ライフサイエンスコーパス: lavage fluid

1 on of TNF-alpha and MIP-2 in bronchoalveolar lavage fluid.

2 cylcarnitines are detectable in normal human lavage fluid.

3 nia were observed in blood, dermis, and lung lavage fluid.

4 crophages and eosinophils in bronchoalveolar lavage fluid.

5 ers of inflammatory cells in bronchoalveolar lavage fluid.

6 racts, nasal secretions, and bronchoalveolar lavage fluid.

7 ells and readily detected in bronchoalveolar lavage fluid.

8 ion using RNA extracted from bronchoalveolar lavage fluid.

9 entage of eosinophils in the bronchoalveolar lavage fluid.

10 triene (cysLT) levels were measured in nasal lavage fluid.

11 ssociated with increased IL-17 production in lavage fluid.

12 nd cytokine concentration in bronchoalveolar lavage fluid.

13 nfiltration and interleukin-5 (IL-5) in lung lavage fluid.

14 rophages were detected in bronchial alveolar lavage fluid.

15 sinophils were enumerated in bronchoalveolar lavage fluid.

16 ng epithelium as detected in bronchoalveolar lavage fluid.

17 and IL-13, but not IL-5, in bronchoalveolar lavage fluid.

18 ammatory cell numbers in the bronchoalveolar lavage fluid.

19 Cells were counted in bronchoalveolar lavage fluid.

20 in saliva correlates with levels in vaginal lavage fluid.

21 IL-33, and eotaxin levels in bronchoalveolar lavage fluid.

22 eduction in Th2 cytokines in bronchoalveolar lavage fluid.

23 lular necrosis, in cell-free bronchoalveolar lavage fluid.

24 d Th1 and Th2 cytokines were analyzed in the lavage fluid.

25 hemoattractant MCP-1 in lung bronchoalveolar lavage fluid.

26 increase in the levels of CCL4 in peritoneal lavage fluid.

27 s, and lower cytokine levels in the alveolar lavage fluid.

28 and KLK14, were assessed in bronchoalveolar lavage fluid.

29 reducing LPA 18:2 content in bronchoalveolar lavage fluid.

30 tion and after culture with broncho-alveolar lavage fluid.

31 imilar results were found in bronchoalveolar lavage fluids.

32 3 measured by ELISA in their bronchoalveolar lavage fluids.

33 gher bacterial burden in the bronchoalveolar lavage fluid 3 h following infection.

34 gnificantly increased in the bronchoalveolar lavage fluid 48 hours after segmental allergen challenge

35 ns (5 cerebrospinal fluid, 7 bronchoalveolar lavage fluid, 5 plasma, 2 serum, and 1 nasopharyngeal as

36 We found that relative bronchioalveolar lavage fluid adenosine levels are progressively elevated

37 L-5, IL-13, and eotaxin-1 in bronchoalveolar lavage fluid after OVA challenge.

38 nusitis (CRS), as well as in bronchoalveolar lavage fluid, after segmental allergen challenge in alle

39 ncreased ALI (as assessed by bronchoalveolar lavage fluid albumin concentration) in both NADPH oxidas

40 ith sHLA-G concentrations in bronchoalveolar lavage fluid among asthmatic subjects with an asthmatic

41 ammatory cytokines and eosinophil numbers in lavage fluid and augmented the histopathologic evidence

42 neutrophil numbers and KC in bronchoalveolar lavage fluid and caused slight peribronchiolar inflammat

43 ges in inflammatory cells in bronchoalveolar lavage fluid and cytokine expression in lung tissue.

44 d expression of TNF-alpha in bronchoalveolar lavage fluid and deteriorates lung functions.

45 Th2 cytokines were found in bronchoalveolar lavage fluid and draining lymph node cells of Nur77-KO m

46 nt inflammation in blood and bronchoalveolar lavage fluid and exacerbations despite high systemic cor

47 DM, the levels of PGP in the bronchoalveolar lavage fluid and infiltration of neutrophils into the lu

48 le reproductive tract tissue, cervicovaginal lavage fluid and its intracellular metabolite (TFV dipho

49 numbers of leukocytes in the bronchoalveolar lavage fluid and lower expression of inflammatory chemok

50 n ILC2s and TH2 cells in the bronchoalveolar lavage fluid and lung tissue were assessed.

51 biochemical measurements in bronchoalveolar lavage fluid and lung tissue.

52 xtracellular traps (EETs) in bronchoalveolar lavage fluid and lung tissue.

53 n and levels of cytokines in bronchoalveolar lavage fluid and lung tissue.

54 monocytes/macrophages, mouse bronchoalveolar lavage fluid and lung tissues, and AHR in mice.

55 Bronchoalveolar lavage fluid and lungs were harvested for histopathologi

56 phocytes, and macrophages in bronchoalveolar lavage fluid and lungs; inflammatory cell infiltrates in

57 ociated with lower lymphocyte percentages in lavage fluid and marginally lower plasma cortisol concen

58 NETs were also measured in bronchoalveolar lavage fluid and plasma from lung transplant patients wi

59 d in BOS versus normal lung tissues and both lavage fluid and plasma HA concentrations were increased

60 to extracellular fluid (eg, bronchoalveolar lavage fluid and plasma).

61 mmatory cell accumulation in bronchoalveolar lavage fluid and proinflammatory cytokines levels in lun

62 IL-8 levels were measured in nasal lavage fluid and serum on randomization, day 8, and day

63 e presence of PRELP in human bronchoalveolar lavage fluid and showed that PRELP can be found in alveo

64 rophilic inflammation in the bronchoalveolar lavage fluid and Th1, Th2, and Th17 inflammation in the

65 ear leukocytes (PMNs) in the bronchoalveolar lavage fluid and the wet-to-dry lung weight ratio, a mea

66 s the major MMP found in the bronchoalveolar lavage fluids and bronchial biopsies from patients with

67 e detected in nasal wash and bronchoalveolar lavage fluids and in sera from mice intranasally inocula

68 uced accumulation of IL-6 in bronchoalveolar lavage fluids and NADPH oxidase (NOX) 2 and NOX4 protein

69 ly the wild-type virus was found in tracheal lavage fluids and urine.

70 morphonuclear recruitment in bronchoalveolar lavage fluid), and lethality were evaluated in a pneumon

71 gher amounts of IL-17 in the bronchoalveolar lavage fluid, and (iii) more neutrophils in the lungs th

72 trophil predominance in the bronchioalveolar lavage fluid, and enhanced airway mucus production.

73 ll counts and neutrophils in bronchoalveolar lavage fluid, and had earlier influx of macrophages.

74 content and cell count in bronchial alveolar lavage fluid, and increased Evans Blue dye infiltration

75 C in lung tissue homogenate, bronchoalveolar lavage fluid, and lung LB was increased significantly in

76 mmation, cytokine levels in broncho-alveolar lavage fluid, and mucus production were determined.

77 odies was measured in tissue extracts, nasal lavage fluid, and sera by using multiplex bead arrays an

78 and challenged with OVA and bronchoalveolar lavage fluid, and the lungs were collected for assessing

79 emokines eotaxin and CCL2 in bronchoalveolar lavage fluid; and decreased pulmonary inflammation, as w

80 ater than 20% lymphocytes in bronchoalveolar lavage fluid; and three had findings on surgical lung bi

81 No changes in bronchoalveolar lavage fluid angiotensin-converting enzyme 2 activity we

82 creased extracellular DNA in bronchoalveolar lavage fluid as well as in lung tissue, confirming the p

83 tissues, including lung and bronchoalveolar lavage fluid, as measured by H2-Db NP366 and PA224 tetra

84 oglobulin E were measured in bronchoalveolar lavage fluid at baseline and 24 h after allergen instill

85 s), and IgE were measured in bronchoalveolar lavage fluid at baseline and 24 hours after allergen ins

86 greater drug levels in lung, bronchoalveolar lavage fluid (BAL), and plasma compared to conventional

87 ecific memory CD8 T cells in bronchoalveolar lavage fluid (BAL), lungs, and spleen.

88 njury was evaluated by using bronchoalveolar lavage fluid (BALF) analysis and pathology.

89 eased concentration in both bronchioalveolar lavage fluid (BALF) and blood of doxycycline-treated CCS

90 cles (EVs) are detectable in bronchoalveolar lavage fluid (BALF) and culture medium of lung epithelia

91 Lung tissue, bronchoalveolar lavage fluid (BALF) and draining lymph node cells were a

92 was to analyze cytokines in bronchoalveolar lavage fluid (BALF) and explore predicting factors of se

93 treptococcus pneumoniae, and bronchoalveolar lavage fluid (BALF) and lung CFU values were determined.

94 ics of GT and bmGT in serum, bronchoalveolar lavage fluid (BALF) and lungs of A. fumigatus-infected c

95 metabolites between matched bronchoalveolar lavage fluid (BALF) and plasma, identifies the degree of

96 ssess airway inflammation in bronchoalveolar lavage fluid (BALF) and Th2 responses in lung explants a

97 BORT reduced bronchoalveolar lavage fluid (BALF) and tissue eosinophils and inflammat

98 s were assessed in lungs and bronchoalveolar lavage fluid (BALF) by multiparametric flow cytometry.

99 Bacterial burdens, bronchoalveolar lavage fluid (BALF) cell counts, cell types, and cytokin

100 characterized by increased broncho-alveolar lavage fluid (BALF) cells and cytokines (IL-6 and TNF-al

101 ed lung bacterial clearance, bronchoalveolar lavage fluid (BALF) characterization, lung histology, lu

102 ity decreased by 80% in COPD bronchoalveolar lavage fluid (BALF) due to serine protease cleavage, pri

103 the proinflammatory role of bronchoalveolar lavage fluid (BALF) exosomes in patients with sarcoidosi

104 We studied bronchoalveolar lavage fluid (BALF) from 36 patients with ARDS (20 survi

105 ng of serum, lung tissue and bronchoalveolar lavage fluid (BALF) from a non-lethal mouse model with i

106 ne profiles were measured in bronchoalveolar lavage fluid (BALF) from children with CF (n = 57; 6.1 +

107 of two biomarkers present in bronchoalveolar lavage fluid (BALF) from chlorine gas exposed mice.

108 on neutrophils in blood and bronchoalveolar lavage fluid (BALF) from mechanically ventilated patient

109 when stimulated with LPS or bronchoalveolar lavage fluid (BALF) from patients with ARDS.

110 mmation and cytokines in the bronchoalveolar lavage fluid (BALF) in a murine model of allergic asthma

111 (HCMV) DNA detection in the bronchoalveolar lavage fluid (BALF) indicates HCMV replication in the pu

112 h the observation that local bronchoalveolar lavage fluid (BALF) levels of vitamin D are severely def

113 CD4(+) T cells in the bronchoalveolar lavage fluid (BALF) of Af5517-aspirated mice displayed d

114 ed controls, ozone increased bronchoalveolar lavage fluid (BALF) protein, a marker of lung permeabili

115 Neutrophils in bronchoalveolar lavage fluid (BALF) served as markers of inflammation.

116 Bronchoalveolar lavage fluid (BALF) was analysed for total protein, lact

117 A04-neutralizing activity of bronchoalveolar lavage fluid (BALF) was observed following secondary CA0

118 Broncho alveolar lavage fluid (BALF) was sampled from donors prior to pro

119 Lung tissue and bronchoalveolar lavage fluid (BALF) were analyzed for inflammation, as w

120 arallel, adenosine levels in bronchoalveolar lavage fluid (BALF) were increased by approximately 3-fo

121 of Th2 and Th17 cytokines in bronchoalveolar lavage fluid (BALF), accompanied by an increment in tran

122 Bronchoalveolar lavage fluid (BALF), airway inflammation and hyperrespon

123 e levels in spleen cells and bronchoalveolar lavage fluid (BALF), and cellular distribution in BALF w

124 r 33 d after exposure, SpO2, bronchoalveolar lavage fluid (BALF), and histologic analyses were perfor

125 mber of cells and macrophages in bronchiolar lavage fluid (BALF), as well infiltrating inflammatory c

126 ice, with fewer cells in Wsh bronchoalveolar lavage fluid (BALF), despite similar levels of cytokines

127 g function measurements, and bronchoalveolar lavage fluid (BALF), serum, and lungs were collected on

128 f aspiration (bile salts) in bronchoalveolar lavage fluid (BALF).

129 ntent in MMP active forms of bronchoalveolar lavage fluids (BALf) from male C57BL/6 mice exposed to u

130 ntration of total protein in bronchoalveolar lavage fluids (BALF) from patients with sepsis-related a

131 activation, was detected in bronchoalveolar lavage fluids (BALF) in a macrophage- and neutrophil-dep

132 ic response were assessed in bronchoalveolar lavage fluids (BALFs) after allergen challenge.

133 spiratory distress syndrome broncho-alveolar lavage fluid by 60%, whereas serum amyloid P replenishme

134 eased lung injury scores and bronchoalveolar lavage fluid cell numbers in control but not TREK-1-defi

135 Total bronchoalveolar lavage fluid cell numbers increased from 13+/-4 to 106+/

136 rease in lung injury scores, bronchoalveolar lavage fluid cell numbers, and cellular apoptosis and a

137 ginal virus titers were detected in tracheal lavage fluid cells of N4-blind MV-infected hosts.

138 munosuppressive functions of bronchoalveolar lavage fluid cells, inhibited bone marrow cell transendo

139 evere asthma whole blood and bronchoalveolar lavage fluid cells.

140 ing cellular accumulation in bronchoalveolar lavage fluid, collagen levels, and histologic changes.

141 endobronchial brushings and bronchoalveolar lavage fluid collected from 39 asthmatic patients and 19

142 Analysis of bronchoalveolar lavage fluid collected from human patients with P. aerug

143 nophil and T cell numbers in bronchoalveolar lavage fluid compared with those in diluent-treated or c

144 F production in the lung and bronchoalveolar lavage fluid compared with wild-type mice, without chang

145 y cytokines/chemokines in bronchial alveolar lavage fluids compared to WT mice.

146 vity), and neutrophils in bronchial alveolar lavage fluids compared with wild-type mice.

147 inflammatory cytokines in perfusate and lung lavage fluid, compared to control.

148 eumonia demonstrated cytotoxic activity, and lavage fluid contained amyloid molecules, including olig

149 y inflammation and function, bronchoalveolar lavage fluid cytokine levels, and flow cytometry of lung

150 Outcome measures included: bronchoalveolar lavage fluid cytology to assess airway eosinophilia, pul

151 kines interleukin-5 (IL-5) and IL-13 in lung lavage fluid, decreased regulatory T cell-associated FOX

152 g cyto-/chemokine profile in bronchoalveolar lavage fluid, decreased TLR2/4 expression and NF-kappaB

153 tive microbiology applied to bronchoalveolar lavage fluid derived from infected segments proved an in

154 the proinflammatory role of bronchoalveolar lavage fluid-derived exosomes in asthmatic progression,

155 use, human CD49d(+) PMNs isolated from nasal lavage fluid during a viral respiratory tract infection

156 d dramatically increased in broncho-alveolar lavage fluid during acute respiratory distress syndrome.

157 frequency was significantly higher in nasal lavage fluid during acute respiratory symptoms in all su

158 okines and chemokines in the bronchoalveolar lavage fluid, enhanced bronchoalveolar cellular influx,

159 roliferation (Ki-67(+)), and bronchoalveolar lavage fluid eosinophil numbers were measured.

160 n important mediator in resolving tissue and lavage fluid eosinophilia in allergic mouse models.

161 ssed, the inflammatory cell content in nasal lavage fluids estimated, and the activation pattern of p

162 ic fibrosis underwent annual bronchoalveolar lavage fluid examination, and chest computed tomography.

163 Our findings validate bronchoalveolar lavage fluid exosomal shuttle RNA as a source for unders

164 Bronchoalveolar lavage fluid exosomes were collected from healthy indivi

165 counts were performed on the bronchoalveolar lavage fluid, followed by histological analysis of lung

166 mucosal IgA was detected in bronchioalveolar lavage fluid for up to 6 weeks.

167 Siglec-F ligand activity in bronchoalveolar lavage fluid fractions containing polymeric secreted muc

168 Mutations originally only detected in lavage fluid fractions were later confirmed to be presen

169 Bronchoalveolar lavage fluid from 23 lean, 12 overweight, and 20 obese s

170 counts, and total protein in bronchoalveolar lavage fluid from acute respiratory distress syndrome 1.

171 Bronchoalveolar lavage fluid from Ad-MD-2s mice transferred into lungs o

172 CG and NE in bronchoalveolar lavage fluid from CF patients both contributed to C5aR c

173 In bronchoalveolar lavage fluid from human lung transplant recipients, NETs

174 In bronchoalveolar lavage fluid from humans with ARDS, gut-specific bacteri

175 inhibited by treatment with bronchoalveolar lavage fluid from inhibitory kappaB kinase beta transact

176 significantly higher in the bronchoalveolar lavage fluid from OVA/OVA than OVA/PBS mice and were una

177 H complexes were detected in bronchoalveolar lavage fluid from patients with acute respiratory distre

178 icant elevation of IL-17A in bronchoalveolar lavage fluid from patients with ARDS, and rIL-17A direct

179 ssion patterns in plasma and bronchoalveolar lavage fluid from patients with ARDS.

180 were quantified in serum and bronchoalveolar lavage fluid from patients with CF, asthmatic patients,

181 ophilia were measured in the bronchoalveolar lavage fluid from patients with mild asthma 48 hours aft

182 s elevated in both serum and bronchoalveolar lavage fluid from Rasgrp1-deficient mice; 4) GM-CSF-spec

183 Additionally, bronchoalveolar lavage fluid from this group of hepSTAT3(-/-) mice allow

184 tokines were compared in the bronchoalveolar lavage fluid from WT and SP-D(-/-) mice after C. neoform

185 veolar epithelial cells with bronchoalveolar lavage fluids from ARDS patients drove betaENaC internal

186 Peritoneal lavage fluids from CREB-inhibited tumor-bearing mice sho

187 Bronchoalveolar lavage fluids from ozone-treated rats reproduced this ef

188 as non-ventilated controls' broncho-alveolar lavage fluid had no effect on fibrocyte differentiation.

189 ate surfactant isolated from LCAD(-/-) mouse lavage fluid had significantly reduced phospholipid cont

190 y elevated total proteins in bronchoalveolar lavage fluid, higher parasitemia and tissue parasite bur

191 ines in lung homogenates and bronchoalveolar lavage fluid; however, PMN recruitment in mice treated w

192 lammatory mediator levels in bronchoalveolar lavage fluid, ii) lung parenchymal leukocyte counts and

193 eosinophilia, and increased bronchoalveolar lavage fluid IL-4 and IL-5, whereas adoptive transfer of

194 but resulted in a greater decrease in nasal lavage fluid IL-8 levels by day 15 (P = .03).

195 Bronchoalveolar lavage fluid IL-9 and IL-10, serum IL-9, and lung IL-17R

196 uction was attenuated in the bronchoalveolar lavage fluid in all factor-deficient mice compared with

197 solute neutrophil numbers in bronchoalveolar lavage fluid in GSTM1+ but not GSTM1null asthmatics.

198 inflammatory protein-2 in bronchial alveolar lavage fluids in LPS-injured lung compared with wild-typ

199 onverting enzyme activity in bronchoalveolar lavage fluid increased 3.2-fold in elderly when compared

200 terleukin-1beta levels obtained from vaginal lavage fluid increased by day 3 onward.

201 spiratory distress syndrome broncho-alveolar lavage fluid inhibited by 71% (55-94) fibrocyte differen

202 delayed (6-8 hr) increase in bronchoalveolar lavage fluid interleukin-6 concentration (p < 0.001) and

203 ation sufficiently decreased bronchoalveolar lavage fluid levels of IFN-gamma at day 7 postinfluenza

204 as associated with increased bronchoalveolar lavage fluid levels of IL-10 and TGF-beta and decreased

205 elial cell proliferation and bronchoalveolar lavage fluid levels of keratinocyte growth factor were o

206 in a remarkable increase in bronchoalveolar lavage fluid levels of LPA enriched in polyunsaturated 2

207 s in infiltrating neutrophils (23% and 68%), lavage fluid levels of TNF-alpha (70% and 80%), and the

208 Bronchoalveolar lavage fluid LT levels were increased in neonatal and ad

209 ne levels were determined in bronchoalveolar lavage fluid, lung homogenates and lung mononuclear cell

210 Remodeling factors in murine bronchoalveolar lavage fluid, lung tissue, or human nasal polyp tissue w

211 ells were detected in blood, bronchoalveolar lavage fluid, lungs, spleen, and brain, demonstrating th

212 cells was increased in lung, bronchoalveolar lavage fluid, lymph nodes, and blood of allergic COX-2(-

213 he study day, and plasma and bronchoalveolar lavage fluid markers of inflammation were measured.

214 on, including cell counts in bronchoalveolar lavage fluid, mucin production, ASM mass, and subepithel

215 tracheal aspirates (n = 71), bronchoalveolar lavage fluid (n = 152), pleural fluid (n = 76), cerebral

216 G (sHLA-G) concentrations in bronchoalveolar lavage fluid (n = 36) and plasma (n = 57) from adult ast

217 Bronchoalveolar lavage fluid neutrophils, total protein, LDH, CXCL1/KC a

218 We analysed cytokine levels in nasal lavage fluid (NLF) in 59 subjects (46 with asthma) with

219 ught to compare MP types and levels in nasal lavage fluids (NLFs) from controls and patients with chr

220 Bronchoalveolar lavage fluid obtained during this bronchospasm contained

221 Similarly, bronchoalveolar lavage fluid obtained from human volunteers exposed to O

222 control lung tissue, (ii) in bronchoalveolar lavage fluid obtained from non-severe and severe asthmat

223 ifferential counts were evaluated from nasal lavage fluid obtained from wild-type and IL-17A-deficien

224 ng could detect somatic mutations in uterine lavage fluid obtained from women undergoing hysteroscopy

225 kines, and growth factors in bronchoalveolar lavage fluid of 20 stable patients, 20 patients sufferin

226 rnithine) were isolated from bronchoalveolar lavage fluid of a patient with idiopathic pulmonary fibr

227 nodes, peripheral blood, and bronchoalveolar lavage fluid of AGMs and rhesus macaques (in which CD4 d

228 ignificantly elevated in the bronchoalveolar lavage fluid of all mice infected with C. neoformans Del

229 evels were also increased in bronchoalveolar lavage fluid of allergic asthmatic patients after segmen

230 tion driver mutations were identified in the lavage fluid of approximately half of the women without

231 tly, the IL-22 expression in bronchoalveolar lavage fluid of asthmatic patients inversely correlated

232 OCS3 protein was elevated in bronchoalveolar lavage fluid of both virus- and bacteria-infected mice,

233 the number of neutrophils in bronchoalveolar lavage fluid of C3aR-deficient mice.

234 e isolated and detected from bronchoalveolar lavage fluid of control and asthmatic mice and were quan

235 ned that cells collected from the peritoneal lavage fluid of E. faecalis-infected mice showed reduced

236 mistry and Luminex assays in bronchoalveolar lavage fluid of healthy and asthmatic patients.

237 found ILC3-like cells in the bronchoalveolar lavage fluid of individuals with asthma, we suggest that

238 ines and chemokines into the bronchoalveolar lavage fluid of Lipa(-/-) mice.

239 e frequently detected in the bronchoalveolar lavage fluid of lung transplant patients diagnosed with

240 MUC1-ED were elevated in the bronchoalveolar lavage fluid of mechanically ventilated patients with P.

241 mined in protein, serum, and bronchoalveolar lavage fluid of mice and lungs and serum of human donors

242 5-HT levels are increased in bronchoalveolar lavage fluid of mice and people with asthma after allerg

243 of B20-treated cells and in bronchoalveolar lavage fluid of mice exposed to B20 were approximately 2

244 ophils, and cytokines in the bronchoalveolar lavage fluid of mice than its mutant counterpart 7 days

245 roteins contained within the bronchoalveolar lavage fluid of mice that are cleaved and/or processed b

246 ophils were increased in the bronchoalveolar lavage fluid of wild-type mice.

247 he levels of chemerin in the bronchoalveolar lavage fluid of WT mice.

248 centrations were reported in bronchoalveolar lavage fluids of asthmatic patients.

249 he level of ATX exoenzyme in bronchoalveolar lavage fluids of human patients with asthma subjected to

250 5-HT levels were measured in bronchoalveolar lavage fluid or serum of animals with AAI and in human a

251 onuclear cell recruitment in bronchoalveolar lavage fluid (p<0.05 for both).

252 measured by respiratory system mechanics and lavage fluid protein.

253 from eyelid skin and peritoneal and uterine lavage fluid provide unprecedented opportunities to diss

254 cluding IL-4 and IL-5 in the bronchoalveolar lavage fluid relative to control mice.

255 say is validated for testing bronchoalveolar lavage fluids, replacing the requirement for culture and

256 of the injury markers in the bronchoalveolar lavage fluid, respectively.

257 spiratory distress syndrome broncho-alveolar lavage fluid restored their full inhibitory effect.

258 Cytologic examination of bronchial alveolar lavage fluid revealed a high proportion of lipid-laden m

259 virulence, while analysis of bronchoalveolar lavage fluid revealed that tumor necrosis factor alpha (

260 onventional culture by using bronchoalveolar lavage fluid samples from patients with underlying respi

261 1, we used paired serum and bronchoalveolar lavage fluid samples obtained within 48 hours of acute r

262 Endobronchial biopsies and bronchoalveolar lavage fluid samples were collected from 13 GSTM1+ and 1

263 Plasma and broncho-alveolar lavage fluid serum amyloid P contents were determined by

264 or systemic health effects, bronchoalveolar lavage fluid, serum metabolic and inflammatory end point

265 y attenuated the increase in bronchoalveolar lavage fluid SOCS3 noted in lungs of mice challenged wit

266 nt, was significantly increased in LCAD(-/-) lavage fluid, suggesting increased epithelial permeabili

267 A3 and intracellular IL-4 in bronchoalveolar lavage fluid T cells, but expression of the TH17 transcr

268 a had increased HA levels in bronchoalveolar lavage fluid that correlated with pulmonary function and

269 tissues and Th2 responses in bronchoalveolar lavage fluid), they also accumulate functions normally a

270 intra-alveolar coagulation (bronchoalveolar lavage fluid thrombin-antithrombin complex levels) and P

271 histopathologic changes and bronchoalveolar lavage fluid total protein (endothelial permeability) an

272 In serum and bronchoalveolar lavage fluid, total anti-IAV IgG and IgA titers and viru

273 .03) and a minor increase in bronchoalveolar lavage fluid tumor necrosis factor-alpha were observed (

274 neutrocytosis and increased bronchoalveolar lavage fluid tumor necrosis factor-alpha, interleukin-6,

275 Ventilated controls' broncho-alveolar lavage fluid was a less potent inhibitor (51% [23-66%] o

276 alyzed for immune cell subsets, and alveolar lavage fluid was analyzed for ILC2-derived cytokines.

277 Bronchoalveolar lavage fluid was analyzed for inflammatory cells, and bl

278 Streptococcus pneumoniae) in bronchoalveolar lavage fluid was associated with clinically significant

279 Bronchoalveolar lavage fluid was collected from 23 steroid-free nonsmoki

280 Bronchoalveolar lavage fluid was collected from patients with severe ast

281 One day after treatment, bronchoalveolar lavage fluid was collected to determine differential cel

282 Mouse bronchoalveolar lavage fluid was harvested for cell counts and TH2 cytok

283 onor; however, pretransplant bronchoalveolar lavage fluid was only available from the donor for patie

284 The lavage fluid was separated into cellular and acellular f

285 g, the median log10 HIV copies/milliliter of lavage fluid was significantly lower in men with ART-ind

286 tion of CXCL12 in plasma and bronchoalveolar lavage fluids was quantified by ELISA.

287 Elevated levels of Cif in bronchoalveolar lavage fluid were correlated with lower levels of 15-epi

288 examination, eosinophils in bronchoalveolar lavage fluid were counted.

289 nts in homogenized lungs and bronchoalveolar lavage fluid were decreased after cranberry proanthocyan

290 cytokines and chemokines in bronchoalveolar lavage fluid were measured by enzyme-linked immunosorben

291 iquots of blood, sputum, and bronchoalveolar lavage fluid were obtained from asthma subjects for medi

292 21, levels of neutrophils and macrophages in lavage fluid were reduced by 49% and increased by 287%,

293 crobial burdens in the organs and peritoneal lavage fluid were similar between mono- and coinfected a

294 differential cell counts in bronchoalveolar lavage fluid were similar between the Syk(flox/flox) and

295 s adenosine levels found in bronchioalveolar lavage fluid, were determined in mouse models of resolva

296 f T cells and neutrophils in bronchoalveolar lavage fluid, whereas expression of Foxp3 and IL-2 and n

297 s and reduce neutrophilia in bronchoalveolar lavage fluid, while IL-6 increases rapidly following LPS

298 umbers of eosinophils in the bronchoalveolar lavage fluid, while simultaneously decreasing the percen

299 Cytologic examination of bronchial alveolar lavage fluid with oil red O staining is a useful diagnos

300 cytologic examination of bronchial alveolar lavage fluid with oil red O staining.