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   1 ing sign during the follow-up of extrafacial lentigo.                                                
     2  Histological melanocytic changes resembling lentigo and lentigo maligna were seen in several skin gr
  
  
  
  
  
  
     9 n AM (44.4%) and occasional amplification in lentigo maligna melanoma (10.5%) and superficial spreadi
  
    11 ing basal cell cancer, squamous cell cancer, lentigo maligna melanoma and cutaneous T-cell lymphoma. 
    12  an axial site, and superficial spreading or lentigo maligna melanoma types (P = .02, P < .001, and P
  
  
    15 preading melanomas, 17 nodular melanomas, 19 lentigo maligna melanomas, 18 AMs, and 12 unclassifiable
  
  
  
    19 l melanocytic changes resembling lentigo and lentigo maligna were seen in several skin grafts treated
    20 l spreading subtype (nodular, 0.5 [0.2-1.0]; lentigo maligna, 0.4 [0.2-0.7]; and unclassified/other, 
  
    22 29.4% vs 8.7%; P < .001) and of the nodular, lentigo maligna, or acral lentiginous histologic subtype
    23   Imiquimod leads to an 80-100% cure rate of lentigo maligna; however, studies of invasive melanoma a
    24 ficial spreading melanoma and some of facial lentigo maligna; however, these features are often absen
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