ライフサイエンスコーパス: learned helplessness

1 osure to inescapable electric shock in rats (learned helplessness).

2 sed feeding (NSF), social defeat stress, and learned helplessness.

3 e Th17 cell function, reduced Th17-dependent learned helplessness.

4 vioral paradigms for anhedonia, despair, and learned helplessness.

5 d induction of escape deficits in a model of learned helplessness.

6 sion, chronic unpredictable stress (CUS) and learned helplessness.

7 Whereas synaptic potentiation was linked to learned helplessness, a depression-like behavior, synapt

8 selectively bred line of rats susceptible to learned helplessness, a model of depression, presents an

9 nce of the transcription factor DeltaFosB on learned helplessness, an animal model of affective disor

10 ibility to social aversion, "anhedonia," and learned helplessness and causes impaired glucocorticoid-

11 Mouse brain Th17 cells were elevated by learned helplessness and chronic restraint stress, two c

12 neuroregulation as a proximate mechanism in learned helplessness and conservation-withdrawal.

13 nregulated in the hippocampus after both the learned helplessness and forced swim test (FST) paradigm

14 digm where no vehicle-treated mice developed learned helplessness, and impaired novelty suppressed fe

15 Animals underwent learned helplessness, and subsequently immobility time w

16 To evaluate learned helplessness behavior, we used an active avoidan

17 threshold unpredictable stress and increased learned helplessness behavior.

18 n brain areas that control the expression of learned helplessness behaviors.

19 in brain slices and can significantly reduce learned helplessness behaviour in rats.

20 Three groups were studied: (1) rats bred for learned helplessness (cLH); (2) rats resistant to learne

21 ed helplessness (cLH); (2) rats resistant to learned helplessness (cNLH); and (3) control Sprague Daw

22 rlier, in agreement with previously reported learned-helplessness effects.

23 heels showed antidepressant-like behavior in learned helplessness, forced-swim (FST) and tail suspens

24 Th17 cell production exhibited resistance to learned helplessness, identifying modulation of RORgamma

25 Th17 cell administration promoted learned helplessness in 89% of mice in a paradigm where

26 unsolvable anagrams have been used to induce learned helplessness in humans, this finding may provide

27 Inescapable stress can induce learned helplessness in many species of animals.

28 r CIS, specifically: struggling, aggression, learned helplessness, inhibitory avoidance, and escape b

29 Learned helplessness is a phenomenon which has some beha

30 fect in behavioral models of depression, the learned helplessness (LH) and forced swim test (FST) par

31 al consequences of uncontrollable stress, or learned helplessness (LH) behaviors, are thought to invo

32 he nucleus (DRN) are implicated in mediating learned helplessness (LH) behaviors, such as poor escape

33 e tested the response of brain FoxO3a in the learned helplessness (LH) paradigm and tested signaling

34 lase promoter, showed protection against the learned helplessness (LH) paradigm, an animal model to t

35 ST), the tail suspension test (TST), and the learned helplessness (LH) paradigm-as well as in the fem

36 g a chronic social defeat (SD) stress model, learned helplessness (LH), and a chronic corticosterone

37 sfer), whereas yoked rats failed to learn (a learned helplessness-like effect).

38 In learned helplessness, mice were exposed to a shuttle box

39 ens CREB-dynorphin influence behavior in the learned helplessness model and suggest that this signali

40 n onto LHb neurons contributes to the rodent learned helplessness model of depression.

41 Here we show that in two learned helplessness models of depression, excitatory sy

42 tidepressant-like behavioural effects in the learned helplessness paradigm and regulates molecular ev

43 ificance of this transcription factor in the learned helplessness paradigm, a behavioral model of dep

44 proximate mediator of escape deficits in the learned helplessness paradigm, suggesting that neuronal

45 In the learned helplessness paradigm, the SCN-Bmal1-KD mice wer

46 rformance following inescapable shock in the learned helplessness paradigm.

47 c-Fos-like immunoreactivity (FLI) using the learned helplessness paradigm.

48 Here we used the learned helplessness procedure in mice to examine the ro

49 ehavioral responses to stress induced by the learned helplessness procedure, in which animals are sub

50 hological (illness behavior, social support, learned helplessness, smoking, drinking), clinical, sero

51 e, progressive ratio responding to food, and learned helplessness task were normal, such avolition-li

52 the Porsolt, novelty induced hypophagia, and learned helplessness tests in rats without exhibiting su

53 ing study of the effects of fluoxetine after learned helplessness training.

54 In foot-shock groups, learned helplessness was more robust in heterozygotes th