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1 at tiny silent acute infarcts are a cause of leukoaraiosis.
2 SIVD), multiple infarcts, mixed dementia and leukoaraiosis.
3 st of potential poor outcomes in people with leukoaraiosis.
4 n and more diffuse ischaemia, referred to as leukoaraiosis.
5 tter changes, with the imaging appearance of leukoaraiosis.
6 s: isolated lacunar infarction and ischaemic leukoaraiosis.
7 ese lesions approached those of pre-existing leukoaraiosis.
8 stronger risk factor in those with ischaemic leukoaraiosis [12.92 (95% CI 4.40-37.98), P < 0.0005) pe
13 risk factor for SVD, particularly ischaemic leukoaraiosis, and this effect may be mediated via endot
14 MRI scans were visually scored for degree of leukoaraiosis, central atrophy, and cortical atrophy.
15 ed lacunar infarction (n = 47) and ischaemic leukoaraiosis, defined as a clinical lacunar stroke and
16 ions that stroke outcomes may be affected by leukoaraiosis differentially depending on stroke subtype
17 lele was a risk factor only in the ischaemic leukoaraiosis group [odds ratio (OR) 2.02 (95% CI 1.31-3
20 (MRI) white matter hyperintensities (WMH; or leukoaraiosis) in patients with high vascular amyloid de
25 althy elderly participants with an aggregate leukoaraiosis lesion volume of more than 25 cm(3) and 18
33 th the number of lacunes (P = 0.008) and the leukoaraiosis score (P = 0.03), but TF levels and the TF
36 ntrol participants with less than 5 cm(3) of leukoaraiosis underwent functional MR imaging to allow c
38 g confirmed lacunar stroke with radiological leukoaraiosis were recruited and completed cognitive tes
39 e patients with lacunar stroke and confluent leukoaraiosis were recruited into the ongoing SCANS stud
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