ライフサイエンスコーパス: leanness

コーパス検索結果 (１語後でソート)

通し番号をクリックするとPubMedの該当ページを表示します

1 of the MCH gene in mice cause hypophagia and leanness.

2 loss of MCH neurons can lead to an acquired leanness.

3 ctile dysfunction were physical activity and leanness.

4 ng a single orexigenic peptide can result in leanness.

5 man brown fat activity levels correlate with leanness.

6 riate physiological conditions is related to leanness, aerobic fitness, and insulin sensitivity.

7 functional analyses of this novel candidate leanness and antidiabetic gene.

8 Body leanness and hypertension are additional etiological fac

9 se) responsible for the Fob3b2 QTL effect on leanness and improved metabolic parameters.

10 nstream gene expression changes that promote leanness and increased energy expenditure.

11 mutation, designated supermodel, that caused leanness and kyphosis associated with myopathy and adipo

12 We show that Tbc1d1, a gene known to mediate leanness and linked to obesity, is down-regulated in the

13 +/- 0.4 y were studied according to parental leanness and overweight or obesity.

14 nditure, and increased satiety, resulting in leanness and reduced body fat.

15 der bidirectional selection for the distinct leanness and reproduction traits between CHD and EUD.

16 in AgRP neurons partially recapitulates the leanness and resistance to diet-induced obesity of RIIbe

17 of NEAT dissipates excess energy to preserve leanness and that failure to activate NEAT may result in

18 We examined the health benefits of leanness and the hazards of obesity while simultaneously

19 The health benefits of leanness are limited to fit men, and being fit may reduc

20 tary patterns or nutrient intakes that favor leanness [BMI (in kg/m(2)) </=25] in free-living populat

21 Leanness can result from deficiency of stored energy or

22 -deficient mice have reduced body weight and leanness due to hypophagia (reduced feeding) and an inap

23 ylcarboxypeptidase (PRCP) is associated with leanness, hypertension, and thrombosis.

24 rons of mice results in reduced food intake, leanness, improved glucose homeostasis, and increased se

25 rons of mice results in reduced food intake, leanness, improved glucose homeostasis, and increased se

26 Mex3c mutation caused leanness in both heterozygous and homozygous transgenic

27 in-1 (UCP1) and BAT activity correlates with leanness in human adults.

28 ncluding obesity, in mice and correlate with leanness in humans.

29 enomic and non-genomic actions of E2 promote leanness in OVX mice independently of reduced energy int

30 Leanness in PTP-1B-deficient mice is accompanied by incr

31 ants, previously shown to be associated with leanness in the HERITAGE Family Study, are also associat

32 l CoA carboxylase-2 (Acc2) reportedly causes leanness in the setting of hyperphagia.

33 Clinical features include leanness, intellectual disability, autistic features and

34 c when maintained on regular chow, and their leanness is a consequence of hyperactivity and altered m

35 otic and dizygotic twin pairs concordant for leanness or obesity, and their mothers, to address how h

36 Leanness secondary to caloric restriction is known to be

37 Because the MCH(-/-) model represents leanness secondary to increased energy expenditure rathe

38 y metabolism evolved to favor adiposity over leanness, the availability of palatable, easily attainab

39 me characterized by low appetite and extreme leanness; this is phenocopied by mice carrying similar m

40 Furthermore, cecal transplantation conferred leanness to germ-free recipients.

41 Leanness was characterized by hypophagia and increased e

42 Instead, leanness was secondary to a marked increase in energy ex

43 adipose-Stra6(-/-) mice fed chow resulted in leanness, which may contribute to their increased insuli

44 erested in determining whether this model of leanness would be associated with beneficial metabolic e

WebLSDに未収録の専門用語（用法）は "新規対訳" から投稿できます。