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1 or progressive symptomatic heart failure and left ventricular dilation.
2 aortic regurgitation complicated by extreme left ventricular dilation.
4 tration of an MMP inhibitor attenuates early left ventricular dilation after experimental MI in mice.
5 ensitized mice to cardiac decompensation and left ventricular dilation after long-term stimulation by
7 raphic analysis demonstrated the presence of left ventricular dilation and decreased systolic functio
8 farction with 3 months follow-up resulted in left ventricular dilation and dysfunction in both wild-t
11 ressure 85+/-1 versus 66+/-2 mm Hg; P<0.01), left ventricular dilation and hypertrophy (mass/body wei
13 fold more than wild type, along with greater left ventricular dilation and increased fibrosis, apopto
15 r changes were associated with reductions in left ventricular dilation and left ventricular mass meas
17 asis during atrial arrhythmia are those with left ventricular dilation and low atrial ejection fracti
19 ion < 3+, fibrillation (vs. type I flutter), left ventricular dilation and mitral valve area < 2.0 cm
21 Thallium lung-to-myocardial ratio (L/M), left ventricular dilation and perfusion defect site were
24 ardiography of homozygous mutant mice showed left ventricular dilation and reduced contractile functi
25 isolated myocytes was depressed and preceded left ventricular dilation and reduced fractional shorten
26 may also enhance late survival by preventing left ventricular dilation and reducing arrhythmias, inde
27 trials of afterload reduction for preventing left ventricular dilation and symptom onset in aortic re
31 e-dependent increase in cardiac hypertrophy, left ventricular dilation, and adverse left ventricular
32 eft ventricular ejection fraction <40%, mild left ventricular dilation, and no symptoms of heart fail
33 also developed greater cardiac hypertrophy, left ventricular dilation, and reduced contractile funct
34 higher rates of cardiac rupture, more severe left ventricular dilation, and suppressed ejection fract
35 ith vascular-specific expression (vGOF) show left ventricular dilation as well as less-markedly incre
36 with db/db animals, dbTSP mice had increased left ventricular dilation associated with mild nonprogre
38 r age, congestive heart failure, and greater left ventricular dilation at diagnosis were independentl
39 ce could not be attributed to differences in left ventricular dilation because end-diastolic volumes
41 -)) mice, loss of cMyBP-C has been linked to left ventricular dilation, cardiac hypertrophy, and syst
42 ddle age, heterozygous individuals developed left ventricular dilation, contractile dysfunction, and
43 ed a decline in cardiac function, attenuated left ventricular dilation, decreased infarct size, and r
44 e was exacerbated, as indicated by increased left ventricular dilation, decreased ventricular functio
46 mmended in aortic regurgitation with extreme left ventricular dilation (diastolic dimension >/= 80 mm
51 cal heart failure therapy indexed by reduced left ventricular dilation, improved left ventricular eje
52 phy up to day 56 after MI revealed increased left ventricular dilation in CD4 KO compared with WT mic
53 onstriction (TAC) for 6 weeks caused greater left ventricular dilation in G6PDX mice than wild-type m
54 ocardiography showed significantly increased left ventricular dilation in male IL-13(-/-) compared wi
57 aled severe cardiac abnormalities, including left ventricular dilation, left ventricular mass reducti
61 entricular ejection fraction (p = 0.006) and left ventricular dilation (p = 0.015) at the follow-up e
62 (diastolic dimension >/= 80 mm), but extreme left ventricular dilation raises concern about irreversi
63 Dilated cardiomyopathy is characterised by left ventricular dilation that is associated with systol
64 ed a rapidly progressive cardiomyopathy with left ventricular dilation, wall thinning, and reduced sy
68 ardial infarction (MI) developed progressive left ventricular dilation with dysfunction and HF at 4 w
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