ライフサイエンスコーパス: left ventricular pressure

1 pressure and maximal rate of development of left ventricular pressure).

2 , and decreased myocardial contractility and left ventricular pressure.

3 inhibited TdP, but caused a 15+/-8% drop of left ventricular pressure.

4 pressure product and the first derivative of left ventricular pressure.

5 A lower dose of verapamil without effects on left ventricular pressure (0.06 mg/kg) was not antiarrhy

6 contrast, have normal heart function despite left ventricular pressures 25% higher than wild type.

7 On the basis of end-diastolic left ventricular pressure, 34 MI rats were classified as

8 hearts transduced with Ad.PL had lower peak left ventricular pressure (58.3 +/- 12.9 mmHg, n = 8) co

9 ed later (30+/-11 ms, P<0.015) and at higher left-ventricular pressure (61+/-9 mm Hg, P<0.001) than i

10 BKPC significantly improved the recovery of left ventricular pressure (73+/-5 versus 51+/-4 mm Hg; P

11 Left ventricular pressure, action potential duration, an

12 astolic pressure and a decrease in developed left ventricular pressure (all P<0.01 versus baseline) i

13 pressure and reduced peak and end-diastolic left ventricular pressures (all P<0.05).

14 affeine restored CcOX activity and increased left ventricular pressure and +/-dP/dt toward sham value

15 Left ventricular pressure and cardiac efficiency improve

16 ght dogs chronically instrumented to measure left ventricular pressure and dimension.

17 This increased left ventricular pressure and increased pressure develop

18 ine increased mean heart rate, peak positive left ventricular pressure and its first time-derivative,

19 Left ventricular pressure and maximal change in pressure

20 y blood flow and the maximum rate of rise in left ventricular pressure and reduced peak and end-diast

21 EA-0400 (0.4 and 0.8 mg/kg) had no effect on left ventricular pressure and suppressed dofetilide-indu

22 We measured the left ventricular pressure and volume continuously with a

23 Left ventricular pressure and volume data were determine

24 Characteristics of left ventricular pressure and volume unloading between t

25 ular diastolic filling pressures (pre-A wave left ventricular pressure) and Doppler mitral inflow at

26 of forearm blood flow, coronary blood flow, left ventricular pressure, and cardiac output were made

27 Aortic pressure, electrocardiogram, left ventricular pressure, and left ventricular pressure

28 yocardial CcOX activity, oxygen consumption, left ventricular pressure, and pressure developed during

29 trumented to measure aortic, left atrial and left ventricular pressures, and regional myocardial func

30 e on overall valve opening-closing dynamics, left ventricular pressure, aortic pressure, blood flow r

31 Because BNP reflects both elevated left ventricular pressure as well as neurohormonal modul

32 ejection fraction, analog differentiation of left ventricular pressure at 40 mm Hg, and rate of maxim

33 esistance index, the first derivative of the left ventricular pressure at a left ventricular pressure

34 idates in humans an equation relating tau to left ventricular pressure at peak -dP/dt (P0), pressure

35 minutes of reperfusion, maintaining CPP and left ventricular pressure at preischemic values.

36 Larger left heart structures and higher left ventricular pressure at the time of intervention we

37 n of maximum of the first time derivative of left ventricular pressure by dobutamine was blunted by i

38 artery (LAD) bypass were instrumented with a left ventricular pressure catheter and 2 subepicardial c

39 ced at 270 beats per minute, and the rate of left ventricular pressure change (LV dP/dt) was monitore

40 ed area produced minimal changes in systolic left ventricular pressure compared with baseline sinus r

41 the exponential time constant of isovolumic left ventricular pressure decay (Tau) and the "stiffness

42 ular pressure of 40 mm Hg (dP/dt40), rate of left ventricular pressure decline (-dP/dt), and a lower

43 ular pressure of 40 mm Hg (dP/dt40), rate of left ventricular pressure decline (-dP/dt), and end-tida

44 ar pressure at 40 mm Hg, and rate of maximal left ventricular pressure decline were continuously meas

45 ressure of 40 mm Hg, and the maximum rate of left ventricular pressure decline were significantly les

46 The rate of left ventricular pressure decrease was unchanged.

47 Peak left ventricular pressure decreased after TAVR (186 +/-

48 phy) and then cardiac catheterization, where left ventricular pressure development (+dP/dt) and decli

49 ignificant reduction in the maximum rates of left ventricular pressure development and pressure decli

50 ll acceleration of cross-bridge kinetics and left ventricular pressure development cannot be achieved

51 rload, fractional shortening and the rate of left ventricular pressure development decreased by 36% a

52 d as a percentage of the zone at risk; ZAR), left ventricular pressure development, and coronary flow

53 l tissue and showed electrical conductivity, left ventricular pressure development, and metabolic fun

54 tamine stress, VS attenuated the increase in left ventricular pressure-diameter area from 235.9 +/- 7

55 conscious dogs chronically instrumented for left ventricular pressure-dimension analysis, PDE5A inhi

56 nd-diastolic pressure (LVEDP), and developed left ventricular pressure (dLVP=LVSP-LVEDP), ischemia-re

57 n consumption (MVO(2)), peak rate of rise of left ventricular pressure (dP/dt(max)), stroke work (SW)

58 ve maximal values of the first derivative of left ventricular pressure (dP/dt) were significantly imp

59 wall stress and positive first derivative of left ventricular pressure (dP/dt).

60 ak positive and negative first derivative of left ventricular pressure (dP/dt).

61 sure (LVSP), the maximum first derivative of left ventricular pressure (dp/dtmax ), and the slope of

62 obutamine increased the peak rate of rise of left ventricular pressure (+dP/dt) by 49 +/- 8% (p < 0.0

63 on) revealed no differences in heart weight, left ventricular pressure, dP/dt, cardiac index, time co

64 Left ventricular pressure, dP/dt40, negative dP/dt and c

65 of discordance between right ventricular and left ventricular pressures during inspiration, a sign of

66 mic contracture as indicated by increases in left ventricular pressure from 9+/-3 to 33+/-6 mm Hg (p

67 ed animal weight, mean impact velocity, mean left ventricular pressure generated by the blow, mean QR

68 In multivariable analysis, mean left ventricular pressure generated by the blow, mean QR

69 Peak left ventricular pressure generated by the chest blow wa

70 decreases in heart rate and rate of rise in left ventricular pressure, improvement of regional coron

71 These previous studies, however, used left ventricular pressure in formulas that assumed the a

72 Left ventricular pressures in vivo and cardiomyocyte sar

73 cardial function, as measured by the rate of left ventricular pressure increase at 40 mm Hg, left ven

74 was significantly less depression in rate of left ventricular pressure increase measured at a left ve

75 Left ventricular pressure, rate of left ventricular pressure increase measured at a left ve

76 tricular end-diastolic pressure, the rate of left ventricular pressure increase measured at a left ve

77 Left ventricular pressure is 2-fold higher than wild typ

78 We demonstrate that left ventricular pressure is closely linked to KATP chan

79 The AHR (maximum rate of left ventricular pressure [LV-dP/dt(max)]) was assessed

80 Left ventricular pressure (LVP) and ECG were monitored d

81 rbital-anesthetized intact dogs arterial and left ventricular pressure (Millar) and left ventricular

82 stimulation parameters, Pt was calculated as left ventricular pressure minus balloon luminal pressure

83 ardiography (n = 4 per group), and right and left ventricular pressure (n = 5 and n = 4 per group, re

84 Significant reductions in left ventricular pressures occurred in vivo and in cardi

85 The volume intercept at left ventricular pressure of 100 mm Hg increased from 43

86 cular end-systolic volume at an end-systolic left ventricular pressure of 100 mm Hg.

87 he rate of left ventricular pressure rise at left ventricular pressure of 40 mm Hg (dP/dt40) and fall

88 ventricular pressure increase measured at a left ventricular pressure of 40 mm Hg (dP/dt40), rate of

89 ventricular pressure increase measured at a left ventricular pressure of 40 mm Hg (dP/dt40), rate of

90 ventricular pressure increase measured at a left ventricular pressure of 40 mm Hg, and the maximum r

91 vative of the left ventricular pressure at a left ventricular pressure of 50 mm Hg, rate-pressure pro

92 rease in LVdP/dtmax (maximal rate of rise of left ventricular pressure) of >/=90% of the maximum LVdP

93 sphorylation increased to 23% in response to left ventricular pressure overload as compared with 7% p

94 normal postnatal cardiac growth, concurrent left ventricular pressure overload hypertrophy did not s

95 ntricular function despite persistent severe left ventricular pressure overload in ascending aortic-b

96 Left ventricular pressure overload in mouse working hear

97 icular (LV) inotropic effects (adjusted peak left ventricular pressure rate of rise (dP/dt)max/P, 21.

98 Left ventricular pressure, rate of left ventricular pres

99 ary bypass, the average intraoperative right/left ventricular pressure ratio was 55% +/- 13%.

100 High-fidelity left atrial and left ventricular pressure recordings with simultaneous D

101 ll thickening as well as the maximal rate of left ventricular pressure rise (+dP/dt) and ventricular

102 he onset of sepsis, the maximal rates of the left ventricular pressure rise (+dP/dtmax) and fall (-dP

103 to LVP and BiVP (% change in maximal rate of left ventricular pressure rise [LVdP/dtmax]) was measure

104 ll thickening as well as the maximal rate of left ventricular pressure rise and fall (+dP/dt and -dP/

105 Cardiac index and the rate of left ventricular pressure rise at left ventricular press

106 ntricular systolic pressure, maximal rate of left-ventricular pressure rise and decline (+dP/dt, -dP/

107 alues of key parameters such as arterial and left ventricular pressures, serum lipoprotein, and other

108 ted with intracardiac transducers to measure left ventricular pressure, sonomicrometer crystals in th

109 Direct curve fitting to the left ventricular pressure trace by Levenberg-Marquardt r

110 rocardiogram, left ventricular pressure, and left ventricular pressure value of 40 mm Hg were continu

111 weeks followed by a terminal measurement of left ventricular pressure volume loops.

112 kit(-/-) mice, and that the leftward shifted left ventricular pressure-volume curve in the MHCsTNF/c-

113 rtery catheterization to define Starling and left ventricular pressure-volume curves.

114 ion without significant myocardial necrosis (left ventricular pressure-volume curves; 1% triphenyltet

115 Left ventricular pressure-volume relations were measured

116 -induced myocardial dysfunction by improving left ventricular pressure-volume relationship.

117 Left ventricular pressure-volume relationships utilizing

118 Left ventricular pressure-volume relationships were asse

119 Left-ventricular pressure-volume analyses in adult homoz

120 lenge (dobutamine 0.3-10 mug/kg/min) using a left ventricular pressure/volume catheter.

121 ronically instrumented to measure aortic and left ventricular pressures, wall thickness, and left cir

122 Left ventricular pressure was monitored.

123 ith either endotoxin or saline, systemic and left ventricular pressures were measured, and the first

124 High fidelity measures of left atrial and left ventricular pressures were obtained simultaneously

125 ed rat model of full thickness scald injury, left ventricular pressures were recorded in vivo followe

126 nce imaging, the rats were catheterized, and left ventricular pressures were recorded.

127 racic echo Doppler studies, and closed-chest left ventricular pressures with direct left ventricular