lavage fluid was only available from the donor for patie

178 The lavage fluid was separated into cellular and acellular f

179 Elevated levels of Cif in bronchoalveolar lavage fluid were correlated with lower levels of 15-epi

180 nts in homogenized lungs and bronchoalveolar lavage fluid were decreased after cranberry proanthocyan

181 tissues and Th2 responses in bronchoalveolar lavage fluid), they also accumulate functions normally a

182 nusitis (CRS), as well as in bronchoalveolar lavage fluid, after segmental allergen challenge in alle

183 trophil predominance in the bronchioalveolar lavage fluid, and enhanced airway mucus production.

184 ll counts and neutrophils in bronchoalveolar lavage fluid, and had earlier influx of macrophages.

185 C in lung tissue homogenate, bronchoalveolar lavage fluid, and lung LB was increased significantly in

186 and challenged with OVA and bronchoalveolar lavage fluid, and the lungs were collected for assessing

187 kines interleukin-5 (IL-5) and IL-13 in lung lavage fluid, decreased regulatory T cell-associated FOX

188 g cyto-/chemokine profile in bronchoalveolar lavage fluid, decreased TLR2/4 expression and NF-kappaB

189 counts were performed on the bronchoalveolar lavage fluid, followed by histological analysis of lung

190 y elevated total proteins in bronchoalveolar lavage fluid, higher parasitemia and tissue parasite bur

191 lammatory mediator levels in bronchoalveolar lavage fluid, ii) lung parenchymal leukocyte counts and

192 Remodeling factors in murine bronchoalveolar lavage fluid, lung tissue, or human nasal polyp tissue w

193 ells were detected in blood, bronchoalveolar lavage fluid, lungs, spleen, and brain, demonstrating th

194 of the injury markers in the bronchoalveolar lavage fluid, respectively.

195 or systemic health effects, bronchoalveolar lavage fluid, serum metabolic and inflammatory end point

196 In serum and bronchoalveolar lavage fluid, total anti-IAV IgG and IgA titers and viru

197 s adenosine levels found in bronchioalveolar lavage fluid, were determined in mouse models of resolva

198 hemoattractant MCP-1 in lung bronchoalveolar lavage fluid.

199 increase in the levels of CCL4 in peritoneal lavage fluid.

200 sinophils were enumerated in bronchoalveolar lavage fluid.

201 s, and lower cytokine levels in the alveolar lavage fluid.

202 and KLK14, were assessed in bronchoalveolar lavage fluid.

203 reducing LPA 18:2 content in bronchoalveolar lavage fluid.

204 tion and after culture with broncho-alveolar lavage fluid.

205 nia were observed in blood, dermis, and lung lavage fluid.

206 on of TNF-alpha and MIP-2 in bronchoalveolar lavage fluid.

207 cylcarnitines are detectable in normal human lavage fluid.

208 ion using RNA extracted from bronchoalveolar lavage fluid.

209 ng epithelium as detected in bronchoalveolar lavage fluid.

210 and IL-13, but not IL-5, in bronchoalveolar lavage fluid.

211 ntration of total protein in bronchoalveolar lavage fluids (BALF) from patients with sepsis-related a

212 ught to compare MP types and levels in nasal lavage fluids (NLFs) from controls and patients with chr

213 ly the wild-type virus was found in tracheal lavage fluids and urine.

214 centrations were reported in bronchoalveolar lavage fluids of asthmatic patients.

215 tion of CXCL12 in plasma and bronchoalveolar lavage fluids was quantified by ELISA.

216 say is validated for testing bronchoalveolar lavage fluids, replacing the requirement for culture and

217 imilar results were found in bronchoalveolar lavage fluids.

218 ound (EUS)-guided chemoablation with ethanol lavage followed by infusion of paclitaxel is effective f

219 ompared with the current standard of alcohol lavage followed by paclitaxel alone.

220 Nasal lavage for flow cytometry and nasal swabs for viral PCR

221 AMs were obtained from bronchoalveolar lavage from healthy donors or patients with COPD and cha

222 AMs were recovered from bronchoalveolar lavage from healthy subjects and patients with severe as

223 Bronchoalveolar lavage from mice expressing CAIKKbeta mice induced CD4(+

224 rophages, and neutrophils in bronchoalveolar lavage from O3-exposed mice.

225 e measured longitudinally in bronchoalveolar lavages from lung transplant recipients who developed br

226 nd neutrophil populations in bronchoalveolar lavages from mice with asthma.

227 dole, and IFN-gamma levels in cervicovaginal lavages from women with either naturally cleared or pers

228 ys postoperatively was 45/159 (28.3%) in the lavage group and 13/148 (8.8%) in the resection group (r

229 of 101 patients (30.7%) in the laparoscopic lavage group and 25 of 96 patients (26.0%) in the colon

230 Laparoscopic lavage has been suggested as a less invasive surgical tr

231 ely correlated with elevated bronchoalveolar lavage IL-8 levels (r(2) = 0.6095, p < 0.0001) and neutr

232 ery high percentages of Th17.1 cells in lung lavage in sarcoidosis compared with controls in two sepa

233 We found that bronchoalveolar lavage interleukin-17A was strongly associated with high

234 cific tendinopathy (local anesthesia, needle lavage, intrabursal steroid injection) was performed in

235 -17A-secreting cells have been found in lung lavage, invoking Th17 immunity in sarcoidosis.

236 Blood, bronchoalveolar lavage, large proximal and small distal airway brushings

237 Blood, bronchoalveolar lavage, large proximal, and small distal airway brushing

238 nflammation was evaluated by bronchoalveolar lavage, lung histology, serology, gene expression in lun

239 ehydrogenase and proteins in bronchoalveolar lavage, lung weight gain, perivascular edema, as well as

240 OVA-induced increases in bronchoalveolar lavage lymphocytes, eosinophils, IL-13, IL-5, and eotaxi

241 Bronchoalveolar lavage macrophages were stimulated in vitro, and iNOS ex

242 ort studies have suggested that laparoscopic lavage may treat perforated diverticulitis with less mor

243 oups, despite a reduction in bronchoalveolar lavage neutrophils.

244 Nasal lavage (NL) samples from 20 subjects who were exposed to

245 mal RNA sequencing data from bronchoalveolar lavage obtained as part of the COMET-IPF (Correlating Ou

246 l RNA were isolated from 203 bronchoalveolar lavages obtained from 112 patients post-lung transplanta

247 l RNA were isolated from 189 bronchoalveolar lavages obtained from 116 patients post lung transplanta

248 (IL-1alpha) was increased in bronchoalveolar lavage of lung transplant recipients growing P. aerugino

249 ture and was also present in bronchoalveolar lavage of lung transplantation patients.

250 tures were also found in the bronchoalveolar lavage of naive mice and were observed on their apical c

251 rated in vivo by challenging bronchoalveolar lavage of SET-M33-treated mice with LPS, which led to a

252 monary nocardiosis proved by bronchoalveolar lavage or biopsy were reviewed by two experienced pulmon

253 activation) and IL-6, in the bronchoalveolar lavage or the lung tissue, and histology.

254 nction of T lymphocytes from bronchoalveolar lavage or the peripheral blood.

255 ), systemic (p = 0.019), and bronchoalveolar lavage (p = 0.011) quantitative bacterial culture.

256 ongly associated with higher bronchoalveolar lavage percent neutrophils (p < 0.001) and bronchoalveol

257 in reduced edema formation (bronchoalveolar lavage protein concentration and lung histology) and imp

258 g control cells with disease bronchoalveolar lavage recapitulated the aberrant functional phenotype,

259 Laparoscopic lavage reduced the need for reoperations, had a similar

260 s with severe asthma in whom bronchoalveolar lavage regulatory T-cell numbers were also reduced.

261 airways (bronchial wash and bronchoalveolar lavage, respectively), as well as mucosal tissue (endobr

262 Laparoscopic lavage resulted in shorter operating time, shorter time

263 ents can be safely managed with laparoscopic lavage, resulting in reduced mortality and stoma formati

264 Blood, bone marrow, and bronchoalveolar lavage sample analyses from juvenile and 20-24-mo-old mi

265 Penile lavage samples collected from the coronal sulcus at base

266 h upper airway and acellular bronchoalveolar lavage samples from 49 subjects from three cohorts witho

267 ces in microbial diversity between brush and lavage samples from asthmatic patients and control subje

268 demonstrated that acellular bronchoalveolar lavage samples from half of the healthy people examined

269 tive) forms of IL-33 can be detected in lung lavage samples from mice challenged with Alternaria extr

270 Vaginal lavage samples from nonpregnant women were tested for GB

271 Bronchoalveolar lavage samples from Ugandan patients with HIV and pneumo

272 olymorphism with microbiota from 338 mucosal lavage samples in the Mucosal Luminal Interface cohort m

273 I mRNA and protein levels in bronchoalveolar lavage samples obtained before and after segmental aller

274 ctomannan assay in serum and bronchoalveolar lavage samples were 61.3% and 57.1%, respectively.

275 sis (MTB) mRNA in sputum and bronchoalveolar lavage samples, in a substantial proportion of adult, HI

276 l DNA was monitored periodically by Q-PCR of lavage samples.

277 Cervicovaginal lavage sequencing (n = 109) resulted in a low-diversity

278 Measurement of cytokines from the pleural lavage showed that mice implanted with MPM cells express

279 tes dates back to its initial development as lavage solutions in preparation for gastrointestinal int

280 conducted annually in serial cervicovaginal lavage specimens obtained over 8-10 years from African A

281 Automated peritoneal lavage system is a safe and ultrarapid method to induce

282 The lavage technique could be applied successfully, and suff

283 Lung lavage Th17.1-cell percentages were also higher than Th1

284 Following lung injury induced by polysorbate lavage, the APRV group was ventilated with a progressive

285 Following lung injury induced by polysorbate lavage, the LTVV group was ventilated with a tidal volum

286 rfactant deactivation induced by polysorbate lavage: the LTVV group (n = 5) and the APRV group (n = 5

287 neutrophils (p < 0.001) and bronchoalveolar lavage total protein (p < 0.01) in acute respiratory dis

288 Laparoscopic lavage used in the management of Hinchey grade III diver

289 Laparoscopic lavage was compared with colon resection and stoma in a

290 ree days after instillation, bronchoalveolar lavage was performed and plastic-adherent cells were cul

291 findings, bronchoscopy with bronchoalveolar lavage was performed.

292 anesthetized and intubated, and saline lung lavage was performed.

293 , the viral load in salivary glands and oral lavage was strongly decreased in the MCMV-infected Ebi3(

294 opsies, and Th1 and Th2 cytokines from nasal lavage were assessed in subjects with severe PAR (n = 46

295 coli colony-forming units in bronchoalveolar lavage were reduced in both cell therapy groups, despite

296 tis was induced by SEB injection; peritoneal lavages were collected after 48 h and analyzed by flow c

297 with IFN-gamma expression in bronchoalveolar lavage, while inducing its expression in vitro in primar

298 ps that underwent EUS-guided pancreatic cyst lavage with either 80% ethanol (control) or normal salin

299 Pretreatment or single intraperitoneal lavage with YC-1 significantly reduced postoperative adh

300 s syndrome was induced combining saline lung lavages with injurious mechanical ventilation.