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1 the heart with a significant decrease in the left ventricular fractional shortening.
2 rdiomyocyte cell size, and modestly impaired left ventricular fractional shortening.
3 f the FHC mice, with a significant change in left ventricular fractional shortening.
4 ar functions, such as peak filling rates and left ventricular fractional shortening.
5 for children who received doxorubicin alone (left ventricular fractional shortening: -0.82, 95% CI -1
6  end-diastolic z-scores (+4.2 vs. +4.2), and left ventricular fractional shortening (16% vs. 17%) at
7 ved cardiac contractile function measured as left ventricular fractional shortening (30 +/- 3% Galpha
8 rcoglycan-null mice showed an improvement in left ventricular fractional shortening (44.3 +/- 13.3% t
9 atrial dimension, left ventricular mass, and left ventricular fractional shortening (adjusted HR, 1.2
10 completion of doxorubicin chemotherapy, mean left ventricular fractional shortening and end-systolic
11                             Vegetation size, left ventricular fractional shortening, and onset of aor
12 s showed dexrazoxane protection (p=0.04) for left ventricular fractional shortening at 5 years in gir
13                 HF resulted in a decrease in left ventricular fractional shortening compared with con
14 iated with significant abnormalities in mean left ventricular fractional shortening, end-diastolic di
15                        Doxorubicin decreased left ventricular fractional shortening from 57+/-2% to 4
16 ebrate rats with myocardial infarction (MI) (left ventricular fractional shortening (FS) = 24 +/- 1%;
17 om presentation to the echocardiogram before left ventricular fractional shortening (FS) improved to
18 iography showed a significant improvement of left ventricular fractional shortening in the minicircle
19 ngham Heart Study population, mildly reduced left ventricular fractional shortening (<30%) was more c
20 rements were abnormal at baseline (decreased left ventricular fractional shortening [LV FS] and contr
21  presence of congestive heart failure, lower left ventricular fractional shortening, or higher left v
22  wild-type mice, LPS significantly decreased left ventricular fractional shortening, velocity of circ
23 otoxin challenge in wild-type mice decreased left ventricular fractional shortening, velocity of circ
24                                     The mean left ventricular fractional shortening, wall thickness,
25        Using transthoracic echocardiography, left ventricular fractional shortening was 47+/-2%, 44+/
26                                              Left ventricular fractional shortening was quantified by
27                                              Left ventricular fractional shortening was significantly
28 icular fractional shortening Z score, higher left ventricular fractional shortening Z score during fo
29 sk of death or transplantation, as was lower left ventricular fractional shortening Z score during fo
30  familial cardiomyopathy, and lower baseline left ventricular fractional shortening Z score were asso
31 e older age, congestive heart failure, lower left ventricular fractional shortening Z score, and caus
32 Normalization was related to higher baseline left ventricular fractional shortening Z score, higher l
33 during follow-up, and greater improvement in left ventricular fractional shortening Z score.
34 ersus 78%, P<0.001), had less-depressed mean left ventricular fractional shortening z scores (-7.85+/

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