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1  areas, industrial drug development has been lethargic.
2 phology, they became severely dehydrated and lethargic after water deprivation for 36 h.
3         Patients given carboplatin were less lethargic and less likely to take time off work than tho
4                             The animals were lethargic and runted; they died within 1 day of birth.
5 ole-genome expression profiling of CGCs from lethargic (beta4-null) mice individually reconstituted w
6                  Another pig became severely lethargic but not dehydrated.
7     The other pig that produced IMTGP became lethargic but not severely diarrheic.
8 ltiple days, we used only the first-alert or lethargic comparison evaluation in each patient.
9 (loxp/loxp); Alb-Cre(+/-) mice were sick and lethargic, especially during the first 2-3 days only.
10                The mouse neurological mutant lethargic (lh) is characterized by ataxia, focal myoclon
11 etermine that the ataxia and seizures in the lethargic (lh) mouse arise from mutation of the beta-sub
12 uncated beta4 isoform in the epileptic mouse lethargic (lh).
13 n both tottering (tg, alpha(1A) subunit) and lethargic (lh, beta(4) subunit) mutant mice.
14 1B and beta isoforms in the epileptic mouse, lethargic (lh/lh), a mutant anticipated to produce a tru
15 s of tottering (tg; Cav2.1/alpha1A subunit), lethargic (lh; beta4 subunit), and stargazer (stg; gamma
16 a gain-of-function mutation, causes severely lethargic locomotion, presumably by inappropriate phosph
17 +) dependence of neurotransmitter release in lethargic mice.
18 ng defects, rescue unc-73 RhoGEF-2 and rab-2 lethargic movement phenotypes.
19                                              Lethargic mutant mice carry a mutation in the CCHB4 gene
20                                          The lethargic mutants provide additional evidence that calci
21     In a genetic screen for suppressors of a lethargic phenotype caused by a gain-of-function isoform
22 genetic screen for mutants that suppressed a lethargic phenotype caused by expressing a gain-of-funct
23 orhabditis elegans mutants that suppressed a lethargic phenotype caused by expressing a gain-of-funct
24 f) in muscle was sufficient to reinstate the lethargic phenotype in slo-1(gf);ctn-1(lf).
25                                          The lethargic phenotype is the first example of a mammalian
26 -3 gene rescued the pag-3 reverse kinker and lethargic phenotypes.
27 ium channel in the mouse neurological mutant lethargic results in a complex neurological disorder tha

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