ライフサイエンスコーパス: left ventricle

1 nal distribution (predominating in the basal left ventricle).

2 ad an intraaortic balloon pump to unload the left ventricle).

3 ded right ventricle and good function of the left ventricle.

4 volving the aorta, aortic valve annulus, and left ventricle.

5 y a typical diffuse spongy appearance of the left ventricle.

6 life-threatening CHD primarily affecting the left ventricle.

7 VH, and these derangements often involve the left ventricle.

8 vered randomly on the basal, mid and lateral left ventricle.

9 vortex ring from the surrounding flow in the left ventricle.

10 lly greater than previously reported for the left ventricle.

11 We studied single myocytes from pig left ventricle.

12 dies, this results in an adversely remodeled left ventricle.

13 he posterior mitral annulus of a pressurized left ventricle.

14 identify the position and orientation of the left ventricle.

15 nteroapical, lateral and septal walls of the left ventricle.

16 age-dependent increase of GAG levels in the left ventricle.

17 acquired at sites distributed throughout the left ventricle.

18 heart tube and ultimately gives rise to the left ventricle.

19 us 21+/-12 ms; P=0.0005) compared with RV or left ventricle.

20 ated from human donor and heart failure (HF) left ventricle.

21 y intervals prolongation in the RVOT, RV, or left ventricle.

22 involvement at the inferolateral wall of the left ventricle.

23 le or posteromedial papillary muscles of the left ventricle.

24 protein were unchanged in MR versus control left ventricles.

25 ventricles and its marked upregulation in MR left ventricles.

26 lt human CF isolated from normal and failing left ventricles.

27 heavy chain 7 mRNA expression is detected in left ventricles.

28 patients with SCD had severe dilation of the left ventricle (124+/-27 vs 79+/-12 mL/m(2)), right vent

29 ring the matched breathing control (end film left ventricle 199.8 ms [SD, 16] versus control 201.6 ms

30 in significantly worse pump function of the left ventricle 28 days after MI.

31 ds, 17 mL/min irrigation) and 3 sites in the left ventricle (40 W, 60 seconds, 30 mL/min irrigation)

32 cortex, renal outer medulla, liver, cardiac left ventricle, adrenal gland, and hypothalamus.

33 t of MI than placebo-control animals (15.7 g left ventricle and 12.0 g left ventricle versus 22.8 g l

34 GE yields good to moderate quality in 74% of left ventricle and 84% of right ventricle acquisitions a

35 c was safe and improved contractility of the left ventricle and atrium in a large animal model of non

36 was increased in the infarcted region of the left ventricle and in the circulation of wild-type mice

37 art tube (HT), with the FHF contributing the left ventricle and part of the atria, and the SHF the re

38 dependent effects of circulating histones on left ventricle and right ventricle function at clinicall

39 heters were inserted, respectively, into the left ventricle and right ventricle of dogs to record end

40 otoxicity and the functional consequences on left ventricle and right ventricle remain unclear.

41 nct cardiac regions (the focal injury in the left ventricle and the border region close to the occlud

42 hibited discordant reverse remodeling in the left ventricle and the left atrium.

43 o not reliably prevent the remodeling of the left ventricle and the progression to heart failure.

44 y mimics pressure-volume changes seen in the left ventricle and to use this system to achieve cardiac

45 onfirmed the absence of sarcolipin in normal left ventricles and its marked upregulation in MR left v

46 f the pulmonary artery, aorta, and right and left ventricles and the severity of reflux of contrast m

47 ements were made at 222 sites (excluding the left ventricle) and compared with measurements from intr

48 Global (left ventricle) and regional (left anterior descending,

49 ith an ischemic pathogenesis, a more dilated left ventricle, and a detectable hs-troponin had lower o

50 FGE acquisitions, respectively, covering the left ventricle, and in 69% and 84%, respectively, coveri

51 position of the great arteries, subpulmonary left ventricle, and left ventricular outflow tract (LVOT

52 rom the release source, down the axis of the left ventricle, and selectively toward the left heart fo

53 favorable energetic effect of unloading the left ventricle, and thus reduction of wall stress, could

54 owed by the left atrium-aorta system and the left ventricle-aorta system (P<0.001).

55 However, the left ventricle-aorta system with norepinephrine still ma

56 cle-aorta, Impella 2.5 system (n=4), and (4) left ventricle-aorta with norepinephrine at 0.1 microg/k

57 atrium-aorta, TandemHeart system (n=4); (3) left ventricle-aorta, Impella 2.5 system (n=4), and (4)

58 Logistic regression identified the left ventricle/aorta angle as an indicator of indexed ao

59 ft ventricular outflow axis and aortic root (left ventricle/aorta angle) in both groups (BAV group: r

60 Early in systole, parts of the left ventricle are being stretched by incoming blood, be

61 creted in acute stages of inflammation after left ventricle assisted device (LVAD) implantation for p

62 In left ventricle at baseline, messenger ribonucleic acid f

63 d component of therapy for HF with a dilated left ventricle because of its effectiveness in inhibitin

64 miRNA expression profiles were measured in left ventricle biopsies from 10 diabetic HF (D-HF) and 1

65 ale ranging from 0 (uptake less than that in left ventricle blood pool) to 4 (diffuse uptake greater

66 ted well with lesion size for lesions in the left ventricle but less well for lesions in the right ve

67 s no change to the number of ICDs within the left ventricle, but there is an almost doubling of the n

68 (99m)Tc-HYNIC annexin V uptake as percentage left ventricle by scanning correlated with caspase stain

69 etic resonance imaging reconstruction of the left ventricle can reproduce the reentrant circuits of i

70 ), we observed no significant differences in left ventricle capillary density between wild-type and S

71 Images were analyzed using left ventricle cavity, left atrial cavity, or inferior v

72 nt of the right ventricle in addition to the left ventricle classically studied.

73 was 8-fold more highly expressed in the male left ventricle compared with females in young adult C57B

74 y exhibited decreased survival with impaired left ventricle contractility and decreased serum adipone

75 rosis, leukocyte infiltration, angiogenesis, left ventricle contractility, and inflammatory gene expr

76 e result in severe cardiac hypertrophy, poor left ventricle contraction and death by postnatal day 16

77 BrS, conduction delay in RVOT, but not RV or left ventricle, correlated to the degree of J-ST point e

78 Stress MBF of the left ventricle decreased by 10% +/- 6% (P = 0.005) after

79 These data support that first unloading the left ventricle despite delaying coronary reperfusion dur

80 ith the ratio of right ventricle diameter to left ventricle diameter (RV/LV ratio).

81 ecific ephrin-B1 KO mice exhibited increased left ventricle diameter and delayed atrioventricular con

82 The median right ventricle to left ventricle diameter ratio was 1.39.

83 otypes under base-line conditions but caused left ventricle dilatation, contractile dysfunction, and

84 expression, direct targets of FXR1 in human left ventricle dilated cardiomyopathy (DCM) biopsy sampl

85 Thereafter, the left ventricle dilates and restores the baseline mass-to

86 nts in anterior wall dimensions and prevents left ventricle dilation compared with hearts treated wit

87 ed myocyte apoptosis and induced significant left ventricle dilation in alpha1(+/-) mice but not in t

88 ablation of MMP-9 attenuated cardiac injury, left ventricle dilation, and fibrosis in 1-y-old mdx mic

89 ptosis, limited infarct expansion, prevented left ventricle dilation, and reduced mortality in Capn4-

90 unctionally, rAAV.hHO-1 and hHO-1 transgenic left ventricles displayed a smaller loss of ejection fra

91 terminant factor in how much blood fills the left ventricle during diastole and thus in the etiology

92 partially mediated by diseases affecting the left ventricle during follow-up (myocardial infarction [

93 CTRP9-KO mice had exacerbated contractile left ventricle dysfunction following intraperitoneal inj

94 Compared with the left ventricle, E12 values were lower in the right ventr

95 s the function of the proximal aorta and the left ventricle (eg, aortic arch pulse wave velocity and

96 ic evidence of left ventricular dysfunction (left ventricle ejection fraction <50%).

97 ume, left ventricle end-systolic volume, and left ventricle ejection fraction (LVEF) were evaluated.

98 The pre-LTx left ventricle ejection fraction did not differ between

99 Median left ventricle ejection fraction was 24% (10%-36%).

100 Nonetheless, the improvement in left ventricle ejection fraction with hMSC therapy persi

101 early, cardiac remodeling, deterioration of left ventricle ejection fraction, and cardiac arrhythmia

102 CMR showed higher left ventricle end-diastolic volume (mean difference: 43

103 Left ventricle end-diastolic volume index and end-systol

104 Patients experiencing MACE showed higher left ventricle end-diastolic volume, higher left ventric

105 All patients underwent TTE and CMR, and left ventricle end-diastolic volume, left ventricle end-

106 lume (mean difference: 43+/-22.5 mL), higher left ventricle end-systolic volume (mean difference: 34+

107 MR, and left ventricle end-diastolic volume, left ventricle end-systolic volume, and left ventricle e

108 left ventricle end-diastolic volume, higher left ventricle end-systolic volume, and lower LVEF with

109 remodeling characterized by dilation of the left ventricle (end-diastolic volume, 156+/-26 versus 17

110 using a 64-electrode basket catheter on the left ventricle endocardium and 54 6-electrode plunge nee

111 Underdeveloped left ventricle exerts biomechanical stress on the right

112 rovement of its volumes and function; 2) the left ventricle experiences improvement of its function;

113 ure work is warranted to investigate whether left ventricle fibrosis affects clinical outcomes.

114 s used to quantify the edema-based AAR (% of left ventricle) following ischemic preconditioning (IPC)

115 valve plane (VP) during segmentation of the left ventricle for SPECT myocardial perfusion imaging (M

116 old; P<0.05), which coincided with decreased left ventricle fractional shortening (-Delta11%; P<0.05)

117 rk to create 3D finite element models of the left ventricle from cardiac ultrasound or magnetic reson

118 adult mouse hearts and was also elevated in left ventricles from patients with dilated cardiomyopath

119 ccording to standard 17-segment model of the left ventricle (fully automatic analysis).

120 gradients, valve effective orifice area) or left ventricle function (i.e., left ventricular ejection

121 patients </=70 y with scarring </=22% of the left ventricle had a greater increase in LV ejection fra

122 ariations in longitudinal deformation of the left ventricle have been suggested to be useful for diff

123 rs for pacemaker placement included systemic left ventricle (hazard ratio [HR], 2.2; P=0.006) and lat

124 as clinically unexplained hypertrophy of the left ventricle, hypertrophic cardiomyopathy (HCM) is tra

125 The ECG showed left ventricle hypertrophy by voltage and slight prolong

126 ed insulin levels, cardiac systole deficits, left ventricle hypertrophy, a predictor of a later onset

127 or linear (38%, nine of 24) and involved the left ventricle in 13 patients and both ventricles in 24

128 s (63%) and in the endocardial inferolateral left ventricle in 3 patients (37%).

129 a-MyHC were found in discrete regions of the left ventricle in 3 patterns: perivascular, in areas wit

130 ns were significantly induced in the excised left ventricle in cholesterol group, whereas significant

131 ferential and longitudinal strain within the left ventricle in healthy Chinese subjects.

132 r cells were injected intraarterially in the left ventricle in nude mice.

133 tive physiology leading to the growth of the left ventricle in parallel with somatic growth.

134 of life, the number of cardiomyocytes in the left ventricle increased 3.4-fold, which was consistent

135 erentiated these groups were right ventricle:left ventricle inflow angle, LV width/LV length, left AV

136 Whether the left ventricle is also affected by VIP gene deletion is

137 Flow entering the left ventricle is reversed toward the outflow tract thro

138 Plasma troponin and left ventricle lactate were higher in controls than that

139 ss disrupts insulin signaling in the cardiac left ventricle leading to adverse cardiac programming.

140 In guinea-pig left ventricle, left atrium, and right atrium, carbenoxo

141 sverse aortic constriction (TAC) to increase left ventricle load.

142 <3.1 cm/m(2), global longitudinal strain of left ventricle <-7%, left atrial area <26 cm(2), right v

143 egeneration after surgical amputation of the left ventricle (LV) (apical resection) and coronary arte

144 cells (BM-MNC) may improve remodeling of the left ventricle (LV) after acute myocardial infarction.

145 se To prospectively evaluate the accuracy of left ventricle (LV) analysis with a two-dimensional real

146 enosis (AS) leads to variable stress for the left ventricle (LV) and consequently a broad range of LV

147 as due to enhanced myofiber work of both the left ventricle (LV) and RV.

148 progenitor populations that give rise to the left ventricle (LV) and sinus venosus (SV) are still amb

149 Ptges(-/-) mice develop more left ventricle (LV) dilation, worse LV contractile funct

150 Although contributors to remodeling of the left ventricle (LV) have been well studied in general po

151 Pathological left ventricle (LV) hypertrophy (LVH) results in reactiv

152 severe cardiac malformation characterized by left ventricle (LV) hypoplasia and abnormal LV perfusion

153 s study was to assess the performance of the left ventricle (LV) in normal, uncomplicated pregnancies

154 evaluate diffuse myocardial fibrosis of the left ventricle (LV) in patients with atrial fibrillation

155 al systolic and diastolic performance of the left ventricle (LV) in patients with heart failure with

156 Remodeling of the left ventricle (LV) in response to pressure overload lea

157 lectrophysiologic remodeling of the atrophic left ventricle (LV) in right ventricular (RV) failure (R

158 with hypoplastic LH syndrome and borderline left ventricle (LV) involves 2 options: SVP or biventric

159 n (MI), overactive inflammation remodels the left ventricle (LV) leading to heart failure coinciding

160 The geometry of the right ventricle (RV) and left ventricle (LV) may alter tricuspid annulus size and

161 an detect morphological abnormalities of the left ventricle (LV) not visualized with echocardiography

162 he aim of this study was to characterize the left ventricle (LV) of tachycardia-mediated cardiomyopat

163 Whether T-cell recruitment to the left ventricle (LV) participates in the development of H

164 of direct parasympathetic innervation of the left ventricle (LV) remain controversial.

165 tributing to post-myocardial infarction (MI) left ventricle (LV) remodeling.

166 s this problem and better understand how the left ventricle (LV) remodels post-MI at both the molecul

167 ar autonomic neuropathy (CAN) and indices of left ventricle (LV) structure and function in patients w

168 ined the reference values of left atrium and left ventricle (LV) structure in a large ethnically dive

169 Purpose To determine if excess greater left ventricle (LV) trabeculation is associated with dec

170 left ventricular hypertrophy, a thick-walled left ventricle (LV) ultimately transitions to a dilated

171 tively and quantitatively in order to assess left ventricle (LV) wall thickness (full width at half m

172 ardiac MR imaging analyses of the RV and the left ventricle (LV) were performed to determine cardiac

173 rse aortic constriction activated FYN in the left ventricle (LV), and FYN-deficient mice displayed ex

174 rast-enhanced ultrasound in skeletal muscle, left ventricle (LV), and kidney.

175 nificant volume and pressure overload on the left ventricle (LV), but such patients typically remain

176 were placed around the epicardium and in the left ventricle (LV), respectively.

177 ining maintains a youthful compliance of the left ventricle (LV), whereas a year of exercise training

178 n time was determined along 4 anatomic axes: left ventricle (LV)-right ventricle (RV), LV:apico-basal

179 rated post-MI that mediate remodeling of the left ventricle (LV).

180 activation waves and optimal filling of the left ventricle (LV).

181 rm describing lethal underdevelopment of the left ventricle (LV).

182 mal RV DCE, >/=1 DCEs were identified in the left ventricle (LV).

183 ed in T2DM patients because of dysfunctional left ventricle (LV).

184 The right (RV) and left ventricles (LV) do not function in isolation, shari

185 Gene array on left ventricles (LV) showed increased fractalkine, a che

186 early after MI is associated with long-term left-ventricle (LV) remodeling in a rat model.

187 Infarct volume (22 +/- 7% vs. 19 +/- 9% of left ventricle [LV]) and LVEF (24 +/- 8% vs. 28 +/- 9%)

188 Infarct size (percent of left ventricle [LV]) by CMR did not differ between the m

189 enting did not reduce final infarct size (9% left ventricle [LV]; interquartile range [IQR]: 3% to 18

190 ipitated with NCX1 in rat cardiomyocytes and left ventricle lysate.

191 recipitated with NCX1 in rat cardiomyocytes, left ventricle lysates, and HEK293 cells.

192 values (r=0.37, P=0.02) and to the change in left ventricle mass (r=0.31, P=0.046).

193 ife was independently associated with higher left ventricle mass index only among women (P=0.001).

194 edian Society of Thoracic Surgeons score and left ventricle mass.

195 odel was then implemented in a 3-dimensional left ventricle model, demonstrating that such early afte

196 racterised by abnormal trabeculations in the left ventricle, most frequently at the apex.

197 aging measurements, we show that the healthy left ventricle moves in tandem with the expanding vortex

198 urs of reoxygenation, plasma troponin level, left ventricle myocardial levels of lipid hydroperoxides

199 Mechanical studies on skinned left ventricle myocardium measured total and titin-based

200 ons of IKr and IKs to repolarizing the human left ventricle (n = 18).

201 og of MIB2, have been found in patients with left ventricle non-compaction (LVNC), we investigated me

202 ortic flow reversal and ejection flow in the left ventricle occurs at optimal AVD.

203 lity were greater for right ventricle versus left ventricle (odds ratio, 1.8; 95% confidence interval

204 s in calcineurin-dependent activities in the left ventricle of healthy C57BL/6 mice.

205 IFNgamma) were significantly elevated in the left ventricle of heart failure patients carrying the MI

206 rsible cysteine oxidation of proteins in the left ventricle of hearts from mice with metabolic syndro

207 as did survival.Cav1.2mRNAexpression in the left ventricle of heterozygous knockout mice was reduced

208 that endoglin expression is increased in the left ventricle of human subjects with heart failure and

209 atrial pulmonary vein junction, and freewall left ventricle of intact animals.

210 Myocytes were isolated from the left ventricle of male C57BL/6J mice after transverse ao

211 c collagen I and titin n2b expression in the left ventricle of mice with HFpEF.

212 s determine Kir6.2 protein expression in the left ventricle of patients with severe mitral dysfunctio

213 6-electrode plunge needles inserted into the left ventricles of 6 dogs.

214 xpression and increased Wisp-1 levels in the left ventricles of patients with ischemic dilated cardio

215 a-adrenergic signaling and sarcolipin in the left ventricles of patients with isolated MR and LV ejec

216 ependently up-regulated in the atria and the left ventricles of RacET mice on mRNA and protein levels

217 mining the cellular growth mechanisms of the left ventricle on a set of healthy hearts from humans ag

218 S AND Twenty-eight consecutive patients with left ventricle outflow tract premature ventricular contr

219 In patients referred for left ventricle outflow tract premature ventricular contr

220 ventricular contractions originating in the left ventricle outflow tract represent a significant sub

221 rium and atrioventricular node compared with left ventricle (P=5.6x10(-6)).

222 in all animals, 42 +/- 12 vs. 35% +/- 12% of left ventricle, P < 0.001).

223 than epinephrine (41 +/- 8 vs. 47% +/- 6% of left ventricle, P = 0.004), whereas defect of a third ca

224 8% [IQR, 4-12] versus 11% [IQR, 7-17] of the left ventricle, P=0.015).

225 onsistent with origin from scar in the basal left ventricle, particularly the septum, but also basal

226 Left ventricle performance was improved in double transg

227 racterized by excessive trabeculation of the left ventricle, progressive myocardial dysfunction, and

228 The posterior-superior process of the left ventricle (PSP-LV) is the most inferior and posteri

229 basal third of both ventricles, and right-to-left-ventricle PTT, LV FWHM, and LV TTP were calculated.

230 rameter to predict lesion depth in right and left ventricle (r=0.47; P<0.0001; multiple regression P=

231 Analysis showed that the volume of the left ventricle receiving 5 Gy (LV-V5) was the most impor

232 s further characterized by dilatation of the left ventricle, reduced systolic strain rate of the post

233 rigins of parasympathetic innervation of the left ventricle remain controversial.

234 Left ventricle remodeling after anterior wall myocardial

235 (MMPs), have been identified in all forms of left ventricle remodeling and can be a contributory fact

236 more efficiently, as determined by improved left ventricle remodeling and ejection fraction.

237 impairs infarct healing, and contributes to left ventricle remodeling and heart failure.

238 continued research about the relationship of left ventricle remodeling in this family of proteases wi

239 g and unpredicted changes in MMP profiles in left ventricle remodeling processes, such as with pressu

240 left ventricular function and attenuation of left ventricle remodeling that were sustained during 6 m

241 e (PVR), effects reverse right ventricle and left ventricle remodeling, improves ventilator efficienc

242 inflammation, arteriosclerotic lesions, and left ventricle remodeling, we performed a cross-sectiona

243 tiation; both are critical events in adverse left ventricle remodeling.

244 ries to the aorta, aortic valve annulus, and left ventricle require open surgical repair.

245 icle and 12.0 g left ventricle versus 22.8 g left ventricle, respectively).

246 Contrary to conventional thinking, the left ventricle responds to exercise with initial concent

247 Transcriptomic profiling of left ventricles revealed three specific genes [natriuret

248 s of whole cardiomyocytes from sheep and rat left ventricle, revealing that the SR forms a continuous

249 icular outflow tract (P<0.001) and higher in left ventricle-right ventricle pairs (P=0.021) and left

250 hanges characterized by echocardiography and left ventricle/right ventricle catheter-derived variable

251 The change in left ventricle rotation was associated with the change i

252 ntricular arrhythmias (VAs) arising from the left ventricle's papillary muscles has been associated w

253 RV/(left ventricle + septum) did not rise directly in propor

254 Overall, 24+/-19% of left ventricle showed HED uptake levels comparable with

255 ft structures, exemplified here by an active left ventricle simulator.

256 Cardiovascular risk factors, CAC scores, left ventricle size and function, and carotid intima-med

257 erfused rat hearts and coronary-perfused pig left ventricles stained with either di-4-ANEPPS or the n

258 d Performance of Electrodes implanted in the Left Ventricle) study is a prospective multicenter non-r

259 anges compared to our previous report of the left ventricle, suggesting there is likely to be a compo

260 nown type of physiological adaptation of the left ventricle that may have important implications for

261 erential strain gradient was observed in the left ventricle that showed universal increment from the

262 In the left ventricle, the Kir6.2 protein/mRNA ratio was also s

263 itral regurgitation, mainly a disease of the left ventricle, the vision for the next 5 years is not n

264 l direction from the base to the apex of the left ventricle, there was a trend of decreasing peak sys

265 islocalization was also evident in autopsied left ventricle tissue from HGPS patients, suggesting int

266 The HIF-1alpha mRNA related in the left ventricle to aortic pressure, in the left atrium to

267 tic resonance imaging reconstructions of the left ventricle to predict VT circuits.

268 blood was collected percutaneously from the left ventricle under ultrasound guidance.

269 After myocardial infarction, the left ventricle undergoes a wound healing response that i

270 ction in left ventricular (LV) function, the left ventricle undergoes structural remodelling under th

271 rough imaging of tissue taken from the sheep left ventricle using serial block face scanning electron

272 ol animals (15.7 g left ventricle and 12.0 g left ventricle versus 22.8 g left ventricle, respectivel

273 all myocardial infarction leads to increased left ventricle volumes, myocardial stress, and ultimatel

274 We tracked the left ventricle wall motion by segmenting the blood from

275 The left ventricle was accessed via transseptal, retrograde

276 Global longitudinal strain of left ventricle was added to standard echocardiographic m

277 n of parametric and functional measures, the left ventricle was analyzed over 200 sectors.

278 lysis of cine multidetector CT images of the left ventricle was optimized and analyzed with feature-t

279 The left ventricle was segmented with standard clinical soft

280 icardial activation mapping of the right and left ventricles was performed on Langendorff perfused he

281 hearts, ischemic zone territory (34+/-1% of left ventricle) was selected so that ischemia evoked VF

282 antly in the basal inferolateral wall of the left ventricle, was observed in 76% of HIV-infected subj

283 Canine left ventricle wedge preparations at 1) control (36 degr

284 our-chamber sections that covered the entire left ventricle were acquired by using simultaneous multi

285 Detailed bipolar voltage maps of the left ventricle were acquired with the use of NavX.

286 rs old), visually identified areas of LGE in left ventricle were analyzed quantitatively for intermed

287 adial, circumferential, longitudinal) of the left ventricle were analyzed using DRA on steady-state f

288 ntricle (RV) body, outflow tract (RVOT), and left ventricle were calculated and analyzed at baseline

289 Regions of interest for TA encompassing the left ventricle were drawn by two blinded, independent re

290 Peak strain and strain rate values of the left ventricle were not significantly different; however

291 epicardially, and epicardial mapping of the left ventricle were performed in all patients during sin

292 opposing myocardial surfaces (in septum and left ventricle), which rapidly closed down excitable gap

293 gy phosphate metabolism in the human cardiac left ventricle, which may lead to a decline in diastolic

294 njected subepicardially into the anterobasal left ventricle with 40 to 75 rhythmically contracting em

295 farction underwent sequential mapping of the left ventricle with both catheters.

296 tion of VA from the papillary muscles of the left ventricle with either cryoenergy or radiofrequency.

297 Three-dimensional tractograms of the left ventricle with no SMS and rate 2 and rate 3 SMS exc

298 plained by secondary causes and a nondilated left ventricle with preserved or increased ejection frac

299 Preterm-born individuals had short left ventricles with small internal diameters and a disp

300 ht ventricle than previously observed in the left ventricle, with potentially clinically significant