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1 lar density, reduced fibrosis, and decreased left ventricular end-diastolic pressure.
2 a normal ejection fraction, and an increased left ventricular end-diastolic pressure.
3 ested by an early and more rapid increase in left ventricular end-diastolic pressure.
4 ulmonary capillary wedge pressure (PCWP) and left ventricular end-diastolic pressure.
5 ecrease of +dP/dt but a significantly higher left ventricular end-diastolic pressure.
6 volume (16 +/- 4%), and increased transmural left ventricular end-diastolic pressure (139 +/- 6%).
7 +/- 19 versus 42 +/- 15 mm Hg (p = 0.07) and left ventricular end-diastolic pressure 14 +/- 5 versus
8 ecovery; P<0.05) and diastolic function (eg, left ventricular end-diastolic pressure 23+/-9 in WT and
9 ugh the rats with heart failure had elevated left ventricular end-diastolic pressures (24.1 +/- 2.6 m
10 ed diastolic abnormalities, including higher left ventricular end-diastolic pressures (24.3+/-4.6 ver
11 of epicardial coronary arteries and elevated left ventricular end-diastolic pressure (7.7+/-0.3 to 19
12 perfusion gradient in patients with elevated left ventricular end-diastolic pressure, a finding large
13 xperiments, CsA blocked neither the elevated left ventricular end-diastolic pressures, a measure of d
14 increases in coronary perfusion pressure and left ventricular end-diastolic pressure and a decrease i
15 on, rats with infarcts >35% had an increased left ventricular end-diastolic pressure and a marked inc
16 hearts treated with 80 micromol/L diazoxide, left ventricular end-diastolic pressure and coronary flo
17 antly, intracoronary cardiospheres decreased left ventricular end-diastolic pressure and increased ca
18 3 also improved diastolic function, lowering left ventricular end-diastolic pressure and increasing t
19 on show significant inverse relationships to left ventricular end-diastolic pressure and infarct size
20 pigs developed HF as evidenced by increased left ventricular end-diastolic pressure and left ventric
21 croembolization-induced HF, CXL-1020 reduced left ventricular end-diastolic pressure and myocardial o
22 unction gain between beat-to-beat changes in left ventricular end-diastolic pressure and SV was used
23 e volume and cardiac output and decreases in left ventricular end-diastolic pressure and systemic vas
24 t ventricular pressure increase at 40 mm Hg, left ventricular end-diastolic pressure, and cardiac ind
25 by CMR strongly correlated with PH severity, left ventricular end-diastolic pressure, and left ventri
26 d, lower plasma norepinephrine levels, lower left ventricular end-diastolic pressure, and lower right
27 died by measuring pulmonary artery pressure, left ventricular end-diastolic pressure, and lower thora
28 sus controls, was not a function of elevated left ventricular end diastolic pressure but was associat
29 ignificantly (p < .05) attenuated transmural left ventricular end-diastolic pressure by 30% to 40%, l
30 ntilation (either mode) decreased transmural left ventricular end-diastolic pressure by 40% to 60% (p
31 r function in the controlled delivery group (left ventricular end-diastolic pressure, cardiac index,
32 ficantly faster rate of relaxation and lower left ventricular end diastolic pressure compared with co
34 fficacy of a new fluid protocol based on the left ventricular end-diastolic pressure for the preventi
35 tral prosthesis were as follows: "a" wave to left ventricular end-diastolic pressure gradient 17 +/-
36 art failure as defined by clinical signs and left ventricular end diastolic pressures > 25 mm Hg.
38 r end-diastolic pressure into postcapillary (left ventricular end-diastolic pressure, >15 mm Hg; n=26
40 occurred less frequently in patients in the left ventricular end-diastolic pressure-guided group (6.
41 s) were randomly allocated in a 1:1 ratio to left ventricular end-diastolic pressure-guided volume ex
42 tor blockade and amlodipine improved in vivo left ventricular end-diastolic pressure in association w
43 ne levels because of increases in transmural left ventricular end-diastolic pressure in both heart co
45 rats (11.5 +/- 1.8 mm Hg) and was similar to left ventricular end-diastolic pressure in the sham-oper
47 atients with PH were further dichotomized by left ventricular end-diastolic pressure into postcapilla
48 pEF, patients with HTN(+)HFpEF had increased left ventricular end-diastolic pressure, left atrial vol
49 , >15 mm Hg; n=269) and precapillary groups (left ventricular end-diastolic pressure, </=15 mm Hg; n=
50 erved an approximately threefold increase in left ventricular end diastolic pressure (LVEDP) and 38%
52 epicardial lidocaine paradoxically decreased left ventricular end-diastolic pressure (LVEDP) and left
53 ic cardioplegic arrest led to an increase of left ventricular end-diastolic pressure (LVEDP) by > or
55 t between diastolic blood pressure (DBP) and left ventricular end-diastolic pressure (LVEDP) to systo
56 hanges in coronary perfusion pressure (CPP), left ventricular end-diastolic pressure (LVEDP), and dev
57 addition, intracoronary enalaprilat reduced left ventricular end-diastolic pressure (LVEDP), but not
58 io (LHR) is a traditional marker of elevated left ventricular end-diastolic pressure (LVEDP), which a
59 on and resulted in increased DCS (isovolumic left ventricular end-diastolic pressure [LVEDP] increase
60 eperfusion injury, indicated by an increased left ventricular end diastolic pressure, myocardial crea
61 , patients with HTN(-)HFpEF had no change in left ventricular end-diastolic pressure, myocardial pass
62 romised myocardial performance with elevated left ventricular end-diastolic pressure, not seen in the
64 No difference was observed among groups for left ventricular end-diastolic pressures or dimensions,
66 postprocedural pulmonary artery systolic and left ventricular end-diastolic pressures (p < 0.01).
67 increase and declines in blood pressure and left ventricular end-diastolic pressure produced by GTN
68 usion gradient was inversely associated with left ventricular end-diastolic pressure (r=-0.728; P<0.0
69 were instrumented and the heart paced until left ventricular end-diastolic pressure reached 25 mm Hg
70 nclude left ventricular dilitation, elevated left ventricular end-diastolic pressure, redo coronary s
72 ntricular volumes, diastolic relaxation, and left ventricular end-diastolic pressures stabilized in t
74 e rate of myocardial relaxation and lowering left ventricular end diastolic pressure to facilitate ve
76 d-stage failure, occurring at 29+/-1.6 days, left ventricular end-diastolic pressure was 25+/-1 mm Hg
80 cardial function (ejection fraction [EF] and left ventricular end-diastolic pressure) was assessed at
81 ference (diastolic pulmonary artery pressure-left ventricular end-diastolic pressure) was normal (<7
83 ody weight, right ventricle/body weight, and left ventricular end-diastolic pressure were increased a
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