ライフサイエンスコーパス: leptomeninges

1 llaries of the cerebral cortex and overlying leptomeninges.

2 ection of cortex, white matter and overlying leptomeninges.

3 d to the implant pocket and to adjacent host leptomeninges.

4 risk of melanoma within the skin, brain, or leptomeninges.

5 four mGATs was present to some degree in the leptomeninges.

6 king of effector T cells from the gut to the leptomeninges after stroke.

7 rogeneity as medulloblastoma metastasizes to leptomeninges and as it evolves in the face of radiation

8 tia of small arteries and capillaries of the leptomeninges and cerebral cortex and is a major cause o

9 ulates predominantly in the small vessels of leptomeninges and cerebral cortex, leading to fatal stro

10 lso was detected in the lung, as well as the leptomeninges and choroid plexus of the fetal brain.

11 econd most common finding was involvement of leptomeninges and cranial nerves, which manifested as ab

12 adherence of different Neisseria species to leptomeninges and cultured cells.

13 It was, however, expressed in the leptomeninges and in a subpopulation of brain blood vess

14 She had extensive amyloid deposition in the leptomeninges and liver as well as the involvement of th

15 d a specific predilection for binding to the leptomeninges and meningeal blood vessels in human brain

16 ly more serotype A than B spirochetes in the leptomeninges and more serotype B than A spirochetes in

17 increased Fos immunoreactivity in the basal leptomeninges and several regions implicated in autonomi

18 ers was then compared to cells cultured from leptomeninges and to two other types of endothelial cell

19 cells produce metastatic lesions only in the leptomeninges and ventricles.

20 ne patient developed a relapse in the spinal leptomeninges and was rendered free of disease with spin

21 l anomalies, and abnormalities of the brain, leptomeninges, and eye.

22 lary-associated pericytes, astroglial cells, leptomeninges, and the choroid plexus.

23 in the brain localized predominantly to the leptomeninges, and those in peripheral tissues localized

24 resident within the parenchyma of the brain, leptomeninges, and vascular beds, as well as through sec

25 Thus, the data demonstrate that cells of the leptomeninges are not inert but are active participants

26 of Neisseria meningitidis with cells of the leptomeninges are pivotal events in the progression of b

27 They were detected in the leptomeninges around the neural tube, and only rarely we

28 in multiple tissues, including the cerebral leptomeninges, brainstem, peripheral nerves from both fo

29 ated with the cerebral blood vessels and the leptomeninges by immune stimuli such as intravenous admi

30 formed on mRNA isolated from the human fetal leptomeninges detected expression of the G protein-coupl

31 ly, TTR amyloid deposits were present in the leptomeninges, especially the leptomeningeal vessels, an

32 uR2/3, mGluR4a, and mGluR7 were expressed in leptomeninges from adult rats.

33 Inflammation in the leptomeninges has been identified as a key feature of se

34 a melatonin receptor expression in the fetal leptomeninges implies that melatonin may play a role in

35 has also been observed in choroid plexus and leptomeninges in normal mouse brain.

36 ells was used to investigate the role of the leptomeninges in the inflammatory response.

37 ed it to amyloid in the neuritic plaques and leptomeninges in the same patients.

38 ecrease in the degree of inflammation of the leptomeninges in these mice.

39 erature shows that amyloid deposition in the leptomeninges is not uncommon in TTR amyloidoses clinica

40 The structural organization of the dura and leptomeninges is reflected in its magnetic resonance (MR

41 1 (SDF1) (CXCL12), which is expressed by the leptomeninges, is necessary and sufficient to cause marg

42 Tumors metastasizing to the CNS and leptomeninges (LM) are associated with significant morta

43 ve, abnormal capillary venous vessels in the leptomeninges of the brain and choroid, glaucoma, seizur

44 with the exception of the choroid plexus and leptomeninges of the brain, where it is expressed from b

45 es except adult liver and the choroid plexus/leptomeninges of the central nervous system where IGF-II

46 alisation of Mel1a mRNA transcripts over the leptomeninges of the fetal brain.

47 nstrated enhancement of the cauda equina and leptomeninges of the lower spinal cord.

48 Rare spirochetes were seen in the leptomeninges of two cases by immunohistochemistry.

49 enchyma but did not induce metastasis to the leptomeninges or ventricles.

50 yma, without a decrease in metastasis to the leptomeninges or ventricles.

51 cells of the pia and arachnoid mater of the leptomeninges over large areas of the cerebral hemispher

52 cortex (P<0.001) and seven times more in the leptomeninges (P = 0.013); among the affected blood vess

53 l wall in the cortex (P = 0.001), and in the leptomeninges (P = 0.015).

54 the unimmunized cases (cortex: P = 0.009 and leptomeninges: P = 0.002).

55 ion was explored in blood vessels located in leptomeninges (pial vessels) and brain parenchyma (paren

56 the subarachnoid space and indicate that the leptomeninges play an important role in experimental aut

57 ly, severe meningitis with thickening of the leptomeninges, prominent vasculitis, and encephalitis wa

58 The leptomeninges, subpial astrocytes and astrocytes ensheat

59 nescence, with rare metastatic spread to the leptomeninges, suggesting roles for MYCN in both progres

60 tonin binding sites were identified over the leptomeninges surrounding the human fetal brain using qu

61 sclerosis have extensive inflammation in the leptomeninges that is associated with increased subpial

62 neuropathy have TTR amyloid deposits in the leptomeninges, the brain parenchyma, and the eye.

63 expression of Igf2 in the choroid plexus and leptomeninges, tissues where the gene is thought not to

64 onsistent with the barrier properties of the leptomeninges to N. meningitidis observed in vivo.