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1 (such as muscle weakness, obesity, and joint laxity).
2 hip between specific features of OA and knee laxity.
3 ight is associated with greater varus-valgus laxity.
4 th and function is weaker in the presence of laxity.
5 an intact ACL owing to increased ligamentous laxity.
6 33 N was considered to be indicative of knee laxity.
7     All signs had a low PPV and high NPV for laxity.
8 assification of FES is proposed based on lid laxity.
9 l MR images for seven signs of anterior knee laxity.
10 ables or the technique of identifying eyelid laxity.
11  correlation between osteophyte grade and AP laxity.
12 insertion site is a response to elevated ACL laxity.
13 loss than did knees without a decrease in AP laxity.
14 d to establish a clinical diagnosis of joint laxity.
15  such as muscle weakness, obesity, and joint laxity.
16                                              Laxity and alignment were measured at baseline in indivi
17                    Quadriceps strength, knee laxity and alignment, and osteoarthritis progression.
18 pe of EDS, a condition marked by gross joint laxity and chronic musculoskeletal pain.
19  altered cell wall properties such as higher laxity and degradability, which are valuable characteris
20                        In addition to tissue laxity and descent, volume loss significantly contribute
21 seal dysplasia, short-limbed dwarfism, joint laxity and early onset osteoarthritis.
22  or IV OI, plus severe large and small joint laxity and early progressive scoliosis.
23 aracterized by variable short stature, joint laxity and early-onset degenerative joint disease.
24  for a null mutation in lumican display skin laxity and fragility resembling certain types of Ehlers-
25 ial knee osteoarthritis (OA) experience knee laxity and instability.
26  it lacks the highly derived tarsometatarsal laxity and inversion in extant African apes that provide
27 at results in dermal lesions with associated laxity and loss of elasticity, arterial insufficiency an
28                                        Joint laxity and low muscle tone contributed to musculoskeleta
29 es, mechanical characteristics such as joint laxity and malalignment, and radiographic severity are d
30                                              Laxity and meniscal extrusion had inconsistent effects.
31                                       Eyelid laxity and ocular surface disease were assessed on bedsi
32 function measures included the local factors laxity and proprioceptive inaccuracy, as well as age, BM
33 cutis laxa, a rare syndrome with marked skin laxity and pulmonary and cardiovascular compromise, is d
34  a shield, and surgery to address horizontal laxity and redundant eyelid tissues.
35 dition characterized by short stature, joint laxity, and advanced carpal ossification.
36 termates and display gait abnormality, joint laxity, and age-dependent osteoarthritis.
37 dy size, grip weakness, abnormal gait, joint laxity, and early-onset osteoarthritis.
38 ed quadriceps angle, generalized ligamentous laxity, and family history.
39 with OA were weaker, had greater medial knee laxity, and had more varus alignment.
40             Quadriceps strength, medial knee laxity, and limb alignment were measured.
41 ma, inguinal and diaphragmatic hernia, joint laxity, and pectus excavatum by age 2 years.
42 trinsic factors such as alignment, strength, laxity, and proprioception have begun to receive more at
43 rders characterized by skin fragility, joint laxity, and skeletal deformities.
44 stis and female neonates with abdominal wall laxity are classified as Pseudo Prune Belly syndrome (PP
45  of little value in predicting anterior knee laxity as demonstrated with mechanical testing.
46                           Patients with knee laxity at arthrometric testing had significantly lower I
47               Ten patients demonstrated knee laxity at arthrometric testing.
48                                           AP laxity at baseline is not predictive of progression of O
49                                           AP laxity at baseline was not a predictor of progression of
50 ibutable to the significant difference in AP laxity between knees with a K/L score of 0-1 and knees w
51 "FES stage 2 (symptomatic)" depending on its laxity capacity.
52 I] of difference 0.38, 1.56; P = 0.004), and laxity correlated modestly with age (r = 0.29, P = 0.04)
53 lihood of a poor WOMAC outcome were baseline laxity (crude odds ratio [OR] 1.48/3 degrees, 95% confid
54                            Knees in which AP laxity decreased had less medial joint space loss than d
55 r age, sex, and BMI) than did knees in which laxity decreased.
56 b support, whereas limb alignment and medial laxity did not.
57 amage, meniscal extrusion, malalignment, and laxity each predicted tibiofemoral cartilage loss after
58  of this study is to present a method of lid laxity evaluation and investigate whether there is an as
59 driceps and hamstring strength, varus-valgus laxity, functional status (Western Ontario and McMaster
60  joint environments, such as malalignment or laxity, greater strength may translate into damaging joi
61                                 High and low laxity groups were defined as above and below the sample
62               Knees without a decrease in AP laxity had a greater loss of medial joint space (0.22 mm
63 gnostic criteria for generalized ligamentous laxity (hypermobility) in children are widely used, thei
64 iving animals exhibited growth failure, skin laxity, hypopigmentation, and seizures because of perina
65 We assessed varus-valgus and anteroposterior laxity in 25 young control subjects, 24 older control su
66 MGD in 58%, lagophthalmos in 80%, and eyelid laxity in 46% were observed.
67 ine the correlation of age and sex with knee laxity in control subjects without OA, compare laxity in
68 compared with controls consistent with joint laxity in EDS patients.
69 use of osteoarthritis (OA) in animal models, laxity in human knee OA has been minimally evaluated.
70 don weakness are the likely causes for joint laxity in the double-nulls.
71            Although MR imaging signs of knee laxity in the presence of an intact ACL graft have a hig
72 , indicating that the prime abnormality is a laxity in the transition of the main sheet of the molecu
73                         Greater varus-valgus laxity in the uninvolved knees of OA patients versus old
74 xity in control subjects without OA, compare laxity in uninvolved knees of OA patients with that in o
75                 In OA patients, varus-valgus laxity increased as joint space decreased (slope -0.34;
76 nee osteoarthritis (OA) and it is known that laxity influences muscle activity, this study examined w
77                                 Varus-valgus laxity is associated with a decrease in the magnitude of
78 t with age; whether this results in clinical laxity is not clear.
79  increased likelihood of progression in high-laxity knees (P = 0.003 when high laxity was defined as
80  Spondyloepimetaphyseal dysplasia with joint laxity, leptodactylic type (lepto-SEMDJL, aka SEMDJL, Ha
81 nfluence of quadriceps strength, medial knee laxity, limb alignment, and self-reported knee instabili
82 ical and neuromuscular factors (varus-valgus laxity, malalignment, proprioceptive inaccuracy, quadric
83      In addition, patients with pelvic organ laxity may be at increased risk if their vaginal walls a
84 ults raise the possibility that varus-valgus laxity may increase the risk of knee OA and cyclically c
85                                   In theory, laxity may predispose to OA and/or result from OA.
86       Hypermobility, also termed ligamentous laxity, may present in different parts of the body at di
87         Blinking rate, lagophthalmos, eyelid laxity, MGD, Schirmer test, tear meniscus height, and dy
88 agen are thinner and weaker causing EDS-like laxity of large and small joints and paraspinal ligament
89 e concept that some portion of the increased laxity of OA may predate disease.
90                Clinical examination revealed laxity of the left abdominal wall.
91 of subtle hypermobility or symptomatic joint laxity on physical examination facilitates optimal manag
92 tween OSA and quantitative markers of eyelid laxity or secondary ocular surface disease in a sleep cl
93                                       Medial laxity (P = 0.003) and instability (P = 0.002) significa
94 ter full adjustment, no factor except medial laxity predicted qualitative outcome.
95                         Despite this genetic laxity, protein B is absolutely required for virus assem
96                             Range of motion, laxity, radiographic severity, and functional status wer
97  observed between OSA severity and an eyelid laxity score (regression coefficient, 0.85; 95% CI, -0.3
98 iabetes were associated with a higher eyelid laxity score.
99 d regarding treatment direction and how much laxity should be considered acceptable.
100 , stratification of analyses by varus-valgus laxity should be considered.
101 gs with progressive neurodegeneration, joint laxity, skin hyperelasticity and bilateral subcapsular c
102  and an age-related increase in varus-valgus laxity support the concept that some portion of the incr
103 the controls, women had greater varus-valgus laxity than did men (3.6 degrees versus 2.7 degrees; 95%
104 ough knees with a K/L score of 4 had less AP laxity than those with a K/L score of 0-1, most of this
105 traction of the medial muscle in response to laxity that appears on the medial side of the joint only
106 nee joint may successfully compensate for AP laxity; the absence of such compensation may have a dele
107 A device was designed to assess varus-valgus laxity under a constant varus or valgus load while maint
108 moral joints, unknown degrees of soft-tissue laxity, variations in the alignment of the knee, and oth
109            In multivariate analyses, greater laxity was consistently associated with a weaker relatio
110 on in high-laxity knees (P = 0.003 when high laxity was defined as > or =6.75 degrees).
111                                 Varus-valgus laxity was greater in the uninvolved knees of OA patient
112                At baseline and 18 months, AP laxity was measured (in millimeters of tibial translatio
113                                Frontal plane laxity was measured from stress radiographs.
114         In knees with a K/L score of 0-2, AP laxity was slightly lower at 18 months than at baseline.
115              At baseline, measurements of AP laxity were lower in knees with a Kellgren/Lawrence (K/L
116 ain, functional status, range of motion, and laxity were measured.
117 ysomnography, quantitative markers of eyelid laxity were not associated with the presence or severity
118 and/or calcifications, and demonstrates more laxity with valgus stress.

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