1 oteomes will be an extremely powerful way to
learn more about a particular protein's structure, its f
2 fills or a desire to receive help refilling,
learn more about asthma control, or speak with an asthma
3 To
learn more about autotrophic growth of methanococci, we
4 onosubstituted dianionic phosphates, both to
learn more about basic properties intrinsic to this impo
5 Two bindin genes were sequenced to
learn more about bindin introns.
6 To
learn more about biological pathways that are potentiall
7 neutrophil entry during lung injury and are
learning more about cellular interactions during inflamm
8 Clinicians are
learning more about chronic pancreatitis but are enterin
9 ination treatment strategies, it is vital to
learn more about coexpression of both inhibitory and sti
10 Finally, we stress the importance of
learning more about coverage measurement to strengthen t
11 ed gain-of-function Cpx* mutants in order to
learn more about Cpx signal transduction.
12 Thus,
learning more about cytokine-mediated regulation of esta
13 The challenge for the future will be to
learn more about dose, duration, and mechanism of action
14 As we
learn more about fMRI related to pain, functional connec
15 To
learn more about FTi and to identify novel regulators in
16 To
learn more about functional contributions made by Blimp1
17 obile elements are becoming useful tools for
learning more about genome evolution and gene function.
18 To
learn more about holocentric chromosome structure and fu
19 To
learn more about how BFP protein expression is induced d
20 To
learn more about how cells regulate production of nitrit
21 seed that contribute to seed dormancy and to
learn more about how dormancy and germination are regula
22 To
learn more about how Dsh can discriminate between these
23 To
learn more about how dyneins are targeted to specific si
24 To
learn more about how FXIIIa selects its targets, a matri
25 To
learn more about how the cytosolic domains regulate chan
26 To
learn more about how these two processes determine wheth
27 More than 85% (175 [87.9%]) wanted to
learn more about how to prevent skin cancer.
28 As we
learn more about how to refine our present resistance ma
29 clinical trials offer a unique resource for
learning more about individual susceptibility and develo
30 To
learn more about islet vulnerability in the immediate po
31 To
learn more about its effect on cellular proliferation an
32 udy, we analyzed the lasB promoter region to
learn more about lasB activation by LasR and PAI.
33 In answering these questions we may hope to
learn more about mechanisms of genome plasticity and ada
34 To
learn more about metals in ALS, we determined the metall
35 To
learn more about MLK3 regulation and its signaling pathw
36 To
learn more about Mot3 function, we have performed a synt
37 To
learn more about Mus101, we have initiated an analysis o
38 To
learn more about mutations that can suppress the spt21De
39 To
learn more about NBD structure and function, we introduc
40 ative genomics of helminth genomes to aid in
learning more about nematode genomes, as well as drug, p
41 To
learn more about p53's transactivator function in vivo,
42 ed with inflammatory bowel diseases (IBD) to
learn more about pathogenesis.
43 To
learn more about pathogenic PV autoantibodies, we isolat
44 at high risk and offer key opportunities to
learn more about pediatric nephrolithiasis, thereby fuel
45 To
learn more about regulation of hilA, we isolated Tn5 mut
46 To
learn more about silencing, we investigated mating type
47 and progression, but also as a biomarker to
learn more about specific treatment responses and pharma
48 To
learn more about Spt3-TBP interactions in vivo, we have
49 To
learn more about such control, we initiated studies of p
50 To
learn more about switching, we measured the fraction of
51 To
learn more about TAO/KIN-18 function, we studied how exp
52 his issue, took an epidemiologic approach to
learn more about the 8q24 region.
53 To
learn more about the action of these important complexes
54 To
learn more about the BAM complex, we solved structures o
55 biologists interested in these topics should
learn more about the basic structure of drug development
56 ther optimization should be possible when we
learn more about the binding requirements at the active
57 ciliate protozoan Tetrahymena thermophila to
learn more about the biochemical characteristics of RNas
58 To
learn more about the biochemical composition and activit
59 To
learn more about the biochemistry of the human enzyme an
60 They also provide the opportunity to
learn more about the biology of DM which had previously
61 creatic surgery is a fascinating field as we
learn more about the biology of the conditions that affl
62 Additional studies are required to
learn more about the biology of these diseases, which ma
63 essing and internal cognitive state, we must
learn more about the brain areas supporting attentional
64 To probe the cross-bridge cycle and to
learn more about the cardioplegic agent BDM (2,3-butaned
65 n view of this continuing controversy and to
learn more about the cell interactions during rib morpho
66 To
learn more about the characteristics, origin, and possib
67 To
learn more about the compensatory mechanism by which vir
68 This evolution continues, as we
learn more about the complex interplay between patient a
69 As we
learn more about the complexities of immune responses, n
70 To
learn more about the cross-talk that must be occurring b
71 To
learn more about the determinants of this increasingly i
72 therapy holds much promise in SLE and as we
learn more about the disease and apply the lessons learn
73 n mediators and receptors also expands as we
learn more about the disease processes that are either i
74 l continue to be reviewed and modified as we
learn more about the disease to better perform this risk
75 To
learn more about the distribution and possible regulator
76 To
learn more about the dynamics of soluble Abeta economy i
77 To
learn more about the effects of repetitive magnetic stim
78 egulation by OxyR and illustrate the need to
learn more about the ensembles of binding sites and tran
79 idual metabolic networks is increasing as we
learn more about the enzymes that are active in particul
80 To
learn more about the epidermal antigens targeted by Ro a
81 To
learn more about the epimerization process, the structur
82 To
learn more about the essential role of Cdc7 kinase in th
83 nd lifetime nonsmokers can be enhanced as we
learn more about the etiologic mechanism(s) of lung canc
84 To
learn more about the evolutionary effects of the duplica
85 To
learn more about the function and regulation of small he
86 individual mutations separately in vitro to
learn more about the function of the kindlin-3 protein.
87 To
learn more about the function of the NTSFs in species wh
88 To
learn more about the function of the Spt4/Spt5 complex a
89 To
learn more about the function of these genes in auxin re
90 erated mice lacking MDC9 (mdc9(-/-) mice) to
learn more about the function of this protein during dev
91 To
learn more about the function of Tim18p, we screened for
92 As we
learn more about the functional expression of nicotinic
93 To
learn more about the functional requirements of this com
94 In an effort to
learn more about the functional role of this protein, we
95 To
learn more about the functions and regulation of CenH3,
96 orker and haploid male honeybees (drones) to
learn more about the genetic architecture of the overall
97 To
learn more about the genomic organization of regions sur
98 To
learn more about the gliding motor, we sheared cells to
99 To
learn more about the importance of ascorbate in the accl
100 To
learn more about the interaction between these proteins,
101 To
learn more about the mechanism and specificity of isoAsp
102 To
learn more about the mechanism of action of PEG, we anal
103 To
learn more about the mechanism of hepadnavirus replicati
104 To
learn more about the mechanism of the DnaB transfer reac
105 the use of this approach we will be able to
learn more about the mechanisms by which methylation pat
106 To
learn more about the mechanisms by which this pathogen c
107 To
learn more about the mechanisms controlling neurite outg
108 6-31+G) study was undertaken in an effort to
learn more about the mechanisms controlling the regiosel
109 To
learn more about the mechanisms involved in establishing
110 nt medical practice mandates that physicians
learn more about the mechanisms of trastuzumab-induced c
111 To
learn more about the mechanisms that regulate entry into
112 To
learn more about the miRNAs of mammals, we sequenced 60
113 e technology, to address these questions and
learn more about the molecular functions of each of the
114 To
learn more about the molecular mechanism of H3K56 acetyl
115 The current studies were carried out to
learn more about the molecular motors responsible for LP
116 We will
learn more about the natural history of the disease and
117 the questions and opportunities it opens to
learn more about the nature of phage-bacterial coevoluti
118 kers rely on available technology and, as we
learn more about the neurobiological correlates of neuro
119 To
learn more about the pathogenesis of Alport disease, we
120 t-partum patients is likely to improve as we
learn more about the pathogenesis of these disorders.
121 Our objective was to
learn more about the pathophysiology of severe meningiti
122 ent research using tissue and cell models to
learn more about the plasma delivery of RONS into biolog
123 To
learn more about the possible function of Hsp93 during p
124 To address this possibility in vivo and to
learn more about the potential physiological roles of CK
125 and provide direction for those desiring to
learn more about the precise topics.
126 To
learn more about the problem, we studied a group of chil
127 To identify and
learn more about the protein encoded by HCMV UL46, we ha
128 To
learn more about the regulation of contraction of collag
129 To
learn more about the regulation of H blood group express
130 To
learn more about the relation between diabetes and cance
131 To
learn more about the requirement for subplate neurons in
132 The goal of this study was to
learn more about the risk factors and short- and long-te
133 As we
learn more about the RNA codes and maps that guide the a
134 To
learn more about the role of chromatin in P cytotype rep
135 To
learn more about the role of denitrification in these an
136 To
learn more about the role of GGT in metabolism in vivo,
137 To
learn more about the role of GR3 in salt-stress toleranc
138 f this heterogeneity will become clear as we
learn more about the role of NK cells in cattle disease
139 To
learn more about the role of protein methylation during
140 To
learn more about the role of STAT-5B in vessel wall remo
141 To
learn more about the role of STATs in the mitogenic and
142 To
learn more about the role of these compounds, we have ex
143 Therefore, to
learn more about the role of this pathway in vivo, we ge
144 To
learn more about the roles of PDK1, we generated mice th
145 To
learn more about the roles of the conserved proline resi
146 To
learn more about the roles of the tentacles in ribosome
147 To
learn more about the shared function, we sought new muta
148 To
learn more about the signaling pathways involved, we did
149 To
learn more about the sites of blocker binding and about
150 ls, further field research is recommended to
learn more about the sources of transmission and the eff
151 To
learn more about the specificity of this intercompartmen
152 In order to
learn more about the STAT-3-cPLA(2) axis in motogenic si
153 To
learn more about the structural and functional significa
154 cal processes, there is an important need to
learn more about the structural features relevant to the
155 Drosophila gene in the erg subfamily and to
learn more about the structure and biological function o
156 To
learn more about the structure and function of eIF3 we h
157 To
learn more about the structure of the DNA terminus at Te
158 To
learn more about the structure, expression, and function
159 To
learn more about the targets of Cn (Cn) and calcium/calm
160 To
learn more about the telomeric function of proteins from
161 The goal of this study was to
learn more about the transfer mechanism by defining the
162 s in humans, providing a strong incentive to
learn more about the underlying causes.
163 Here we set out to
learn more about the underlying mechanism by which Cat-p
164 n and development is expanding rapidly as we
learn more about the unique properties associated with t
165 The reasons are simple: 1) we need to
learn more about the world around us; and 2) a robust an
166 In recent years, we have
learned more about the complex interactions that occur b
167 armers who had attended a brief workshop and
learned more about the forecasts were significantly more
168 four decades, it is fair to say that we have
learned more about the lysosomal system of cells through
169 At the same time, we
learned more about the regulatory networks of H. influen
170 indicate some of the resources available for
learning more about the advanced capabilities of these p
171 Learning more about the cells that enable parasitic worm
172 In this regard,
learning more about the immune components of these disea
173 osomic humans (45,X) and mice (39,X), we are
learning more about the influences of X-linked imprinted
174 While we are still
learning more about the myriad clinical presentations in
175 brains of 12 human subjects, with the aim of
learning more about the position and variability of the
176 imed at nonspecialists who are interested in
learning more about the potential and impact of NMR spec
177 Learning more about the regulation of TEMs by the tumor
178 Interested in
learning more about the starvation response, we cloned a
179 logical disorders is likely to improve as we
learnt more about the spectrum of this entity in the las
180 sought to study their C. elegans homolog to
learn more about their functions.
181 remain a vexing treatment dilemma, but as we
learn more about their natural history, more thoughtful
182 19 genes implicated in early development, to
learn more about their potential role in specifying and
183 isparities, health care professionals should
learn more about them and the roles they can play in eli
184 As we
learn more about this disorder, what is emerging is that
185 However, as we
learn more about this evolutionarily ancient process, we
186 As we
learn more about this family, more therapeutics will pro
187 To
learn more about this isoform, we isolated Chlamydomonas
188 To
learn more about this remarkable phenomenon, we examined
189 Oncologists therefore need to
learn more about this subject.
190 To
learn more about which factors and/or events end the imp
191 To
learn more about why loss of c-Rel led to earlier onset