ライフサイエンスコーパス: led us to hypothesize that

1 The prevalence of polyploids in adverse climates led us to hypothesize that abiotic stress conditions can indu

2 These and other considerations led us to hypothesize that AGPHD1 corresponded to 5-hydroxy-L

3 These findings led us to hypothesize that altered ERO1 activity may affect c

4 These findings led us to hypothesize that AML cells will be hypersensitive t

5 The requirement for UL135 but not the entire locus led us to hypothesize that another gene in this locus suppres

6 These data led us to hypothesize that AR may directly affect PTEN transc

7 This led us to hypothesize that Bim is required for the latter.

8 This finding led us to hypothesize that call rate and duration are regulat

9 This led us to hypothesize that correlated variation in functional

10 dent kinesin family show Brownian motion along microtubules led us to hypothesize that diffusion along microtubules could

11 file of gene expression changes in end-stage cardiomyopathy led us to hypothesize that distinct global patterns of the ep

12 er and activator of transcription (STAT) signaling pathways led us to hypothesize that ETP-ALL is dependent on JAK/STAT s

13 This finding led us to hypothesize that FKBP52 has functions in addition t

14 egulation of actin reorganization in various cell types has led us to hypothesize that fluid shear stress-induced Vegf up

15 This finding led us to hypothesize that Foxo1 regulates Na(V)1.5 expressio

16 These observations led us to hypothesize that helminths can regulate GVHD and ma

17 group and previously published molecular epidemiologic data led us to hypothesize that hvKP strains produced more siderop

18 as specific to the conditioning amplitude, and this concept led us to hypothesize that if the amplitude of a vibrotactile

19 These observations led us to hypothesize that inflammation that accompanies earl

20 to recombinant fragments of the Fn integrin binding domain led us to hypothesize that integrin alpha3beta1 binding may a

21 humans and other species that benefit from free mate choice led us to hypothesize that it also confers reproductive benef

22 y of LRBA to proteins controlling intracellular trafficking led us to hypothesize that it regulates CTLA4, a potent inhib

23 Its dysregulated expression in other tumors led us to hypothesize that latexin may have tumor suppressor

24 This led us to hypothesize that MIF may have the capacity to inter

25 ved mouse models, including calorie restriction (CR), which led us to hypothesize that MIF might be important in the cont

26 The role of miRNAs and TICs in cancer progression led us to hypothesize that miRNAs may regulate genes involved

27 These observations led us to hypothesize that more iron is absorbed in beta-thal

28 These correlations led us to hypothesize that polymorphisms in open reading fram

29 ed to dissociate from mucus and presumably utilize peptides led us to hypothesize that protease activity is integral to t

30 This result led us to hypothesize that REV-ERBalpha may mediate the circa

31 a)-alpha is highly expressed in lungs of patients with COPD led us to hypothesize that Rora may contribute to the pathoge

32 trigger zinc release from cytosolic zinc binding proteins, led us to hypothesize that ROS can transactivate TrkB in CNS

33 sis) and that TCA cycle activity regulates PIA biosynthesis led us to hypothesize that S. epidermidis is "sensing" dispar

34 This led us to hypothesize that skeletal deterioration following H

35 restore infectivity to maturation-defective mutant viruses led us to hypothesize that SP may play an important role in p

36 ate cholylglycine and exhibits characteristics in vivo that led us to hypothesize that the (11)C-labeled form, that is, [

37 This led us to hypothesize that the cellular functions of these ge

38 Our results led us to hypothesize that the enrichment of C16:0 SM could d

39 These observations led us to hypothesize that the exposure of the maternal gamet

40 ions that EvC and BPI do not occur in the same individuals, led us to hypothesize that the genetic defect causing EvC in

41 des nor deleting the GLD of AGAP1 affected catalysis, which led us to hypothesize that the GLD is a protein binding site

42 These observations led us to hypothesize that the imidazole ring of His(229) may

43 These observations led us to hypothesize that the reported relationship of perio

44 These observations led us to hypothesize that the vhs-mediated inhibition was de

45 haviors and their specific expression in larval nociceptors led us to hypothesize that these DEG/ENaC subunits form an io

46 Recent work led us to hypothesize that this tropism may reflect pathogeni

47 This led us to hypothesize that Tregs may affect the trafficking o

48 This finding led us to hypothesize that vesicle trafficking contributes to

49 ellular distribution differences between the two CUL4 types led us to hypothesize that Vpr and Vpx employ these in a func

50 Our analyses of the exon4 sequence led us to hypothesize that when spliced out, exon4 may genera