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1 rtery stenoses (left anterior descending, 9; left circumflex, 2; mean, 59 +/- 23% diameter stenosis)
2 ry angiography (left anterior descending, 4; left circumflex, 2; right coronary artery, 4; and right
4 y for the detection of > 50% stenosis in the left circumflex (74% and 96%; 50% and 100%; 63% and 91%,
5 swine fed a high-cholesterol diet underwent left circumflex ameroid constrictor placement to induce
9 nce interval, 1.00 to 1.64; P=0.046) for the left circumflex, and 1.32 (95% confidence interval, 1.03
10 th those in distal left anterior descending, left circumflex, and right coronary arteries (0.80+/-0.0
11 cle) and regional (left anterior descending, left circumflex, and right coronary arteries) MBF and fl
14 r the detection of left anterior descending, left circumflex, and right coronary artery stenosis, sen
15 automatically for left anterior descending, left circumflex, and right coronary artery territories a
16 defect size in the left anterior descending, left circumflex, and right coronary artery territories,
17 artery territories-left anterior descending, left circumflex, and right-as well as left ventricular (
22 left anterior descending arteries and 86% of left circumflex arteries, whereas calcified nodules with
23 anterior descending artery (LAD) (n = 27) or left circumflex artery (LCx) (n = 29) were evaluated wit
25 detection of right coronary artery (RCA) and left circumflex artery (LCX) lesions (0.84 +/- 0.08 vs.
26 In the 6 critical-stenosis dogs, the LAD-to-left circumflex artery (LCx) microsphere flow ratio was
28 ries: left anterior descending artery (LAD), left circumflex artery (LCX), and right coronary artery
30 t anterior descending coronary artery [20%], left circumflex artery [12%] and right coronary artery [
31 .2% +/- 1%), 123I-IPPA defect magnitude (LAD/left circumflex artery [LCX] count ratios) decreased fro
32 itory (left anterior descending artery [LAD] left circumflex artery and posterior descending artery [
33 nary collateral channels were induced in the left circumflex artery bed of 12 chronically instrumente
34 rescent microsphere-derived MBF ratio in LAD/left circumflex artery beds demonstrated close correlati
37 illation (VF) was evaluated using a 2-minute left circumflex artery occlusion during the last minute
38 ctive CFI differences during either IMA with left circumflex artery occlusion were inconsistently pos
42 1 month after the surgery, occlusion of the left circumflex artery regularly produced ventricular fi
45 myocardial flow reserve were reduced in the left circumflex artery territory (both P<0.001), and hib
46 placement of an ameroid constrictor into the left circumflex artery to induce chronic myocardial isch
47 ients selected for coronary angiography, the left circumflex artery was the culprit vessel in 34.6% o
48 nary artery, 10 right coronary artery, and 5 left circumflex artery) retrieved by using directional c
49 erior descending artery, 90% and 70% for the left circumflex artery, and 74% and 79% for the right co
50 lium chloride staining demonstrated that the left circumflex artery, and not the LAD, group had atria
62 terior descending branch, 115.9 mm +/- 19.7; left circumflex branch, 97.2 mm +/- 12.5; and right coro
63 retch of normal left anterior descending and left circumflex coronary arteries dose dependently reduc
64 two Yorkshire pigs with chronically occluded left circumflex coronary arteries were randomly assigned
66 rior descending coronary artery, 96% for the left circumflex coronary artery (22/23), and 100% for th
67 680 were then separately infused to maximize left circumflex coronary artery (LCx) flow velocity.
69 oronary artery (RCA) in six subjects and the left circumflex coronary artery (LCX) in one patient.
72 rogressive ameroid occlusion of the proximal left circumflex coronary artery (LCx); after 2 months, a
73 +/- 0.01; P < 0.01) defect count ratios (LAD/left circumflex coronary artery [LCx]) differentiated be
74 artery [LAD]: n = 13, r = 0.89, p < 0.0001; left circumflex coronary artery [LCx]: n = 11, r = 0.7,
75 gs underwent operative placement of proximal left circumflex coronary artery ameroid constrictors.
76 cending coronary artery, 44% and 90% for the left circumflex coronary artery and 75% and 77% for the
77 ubjected to ameroid-induced occlusion of the left circumflex coronary artery and randomized to bFGF (
78 ubjected to ameroid-induced occlusion of the left circumflex coronary artery and randomized to bFGF 1
79 ulse Doppler flow probe implanted around the left circumflex coronary artery and with catheters in le
80 es along the left anterior descending and/or left circumflex coronary artery by intracoronary ultraso
81 n was provoked by transient occlusion of the left circumflex coronary artery during submaximal exerci
83 gs (left anterior descending coronary artery/left circumflex coronary artery flow 0.53+/-0.16 in the
86 sed at a rate of 2 ng/kg per minute into the left circumflex coronary artery in normal dogs (n = 5) a
87 e from the infarction zone eight weeks after left circumflex coronary artery ligation in pigs, demons
90 (open chest) before and continuously during left circumflex coronary artery occlusion to induce acut
91 ht of early passage autologous MSCs into the left circumflex coronary artery of anaesthetised dogs.
92 gher occurrence of TLR for restenosis of the left circumflex coronary artery ostium (LCX-ISR) (HR, 2.
93 chest dogs with left anterior descending and left circumflex coronary artery stenoses that reduced hy
94 erritories: the left anterior descending and left circumflex coronary artery territories and the righ
95 left anterior descending coronary artery and left circumflex coronary artery territories, whereas AC
97 A) and coronary sinus (CS) as well as CS and left circumflex coronary artery using cardiac computed t
98 this hypothesis, a 2-minute occlusion of the left circumflex coronary artery was made during the last
99 dogs, either the left anterior descending or left circumflex coronary artery was occluded for 90 minu
101 three segments of LAD and three segments of left circumflex coronary artery) increased by 19.3% (3.2
102 vena cava, a flow probe around the proximal left circumflex coronary artery, and catheters in the le
103 on in either the left anterior descending or left circumflex coronary artery, and the S-VF DFT was de
104 s with an ameroid constrictor applied to the left circumflex coronary artery, in each pig, peak beta-
115 internal diameter and wall thickness, and a left circumflex coronary blood flow velocity transducer.
117 D above normal (P<0.0001), and single-vessel left circumflex disease (P<0.0007; odds ratio, 7.6).
118 of false-negative studies were single-vessel left circumflex disease, increased wall thickness, small
119 gher resting left anterior descending artery/left circumflex flow ratio compared with placebo (P<0.03
120 microsphere-derived LAD flow, normalized to left circumflex flow, correspondingly increased between
123 f adenosine infusion via a surgically placed left circumflex (LCx) catheter (n=11) or via a right atr
124 ree models of MI were generated: 1) proximal left circumflex (LCx) coronary artery occlusion involvin
126 e-related left anterior descending (LAD) and left circumflex (LCx) coronary artery vasodilatation wit
130 in chronically instrumented dogs with either left circumflex (LCx) infusion of adenosine or partial L
132 te loss was significantly greater within the left circumflex (LCX) ostium compared to the parent vess
135 -line for the LAD risk area and the adjacent left circumflex (LCx) territory, and peak background-sub
136 e from ostium of CS to the intersection with left circumflex (LCX), and anatomical relation of LCX an
137 f the left anterior descending (LAD, n = 5), left circumflex (LCx, n = 5), and carotid (n = 5) arteri
139 ed in the left anterior descending (n = 11), left circumflex (n = 7) or right (n = 1) coronary artery
141 left versus right dominance among those with left circumflex or left main culprit lesions was 1.25 (9
143 n the left anterior descending (P=0.038) and left circumflex (P=0.009) regions persisted, which indic
144 sty in both the left anterior descending and left circumflex plus stent implantation in the right cor
145 pressed as lower Ecc in the RCA (P<0.01) and left circumflex regions (P<0.05) measured in the subendo
147 ght coronary segments were reassigned to the left circumflex territory (39% of reassigned segments),
148 80%, P<0.0001) and was more accurate for the left circumflex than for the left anterior descending co
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