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1 ity limit of 8 fmol has been established for leucine enkephalin.
2 a transmitters gastrin-releasing peptide and leucine-enkephalin.
3 des tested, only N, N-dibenzyl[Phe(p-NCS)(4)]leucine enkephalin (2) exhibited wash-resistant inhibiti
4 ne exception was N, N-dibenzyl[Phe(p-NCS)(4)]leucine enkephalin (2) which exhibited a 2-fold increase
8 d as the biologically active conformation of leucine enkephalin and its methyl ester in the nonpolar
10 molecules in droplets containing bradykinin, leucine enkephalin and myoglobin, but loss of the heme g
12 FPI attenuated PACAP, methionine enkephalin, leucine enkephalin, and dynorphin induced elevations in
17 s opiate receptor to the agonist D-alanine-5-leucine-enkephalin (DADLE) in wild-type and T149A mutant
18 ons of the opioids methionine enkephalin and leucine enkephalin during hypoxia in the newborn pig.
21 ine enkephalin and N,N-diallyl[Aib(2),Aib(3)]leucine enkephalin (ICI-174, 864) were substituted with
22 ion as shown by Perls' iron stain as well as leucine-enkephalin immunoreactivity, both markers for th
24 port, and chromatographic data indicate that leucine enkephalin is not hydrolyzed in the extracellula
28 er methionine enkephalin (met-enkephalin) or leucine enkephalin (leu-enkephalin), we observed enkepha
29 stigated the effect of exposure to exogenous leucine-enkephalin (Leu-Enk), methionine-enkephalin (Met
30 ion in levels of the endogenous DOR peptide, leucine enkephalin, normally seen during SH, thus protec
32 tapeptides, including the endogenous opioids leucine enkephalin (Tyr-Gly-Gly-Phe-Leu) and methionine
34 ptimal derivatization conditions for 1microM leucine-enkephalin were achieved when 10mM cysteine and
36 glycine and the fluorescamine derivative of leucine enkephalin, with the peak excitation cross secti
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