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1 25), including after adjustment for baseline left ventricular end-diastolic volume.
2 defined as an increase of 15% or more in the left ventricular end-diastolic volume.
3 tolic dysfunction by reducing the unstressed left ventricular end-diastolic volume.
4  Greater mean LVMT was associated with lower left ventricular end-diastolic volume (0.01 mm/mL; P<0.0
5 ic response (defined as percent reduction in left ventricular end-diastolic volume 1 year after CRT-D
6 se in stroke volume (SV) without a change in left ventricular end diastolic volume (102+/-16% change
7 me (10.4 +/- 2 to 7.7 +/- 4 mL; p < .05) and left ventricular end-diastolic volume (18.5 +/- 3 to 14.
8 ared with placebo, a significant decrease in left ventricular end-diastolic volume (-18 mL; P=0.009)
9 ng as shown by increased E-wave velocity and left ventricular end-diastolic volume, 2) exhibit a high
10  left ventricular remodeling (>20% change in left ventricular end-diastolic volume; 21.91 [2.75-174.2
11 -diastolic pressure by 40% to 60% (p < .05), left ventricular end-diastolic volume 25 +/- 8%, and str
12 hysema was linearly related to reductions in left ventricular end-diastolic volume (-4.1 ml; 95% conf
13  significantly (P<0.05) greater reduction in left ventricular end-diastolic volume (-49+/-16% versus
14                                              Left ventricular end-diastolic volume (72.5 to 77.4 mL/m
15 ated with change in RV ejection fraction and left ventricular end-diastolic volume, although correlat
16                         On echocardiography, left ventricular end diastolic volume and deceleration t
17 icity was evaluated as the change in indexed left ventricular end diastolic volume and LVEF.
18 d venodilation reflected by a 10% decline in left ventricular end-diastolic volume and a 30% fall in
19 raction compared with placebo did not change left ventricular end-diastolic volume and left ventricul
20  Secondary end points, including structural (left ventricular end-diastolic volume and left ventricul
21                                      Indexed left ventricular end-diastolic volume and mass correlate
22  1 and 10 weeks after infarction showed that left ventricular end-diastolic volume and mass increased
23 +/-7 and 24+/-5 g/m(2); P<0.001) and indexed left ventricular end-diastolic volume and right ventricu
24 ality-of-life score, 6-minute walk distance, left ventricular end-diastolic volume, and left ventricu
25 d rats showed a borderline (16%) increase in left ventricular end-diastolic volume (angiography), whe
26          The limits of agreement between the left ventricular end-diastolic volume as estimated by bi
27 r ejection fraction (beta=-0.02/%; P=0.015), left ventricular end-diastolic volume (beta=0.01/mL; P<0
28  and right ventricular end-diastolic volume (left ventricular end-diastolic volume/body surface area,
29 icular end-diastolic pressure by 30% to 40%, left ventricular end-diastolic volumes by 33 +/- 9%, and
30      Pulmonary capillary wedge pressures and left ventricular end-diastolic volumes by use of echocar
31       The relation between stroke volume and left ventricular end-diastolic volume, by the Frank-Star
32 dence interval, -4.02 to -0.23; P=0.028) and left ventricular end-diastolic volume (coefficient, 7.85
33 ular end-diastolic pressure (LVEDP), but not left ventricular end-diastolic volume, consistent with i
34                                              Left ventricular end-diastolic volume decreased (from 12
35                  Cardiac index increased and left ventricular end-diastolic volume decreased signific
36 al clipping led to a significant increase in left ventricular end-diastolic volume, demonstrating inc
37                           The mean change in left ventricular end-diastolic volume differed significa
38                       Patients who increased left ventricular end-diastolic volume during lower-body
39 ic resonance imaging demonstrated increasing left ventricular end-diastolic volumes, end-systolic vol
40 ds, prior hospitalization for heart failure, left ventricular end-diastolic volume &gt;/=125 mL/m(2), an
41 rse remodeling was defined as an increase in left ventricular end-diastolic volume &gt;/=20% at 6 months
42                                  CMR-derived left ventricular end-diastolic volume &gt;246 mL had good,
43 icular pressure increase and dilatation, but left ventricular end-diastolic volume improved because o
44                                              Left ventricular end-diastolic volume improved from 172
45 atients, there was a paradoxical increase in left ventricular end-diastolic volume in association wit
46 by 30 mm Hg lower-body suction, on right and left ventricular end-diastolic volumes in 21 patients wi
47 +/-33 to 43+/-14 mL/m(2), P<0.0001), whereas left ventricular end diastolic volume increased (66+/-12
48      After 17 cycles of trastuzumab, indexed left ventricular end diastolic volume increased in patie
49                                              Left ventricular end-diastolic volume increased in the r
50 urviving without surgery at 5 years, 90% for left ventricular end-diastolic volume index <100 mL/m(2)
51 RT-plus-ICD group had greater improvement in left ventricular end-diastolic volume index (-26.2 versu
52 plasma brain natriuretic peptide (79 pg/mL), left ventricular end-diastolic volume index (110 mL/m2),
53  did not correlate with six-month changes in left ventricular end-diastolic volume index (p = 0.26),
54 iography showed a small, stable reduction in left ventricular end-diastolic volume index (P<0.001), w
55 ely), RV stroke volume index (P<0.0001), and left ventricular end-diastolic volume index (P=0.0015).
56 ilure 3.2 +/- 2.9 years, LVEF 0.21 +/- 0.07, left ventricular end-diastolic volume index 180 +/- 64 m
57                     The relationship between left ventricular end-diastolic volume index and stroke v
58                                          The left ventricular end-diastolic volume index decreased (9
59 ex, pulmonary artery occlusion pressure, and left ventricular end-diastolic volume index failed to co
60                                      Resting left ventricular end-diastolic volume index increased (P
61 ors of SCA were PET sympathetic denervation, left ventricular end-diastolic volume index, creatinine,
62 vated B-type natriuretic peptide, and larger left ventricular end-diastolic volume index.
63  larger left atrial volume index, and larger left ventricular end-diastolic volume index.
64 farct zone and its percentage of myocardium, left ventricular end-diastolic volume, left ventricular
65 myocardial scar volume by CMR is superior to left ventricular end-diastolic volume, left ventricular
66 gnificant improvements in ejection fraction, left ventricular end-diastolic volume, left ventricular
67 eft ventricular ejection fraction (LVEF) and left ventricular end diastolic volume (LVEDV) on cardiac
68 antagonist tolvaptan (30 mg/day) on reducing left ventricular end-diastolic volume (LVEDV) compared w
69                 Gated SPECT measurements for left ventricular end-diastolic volume (LVEDV) index, lef
70 lmonary capillary wedge pressure (PCWP), SV, left ventricular end-diastolic volume (LVEDV), and left
71                                              Left ventricular end-diastolic volume (LVEDV), left vent
72 ponse with CRT, defined as percent change in left ventricular end-diastolic volume (LVEDV), was analy
73  of left ventricular end-systolic volume and left ventricular end-diastolic volume (LVEDV).
74 odds ratio, 1.16; P=0.002), exercise indexed left ventricular end-diastolic volume (odds ratio, 1.04;
75 ients) was the only predictor of increase in left ventricular end-diastolic volume (p < 0.001).
76 cular end-diastolic volume, as expected, but left ventricular end-diastolic volume paradoxically incr
77 ntricular end-diastolic pressure (LVEDP) and left ventricular end-diastolic volume (preload) in CHF r
78 ce method (n=53, r=0.85, P<0.0001), and with left ventricular end-diastolic volume (r=0.81, P<0.0001)
79 ctron-beam computed tomography for measuring left ventricular end-diastolic volume (r=0.96; standard
80 ts displayed an additional decline in the RV/left ventricular end-diastolic volume ratio (P=0.05) and
81 airflow obstruction are inversely related to left ventricular end-diastolic volume, stroke volume, an
82 ing hearts increased relaxation velocity and left ventricular end diastolic volume to produce higher
83 ena caval occlusion (VCO) was used to reduce left ventricular end-diastolic volume to 70 +/- 5% of ba
84 ng was minor, and a decrease in the ratio of left ventricular end-diastolic volume to body weight ref
85 c volume (angiography), whereas the ratio of left ventricular end-diastolic volume to body weight was
86                                     The mean left ventricular end-diastolic volume was 108 +/- 47 mL,
87       At 6 months follow-up, left atrial and left ventricular end-diastolic volumes were significantl
88 hat heat stress did not significantly change left ventricular end-diastolic volume, while ventricular
89 n the most recent echocardiogram, the median left ventricular end-diastolic volume z score was +1.7 (
90  dimension z-score (OR=2.2, P=0.02) or lower left ventricular end-diastolic volume z-score (OR=1.9, P

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