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1 (such as muscle weakness, obesity, and joint laxity).
2 hip between specific features of OA and knee laxity.
3 ight is associated with greater varus-valgus laxity.
4 th and function is weaker in the presence of laxity.
5 an intact ACL owing to increased ligamentous laxity.
6 33 N was considered to be indicative of knee laxity.
7 All signs had a low PPV and high NPV for laxity.
8 assification of FES is proposed based on lid laxity.
9 l MR images for seven signs of anterior knee laxity.
10 ables or the technique of identifying eyelid laxity.
11 correlation between osteophyte grade and AP laxity.
12 insertion site is a response to elevated ACL laxity.
13 loss than did knees without a decrease in AP laxity.
14 d to establish a clinical diagnosis of joint laxity.
15 such as muscle weakness, obesity, and joint laxity.
19 altered cell wall properties such as higher laxity and degradability, which are valuable characteris
24 for a null mutation in lumican display skin laxity and fragility resembling certain types of Ehlers-
26 it lacks the highly derived tarsometatarsal laxity and inversion in extant African apes that provide
27 at results in dermal lesions with associated laxity and loss of elasticity, arterial insufficiency an
29 es, mechanical characteristics such as joint laxity and malalignment, and radiographic severity are d
32 function measures included the local factors laxity and proprioceptive inaccuracy, as well as age, BM
33 cutis laxa, a rare syndrome with marked skin laxity and pulmonary and cardiovascular compromise, is d
42 trinsic factors such as alignment, strength, laxity, and proprioception have begun to receive more at
44 stis and female neonates with abdominal wall laxity are classified as Pseudo Prune Belly syndrome (PP
50 ibutable to the significant difference in AP laxity between knees with a K/L score of 0-1 and knees w
52 I] of difference 0.38, 1.56; P = 0.004), and laxity correlated modestly with age (r = 0.29, P = 0.04)
53 lihood of a poor WOMAC outcome were baseline laxity (crude odds ratio [OR] 1.48/3 degrees, 95% confid
57 amage, meniscal extrusion, malalignment, and laxity each predicted tibiofemoral cartilage loss after
58 of this study is to present a method of lid laxity evaluation and investigate whether there is an as
59 driceps and hamstring strength, varus-valgus laxity, functional status (Western Ontario and McMaster
60 joint environments, such as malalignment or laxity, greater strength may translate into damaging joi
63 gnostic criteria for generalized ligamentous laxity (hypermobility) in children are widely used, thei
64 iving animals exhibited growth failure, skin laxity, hypopigmentation, and seizures because of perina
65 We assessed varus-valgus and anteroposterior laxity in 25 young control subjects, 24 older control su
67 ine the correlation of age and sex with knee laxity in control subjects without OA, compare laxity in
69 use of osteoarthritis (OA) in animal models, laxity in human knee OA has been minimally evaluated.
72 , indicating that the prime abnormality is a laxity in the transition of the main sheet of the molecu
74 xity in control subjects without OA, compare laxity in uninvolved knees of OA patients with that in o
76 nee osteoarthritis (OA) and it is known that laxity influences muscle activity, this study examined w
79 increased likelihood of progression in high-laxity knees (P = 0.003 when high laxity was defined as
80 Spondyloepimetaphyseal dysplasia with joint laxity, leptodactylic type (lepto-SEMDJL, aka SEMDJL, Ha
81 nfluence of quadriceps strength, medial knee laxity, limb alignment, and self-reported knee instabili
82 ical and neuromuscular factors (varus-valgus laxity, malalignment, proprioceptive inaccuracy, quadric
84 ults raise the possibility that varus-valgus laxity may increase the risk of knee OA and cyclically c
88 agen are thinner and weaker causing EDS-like laxity of large and small joints and paraspinal ligament
91 of subtle hypermobility or symptomatic joint laxity on physical examination facilitates optimal manag
92 tween OSA and quantitative markers of eyelid laxity or secondary ocular surface disease in a sleep cl
97 observed between OSA severity and an eyelid laxity score (regression coefficient, 0.85; 95% CI, -0.3
101 gs with progressive neurodegeneration, joint laxity, skin hyperelasticity and bilateral subcapsular c
102 and an age-related increase in varus-valgus laxity support the concept that some portion of the incr
103 the controls, women had greater varus-valgus laxity than did men (3.6 degrees versus 2.7 degrees; 95%
104 ough knees with a K/L score of 4 had less AP laxity than those with a K/L score of 0-1, most of this
105 traction of the medial muscle in response to laxity that appears on the medial side of the joint only
106 nee joint may successfully compensate for AP laxity; the absence of such compensation may have a dele
107 A device was designed to assess varus-valgus laxity under a constant varus or valgus load while maint
108 moral joints, unknown degrees of soft-tissue laxity, variations in the alignment of the knee, and oth
117 ysomnography, quantitative markers of eyelid laxity were not associated with the presence or severity
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