ライフサイエンスコーパス: leucyl-phenylalanine

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1 ants, such as the peptide N-formyl-methionyl-leucyl-phenylalanine.

2 myristate acetate (PMA) and formyl-methionyl-leucyl-phenylalanine.

3 tase after stimulation with formyl-methionyl-leucyl-phenylalanine.

4 ence of the chemoattractant formyl-methionyl-leucyl-phenylalanine.

5 er muscle in response to N-formyl- methionyl-leucyl-phenylalanine.

6 and after stimulation with formyl-methionyl-leucyl-phenylalanine (100 nM).

7 surface of unactivated and formyl methionyl leucyl phenylalanine-activated PMN as determined by indi

8 We found that N-formyl-methionyl-leucyl-phenylalanine, an FPR agonist peptide, rapidly in

9 ses upon stimulation with N-formyl methionyl leucyl phenylalanine and CC chemokine ligand (CCL) 3 (ne

10 multaneously treated with N-formyl-methionyl-leucyl-phenylalanine and IgE plus polyvalent antigen.

11 ogically relevant agents, N-formyl-methionyl-leucyl-phenylalanine and leukotriene B4, by approximatel

12 tants interleukin 8, C5a, N-formyl-methionyl-leucyl-phenylalanine, and interleukin 15, adhesion molec

13 ads, Staphylococcus aureus, formyl-methionyl-leucyl-phenylalanine, and zymosan were reduced by approx

14 activity and adhesiveness of formylmethionyl-leucyl-phenylalanine- and arachidonic acid-stimulated ne

15 or necrosis factor (TNF) or formyl-methionyl-leucyl-phenylalanine, as described for human PMNs.

16 of the chemotactic peptide formyl-methionyl-leucyl-phenylalanine at 10(-10)-10(-8) M and to the chem

17 CAM-1, and stimulation with formyl-methionyl-leucyl-phenylalanine boosted capture efficiency through

18 non-fluorescent peptide ligand CHO-methionyl-leucyl-phenylalanine (CHO-MLF).

19 ammatory process induced by formyl-methionyl-leucyl-phenylalanine exposure.

20 ng the transmucosal flux of formyl-methionyl-leucyl-phenylalanine (f-MLP), a ubiquitous neutrophilic

21 neutrophil stimulation with formyl-methionyl-leucyl-phenylalanine, FcgammaR cross-linking, or phospha

22 s significantly increased N-formyl-methionyl-leucyl phenylalanine (fMLF)-stimulated superoxide releas

23 the plasma membrane upon N-formyl-methionyl-leucyl-phenylalanine (fMLF) stimulation and colocalizes

24 licited by phorbol ester or formyl-methionyl-leucyl-phenylalanine (fMLF) was unaffected.

25 Formyl methionyl leucyl phenylalanine (fMLP) stimulates neutrophils to ad

26 ly 10-fold in response to N-formyl methionyl leucyl phenylalanine (fMLP).

27 nd ex vivo perfusion with n-formyl-methionyl-leucyl-phenylalanine (fMLP) (10(-)(7) M).

28 PMN chemotactic responses to formylmethionyl-leucyl-phenylalanine (fMLP) and IL-8 were dose-dependent

29 neutrophils, which binds to formyl-methionyl-leucyl-phenylalanine (fMLP) and plays a role in neutroph

30 and elastase) exposed to N-formyl-methionyl-leucyl-phenylalanine (fMLP) and/or multivalent immune co

31 of the chemotactic peptide formyl-methionyl-leucyl-phenylalanine (fMLP) caused a dose-dependent (10(

32 state 13-alpha-acetate or N-formyl methionyl-leucyl-phenylalanine (fMLP) for stimulus.

33 cells were exposed to an N-formyl-methionyl-leucyl-phenylalanine (FMLP) gradient whose source was pe

34 chemoattractant signal of N-formyl-methionyl-leucyl-phenylalanine (FMLP) in the absence of a spatial

35 his paradigm, we injected N-formyl-methionyl-leucyl-phenylalanine (FMLP) intradermally in guinea pigs

36 e primary chemoattractant N--formylmethionyl-leucyl-phenylalanine (fMLP) is mediated by leukotriene B

37 Microinjection of formyl-methionyl-leucyl-phenylalanine (fMLP) or macrophage inflammatory p

38 n secondary activation by N-formyl-methionyl-leucyl-phenylalanine (FMLP) or phorbol myristate acetate

39 Formyl-methionyl-leucyl-phenylalanine (FMLP) rapidly and transiently acti

40 ed human neutrophils with N-formyl-methionyl-leucyl-phenylalanine (fMLP) results in biphasic activati

41 The chemoattractant formyl-methionyl-leucyl-phenylalanine (fMLP) stimulated p38-MAPK-dependen

42 followed by treatment with formyl-methionyl-leucyl-phenylalanine (fMLP) stimulates cells in a physio

43 Formyl-methionyl-leucyl-phenylalanine (FMLP) stimulation (10(-7) M) resul

44 e assessed in response to N-formyl-methionyl-leucyl-phenylalanine (fMLP) stimulation.

45 rom cells with or without N-formylmethionine leucyl-phenylalanine (fMLP) stimulation.

46 lation of PMNs with 1 muM N-formyl-methionyl-leucyl-phenylalanine (fMLP) triggered earlier and more s

47 -8), formylpeptides (e.g. N-formyl-methionyl-leucyl-phenylalanine (fMLP)), and platelet-activating fa

48 ing N-formylated peptide (N-formyl-methionyl-leucyl-phenylalanine (fMLP)), platelet activating factor

49 coincubated with 0.5 microM formyl-methionyl-leucyl-phenylalanine (fMLP), 1.3 microM 22:6OOH, or 5.0

50 lls permits absorption of N-formyl-methionyl-leucyl-phenylalanine (fMLP), as occurs in hPepT1 express

51 ) and then activated with N-formyl-methionyl-leucyl-phenylalanine (FMLP), C(2)-ceramide (10 microM) c

52 to the chemotactic peptide formyl-methionyl-leucyl-phenylalanine (FMLP), colony stimulating factor-1

53 dition of PMN stimulants, N-formyl-methionyl-leucyl-phenylalanine (FMLP), or phorbol myristate acetat

54 neutrophils were exposed to formyl-methionyl-leucyl-phenylalanine (fMLP), PKCbetaII was rapidly phosp

55 actic bacterial peptide, N-formyl- methionyl-leucyl-phenylalanine (fMLP), was able to specifically at

56 CNS, and also reduces the N-formyl-methionyl-leucyl-phenylalanine (fMLP)-induced neutrophil respirato

57 examine the mechanism of N-formyl-methionyl-leucyl-phenylalanine (fMLP)-mediated formation of CysLT.

58 ion of FPRwt reconstituted N-formylmethionyl-leucyl-phenylalanine (FMLP)-stimulated extracellular sig

59 Our results show that formyl-methionyl-leucyl-phenylalanine (fMLP)-stimulated respiratory burst

60 polysaccharide (LPS)- and N-formyl-methionyl-leucyl-phenylalanine (FMLP)-stimulated U937 adhesion to

61 d their chemotaxis toward N-formyl-methionyl-leucyl-phenylalanine (FMLP).

62 bacterial chemoattractant, formyl-methionyl-leucyl-phenylalanine (fMLP).

63 ted upon stimulation with N-formyl-methionyl-leucyl-phenylalanine (fMLP).

64 with LPS, RPMI alone, and N formyl-methionyl-leucyl-phenylalanine (FMLP).

65 lammatory stimuli such as N-formyl-methionyl-leucyl-phenylalanine (fMLP).

66 ponse to the formyl peptide formyl-norleucyl-leucyl-phenylalanine (fNLLP).

67 e, opsonized zymosan, and N-formyl-methionyl-leucyl-phenylalanine) induce p22(phox) phosphorylation i

68 atelet-activating factor or formyl-methionyl-leucyl-phenylalanine induced beta(2)-integrin-dependent

69 bolish the TNF-alpha- and N-formyl-methionyl-leucyl-phenylalanine-induced activation of acetyltransfe

70 Since the kinetics of formyl-methionyl-leucyl-phenylalanine-induced F-actin response were highl

71 quantitative analysis of N-formyl-methionyl-leucyl-phenylalanine-induced increase in binding of (35)

72 icient in spontaneous and N-formyl-methionyl-leucyl-phenylalanine-induced polarization, 0.5 microM pe

73 ine before stimulation with formyl methionyl-leucyl-phenylalanine inhibited A3 receptor expression an

74 nse to multiple agonists (N-formyl-methionyl-leucyl-phenylalanine, interleukin-8, and C5a).

75 ntact cells stimulated with formyl-methionyl-leucyl-phenylalanine, intermediate filament assembly is

76 s activated to secrete with formyl-methionyl-leucyl-phenylalanine, intermediate filaments are phospho

77 peptide receptor agonist (N-formyl-methionyl-leucyl-phenylalanine-lysine; N-For-MLFK) were compared w

78 phils under conditions of N-formyl-methionyl-leucyl-phenylalanine-mediated cPLA(2)alpha activation.

79 cells were stimulated by N-formyl-methionyl-leucyl-phenylalanine or opsonized zymosan.

80 h configuration with PMA, N-formyl-methionyl-leucyl-phenylalanine, or anti-IgE greatly enhanced proto

81 bol myristate acetate (PMA), formylmethionyl-leucyl-phenylalanine, or Escherichia coli.

82 stimulated with anti-IgE, N-formyl-methionyl-leucyl-phenylalanine, or phorbol 12-myristate 13-acetate

83 hil activation induced by N-formyl-methionyl-leucyl-phenylalanine, phorbol 12-myristate 13-acetate, o

84 od extension induced with N-formyl-methionyl-leucyl-phenylalanine, platelet activating factor, and le

85 ty in cells stimulated with formyl-methionyl-leucyl phenylalanine plus dihydrocytochalasin B.

86 without the high-affinity N-formylmethionyl-leucyl-phenylalanine receptor antagonist N-tert-butoxyca

87 were assayed in vitro for N-formyl-methionyl-leucyl-phenylalanine receptor binding and superoxide pro

88 mannose receptor, and the N-formyl-methionyl-leucyl-phenylalanine receptor) did not have demonstrated

89 d doubling of the number of formyl-methionyl-leucyl-phenylalanine receptors on the cells.

90 on and O(2)(-) responses to formyl-methionyl-leucyl-phenylalanine, reflecting the same cellular pheno

91 ation, and TNFalpha-primed, formyl-methionyl-leucyl-phenylalanine-stimulated respiratory burst.

92 rachidonate production in N-formyl-methionyl-leucyl-phenylalanine-stimulated U937 cells.

93 Mbeta2 integrin following N-formyl-methionyl-leucyl phenylalanine stimulation.

94 generation before and after formyl-methionyl-leucyl-phenylalanine stimulation was significantly reduc

95 onic saline-treatment after formyl methionyl-leucyl-phenylalanine-stimulation augmented A3 receptor e

96 e-colony-stimulating factor/formyl-methionyl-leucyl-phenylalanine stimuli, which can induce eicosanoi

97 ha (TNF-alpha) as well as N-formyl-methionyl-leucyl-phenylalanine treatment leads to increased phosph

98 retains coupling between N-formyl-methionyl-leucyl-phenylalanine tripeptide (FMLP) receptor stimulat

99 eness upon stimulation with formyl-methionyl-leucyl phenylalanine was found to identify sputum eosino

100 y the chemoattractant fMLF (formyl methionyl leucyl phenylalanine) was observed by RICM (reflection i

101 by neutrophils, induced by N-formylmethionyl-leucyl-phenylalanine, was strongly inhibited by inhibito

102 otype N-formylpeptide fMLF (formyl-methionyl-leucyl-phenylalanine) were both absent in FPR-/- mice.

103 also seen in response to N-formyl-methionyl-leucyl-phenylalanine, zymosan-activated serum, or macrop

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