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1 r appropriate flushing procedures to prevent lead poisoning.
2 e in need of emergency chelation therapy for lead poisoning.
3 s either familial or can be acquired through lead poisoning.
4 een addressed to the prevention of childhood lead poisoning.
5 ilinogen synthase (PBGS) is a main target in lead poisoning.
6 ith differential susceptibility of humans to lead poisoning.
7 ron is routinely prescribed in children with lead poisoning.
8 , and breastfed infants are also at risk for lead poisoning.
9 ly to have >/= 1 child < 5 years of age with lead poisoning (68% vs. 50%, p = 0.17) or death followin
11 subclinical disease; the fact that childhood lead poisoning affected mostly families that were politi
12 sociated with reduced PBGS activity, such as lead poisoning and ALAD porphyria, the latter of which i
14 the brink of extinction, in part, because of lead poisoning, and lead poisoning remains a significant
15 phrenia, post-traumatic stress disorder, and lead poisoning--conditions characterized by impoverished
16 llages with suspected or confirmed childhood lead poisoning continued to be identified in Zamfara Sta
18 vironmental remediation to address childhood lead poisoning epidemic due to artisanal gold mining in
21 We investigated the extent of childhood lead poisoning [>/= 1 child with a blood lead level (BLL
22 s given to children with iron deficiency and lead poisoning have been demonstrated to improve develop
23 s Sans Frontieres (MSF) discovered extensive lead poisoning impacting several thousand children in ru
25 children (R(2) = 0.68) and the incidence of lead poisoning in children under age 1.3 years (R(2) = 0
27 ities are using local policy tools to reduce lead poisoning in high-risk areas, but little is known a
28 made in reducing the incidence of childhood lead poisoning in the United States in the past three de
31 or death in the previous year from suspected lead poisoning included the age of the child, the mother
32 recommended frequently to reduce the risk of lead poisoning, its role in preventing lead absorption h
33 lopmental toxicity associated with childhood lead poisoning may be attributable to interactions of Pb
35 2010, Medecins Sans Frontieres discovered a lead poisoning outbreak linked to artisanal gold process
37 was to determine the cause of the childhood lead poisoning outbreak, investigate risk factors for ch
38 Rochester experience may inform future local lead poisoning prevention policies in other communities.
42 ts have reduced lead exposure significantly, lead poisoning remains the most common environmental hea
43 matic activity due to genetic aberrations or lead poisoning results in a mild to moderate nonspherocy
44 in blood lead in iron-replete children with lead poisoning secondary to iron supplements have not be
46 hese factors, the use of iron supplements in lead poisoning should be individualized, and the supplem
48 The present study used an animal model of lead poisoning to examine the extent to which different
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