ライフサイエンスコーパス: left

1 r in the bilateral superior temporal cortex (left: +10.0%; P = .03 and right: +10.8%; P = .01), super

2 03 and right: +9.5%; P = .02), and thalamus (left: +11.6%; P = .002 and right: +11.1%; P = .001).

3 ed obviously on 4th day and only 15% dye was left 12days later.

4 right: 10.2%, 95% CI 2.0% to 18.4%, p=0.017; left: 14.1%, 95% CI 3.4% to 24.9%, p=0.013).

5 %; P = .01), superior temporal white matter (left: +14.6%; P = .003 and right: +9.5%; P = .02), and t

6 proach to selecting the PERSEVERE biomarkers left 68 genes unconsidered.

7 en oases' under a reducing atmosphere, which left a characteristic oxidative weathering signal.

8 ivity and subsequent error-prone repair have left a mutational footprint on the insertion environment

9 Clinical examination revealed laxity of the left abdominal wall.

10 training during the study period: 15 (17.6%) left after postgraduate year 1, 34 (40.0%) after postgra

11 d to positively modulate connectivity of the left amygdala to the posterior thalamus in male but not

12 lomere length was positively associated with left and right hippocampal volume and with delayed recal

13 The most frequent patterns were left and right posterior-temporal delta brushes which we

14 , extent of late gadolinium enhancement, and left and right ventricular ejection fractions.

15 cipital and posterior cingulate cortex (each left and right).

16 emonstrate that in those deaf from birth the left and the right STC have altered and dissociable func

17 We designed a metasurface that converts left- and right-circular polarizations into states with

18 emonstrated that temporary disruption of the left angular gyrus leads to impairments in simulation an

19 e hippocampus, medial prefrontal cortex, and left angular gyrus, among other regions.

20 e hippocampus, medial prefrontal cortex, and left angular gyrus.

21 formed in 1.4 lesions per patient, mostly in left anterior descending (58%), with a mean percent sten

22 iate-severity stenoses were generated in the left anterior descending artery (LAD), and 20 contrast m

23 intravascular ultrasonography (IVUS) of the left anterior descending coronary artery, within 8 weeks

24 imension and applied a virtual lesion to the left anterior temporal lobe.

25 ral lobe and angular gyrus; semantic memory: left anterior temporal regions; language: left posterior

26 tion of the subsample permutation tests: the left anterior ventrolateral prefrontal cortex/insula, th

27 ponse patterns were similar across right and left arm movements to identical targets (extrinsic coord

28 and a lump in the subcutaneous tissue of the left arm.

29 entially regulates PRRX1 expression in human left atria.

30 Although left atrial (LA) dysfunction is common in heart failure

31 Left atrial appendage closure (LAAC) was approved by the

32 Over the past decade, percutaneous left atrial appendage closure has emerged as a valid alt

33 o [HR] per decade, 1.55; 95% CI, 1.11-2.15), left atrial dimension (HR per centimeter diameter, 1.43;

34 ntricular (LV) dysfunction, ischemic MR, and left atrial infarction (LAI); and 2) to analyze how LA r

35 antly associated with gene expression in 329 left atrial samples.

36 After ablation of 17 +/- 10% of the left atrial surface and 18 months of follow-up, the atri

37 with diastolic dysfunction (E/e') and 5 with left atrial volume index.

38 1 +/- 36 ml to 122 +/- 30 ml; p < 0.001) and left atrial volumes (106 +/- 36 ml to 69 +/- 24 ml; p <

39 Remarkably, many of the Pnmt(+) cells in the left atrium and ventricle appeared to be working cardiom

40 was placed on the epicardial surface of the left atrium in dogs.

41 ient; 7 in the right atrium versus 12 in the left atrium; 15 extrapulmonary).

42 nsitive response in the right and an area in left auditory cortex that is sensitive to individual dif

43 e response to sound onset, which is found in left auditory cortex.

44 absolute difference in ADCs (right branch - left branch) was significantly different between the two

45 In contrast to patients with left bundle branch block (LBBB), heart failure patients

46 ges and in a test area where 13 bridges were left by the pipeline construction company.

47 er-defibrillators alone in 25 (4%) patients, left cardiac sympathetic denervation alone in 18 (3%) pa

48 Perfusion measurements in the LAD, left circumflex artery (LCx), right coronary artery, and

49 ute proximal right coronary artery (RCA) and left coronary artery balloon occlusion at baseline befor

50 Adult B6sv129-mice were subjected to in vivo left coronary artery ligation for 30 minutes followed by

51 l tip, transverse forces are attractive with left-CPL and repulsive with right-CPL.

52 ealed significant subgrouping effects on the left (d1.48) and right NAc (d0.95) with larger bilateral

53 demonstrate functional specialization of the left dHb-IPN pathway in attenuating the response to fear

54 Increased left-dominant activation in the anterior frontal structu

55 , particularly in advanced disease, and that left-dominant forms occur.

56 tamine (Glx), were measured by 1H MRS in the left dorsolateral prefrontal cortex (l-DLPFC) and bilate

57 ial alternating current stimulation over the left dorsolateral prefrontal cortex of human participant

58 rtic and common iliac) and pelvic (right and left external iliac and obturator) LN regions with patho

59 sudden painless profound visual loss in the left eye (LE) two hours after embolization.

60 r periodic acid-Schiff staining and from the left eye for MUC5AC mucin immunostaining at baseline and

61 oscopy showed conjunctival hyperaemia in the left eye with a slight elevation, suggesting a subconjun

62 hy (OCT) revealed the presence of CME in the left eye.

63 , those larvae that previously favored their left-eye exhibited higher survival.

64 between EZW constriction rates of right and left eyes (rs = 0.627, P = .0002) and between EZW constr

65 .6% and 97.9% of CT scans from the right and left femoral arteries, respectively.

66 heterization of the right femoral artery and left femoral vein.

67 ther ancient Near Easterners of this period, left few surviving textual records and thus their origin

68 It was found that THC left firing rate unaltered and only slightly reduced the

69 responses to concurrent thermal stimulation (left forearm) and visual attention tasks of titrated dif

70 Moreover, the MMN amplitudes evoked from the left forebrain and midbrain were typically larger than t

71 hen reading optical density, and the residue left from the exposure mixture after dilution was confir

72 al direct current stimulation applied to the left frontal cortex.

73 ateral sensorimotor regions coupled with the left frontoparietal network and visual areas.

74 plants 10 and 19 km upstream of a reservoir left geochemical signatures in sediments and porewaters

75 to the neutral state, as seen in WT M2, but left half of all histidines cationic, unambiguously demo

76 l enantiomeric excess (EE) between right and left-handed biomolecules.

77 phenotype in POMT mutant embryos that shows left-handed body torsion.

78 In particular, for a left-handed chiral tip, transverse forces are attractive

79 e direction of moving charged particles in a left-handed material.

80 These show that Cdt1 stabilizes MCM in a left-handed spiral open at the Mcm2-5 gate.

81 They were less likely to smoke or to be left-handed.

82 eased pulmonary venous pressure secondary to left heart disease is the most common cause of pulmonary

83 urs in 12% to 13% of patients with PH due to left heart disease.

84 ympathetic stimulation have been reported in left heart failure, but whether it would be beneficial f

85 Here, we show that hypoplastic left heart syndrome (HLHS), a severe CHD, is multigenic

86 espread temporal/frontal lobe regions of the left hemisphere and expressive aphasia; and (iv) bilater

87 indings challenge consensus that because the left hemisphere in neglect is pathologically over-excite

88 l delta brushes which were associated in the left hemisphere with ipsilateral BOLD activation in the

89 gions of the fronto-thalamic circuits in the left hemisphere.

90 There was a left-hemisphere association of motor ability in the cont

91 sentences and word lists to investigate how left-hemispheric brain activity varies during the format

92 culus (AF), a fiber pathway interlinking the left-hemispheric language areas.

93 d action representation system that includes left-hemispheric premotor, parietal, and posterior tempo

94 adverse events was inversely associated with left hippocampal volume.

95 e receptor-1 (Crhr1) gene expression, in the left hippocampus specifically, which co-occurred with ep

96 ol of syllables is mostly lateralized to the left HVC, whereas transition probabilities between the s

97 erial" levels of the normal eye surface have left immunologists wondering whether a true microbiome e

98 o hypothesize the direction of contacts, has left important aspects of ancient exchange open to specu

99 Stents were left in place for 2 months and removed with a stent retr

100 ients with the logopenic variant, within the left inferior frontal cortex in patients with the non-fl

101 me reduction (pre- to post-treatment) in the left inferior parietal cortex, as well as a positive par

102 ng a conventional MIL design, connecting the left inferior pulmonary vein with the mitral annulus.

103 e that the microstructural properties of the left inferior-longitudinal fasciculus predict contextual

104 He underwent left inguinal orchiectomy, which disclosed testicular ca

105 internal thoracic artery (BITA) over single left internal thoracic artery (SITA).

106 y (SRC) effect: When subjects press, e.g., a left key to report stimuli, their reaction time is short

107 e right kidney was slightly enlarged and the left kidney could not be localized.

108 recuneus, left supplementary motor area, and left lingual gyrus were identified as predictors of ECT

109 (IVUS) guidance when performing unprotected left main coronary artery (LMCA) percutaneous coronary i

110 ac Surgery) trial, patients with 3-vessel or left main coronary artery disease (CAD) had improved lon

111 d longitudinal changes in ADAS-cog score and left middle temporal thickness and amygdalar volume (Pon

112 22-year-old African gentleman presented with left nasal obstruction and epistaxis for 2 years and was

113 ons; visuospatial functions: right more than left occipitotemporal regions).

114 However, some wounds may be left open to heal (if there is a risk of infection, or i

115 A diagnosis of left ophthalmic artery occlusion was made.

116 e, high-gamma activity was attenuated in the left orbitofrontal gyrus.

117 The reduced functional connectivity in left orbitofrontal-both thalamic regions with suicidal i

118 s performed where each independent study was left out during model building and later used for valida

119 pant level, correlating abdominal (right and left para-aortic and common iliac) and pelvic (right and

120 Mean diffusivity in the left parahippocampal region of the cingulum was negative

121 elated with WM microstructural damage in the left peritrigonal WM (p<0.0001).

122 gyrus, right inferior temporal gyrus (ITG), left postcentral gyrus/precuneus, left supplementary mot

123 y: left anterior temporal regions; language: left posterior superior temporal lobe and supramarginal

124 ssive aphasia; and (iv) bilateral precentral/left posterior superior-frontal regions and speech arres

125 hrony of alpha and beta frequency within the left posterior temporal and occipital cortices in patien

126 alysis revealed that the fALFF values of the left precuneus and right ITG/IOG were positively correla

127 t decrease in rsFC between the DLPFC and the left putamen and insula.

128 ad significantly increased activation in the left putamen compared with TD patients and healthy contr

129 ) displayed a titration curve shifted to the left relative to wild type channels and the ICl was near

130 quent impact on dand5 expression pattern and left-right (L-R) axis establishment.

131 dal orthologs in zebrafish; southpaw directs left-right asymmetries, while squint and cyclops functio

132 n a split-belt treadmill, which can impose a left-right asymmetry in step lengths.

133 he commissural projections of V0s results in left-right desynchronized inspiratory motor commands in

134 y commissural interneurons (CINs), providing left-right interactions, and LPNs, mediating homolateral

135 heart disease and heterotaxy, a disorder of left-right patterning, we previously identified the guan

136 Four experiments used pointing or left/right 2-alternative forced choice tasks to examine

137 s exhibit development defects in stereotyped left/right axon guidance choices within the GABAergic mo

138 t dorsal anterior cingulate cortex (ACC) and left rostral ACC.

139 Excitation of left sensorimotor circuits, during an adaptive cognitive

140 focal application of acetylcholine induced a left shift of the input/output curve and persistent firi

141 ygen saturation (indicating greater right-to-left shunting); higher transferrin iron saturation index

142 ide) and allocentric neglect (neglect of the left side of each object, regardless of the position of

143 amage has occurred in the brain, usually the left side of space.

144 established within mesodermal tissue on the left side only.

145 t of information falling on the individual's left side) and allocentric neglect (neglect of the left

146 er a bias restricted to the smaller numbers (left side) or no significant number bias (right side).

147 ht heart remodeling differ between AF-TR and left-sided heart disease-TR.

148 e, reflected by increased ratio of right- to left-sided heart filling pressures (0.64+/-0.17 versus 0

149 s cetuximab clearly benefitted patients with left-sided tumors (vs FOLFIRI or FOLFIRI plus bevacizuma

150 lations of CRYSTAL and FIRE-3, patients with left-sided tumors had a markedly better prognosis than t

151 vanced heart failure, VT cycle length, and a left-sided-only procedure.

152 of maxillary sinuses (right sinus: p=0.005; left sinus: p=0.048).

153 Historically, follow-up care has left some patients with insufficient understanding of th

154 ry causes visual neglect - lost awareness of left space.

155 t decrease in rsFC between the DLPFC and the left superior frontal gyrus (SFG) and anterior cingulate

156 ferior parietal lobe post-cocaine and in the left superior frontal gyrus post-saline.

157 hemisphere and auditory hallucination; (ii) left superior-/middle-temporal gyri and receptive aphasi

158 rus (ITG), left postcentral gyrus/precuneus, left supplementary motor area, and left lingual gyrus we

159 on-fluent/agrammatic variant, and within the left temporo-parietal junction in patients with the sema

160 On the pathological examination, all left testes in the torsion group were recoverable after

161 On ultrasound, the left testis was located in the inguinal canal, the right

162 strated a reduction in glucose uptake in the left thalamus and bilateral inferior parietal lobe.

163 stance (thickness) of the right amygdala and left thalamus, and localized increases and decreases in

164 n time is shorter when stimuli appear to the left than to the right of the fixation.

165 ged after imaging, whereas SPECT/CT patients left the department earlier, just after radiopharmaceuti

166 gas column density, luminosity and mass, has left the main physical mechanism that regulates obscurat

167 on transcription of protein-coding genes has left the roles of Pol III in organismal physiology relat

168 chemical synthesis, out of 593 hits, 39 were left top hits.

169 Overall, 85 of 966 residents (8.8%) left training during the study period: 15 (17.6%) left a

170 Left unabated, these challenges will drive many AMCs to

171 When left unchecked, algae can cause declines in seagrass pro

172 ctional anisotropy (FA) was lower within the left uncinate fasciculus, right caudate and occipital re

173 lus predict contextual learning, whereas the left uncinate was associated with cross-situational lear

174 ce is consistent with many food web patterns left unexplained by the simplest prey-dependent models.

175 imately half of DCIS may progress to IDC, if left untreated.

176 lic disorder that can be life-threatening if left untreated.

177 Mild atrophy in the left upper and lower limbs without pain, swelling, or sk

178 re, hyperparathyroidism, or even symptoms of left upper limb ischemia.

179 iform gyrus and sources in a control region (left V1) yielded successful classification of facial ide

180 and amygdala functional connectivity to the left ventral and right rostral prefrontal cortex.

181 Remodeling of the left ventricle (LV) in response to pressure overload lea

182 was increased in the infarcted region of the left ventricle and in the circulation of wild-type mice

183 art tube (HT), with the FHF contributing the left ventricle and part of the atria, and the SHF the re

184 ferential and longitudinal strain within the left ventricle in healthy Chinese subjects.

185 og of MIB2, have been found in patients with left ventricle non-compaction (LVNC), we investigated me

186 S AND Twenty-eight consecutive patients with left ventricle outflow tract premature ventricular contr

187 ventricular contractions originating in the left ventricle outflow tract represent a significant sub

188 BrS, conduction delay in RVOT, but not RV or left ventricle, correlated to the degree of J-ST point e

189 Compared with the left ventricle, E12 values were lower in the right ventr

190 icular outflow tract (P<0.001) and higher in left ventricle-right ventricle pairs (P=0.021) and left

191 ated from human donor and heart failure (HF) left ventricle.

192 heavy chain 7 mRNA expression is detected in left ventricles.

193 of cardioprotective gene networks to prevent left ventricular (LV) adverse remodeling.

194 d restoring forces are known determinants of left ventricular (LV) diastolic function.

195 I swine models recapitulating the effects of left ventricular (LV) dysfunction, ischemic MR, and left

196 mean 148 mL/m(2)) volumes, and lower RV and left ventricular (LV) ejection fractions compared with c

197 erved ejection fraction develop increases in left ventricular (LV) end-diastolic pressures during exe

198 AART exposure was positively associated with left ventricular (LV) fractional shortening (z-score for

199 Left ventricular (LV) global longitudinal strain (GLS) i

200 Left ventricular (LV) hypertrophy and abnormal myocardia

201 Left ventricular (LV) morphology and systolic and diasto

202 tional implications beyond the reflection of left ventricular (LV) pathology are not well understood.

203 The analysis included 16 traits of left ventricular (LV) structure, and systolic and diasto

204 3% male, age 54 +/- 12 years) complicated by left ventricular (LV) systolic dysfunction; (2) an age-

205 After aortic valve replacement, left ventricular afterload is often characterized by the

206 of atrial fibrillation (AF) risk, including left ventricular and pulmonary pathology, systemic infla

207 Blinded quantification of left ventricular and right ventricular (RV) volumes was

208 giodysplasia develops during continuous-flow left ventricular assist device (LVAD) support.

209 activity in patients with end-stage HF after left ventricular assist device (LVAD)-induced remodeling

210 ence interval, 4.19-8.61; P<0.001), need for left ventricular assist device (odds ratio, 3.48; 95% co

211 biopsies of multiple HF patients undergoing left ventricular assist device implantation surgery.

212 ective analysis evaluated 51 continuous-flow left ventricular assist device patients who received sec

213 Left ventricular assist device-supported patients are us

214 Improved safety of left ventricular assist devices means that these are bec

215 t failure receiving mechanical unloading via left ventricular assist devices show increased CTCF abun

216 pression correlated with cardiac fibrosis on left ventricular biopsy (P=0.63; P<0.01).

217 pertrophy is also frequently associated with left ventricular diastolic dysfunction.

218 of CD4(+) T cells and prevented progressive left ventricular dilatation and hypertrophy, whereas ado

219 Left ventricular dilation and loss of heart function was

220 r age, congestive heart failure, and greater left ventricular dilation at diagnosis were independentl

221 Trpc6 deletion also ameliorated left ventricular dilation, improved cardiac function, an

222 ablated the interferon response and improved left ventricular dysfunction and survival.

223 diomyocytes provoked cardiac hypertrophy and left ventricular dysfunction in vivo, whereas genetic kn

224 S10) intracoronary infusion in patients with left ventricular dysfunction post STEMI.

225 s) from donor mice with HF induced long-term left ventricular dysfunction, fibrosis, and hypertrophy

226 ve generally a normal coronary angiogram and left ventricular dysfunction, which extends beyond the t

227 ventricular transmural pressure, and greater left ventricular eccentricity index (1.10+/-0.19 versus

228 t Association class II to IV symptoms, and a left ventricular EF of 40% or less to treatment with ena

229 nt effects were found on secondary outcomes: left ventricular EF, peak aerobic exercise capacity, and

230 In HFpEF, defined as left ventricular ejection fraction >/=40%, we derived pr

231 Two hundred sixty-one patients with left ventricular ejection fraction </=35% and New York H

232 in patients with coronary artery disease and left ventricular ejection fraction </=40%.

233 ng ViV, 72 patients undergoing ViR had lower left ventricular ejection fraction (45.6 +/- 17.4% vs. 5

234 .14, SE=0.23), % females (B=-0.38, SE=0.04), left ventricular ejection fraction (B=-0.81, SE=0.20), a

235 Improvement in left ventricular ejection fraction (EF) to >35% occurs i

236 etermination of left ventricular volumes and left ventricular ejection fraction (LVEF).

237 tic relative area change was associated with left ventricular ejection fraction (P=0.045) and ventric

238 e prospectively enrolled (age 62+/-11 years, left ventricular ejection fraction 27+/-7%).

239 vely reduced LV systolic function (mean+/-SD left ventricular ejection fraction = 52+/-11% versus 63+

240 , and identified 472 donor hearts with LVSD (left ventricular ejection fraction [LVEF] </=40%) on ini

241 ed significantly with MR imaging measures of left ventricular ejection fraction and end-systolic volu

242 [15.8], P=0.02) and no significant change of left ventricular ejection fraction in the cell group.

243 nd safety of levosimendan in patients with a left ventricular ejection fraction of 35% or less who we

244 However, PPM is associated with impaired left ventricular ejection fraction recovery post-transca

245 onischemic dilated cardiomyopathy), the mean left ventricular ejection fraction was 32+/-12% (range,

246 The left ventricular ejection fraction was consistently decr

247 ic frequency methods can be used to document left ventricular ejection fraction with accuracy compara

248 Ivabradine treatment improved left ventricular ejection fraction, and clinical status

249 ond traditional cardiovascular risk factors, left ventricular ejection fraction, myocardial scar and

250 justment were increasing age, lower baseline left ventricular ejection fraction, worse post-procedura

251 for patients with heart failure with reduced left ventricular ejection fraction.

252 nterval, 1.09-2.07), but not with decline in left ventricular ejection fraction.

253 congestion, but no significant difference in left ventricular ejection fraction.

254 or mean age=49-80 years, sex=0%-92% females, left ventricular ejection fraction=26%-61%).

255 cardial function (ejection fraction [EF] and left ventricular end-diastolic pressure) was assessed at

256 age, congestive heart failure, and increased left ventricular end-systolic dimension zscore at diagno

257 entricle-right ventricle pairs (P=0.021) and left ventricular epicardium (P=0.08).

258 ession and CVB3 copy number, and an improved left ventricular function in NOD2(-/-) CVB3 mice compare

259 ymptomatic severe aortic stenosis and normal left ventricular function, current practice guidelines e

260 Improvements in left ventricular function, functional status, and qualit

261 perior cardiac repair in vivo with regard to left ventricular function, vascularization, and ameliora

262 tural and functional remodeling and improves left ventricular functional reserve.

263 mbrane-permeabilized cardiomyocytes in human left ventricular heart tissue.

264 ographic (ECG) criteria for the diagnosis of left ventricular hypertrophy (LVH) have low sensitivity.

265 d would lead to more lowering of the risk of left ventricular hypertrophy (LVH) in patients with hype

266 cretion, has been inconsistently linked with left ventricular hypertrophy.

267 icle predominate, it is well recognized that left ventricular involvement is common, particularly in

268 54.1 years; p = 0.002) and had lower indexed left ventricular mass (5.1 g/m(2) reduction; padjusted =

269 (bioimpedance spectroscopy), 24-hour BP, and left ventricular mass (cardiac magnetic resonance imagin

270 secondary objective was to assess changes in left ventricular mass.

271 y, compared with dimension and area methods, left ventricular measurements by volume method have the

272 vator of transcription (STAT)5 activation in left ventricular myocardium is associated with RIPC s ca

273 Left ventricular NOD2 mRNA expression was also induced i

274 al heart defect (CTD) case-parent trios, 317 left ventricular obstructive tract defect (LVOTD) case-p

275 e lower in the right ventricle (P=0.037) and left ventricular outflow tract (P<0.001) and higher in l

276 myofibers normally run in parallel along the left ventricular outflow tract, but in the Nkx2-5(+/-)/S

277 e pacing, His bundle pacing, and endocardial left ventricular pacing.

278 ic expansion during systole, which modulates left ventricular performance and impacts systemic hemody

279 nce imaging, the rats were catheterized, and left ventricular pressures were recorded.

280 dial injury, and it is a strong predictor of left ventricular remodeling in ST-segment-elevation myoc

281 HIIT was not superior to MCT in changing left ventricular remodeling or aerobic capacity, and its

282 L [2380-3006 mL]; P<0.0001), more concentric left ventricular remodeling, greater right ventricular d

283 e biological changes responsible for adverse left ventricular remodeling, the relationship between in

284 ocardial injury and its predictive value for left ventricular remodeling.

285 of the heart were determined from CT and the left ventricular ROI, and mean counts were calculated us

286 ereas mice producing GM-CSF can succumb from left ventricular rupture, a complication mitigated by an

287 linical CVD was defined by 10-year change in left ventricular structure and function.

288 Atrial fibrillation (AF) and left ventricular systolic dysfunction (LVSD) frequently

289 mortality in patients with AMI without HF or left ventricular systolic dysfunction (LVSD).

290 te gadolinium enhancement in phenotyping the left ventricular to identify those at highest risk for S

291 higher pulmonary venous pressure relative to left ventricular transmural pressure, and greater left v

292 There was a significant reduction of left ventricular volumes (end-systolic volume: -4.3 [11.

293 e and post-cycle 17 for the determination of left ventricular volumes and left ventricular ejection f

294 que were implanted into the anterior-lateral left ventricular wall in C57BL/6J (allogeneic model, n =

295 characterize microstructural dynamics during left ventricular wall thickening, and apply the techniqu

296 ined ventricular tachycardia (nsVT), maximum left ventricular wall thickness and obstruction were sig

297 stigated the distribution characteristics of left-ventricular myocardial strain using a novel cine MR

298 cortex/insula, the dorsal midbrain, and the left ventromedial prefrontal cortex.

299 mation on the location of the primary tumor (left vs right location site of CC) independent of other

300 d evidence for g in nonhuman animals, we are left with major questions about how the disposition to t