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1 areas, industrial drug development has been lethargic.
5 ole-genome expression profiling of CGCs from lethargic (beta4-null) mice individually reconstituted w
9 (loxp/loxp); Alb-Cre(+/-) mice were sick and lethargic, especially during the first 2-3 days only.
11 etermine that the ataxia and seizures in the lethargic (lh) mouse arise from mutation of the beta-sub
14 1B and beta isoforms in the epileptic mouse, lethargic (lh/lh), a mutant anticipated to produce a tru
15 s of tottering (tg; Cav2.1/alpha1A subunit), lethargic (lh; beta4 subunit), and stargazer (stg; gamma
16 a gain-of-function mutation, causes severely lethargic locomotion, presumably by inappropriate phosph
21 In a genetic screen for suppressors of a lethargic phenotype caused by a gain-of-function isoform
22 genetic screen for mutants that suppressed a lethargic phenotype caused by expressing a gain-of-funct
23 orhabditis elegans mutants that suppressed a lethargic phenotype caused by expressing a gain-of-funct
27 ium channel in the mouse neurological mutant lethargic results in a complex neurological disorder tha
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