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1 clusion on the velocity-time integral of the left ventricular outflow tract.
2 ccurately identified the focal origin in the left ventricular outflow tract.
3 obtain velocity information in the aorta and left ventricular outflow tract.
4 e pace mapping was observed in the right and left ventricular outflow tracts.
6 e 3-D Doppler echocardiography images of the left ventricular outflow tract and aortic valve were obt
8 lage introduced reciprocal constraint to the left ventricular outflow tract and mitral annulus that e
12 dictable fashion between the conduit and the left ventricular outflow tract, and AVB surgery reliably
13 volume in mitral valve disease, area of the left ventricular outflow tract, and tricuspid annular ge
15 e the echocardiographic abnormalities of the left ventricular outflow tract associated with subaortic
16 myofibers normally run in parallel along the left ventricular outflow tract, but in the Nkx2-5(+/-)/S
17 tricular septal defect, conotruncal defects, left ventricular outflow tract defect, and right ventric
19 ardiographic studies were performed to image left ventricular outflow tract forward and aortic regurg
20 al and temporal color flow velocity data for left ventricular outflow tract forward flow and ascendin
22 Although the efficacy of both SA and SM in left ventricular outflow tract gradient (LVOTG) reductio
24 hough early reports documented a decrease in left ventricular outflow tract gradient and symptomatic
29 .53; 95% confidence interval, 1.02-2.30) and left ventricular outflow tract gradient progression (haz
30 new therapy that has been shown to result in left ventricular outflow tract gradient reduction and re
34 mitral valve (58%), and, in 11 individuals, left ventricular outflow tract gradients (average, 63+/-
35 rophic cardiomyopathy (HCM) exhibit elevated left ventricular outflow tract gradients (LVOTGs) and ap
36 study, myectomy offered greater reduction in left ventricular outflow tract gradients and larger impr
40 t arteries, subpulmonary left ventricle, and left ventricular outflow tract (LVOT) conduit dysfunctio
42 rdiography (ECG) criteria for distinguishing left ventricular outflow tract (LVOT) from right ventric
43 al septal reduction therapy (NSRT) decreases left ventricular outflow tract (LVOT) gradient and impro
44 relation between increasing severity of the left ventricular outflow tract (LVOT) gradient and outco
46 k Heart Association (NYHA) functional class, left ventricular outflow tract (LVOT) gradient at rest o
49 onal class, angina class, exercise duration, left ventricular outflow tract (LVOT) gradient, ejection
52 s plication of the mitral valve could reduce left ventricular outflow tract (LVOT) obstruction and as
53 therapy results in significant reduction in left ventricular outflow tract (LVOT) obstruction and sy
54 ical septal reduction therapy (NSRT) reduces left ventricular outflow tract (LVOT) obstruction in pat
56 atients with hypertrophic cardiomyopathy and left ventricular outflow tract (LVOT) obstruction, but w
58 Venturi mechanism, high flow velocity in the left ventricular outflow tract (LVOT) should be found at
59 he resting pressure gradient (PG) across the left ventricular outflow tract (LVOT) significantly decr
60 cular arrhythmias (VAs) originating from the left ventricular outflow tract (LVOT) sometimes require
61 cular arrhythmias (VAs) originating from the left ventricular outflow tract (LVOT) sometimes require
62 cular arrhythmias (VAs) originating from the left ventricular outflow tract (LVOT), an alternative ap
63 .3 years; 92 men), ACOM was performed in the left ventricular outflow tract (LVOT), with the color ba
65 rty-one consecutive patients who experienced left ventricular outflow tract (LVOT)/annular/aortic con
66 ophic cardiomyopathy with severe symptomatic left ventricular outflow tract obstruction (47+/-11 year
68 ansposition of the great arteries (TGA) with left ventricular outflow tract obstruction (LVOTO) may b
69 ertension (n = 24 [0.8%]) and hypotension or left ventricular outflow tract obstruction (n = 112 [3.8
71 mediate-term results indicate good relief of left ventricular outflow tract obstruction and need for
72 th d-transposition of the great arteries and left ventricular outflow tract obstruction and results i
74 n patients with hypertrophic cardiomyopathy, left ventricular outflow tract obstruction at rest is a
77 atients undergoing surgery for the relief of left ventricular outflow tract obstruction have low even
78 ter-based and surgical procedures to relieve left ventricular outflow tract obstruction in HCM, but i
79 Despite the association of symptoms with left ventricular outflow tract obstruction in HCM, there
80 ir of congenital heart disease, reduction of left ventricular outflow tract obstruction in hypertroph
81 myectomy and alcohol septal ablation relieve left ventricular outflow tract obstruction in severely s
83 compression wave, whereas proximally, severe left ventricular outflow tract obstruction is associated
88 s with suprasystemic pulmonary hypertension, left ventricular outflow tract obstruction or dilated ca
89 A d-transposition of the great arteries with left ventricular outflow tract obstruction represents a
90 FG+ and G+ probands were younger with less left ventricular outflow tract obstruction than G- proba
91 mptoms at study entry, including 249 in whom left ventricular outflow tract obstruction was absent bo
92 remaining 25 patients had multiple levels of left ventricular outflow tract obstruction, 12 of whom h
93 plasia is typically seen in association with left ventricular outflow tract obstruction, itself part
94 diagnosis, left ventricular wall thickness, left ventricular outflow tract obstruction, or family hi
95 e pericardial tamponade, pulmonary embolism, left ventricular outflow tract obstruction, unexplained
104 ar death (13.02%, 95% CI 3.60-25.91%), while left ventricular outflow tract obstruction/mid-ventricul
106 axy, atrial septal defects, conotruncal, and left ventricular outflow tract obstructive lesions are u
108 e lower in the right ventricle (P=0.037) and left ventricular outflow tract (P<0.001) and higher in l
110 on, AVA(CMR3) simplified continuity equation=left ventricular outflow tract peak flow rate/aortic pea
111 efining the morphologic abnormalities of the left ventricular outflow tract present in patients who d
113 spid valve, a large ASD, a VSD, an elongated left ventricular outflow tract, rightward displacement o
119 ) was calculated as ([LA emptying fraction x left ventricular outflow tract-velocity time integral] /
120 ence of overt structural heart disease, most left ventricular outflow tract ventricular tachycardias
121 iography, of the maximal Doppler velocity in left ventricular outflow tract (VmaxAo) measured using e
122 hat multiple morphologies (MMs) of inducible left ventricular outflow tract VT may indicate a scar-re
123 thout overt structural heart disease, 24 had left ventricular outflow tract VT, 10 had MM VT, and 14
126 creases in pre-ejection flow velocity in the left ventricular outflow tract, with consequent loss of
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