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1 , included improved contractile function and left ventricular remodeling.
2 ardiac functional recovery and prevention of left ventricular remodeling.
3 studies support a key role for cytokines in left ventricular remodeling.
4 t role in the development and progression of left ventricular remodeling.
5 to provoke left ventricular dysfunction and left ventricular remodeling.
6 duce morbidity and mortality associated with left ventricular remodeling.
7 cies may participate in mechanically induced left ventricular remodeling.
8 improve understanding of the role of MMPs in left ventricular remodeling.
9 t noninfarcted regions, as well as attenuate left ventricular remodeling.
10 me constant of relaxation or any variable of left ventricular remodeling.
11 me that regulates post-myocardial infarction left ventricular remodeling.
12 stolic and diastolic function, and decreased left ventricular remodeling.
13 art failure-related urgent care, and adverse left ventricular remodeling.
14 mprove clinical outcomes and prevent adverse left ventricular remodeling.
15 ocardial injury and its predictive value for left ventricular remodeling.
16 and exerted a sustained beneficial effect on left ventricular remodeling.
17 MAD-3 largely prevented fibrosis and limited left ventricular remodeling.
18 ment option for patients with postinfarction left ventricular remodeling.
19 after myocardial infarction (MI) accelerates left ventricular remodeling.
20 ic myocardium and has been linked to adverse left ventricular remodeling.
21 ted infarct inflammation, and curbed post-MI left ventricular remodeling.
22 cardiac pathologies, including post-infarct left ventricular remodeling.
23 in murine infarcts and consequently enhances left ventricular remodeling.
24 escued the angiogenic defect and ameliorated left ventricular remodeling.
25 ounding tissue, which often leads to adverse left ventricular remodeling.
26 triuretic peptide (BNP), and fully prevented left-ventricular remodeling.
27 on of 59.3+/-7.9%, with prevalent concentric left ventricular remodeling (34%) and hypertrophy (43%),
33 trix metalloproteinases (MMPs) contribute to left ventricular remodeling after myocardial infarction
34 matrix metalloproteinases (MMPs) attenuates left ventricular remodeling after myocardial infarction
35 in plasma RNA from patients with or without left ventricular remodeling after myocardial infarction.
36 amine BP, the incidence of hypertension, and left ventricular remodeling among collegiate ASF athlete
37 species production and again observed worse left ventricular remodeling and a lower ejection fractio
40 the post-infarction progression of negative left ventricular remodeling and decline in cardiac funct
41 yocardium is directly cardiotoxic and causes left ventricular remodeling and dilated cardiomyopathy.
43 old (IK-5001), to prevent or reverse adverse left ventricular remodeling and dysfunction in patients
44 hypertension, and renal insufficiency drive left ventricular remodeling and dysfunction through syst
45 e MSC(LacZ) group; MSC(HO-1) also attenuated left ventricular remodeling and enhanced the functional
46 greatest negative effect on infarct size and left ventricular remodeling and function, as well as a s
47 a alone or in combination with parameters of left ventricular remodeling and function, yielded an imp
48 s provide evidence for beneficial effects on left ventricular remodeling and functional capacity.
50 acellular biomaterials hold promise to limit left ventricular remodeling and heart failure precipitat
51 te or greater MR was associated with adverse left ventricular remodeling and increased likelihood of
53 vation of inflammation in the heart provokes left ventricular remodeling and left ventricular dysfunc
55 ling pathways that may contribute to adverse left ventricular remodeling and mitochondrial dysfunctio
56 PMA exerted a long-term beneficial effect on left ventricular remodeling and more effectively restore
57 e found to be most effective in ameliorating left ventricular remodeling and preserving function.
58 dium in the risk region, along with improved left ventricular remodeling and regional and global left
60 therapy did not improve clinical outcomes or left ventricular remodeling and was associated with pote
61 post-PPCI, and it is associated with adverse left ventricular remodeling and worse clinical outcomes.
62 tective response and inhibiting pathological left ventricular remodeling and, therefore, may be a use
63 is principally caused by global or regional left ventricular remodeling and/or severe left atrial di
64 ophy, left ventricular dilation, and adverse left ventricular remodeling, and a significant decrease
66 ric analysis revealed maximal attenuation of left ventricular remodeling, and echocardiography showed
72 MCP-1 results in attenuated post-infarction left ventricular remodeling, at the expense of a prolong
73 MO, iron deposition, infarct resorption, and left ventricular remodeling between day 7 (acute) and we
74 Ethnicity is an established determinant of left ventricular remodeling; black athletes (BAs) exhibi
76 cardiomyocytes, MCP-1-/- mice had attenuated left ventricular remodeling, but similar infarct size wh
78 ves hemodynamic profiles and induces reverse left ventricular remodeling by reducing left ventricular
79 ped larger infarct scars and more pronounced left ventricular remodeling compared with wild-type mice
81 ve favorable cardiac effects associated with left ventricular remodeling early after myocardial infar
83 h lack of functional recovery and adverse LV left ventricular remodeling extending to remote myocardi
85 ects with high-output HF displayed eccentric left ventricular remodeling, greater natriuretic peptide
86 L [2380-3006 mL]; P<0.0001), more concentric left ventricular remodeling, greater right ventricular d
87 .72]; P=0.004) and the likelihood of adverse left ventricular remodeling (>20% change in left ventric
90 icantly affect postinfarct myocardial and LV left ventricular remodeling; hemorrhagic infarcts behave
91 of metoprolol) attenuates the progression of left ventricular remodeling in a rat model of myocardial
93 ted whether beta-blocker therapy ameliorates left ventricular remodeling in asymptomatic patients wit
95 ible interaction of ASA and beta-blockers on left ventricular remodeling in patients with heart failu
96 d c-kit(+) cardiac stem cells (CSCs) improve left ventricular remodeling in porcine models and clinic
97 roves functional status, and induces reverse left ventricular remodeling in selected populations with
98 roves functional status, and induces reverse left ventricular remodeling in selected populations with
100 dial injury, and it is a strong predictor of left ventricular remodeling in ST-segment-elevation myoc
102 tory burden, infarct resorption, and adverse left ventricular remodeling in the chronic phase of MI i
103 tudy was to describe the long-term course of left ventricular remodeling induced by cardiac resynchro
108 itral regurgitation and enhanced reversal of left ventricular remodeling is possible when subvalvular
110 ease, such as hypertrophy and other types of left ventricular remodeling, ischemia/reperfusion injury
111 vided important insights into postinfarction left ventricular remodeling, it has not been possible to
112 onic mitral regurgitation results in adverse left ventricular remodeling, its effect on the mitral va
115 ion in pigs, demonstrating either successful left ventricular remodeling (LVR, n = 8) or congestive h
116 h better systolic valve performance, similar left ventricular remodeling, more paravalvular regurgita
117 ids was associated with reduction of adverse left ventricular remodeling, noninfarct myocardial fibro
118 HIIT was not superior to MCT in changing left ventricular remodeling or aerobic capacity, and its
119 etween mutation carriers and noncarriers (no left ventricular remodeling or fibrosis, normal left ven
121 s of insulin-like growth factor I (IGF-I) on left ventricular remodeling, partly through its antiapop
123 to test the hypothesis that the severity of left ventricular remodeling predicts the response to tre
124 contractile parameters were associated with left ventricular remodeling, recapitulation of fetal gen
125 n, hemodynamic severity of stenosis, adverse left ventricular remodeling, reduced left ventricular lo
126 r biomaterial treatment of MI and subsequent left ventricular remodeling remain the same, namely, lef
129 ever, we found only a modest acceleration of left ventricular remodeling, suggesting that, in individ
130 viability studies are predictive of reverse left-ventricular remodeling, symptom improvement, and pa
131 ve beta-blockade was associated with adverse left ventricular remodeling, systolic dysfunction, and a
132 ral valve in the setting of advanced adverse left ventricular remodeling that alters the alignment ch
133 t at 12 months of age the T2DN(mtFHH) showed left ventricular remodeling that was verified by histolo
134 e biological changes responsible for adverse left ventricular remodeling, the relationship between in
135 lines in LVEF; however, trastuzumab-mediated left ventricular remodeling-the primary outcome-was not
138 n in another 6 sheep (tethered plus MI), and left ventricular remodeling was limited by external cons
139 ar weights in proportion to body growth, but left ventricular remodeling was minor, and a decrease in
140 tudy of asymptomatic individuals, concentric left ventricular remodeling was related to decreased reg
142 improved cardiac contractility and reversed left ventricular remodeling, which was accompanied by a
143 descending artery occlusion in swine caused left ventricular remodeling with a decrease of ejection
144 reas nontransgenic mice exhibited concentric left ventricular remodeling with maintained ejection per
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