ライフサイエンスコーパス: left ventricular function

1 dity and mortality in patients with impaired left ventricular function.

2 al teaching of cardiac physiology focuses on left ventricular function.

3 independent of the concurrent improvement in left ventricular function.

4 ICD implantation on the basis of symptoms or left ventricular function.

5 ich improved in parallel with improvement in left ventricular function.

6 patients with symptoms at rest and impaired left ventricular function.

7 ism type 1, but rather an improved diastolic left ventricular function.

8 xtensive coronary artery disease or impaired left ventricular function.

9 h image acquisition plane, demographics, and left ventricular function.

10 ts are correlated with sensitive measures of left ventricular function.

11 upling, decreases infarct size, and improves left ventricular function.

12 sis in the noninfarcted region, and improved left ventricular function.

13 reat acute myocardial infarction and improve left ventricular function.

14 who had stable heart failure with depressed left ventricular function.

15 creased risk for sudden death from depressed left ventricular function.

16 yocardial infarction patients with preserved left ventricular function.

17 l ventricular remodeling and improved global left ventricular function.

18 ischemia and ventricular arrhythmias, and on left ventricular function.

19 tion (AMI) has led to limited improvement in left ventricular function.

20 from AC6 deletion underlies abnormalities in left ventricular function.

21 anges curbed infarct expansion and preserved left ventricular function.

22 esolution of pulmonary edema and recovery of left ventricular function.

23 coronary artery disease, angina, and normal left ventricular function.

24 elated to the severity of CAD and diminished left ventricular function.

25 ng/body weight ratios, but no improvement in left ventricular function.

26 % of patients with in-hospital assessment of left ventricular function.

27 unction assessed, 21,149 (51%) had preserved left ventricular function.

28 ntricular ectopy results in normalization of left ventricular function.

29 a previous myocardial infarction and reduced left ventricular function.

30 sepsis-related lactic acidosis, and improved left ventricular function.

31 c variables but have not considered baseline left ventricular function.

32 evaluate right ventricular pacing effects on left ventricular function.

33 No changes were observed in left ventricular function.

34 bined assessment of myocardial perfusion and left ventricular function.

35 tion of ventilation, duration of bypass, and left ventricular function.

36 t of cardiac and great vessel morphology and left ventricular function.

37 stained ventricular tachycardia, and reduced left ventricular function.

38 ical death rates of CAD patients with normal left ventricular function.

39 ssociated enhancement of regional and global left ventricular function.

40 tients with coronary artery disease and poor left ventricular function.

41 ntricular remodeling and regional and global left ventricular function.

42 not adequately address the effect of PVCs on left ventricular function.

43 r relationship with age, sex, body size, and left ventricular function.

44 e to sudden death, and in patients with poor left ventricular function.

45 r interstitial fibrosis, leading to improved left ventricular function.

46 he degradation pattern differed depending on left ventricular function.

47 ffect blood pressure, systolic, or diastolic left ventricular function.

48 te mortality, imparting a negative impact on left ventricular function.

49 with recent-onset heart failure and reduced left ventricular function.

50 t, in terms of clinical events or changes in left ventricular function.

51 lecting both improved systolic and diastolic left ventricular function.

52 mal experimental studies metformin preserves left ventricular function.

53 reverse remodeling without adverse effect on left ventricular function.

54 luate predictors of recurrence and impact on left ventricular function.

55 response to post-acute myocardial infarction left ventricular functions.

56 ed as the gold standard in the evaluation of left ventricular functions.

57 nt MR software programs were used to compare left ventricular functions.

58 the left ventricle and reduced impairment of left ventricular function 2 weeks after I/R injury.

59 much weaker in severe CHF compared to normal left ventricular function; 2) increasing HRR using rate-

60 n in cultured endothelial cells, and improve left ventricular function after global ischemia and repe

61 n scar formation, thereby augmenting cardiac left ventricular function after ischemia/reperfusion.

62 e in humans that myocardial infarct size and left ventricular function after STEMI have a circadian d

63 sis, a large infarct area, and a decrease in left ventricular function (all, p < 0.01 vs. sham contro

64 diographic assessment of myocardial mass and left ventricular function also did not suggest myocardia

65 FOG-2(R3K5A) mice leads to an improvement in left ventricular function and a partial rescue of left v

66 use a state of lipotoxicity that compromises left ventricular function and aggravate heart failure.

67 ouse myocardial infarction model can improve left ventricular function and attenuate cardiac remodeli

68 k region were accompanied by improvements in left ventricular function and attenuation of left ventri

69 ls, cardiopoietic stem cell therapy improves left ventricular function and blunts pathological remode

70 nts with recent-onset heart failure, reduced left ventricular function and clinically suspected myoca

71 For patients in cardiogenic shock, left ventricular function and culprit vessel were indepe

72 ecurrent tachycardia causes rapid decline in left ventricular function and development of heart failu

73 CVB3-infected mice developed an impaired left ventricular function and displayed an increased lef

74 of left ventricular volumes, mass, regional left ventricular function and dyssynchrony.

75 cts of iC-BCP were associated with preserved left ventricular function and endothelium-dependent rela

76 timated dopamine pharmacokinetics as well as left ventricular function and global hemodynamics after

77 n limited myocardial infarct size, preserved left ventricular function and had no influence on haemod

78 myocardial infarction complicated by reduced left ventricular function and HF.

79 ited more adverse cardiac remodeling, poorer left ventricular function and higher mortality by increa

80 2 years vs. 52.3 years), had worse right and left ventricular function and higher pulmonary artery pr

81 ement of inotropic and lusitropic effects on left ventricular function and improved cardiac output.

82 at significant cardiovascular alterations in left ventricular function and in aortic stiffness occur

83 s and is associated with early impairment of left ventricular function and increased myocardial oxida

84 been beneficial for adult patients with poor left ventricular function and intraventricular conductio

85 Regional left ventricular function and late-gadolinium enhancemen

86 genic mice, Tg-GSK-3beta-DN exhibited better left ventricular function and less fibrosis and apoptosi

87 on deficit in human HCM with normal systolic left ventricular function and may contribute to HCM dise

88 mbolic complications; and (5) stable, normal left ventricular function and New York Heart Association

89 mouse IL-10 administration markedly improved left ventricular function and not only inhibited but als

90 In isolated perfused heart experiments, left ventricular function and oxygen consumption were si

91 ents with unexplained cardiac arrest (normal left ventricular function and QT interval) and selected

92 t failure (HF) therapy documented to improve left ventricular function and reduce mortality.

93 ion, reduce its hemodynamic repercussions on left ventricular function and remodeling, and improve cl

94 ssessed by echocardiographic measurements of left ventricular function and structure.

95 er time, whereas other risk factors, such as left ventricular function and surgery on the thoracic ao

96 It also improves left ventricular function and survival in mice (n=20-22

97 yte apoptosis by caspase inhibition improved left ventricular function and survival in pregnant Galph

98 her inhibiting this cell death would improve left ventricular function and survival in the Galpha(q)

99 trates that metformin significantly improves left ventricular function and survival via activation of

100 duced cardiomyocyte hyperplasia had improved left ventricular function and survival.

101 ardiovascular events in women with preserved left ventricular function and suspected myocardial ische

102 UcnII and CRFR2 deficiency on left ventricular function and the systemic vasculature.

103 th a particular focus on those with improved left ventricular function and those who have not experie

104 Secondary end points were left ventricular function and volumes by magnetic resona

105 cardiography was performed for assessment of left ventricular function and wall motion index (WMI).

106 vascularization priority, diabetes mellitus, left ventricular function, and 3-vessel disease.

107 clinical variables, perfusion defect scores, left ventricular function, and absolute myocardial flow

108 k Heart Association functional class 3, poor left ventricular function, and age (all p < 0.001) along

109 died, and the remaining patients had normal left ventricular function, and all but 1 have New York H

110 icity, sex, comorbidities, insurance status, left ventricular function, and aortic stenosis severity

111 toperative cine MRI for assessment of global left ventricular function, and contrast-enhanced CMRI fo

112 to account the angiographic severity of CAD, left ventricular function, and exercise capacity, HRR is

113 ssion attenuates cardiac fibrosis, preserves left ventricular function, and improves survival in a mo

114 atched with regard to hemodynamic variables, left ventricular function, and infarct size.

115 transvenous dual-chamber pacemakers, normal left ventricular function, and intact atrioventricular (

116 d similar improvements in clinical symptoms, left ventricular function, and left ventricular reverse

117 , cardiovascular parameters (carotid plaque, left ventricular function, and myocardial late gadoliniu

118 ents with coronary artery disease, depressed left ventricular function, and nonsustained ventricular

119 total of 74 patients with known CAD, normal left ventricular function, and normal resting levels of

120 hyperlipidemia, chronic renal insufficiency, left ventricular function, and plasma F11R levels.

121 goal becomes limiting remodeling, preserving left ventricular function, and preventing heart failure.

122 istensibility by high-resolution ultrasound, left ventricular function, and quality of life were meas

123 assessment reveals normal coronary arteries, left ventricular function, and resting ECG.

124 ll evolution, thromboembolic risk, preserved left ventricular function, and severely decreased levels

125 actors in women, gender-specific measures of left ventricular function, and the relationship between

126 mptoms, quality of life, exercise tolerance, left ventricular function, and the survival of patients

127 Hemodynamic variables, indices of left ventricular function, and tumor necrosis factor-alp

128 P-2 precommitted mES cells, improved post-MI left ventricular functions, and enhanced capillary densi

129 iac magnetic resonance imaging and change in left ventricular function as affected by timing of treat

130 HIV-infected patients showed alterations in left ventricular function as demonstrated by a lower eje

131 ed animals recovered 85.4% of their baseline left ventricular function as measured by 2-dimensional t

132 The left ventricular function as measured by two-dimensional

133 BMCs; secondary end points included regional left ventricular function assessed by magnetic resonance

134 Of the 41,267 patients with left ventricular function assessed, 21,149 (51%) had pre

135 versus 10.9+/-2.3 cm/s; P=0.01) and regional left ventricular function, assessed by peak systolic vel

136 converting enzyme inhibitor prescription and left ventricular function assessment; no other HF qualit

137 IGF-1/HGF reduced infarct size and improved left ventricular function at 2 months after AMI.

138 Left ventricular function at 30 days was the same in the

139 and iPSC-EV-treated mice exhibited improved left ventricular function at 35 d after myocardial infar

140 m to determine the effects of this system on left ventricular function at 4 months after AMI.

141 ll), which translated into impaired systolic left ventricular function at 6 to 12 months post TAVR (p

142 s postreperfusion and significantly improved left ventricular function at 7 days after myocardial isc

143 ith new LE feature a significant decrease in left ventricular function at discharge.

144 me to return of spontaneous circulation, and left ventricular function at return of spontaneous circu

145 ventricular dilatation and better-preserved left ventricular function at up to 8 weeks post-MI despi

146 /-0.8 mm Hg/mL/m(2)), and worse longitudinal left ventricular function (basal longitudinal strain=-11

147 significant difference in global or regional left ventricular function between groups.

148 sonance imaging data showed no difference in left ventricular function but more RV dilation among PVR

149 myocardial infarction with severely impaired left ventricular function but no inducible ventricular t

150 3D-ST) has been used extensively to quantify left ventricular function, but its value for RV assessme

151 ere was an increase in frequency of impaired left ventricular function, but there was no change in Lo

152 nt in DM2 is already detectable in preserved left ventricular function by cardiovascular magnetic res

153 d by Doppler-derived pulse-wave velocity and left ventricular function by echocardiography.

154 d infarct size, microvascular perfusion, and left ventricular function by histopathology, microsphere

155 the ability of echocardiography to evaluate left-ventricular function by improving delineation of th

156 ve also been observed in the manner in which left ventricular function changes across the lifespan, w

157 ) subjects underwent assessment of diastolic left ventricular function (cine magnetic resonance imagi

158 ds that is incremental to clinical, ECG, and left ventricular function combined.

159 xhibited a significantly greater increase in left ventricular function compared with all the other gr

160 reased valve-related morbidity, and improved left ventricular function compared with MVR.

161 ant effect on recovery of global or regional left ventricular function compared with placebo.

162 ymptomatic severe aortic stenosis and normal left ventricular function, current practice guidelines e

163 Although systolic right and left ventricular function declined, peak exercise capaci

164 nsplantation may be associated with improved left ventricular function, decreased N-terminal pro B-ty

165 In diabetic animals, ALT-711 improved left ventricular function, decreased ventricular collage

166 rtery disease and normal or slightly reduced left ventricular function derive therapeutic benefit fro

167 ation functional class 3 or 30-d death), and left ventricular function did not differ between two gro

168 turn plays an essential role in maintaining left ventricular function during starvation.

169 OPCABG results in significantly better left ventricular function early after surgery but does n

170 scitation results in worse postresuscitation left ventricular function early but did not compromise 2

171 Left ventricular function (echocardiography), clinical s

172 Left ventricular function, edema, vascular permeability,

173 Importantly, SIM improved left ventricular function (EF, 32.4+/-4.1% to 51.7+/-3.2

174 moderate aortic regurgitation and preserved left ventricular function (ejection fraction >/=55%).

175 ase, and a nontransfemoral approach; whereas left ventricular function (ejection fraction <30%), the

176 Patients with new LE had a decreased left ventricular function (ejection fraction: pre, 55.5

177 raphics, risk factors, coronary anatomy, and left ventricular function, end-systolic volume index and

178 s like acute cor pulmonale determination and left ventricular function estimation.

179 to rupture and the significant reduction in left ventricular function evident as early as 1 week pos

180 peremia was associated with reduced regional left ventricular function expressed as lower Ecc in the

181 myocardial infarct size by 45%, and improved left ventricular function (first derivative of pressure

182 species generation, myocardial necrosis, and left ventricular function following ischemia/reperfusion

183 significant differences in change in global left ventricular function for patients treated at day 3

184 al and demographic predictors of recovery of left ventricular function for subjects with recent onset

185 The ability to record changes in left ventricular function from rest to peak stress and t

186 Improvements in left ventricular function, functional status, and qualit

187 the timing of cell delivery on the effect on left ventricular function has not been analyzed.

188 patients with heart failure (HF) who recover left ventricular function (HF-Recovered) have a distinct

189 Left ventricular function, histochemical injury, and dif

190 However, activity reduced left ventricular function (ie, ejection and shortening f

191 -flow ischemia and reperfusion, postischemic left ventricular function improved commensurately with a

192 ic resonance imaging was performed to assess left ventricular function in 20 women with signs and sym

193 y, decreases apoptosis, and greatly enhances left ventricular function in a model of ischemic cardiom

194 of endothelial progenitor cells and improves left ventricular function in a rat model of myocardial i

195 relationship between Hcy levels and regional left ventricular function in an asymptomatic population.

196 ically managed aortic stenosis and preserved left ventricular function in an era where surgical aorti

197 sed-chest hemodynamic assays revealed normal left ventricular function in beta-isoproterenol-stimulat

198 This resulted in preservation of left ventricular function in both acute and chronic myoc

199 T cells was correlated with the decrease in left ventricular function in CHD patients (r=0.629, P=0.

200 eukin-1 receptor antagonist, on coronary and left ventricular function in coronary artery disease (CA

201 eactivity of micro- and macrocirculation and left ventricular function in diabetic patients.

202 has no beneficial effects on endothelial or left ventricular function in diabetic patients.

203 In particular, stress exercise disturbs left ventricular function in dysferlin-null mice and inc

204 in global (LVEF) and regional (wall motion) left ventricular function in infarct and border zones at

205 ynchronization reduces symptoms and improves left ventricular function in many patients with heart fa

206 Tissue Doppler showed better diastolic left ventricular function in mutation carriers (e', P=0.

207 ession and CVB3 copy number, and an improved left ventricular function in NOD2(-/-) CVB3 mice compare

208 hese data also demonstrate that SIM improves left ventricular function in pacing-induced CHF rabbits.

209 free fatty acids and insulin sensitivity on left ventricular function in patients with clinically se

210 (Evaluating Xience and Left Ventricular Function in Percutaneous Coronary Inter

211 The EXPLORE (Evaluating Xience and Left Ventricular Function in Percutaneous Coronary Inter

212 ted information exists regarding the role of left ventricular function in predicting exercise capacit

213 (CPCs) 4 hours after reperfusion ameliorates left ventricular function in rats with acute myocardial

214 The full scorpion venom increased left ventricular function in sedated mice in vivo, prolo

215 ng with determination of regional and global left ventricular function in the same examination.

216 t orifice area, 38+/-18 mm(2)) and preserved left ventricular function in whom initial management was

217 used loss of myocardial tissue and depressed left ventricular function in WT mice.

218 Indices of left ventricular function, including ejection fraction a

219 chocardiography revealed improved indices of left ventricular function, including ejection fraction a

220 hase I clinical trials, MSC therapy improved left ventricular function, induced reverse remodeling, a

221 In vivo secretoneurin improved left ventricular function, inhibited remodeling, and red

222 Baseline left ventricular function is a marker for prognosis rega

223 Left ventricular function is a principal determinant of

224 Of note, measurement of baseline left ventricular function is the single most powerful pr

225 rtrophy but exhibited significantly improved left ventricular function, less cardiac fibrosis and car

226 tration of NaHS improved survival, preserved left ventricular function, limited infarct size, and imp

227 of life and exercise capacity and to improve left ventricular function long-term.

228 33% vs. 14%; p = 0.0027, respectively), and left ventricular function </=35% (26% vs. 10%; p = 0.007

229 Left ventricular function <30% was observed in 44.1% of

230 Depressed left ventricular function (LVF) and low heart rate varia

231 for ablation before ICD implantation because left ventricular function may improve, consequently decr

232 In nonsurgical candidates with compromised left ventricular function, MitraClip therapy could be co

233 hics, acuity of presentation, and measure of left ventricular function, models with better discrimina

234 cell transplantation significantly improved left ventricular function, myocardial metabolism, and ar

235 ents with coronary artery disease and normal left ventricular function (n=79).

236 All animals demonstrated vigorous left ventricular function, no outflow tract obstruction,

237 >50% of peripartum cardiomyopathy patients, left ventricular function normalizes with pharmacologic

238 hes (e.g., beta-blockers) partially preserve left ventricular function, novel strategies are needed t

239 no significant treatment effect on regional left ventricular function observed in either infarct or

240 Whether the improved left ventricular function observed in the Aldo-DHF trial

241 (HCRTR2), were less likely to have improved left ventricular function (odds ratio: 0.40 per minor al

242 al status or that the deleterious effects on left ventricular function offset any salutary vascular e

243 he only significant predictor of recovery of left ventricular function on the basis of the percent ch

244 E) inhibitor therapy in patients with normal left ventricular function on the progression of myocardi

245 t data reported no effect of cell therapy on left ventricular function or clinical outcome.

246 jection of autologous BMNCs does not improve left ventricular function or quality of life in patients

247 essel, cardiogenic shock, severely depressed left ventricular function, or ST segment elevation myoca

248 wever, new PPI did have a negative effect on left ventricular function over time.

249 s was the most important factor in improving left ventricular function (p < 0.001) and 30-day combine

250 Worse left ventricular function (P<0.0001), diabetes (P<0.0001

251 less myocardial salvage (P<0.001), impaired left ventricular function (P<0.001), and more pronounced

252 listing status (P<0.0001), severely impaired left ventricular function (P<0.01), pulmonary hypertensi

253 reas placebo pigs had persistently depressed left ventricular function (P<0.05).

254 (MR) imaging findings were used to quantify left ventricular function, perfusion, and infarct size.

255 art markedly limits remodeling and preserves left ventricular function post-MI.

256 stheses may allow for greater improvement in left ventricular function postoperatively.

257 Left ventricular function predicts cardiovascular mortal

258 linical (older age, diabetes, renal disease, left ventricular function, recent myocardial damage, and

259 atment had no significant effect on regional left ventricular function recovery.

260 L-10-treated mice corroborated with improved left ventricular function, reduced infarct size, and fib

261 Left ventricular function, remodeling parameters, and he

262 l prohormone brain natriuretic peptide), and left ventricular function/remodeling (left ventricular e

263 quantify myocardial triglyceride content and left ventricular function, respectively.

264 AFCA provides long-term benefits on left ventricular function, significantly reducing the nu

265 ronary artery disease and severely depressed left ventricular function to intra-aortic balloon pump (

266 oncomitant coronary artery surgery, impaired left ventricular function, urgent or emergency status, p

267 se treadmill testing, in vivo assessments of left ventricular function using Mikro-Tip catheter trans

268 onal activation in CHF patients with reduced left-ventricular function using SPECT to precisely quant

269 ft ventriculography is a method of measuring left ventricular function usually performed at the discr

270 perior cardiac repair in vivo with regard to left ventricular function, vascularization, and ameliora

271 ian follow-up of 36 months, normalization of left ventricular function was achieved in >90% of patien

272 Longitudinal left ventricular function was assessed by 2D-strain and

273 Left ventricular function was assessed by conventional a

274 Left ventricular function was assessed by measuring deve

275 In patients +CAN, left ventricular function was decreased in both diastole

276 Left ventricular function was evaluated using echocardio

277 Left ventricular function was evaluated using transthora

278 The decline in left ventricular function was further supported by signi

279 Diastolic left ventricular function was graded as normal, mild, mo

280 Left ventricular function was impaired (ejection fractio

281 Consequently, infarct size was increased and left ventricular function was impaired 28 days after myo

282 Left ventricular function was measured by Millar pressur

283 Left ventricular function was measured by the pressure-v

284 Left ventricular function was measured continuously over

285 t time (71%); left atrium was 43+/-6 mm; and left ventricular function was normal (59+/-9%).

286 Left ventricular function was normal in >50% of patients

287 A greater improvement in left ventricular function was observed after intramyocar

288 Myocardial perfusion and left ventricular function were also investigated using a

289 aline, and echocardiographic measurements of left ventricular function were greater in the AMD3100-tr

290 lectrolytes, glomerular filtration rate, and left ventricular function were performed on all patients

291 istics, vascular stiffness measurements, and left ventricular function were similar between participa

292 Left ventricular functions were consistently depressed i

293 farct vessel densities, and better preserved left ventricular function when compared with MSCs transf

294 ardiac MRI assessed myocardial viability and left ventricular function, whereas bioluminescence imagi

295 depends on the actual stenosis severity and left ventricular function, which is of prognostic import

296 g a reduced infarct size and preservation of left ventricular function, which suggests a role for thi

297 S100A8 on protein level, showed an improved left ventricular function, which was associated with a r

298 stable coronary heart disease and preserved left ventricular function who are receiving "current sta

299 comparison group of 131 patients with normal left ventricular function who underwent coronary artery

300 dictors for fast-track failure were impaired left ventricular function with or without recent acute c