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1 ree M. bovis strains were recovered from non-lesioned animals.
2 rats and in the intact hemispheres of 6-OHDA-lesioned animals.
3 m or in the unlesioned hemispheres of 6-OHDA-lesioned animals.
4 ic activity were observed only in the 6-OHDA lesioned animals.
5 patterns in forebrain regions of intact and lesioned animals.
6 ion patterns in the forebrains of intact and lesioned animals.
7 lent levels of True Blue label in intact and lesioned animals.
8 putamen, thalamus, and amygdala of the MPTP-lesioned animals.
9 nimals, and CDP improving the performance of lesioned animals.
10 nding appeared by day 15 in both control and lesioned animals.
11 into the lateral ventricle of fimbria-fornix lesioned animals.
12 ffect on impaired performance in hippocampal lesioned animals.
13 de was collected from a random sample of non-lesioned animals.
14 on in REM sleep time was observed in the SOM-lesioned animals.
15 of correlations at large time scales in SCN-lesioned animals.
16 t as severe as those shown by the dorsal CA1 lesioned animals.
17 ocations relative to control and ventral CA1 lesioned animals.
18 both D2R and D3R relative to vehicle-treated lesioned animals.
19 y was not observed in either the LMN- or DTN-lesioned animals.
20 trast, LTD was preserved in nicotine-treated lesioned animals.
21 duced in BLA-lesioned animals but not in CeA-lesioned animals.
22 ess, a small day/night rhythm was present in lesioned animals.
23 s mild compared with cells recorded from PoS-lesioned animals.
24 re likely to choose the LR arm than the sham-lesioned animals.
25 essed relative to that in the shell- or sham-lesioned animals.
26 mRNA, but NPY mRNA was reduced in the ARC of lesioned animals.
27 anges in DA similar to those seen in the non-lesioned animals.
28 al root ganglion (DRG) neurons in intact and lesioned animals.
29 iate in low-lesioned, and smallest in highly lesioned animals.
30 atic induction of c-Fos in the CPu in 6-OHDA-lesioned animals.
31 ng) was quantified by autoradiography in DRN-lesioned animals.
32 representations in PoS-lesioned, but not ADN-lesioned, animals.
34 n throughout the dorsal striatum of neonatal lesioned animals, a response not observed within the int
37 control groups consisted of a group of sham lesioned animals and a group of animals that had unilate
38 mmatory responses were similar in normal and lesioned animals and the acetylcholinesterase inhibitor,
39 formation relative to control and dorsal CA1 lesioned animals, and a mild deficit for the temporal or
42 VC lesions produced behavior similar to VLF-lesioned animals but did not significantly affect tcMMEP
44 oral arrest could be reconstituted in fornix-lesioned animals by inducing synchronous activity in the
46 in neocortical areas of both hemispheres of lesioned animals compared with protein levels of sham-op
47 e, was significantly impaired in hippocampus-lesioned animals compared with sham control animals.
50 ntly altered, suggesting that smaller CPs in lesioned animals could be largely attributable to greate
52 ransplanting fetal SCN into the brain of the lesioned animal, demonstrating the first two of the esse
55 pattern to avoid the satiated food, whereas lesioned animals did not, suggesting that neonatal hippo
56 nucleus (VPL) in thalamic brain slices from lesioned animals displayed an increased probability of b
59 administration of apomorphine to the 6-OHDA lesioned animal evoked a robust and long-lasting excitat
64 ippocampal deafferentation in both groups of lesioned animals failed to prevent the accumulation of G
68 As compared with control animals, amygdala-lesioned animals had blunted responding to both positive
72 tion of nerve growth factor (NGF) to aged or lesioned animals has been shown to reverse the atrophy o
74 i.p.) improved passive avoidance behavior in lesioned animals in a mecamylamine-sensitive manner.
76 b in the food-restricted, methyl bromide gas-lesioned animals indicates that the mechanisms that guid
81 spatial correlates of complex spike cells in lesioned animals may rely on a more limited set of senso
82 e of CS preexposure), Novelty in hippocampal lesioned animals might be larger, equal, or smaller (cor
84 uced by apomorphine in the 6-hydroxydopamine-lesioned animal models of Parkinson's disease were studi
85 ibited long-term memory; in contrast, the TA-lesioned animals no longer showed target quadrant prefer
87 animals with mPFC lesions compared with sham-lesioned animals on the first day of retesting in all fi
89 DP in the septal lesioned subjects, with the lesioned animals performing worse than control animals,
90 ral asymmetries (compared to nonimmobilized, lesioned animals), presumably attributable to mild disus
91 to the increased locomotion observed in non-lesioned animals, rats pretreated with 6-OHDA showed no
94 elay-discounting curves was observed in wOFC-lesioned animals relative to shams--differences that dis
95 resentation of CS alone), onset responses in lesioned animals returned to their preconditioning firin
96 was identical to that of the experimentally lesioned animals revealed a bilateral ischemic lesion re
99 -term memory was examined, both sham- and TA-lesioned animals showed a significant preference for the
100 following conditioned fear stress, habenula-lesioned animals showed decreased PPI which normalized w
103 by damage to the auditory nerve, because the lesioned animals showed intact compound action potential
113 l dopamine release from synaptosomes of MPTP-lesioned animals, suggesting that nicotine treatment had
117 alysis experiments indicated that only those lesioned animals that received the mixture of MD-TH and
118 ies, revealed important factors operating in lesioned animals that were either absent or insignifican
121 alamic nucleus HD cells was still present in lesioned animals; thus, this property cannot be attribut
122 of ventrolateral PFC and the failure of LPFC-lesioned animals to disengage from the immediately prece
124 2 activation was confirmed in vivo in 6-OHDA-lesioned animals treated systemically with SKF38393.
125 tic assumptions of decision theory, the mOFC-lesioned animals' value comparisons were no longer indep
126 rection remained stable across days when the lesioned animal was placed into a novel environment.
127 halamic nucleus, c-fos mRNA induction in the lesioned animals was abolished in the bed nucleus of the
128 read of visual corticopontine connections in lesioned animals was greatly increased relative to unles
129 he performance of both the lesioned and sham-lesioned animals was impaired by the presentation of a v
131 g followed by surgery, it was found that all lesioned animals were able to remember the sequence.
133 quisition and overall response rates in core-lesioned animals were depressed relative to that in the
134 simultaneous olfactory discrimination; PRER-lesioned animals were dramatically and persistently impa
135 roximately 3 weeks of age, lesioned and sham-lesioned animals were either tested for the display of p
138 ment with our prediction, postsurgery, VLPFC lesioned animals were impaired in performing a series of
141 ndmark cue showed that place fields from PoS-lesioned animals were not controlled by the cue and shif
142 e third and fourth days of retraining, these lesioned animals were performing at a level comparable t
144 e assessed for neuronal differentiation, and lesioned animals were tested for amphetamine-induced rot
147 responses were significantly impaired in BLA-lesioned animals, whereas CeA-lesioned animals exhibited
148 tions of muscimol was markedly attenuated in lesioned animals, whereas the inhibition by VTA injectio
150 nition or motor function in symptomatic MPTP-lesioned animals with deficits in both of these areas.
153 ditioned response (CR) more slowly than sham-lesioned animals with either the 1.5-s ISI or with the 2
154 R and D3R binding in the ventral striatum of lesioned animals with lentiviral over-expression of both
155 irole generated only modest motor effects in lesioned animals with sole over-expression of D2R or D3R
156 imb and produced robust circling behavior in lesioned animals with striatal over-expression of both D
157 in expression was significantly increased in lesioned animals within proximal and distal regions of p
158 ute and transient working memory deficits in lesioned animals without effect in unlesioned controls.
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