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1  by integrin-dependent sickle-red blood cell-leukocyte adhesion.
2 m by which LA1-activated CD11b/CD18 mediates leukocyte adhesion.
3  and is critical for firm integrin-dependent leukocyte adhesion.
4 t captures CTCs while minimizing nonspecific leukocyte adhesion.
5                          LFA-1 also mediates leukocyte adhesion.
6  of mobility for the onset of LFA-1 mediated leukocyte adhesion.
7 the inhibition of exocytosis, and suppresses leukocyte adhesion.
8 othelium (P = 0.03) matching the increase in leukocyte adhesion.
9 lammatory signals with those associated with leukocyte adhesion.
10  microglial activation, and reduced cerebral leukocyte adhesion.
11 ed ICAM-1 in endothelial cells (EC) promotes leukocyte adhesion.
12 ation into plaques through platelet-mediated leukocyte adhesion.
13 s an important regulator of CX3CL1-dependent leukocyte adhesion.
14  a preceding activation step to trigger firm leukocyte adhesion.
15 for antithrombotic therapy based on blocking leukocyte adhesion.
16 on and digestion to regulating bacterial and leukocyte adhesion.
17 lar adhesion molecule-1 dependent intestinal leukocyte adhesion.
18 finity triggering mediates chemokine-induced leukocyte adhesion.
19 tion of HC-HA cable structures and abolishes leukocyte adhesion.
20 ry factor, which inhibits integrin-dependent leukocyte adhesion.
21 the markers of thrombosis, inflammation, and leukocyte adhesion.
22 onal change in beta2 integrins, facilitating leukocyte adhesion.
23         APN-KO mice also exhibited increased leukocyte adhesion (2.3+/-0.4-fold) and tumor necrosis f
24 e (CX3CL1) and its receptor, CX3CR1, mediate leukocyte adhesion, activation, and trafficking.
25 ional biology: a mechanics-based modeling of leukocyte adhesion (adhesive dynamics) and signal transd
26 and partly prevented increases in VCAM-1 and leukocyte adhesion after treatment with tumour necrosis
27 creased permeability, ICAM-1 expression, and leukocyte adhesion, all of which are features of an infl
28 ated a crucial role for CatG during arterial leukocyte adhesion, an effect not found during venular a
29 uorescence microscopy, we observed excessive leukocyte adhesion and accelerated atherosclerotic plaqu
30 nflammation, vascular lesions with increased leukocyte adhesion and capillary degeneration.
31               In contrast, polymorphonuclear leukocyte adhesion and chemotaxis were normal.
32 gulates many biological processes, including leukocyte adhesion and chemotaxis.
33 clerosis in ApoE(-/-) mice, there is reduced leukocyte adhesion and decreased CD18 expression on Gr1(
34 9)/L platelets, showing reduced signaling in leukocyte adhesion and diapedesis and increased compleme
35  DPA and RvD5n-3 DPA decreased the extent of leukocyte adhesion and emigration following ischemia-rep
36 ntified role for Panx1 channels in promoting leukocyte adhesion and emigration through the venous wal
37 ade body secretion, and in the regulation of leukocyte adhesion and extravasation during inflammation
38 ion in the genetic absence of Coro1A impairs leukocyte adhesion and extravasation in inflamed cremast
39 muscle venules revealed severely compromised leukocyte adhesion and extravasation in MK(-/-) mice com
40  control endothelial cell (EC) permeability, leukocyte adhesion and inflammation are pivotal for athe
41                  JAM-C thereby mediated both leukocyte adhesion and leukocyte transendothelial migrat
42 show that N-glycosylation of JAM-A regulates leukocyte adhesion and LFA-1 binding.
43  ligands and their ability to intervene with leukocyte adhesion and migration across brain endothelia
44   CX3CR1 is a chemokine receptor involved in leukocyte adhesion and migration and hence a mediator of
45 egment of the microvasculature that supports leukocyte adhesion and migration expands through remodel
46                                 Constitutive leukocyte adhesion and migration in murine bone marrow (
47 l antibody against alpha4 integrin, inhibits leukocyte adhesion and migration into inflamed tissue.
48    The finding that Cdk inhibitors can block leukocyte adhesion and migration may expand the clinical
49 t human fetal HSCs employ mechanisms used in leukocyte adhesion and migration to mediate HSC self-ren
50 nd its ligand CX3CL1 have been implicated in leukocyte adhesion and neuronal protection.
51 ultiple integrin-induced events important in leukocyte adhesion and phagocytosis.
52 dothelial activation manifested as increased leukocyte adhesion and plasma extravasation in response
53 ta2-integrin activation is indispensable for leukocyte adhesion and recruitment to sites of infection
54                         L-selectin regulates leukocyte adhesion and rolling along the endothelium.
55  new role of fibrinogen in integrin-mediated leukocyte adhesion and suggest that this mechanism may p
56 tify alpha 4 integrin/CD49d as a mediator of leukocyte adhesion and the resultant early signature abn
57 protein I (beta2GPI) induce endothelial cell-leukocyte adhesion and thrombus formation via unknown me
58 ould be used as a therapeutic tool to reduce leukocyte adhesion and thus reduce neuroinflammation.
59 or investigation of lymphatic involvement in leukocyte adhesion and trafficking during the immune res
60 the effects of hemodynamics and cytokines on leukocyte adhesion and trans-endothelial migration (TEM)
61 t human JAM-C to determine its role in human leukocyte adhesion and transendothelial cell migration u
62 ion of endothelial ABPs to the regulation of leukocyte adhesion and transendothelial migration remain
63 e their endothelial cell ligands and support leukocyte adhesion and transendothelial migration.
64                                Consequently, leukocyte adhesion and transmigration are significantly
65 l in vivo, elastic laminae were resistant to leukocyte adhesion and transmigration compared with the
66 synthase and fibrinolytic activity, decrease leukocyte adhesion and transmigration, and enhance resis
67  functions including macrophage recognition, leukocyte adhesion and transmigration.
68 beta while an anti-ICAM-1 mAb inhibited both leukocyte adhesion and transmigration.
69 F-induced proliferation and thrombin-induced leukocyte adhesion and triggers a STAT3-dependent increa
70 dase is positively correlated with increased leukocyte adhesion and vascular leakage in diabetes and
71 s and reduced significantly diabetes-induced leukocyte adhesion and vascular leakage.
72  and smooth muscle cell apoptosis as well as leukocytes adhesion and proliferation.
73 is, cell growth and apoptosis, angiogenesis, leukocyte adhesion, and cytokine activation and inhibiti
74 hamber protein concentration, retinal vessel leukocyte adhesion, and protein leakage were measured 24
75 at plays a role in tight junction formation, leukocyte adhesion, and transendothelial migration.
76 cells is essential for platelet aggregation, leukocyte adhesion, and transmigration through endotheli
77 a-AR stimulation can promote both SS RBC and leukocyte adhesion as well as vaso-occlusion, suggesting
78              AnxA1Ac2-26 treatment decreased leukocyte adhesion at 40 min and all L-E interactions at
79 sing IVM, we could detect instant changes in leukocyte adhesion behavior in the synovial vessels of t
80 ies in the murine inflamed microcirculation (leukocyte adhesion being the readout) and in skin traffi
81 , the retinas were extracted and assayed for leukocyte adhesion; blood-retinal barrier breakdown; VEG
82 hoG by small interfering RNA does not affect leukocyte adhesion but decreases cup formation and inhib
83 tyrosine phosphatase 1 (Shp1) show increased leukocyte adhesion, but the interpretation of these data
84  of the immunoglobulin superfamily, supports leukocyte adhesion by binding alpha4beta1 integrins and
85 3 cells, suggesting that these miRs regulate leukocyte adhesion by modulating the expression of adhes
86                         Understanding of the leukocyte adhesion cascade and interactions of leukocyte
87           Despite expanded definition of the leukocyte adhesion cascade and mechanisms underlying ind
88    These data provide unique insights in the leukocyte adhesion cascade and the potential for time-ba
89               Current in vitro models of the leukocyte adhesion cascade cannot be used for real-time
90                                          The leukocyte adhesion cascade is important in chronic lymph
91 tion in vivo and add additional steps to the leukocyte adhesion cascade that supports leukocyte traff
92  be used for real-time studies of the entire leukocyte adhesion cascade, including rolling, adhesion,
93 N-glycans contributing to all aspects of the leukocyte adhesion cascade, O-glycans only being importa
94 sses have been identified in the endothelial-leukocyte adhesion cascade.
95 llular MRP8/14 as an autocrine player in the leukocyte adhesion cascade.
96    In view of the capacity of FKN to mediate leukocyte adhesion, chemoattraction, and transmigration,
97 neally) resulted in a three-fold increase in leukocyte adhesion, compared with ischemia/reperfusion a
98                              This diminished leukocyte adhesion correlated with the magnitude of gran
99                                              Leukocyte adhesion, crawling, and transmigration are reg
100 s in this disappearance, a dog having canine leukocyte adhesion deficiency (CLAD) or CLAD dogs who ha
101                                       Canine leukocyte adhesion deficiency (CLAD) represents the cani
102 e use of foamy virus vectors to treat canine leukocyte adhesion deficiency (CLAD).
103 umans, mutations in beta(2) integrin lead to leukocyte adhesion deficiency (LAD) syndrome and mutatio
104 activation on neutrophils is the hallmark of leukocyte adhesion deficiency (LAD) syndrome in humans,
105                       PURPOSE OF REVIEW: The leukocyte adhesion deficiency (LAD) syndromes are rare g
106 the canine counter-part of the human disease leukocyte adhesion deficiency (LAD).
107 in inherited and acquired disorders, such as leukocyte adhesion deficiency and myelodysplasia.
108            Chronic granulomatous disease and leukocyte adhesion deficiency are the major primary immu
109 autosomal recessive disease characterized by leukocyte adhesion deficiency as well as severe neurolog
110 ontrasts with the phenotype seen in type III leukocyte adhesion deficiency caused by the absence of k
111 nd identify CF as a new, cell type-selective leukocyte adhesion deficiency disease, providing new ins
112      The protein kindlin 3 is mutated in the leukocyte adhesion deficiency III (LAD-III) disorder, le
113                              In the disorder leukocyte adhesion deficiency III (LAD-III), integrins o
114 e severe than those reported for people with leukocyte adhesion deficiency III (LAD-III).
115 de an effective, but less toxic approach for leukocyte adhesion deficiency in children.
116 SCs, and thus lack of CD18 expression in the leukocyte adhesion deficiency patients may predispose th
117 ivation on leukocytes is the hallmark of the leukocyte adhesion deficiency syndrome in humans, charac
118 f young dogs with the genetic disease canine leukocyte adhesion deficiency that were treated with a n
119                               A patient with leukocyte adhesion deficiency type 1 (LAD1) had severe p
120 e the contribution of CD18 on neutrophils to leukocyte adhesion deficiency type I (LAD-I), a complex
121 se), severe congenital neutropenia (SCN) and leukocyte adhesion deficiency type I confer a predisposi
122                                       LADII (leukocyte adhesion deficiency type II)/CDGIIc (congenita
123  hypofucosylated in PMNs from a patient with leukocyte adhesion deficiency type II, suggesting that i
124 lts imply that the immunodeficiency found in leukocyte adhesion deficiency type III patients, in addi
125  accumulation of HSPCs in the circulation of leukocyte adhesion deficiency type III patients, who lac
126 hat is mutated in the rare genetic disorder, leukocyte adhesion deficiency type III, a disorder chara
127 n-3 is mutated in the rare genetic disorder, leukocyte adhesion deficiency type III, which is charact
128                                              Leukocyte adhesion deficiency type-1 (LAD-1) is an autos
129 enital origin (e.g., congenital neutropenia, leukocyte adhesion deficiency, and Chediak-Higashi syndr
130 cell activity is impaired, but not absent in leukocyte adhesion deficiency, may lead to the developme
131 which die perinatally of severe bleeding and leukocyte adhesion deficiency, mice expressing as little
132                                Subjects with leukocyte adhesion deficiency-1 (LAD-I) do not express b
133                            A canine model of leukocyte adhesion deficiency-I facilitated development
134         Preimplantation genetic diagnosis of leukocyte adhesion deficiency-I led to the birth of a no
135 adhesion deficiency-I; prenatal diagnosis of leukocyte adhesion deficiency-I; and association of auto
136 e adhesion deficiency-I; transplantation for leukocyte adhesion deficiency-I; prenatal diagnosis of l
137 herapy for chronic granulomatous disease and leukocyte adhesion deficiency-I; transplantation for leu
138  been advances in understanding the basis of leukocyte adhesion deficiency-II and III.
139 ng kindlin-3, the protein that is mutated in leukocyte adhesion deficiency-III patients.
140    Patients with Glanzmann thrombasthenia or Leukocyte Adhesion Deficiency-III syndrome (LAD-III or L
141 tudying a patient with kindlin-3 deficiency (leukocyte adhesion deficiency-III).
142 nderlie the human immune deficiency known as leukocyte adhesion deficiency-III.
143 ll transplant and gene therapy approaches to leukocyte adhesion deficiency.
144 E) abolished the inhibitory effect of gAd on leukocyte adhesion, demonstrating the obligatory role of
145                                              Leukocyte adhesion during inflammation is initiated by t
146 electin, ICAM-1, and EphB4 and were sites of leukocyte adhesion during lipopolysaccharide-induced acu
147           Superfusion with IS induced strong leukocyte adhesion, enhanced extravasation, and interrup
148 lence at least in part by impeding Fg-driven leukocyte adhesion events.
149               When MDMECs were evaluated for leukocyte adhesion, exposure to CLP serum caused increas
150                                   Similarly, leukocyte adhesion, extravasation, and bacterial clearan
151 of temporal patterns of rhodamine-6G-labeled leukocyte adhesion/extravasation, was used.
152 molecules, including interleukin (IL)-6, the leukocyte adhesion factor ICAM1, and chemokines that att
153 ter B lymphocytes from mice with a defect in leukocyte adhesion fail to divide asymmetrically.
154 explain the shear threshold effect, in which leukocyte adhesion goes through a maximum with increasin
155 ctionally important endogenous inhibitors of leukocyte adhesion have not been identified.
156           Intravitreal IL-6 directly induced leukocyte adhesion in both wild-type and IL-6-deficient
157 muscle confirmed the defect of CXCL1-induced leukocyte adhesion in HPK1-deficient mice.
158  rolling on unstimulated veins and increased leukocyte adhesion in inflamed veins.
159 ate the complex roles this integrin plays in leukocyte adhesion in inflammation.
160 ceded clinical signs of anterior uveitis and leukocyte adhesion in iris vasculature.
161 eceptors in endothelial cells also increased leukocyte adhesion in mesenteric venules and increased t
162 tin ligands act as competitive inhibitors of leukocyte adhesion in models of inflammation.
163 rteriogenesis, thrombosis, inflammation, and leukocyte adhesion in patients with intermittent claudic
164 rupts capillary fine structure and increases leukocyte adhesion in postcapillary venules.
165      Insulin decreased VCAM-1 expression and leukocyte adhesion in quiescent tumour endothelial cells
166 1INH significantly inhibit selectin-mediated leukocyte adhesion in several in vitro and in vivo model
167     This article discusses the importance of leukocyte adhesion in sickle cell disease, and how this
168 ]-3-methyl-cyclohexanol), greatly attenuated leukocyte adhesion in surface pial vessels and in deep a
169 n leukocyte rolling and a 5-fold increase in leukocyte adhesion in the microcirculation.
170 nd cytokine response with a marked degree of leukocyte adhesion in the microcirculation.
171                                              Leukocyte adhesion in the microvasculature influences bl
172 d protein content in the aqueous fluid, firm leukocyte adhesion in the retinal vessels, and the numbe
173 d the mechanistic roles of hyperglycemia and leukocyte adhesion in this process.
174 l Del-1 deficiency increased LFA-1-dependent leukocyte adhesion in vitro and in vivo.
175 ion molecules (ICAMs) in cis, which inhibits leukocyte adhesion in vitro and in vivo.
176                                      Using a leukocyte adhesion in vitro assay under shear forces mim
177 pproximately 33 mumol) mitigated LPS-induced leukocyte adhesion in WT and AnxA1-null animals without
178 h soluble fibrinogen exerts its influence on leukocyte adhesion indicated that it did not block integ
179 studied endothelial permeability, intravital leukocyte adhesion, involvement of the Akt/WNT/beta-cate
180                            Integrin-mediated leukocyte adhesion is central to this process.
181                                              Leukocyte adhesion is determined by the balance between
182                                              Leukocyte adhesion is mediated mainly by selectins, cell
183 organization of endothelial ICAM-1 regulates leukocyte adhesion is not well understood.
184                                              Leukocyte adhesion is regulated through the modulation o
185                  A key parameter influencing leukocyte adhesion is the shear stress acting on the leu
186                                              Leukocyte adhesion is triggered by the interaction of va
187 platelet dysfunction in addition to impaired leukocyte adhesion, is now known to be due to absence of
188 m from obese mice expressed higher levels of leukocyte adhesion markers and lower levels of cell-cell
189 ignificantly increased the expression of the leukocyte adhesion markers LFA-1 and VLA-4, consistent w
190                                     Platelet-leukocyte adhesion may contribute to thrombosis and infl
191            Evidence suggests that disrupting leukocyte adhesion may prevent the impairment of renal m
192 macrophage-1 antigen complex, which mediates leukocyte adhesion, migration and phagocytosis as part o
193  and functions in diverse capacities such as leukocyte adhesion, migration, and cell survival on liga
194 dose of natalizumab, an antibody against the leukocyte adhesion molecule alpha4 integrin, in patients
195  this report, we have determined that the EC-leukocyte adhesion molecule E-selectin is a key target f
196                                E-selectin, a leukocyte adhesion molecule expressed on endothelium, is
197 irectly suppressed endothelial activation of leukocyte adhesion molecule expression and inflammation.
198 or the synthesis of functional selectin-type leukocyte adhesion molecule ligands.
199 ical surface, where JAM-A played a role as a leukocyte adhesion molecule participating in transendoth
200                     E-selectin, an inducible leukocyte adhesion molecule specifically expressed by en
201                                    Increased leukocyte adhesion molecule VCAM-1 expression and leukoc
202  observed elevated expression of endothelial leukocyte adhesion molecule-1, a human glaucoma marker,
203  but under inflammatory conditions acts as a leukocyte adhesion molecule.
204 ulatory role in cytokine induction of the EC-leukocyte adhesion molecules (ELAM) E-selectin and vascu
205                 Therefore, the expression of leukocyte adhesion molecules and secretion of proinflamm
206 eta2 integrins (CD11/CD18) are heterodimeric leukocyte adhesion molecules expressed on hematopoietic
207                A recent study shows that the leukocyte adhesion molecules known as selectins form 'ca
208                            TNF-alpha induces leukocyte adhesion molecules on endothelial cells (ECs),
209 t, Rho GTPases affect the expression of some leukocyte adhesion molecules on endothelial cells, such
210                   In contrast, the genes for leukocyte adhesion molecules showed a significant upregu
211 esion, and extravasation by up-regulation of leukocyte adhesion molecules such as E-selectin and P-se
212 e-inducible, NF-kappaB-dependent endothelial-leukocyte adhesion molecules that participate in the leu
213 tigated the role of p75 in TNF-alpha-induced leukocyte adhesion molecules using cultured ECs derived
214 elial cells, their expression of ligands for leukocyte adhesion molecules, and vaso-occlusion.
215 ir ability to regulate the expression of key leukocyte adhesion molecules, on both leukocytes and end
216 capillaries enlarged into venules expressing leukocyte adhesion molecules, sprouting angiogenesis and
217 that the phenolic content of RW may modulate leukocyte adhesion molecules, whereas both ethanol and p
218  T cells (Tn) are expanded in mice that lack leukocyte adhesion molecules, which have neutrophilia an
219  associated with endothelial upregulation of leukocyte adhesion molecules, which persist even after i
220                               Transient mild leukocyte adhesion occurred in mCMV-ND venules at 7 and
221 or IL-6, and their retinas were analyzed for leukocyte adhesion or for the expression and localizatio
222 a(2+)]i in endothelial cells does not affect leukocyte adhesion or locomotion but selectively blocks
223 strains lacking Pra1p were unable to support leukocyte adhesion or migration.
224                               No significant leukocyte adhesion or rolling, nor changes in capillary
225 ndogenous inhibitors, as well as the role of leukocyte adhesion-promoting molecules) has provided new
226                       Mechanisms controlling leukocyte adhesion, propulsion and directional migration
227 matory markers, neutrophil chemoattractants, leukocyte adhesion proteins, and matrix metalloproteases
228 coli strains, targets a broad range of human leukocyte adhesion proteins.
229 form to the TNFalpha-induced upregulation of leukocyte adhesion proteins.
230     We report that in addition to supporting leukocyte adhesion, provision of specific substrate to V
231 shear stress responses, and TNFalpha-induced leukocyte adhesion rates characteristic of arterial endo
232 y increases in endothelial expression of the leukocyte adhesion receptor E-selectin and in microvascu
233 xide production, and by direct modulation of leukocyte adhesion receptor expression.
234                                    CD44, the leukocyte adhesion receptor for hyaluronan, has been con
235                                          The leukocyte adhesion receptor L-selectin forms bonds with
236 Pases are activated by engagement of several leukocyte adhesion receptors and contribute to both earl
237 lls (LECs), leading to expression of the key leukocyte adhesion receptors intercellular adhesion mole
238                       The selectin family of leukocyte adhesion receptors is principally recognized f
239 G, interact with ICAM-1 and are recruited to leukocyte adhesion sites.
240 tivity C-reactive protein, osteopontin), and leukocyte adhesion (soluble vascular cell adhesion molec
241  addressed this problem, two key features of leukocyte adhesion, such as cell deformation and rolling
242 n endothelial cells and, in turn, to augment leukocyte adhesion, thus exacerbating S1P-mediated proad
243 chronic inflammatory disease associated with leukocyte adhesion to and extravasation through vascular
244                                              Leukocyte adhesion to brain endothelial cells, the blood
245 a inhibition in vivo substantially decreased leukocyte adhesion to brain endothelium under inflammato
246                 PolyI:C-induced, HA-mediated leukocyte adhesion to colon SMCs from STAT1-null mice wa
247                                              Leukocyte adhesion to endothelium and platelets plays an
248 treated SS RBCs induced activation of murine leukocyte adhesion to endothelium as well.
249 ich SS RBCs act via LW and CD44 to stimulate leukocyte adhesion to endothelium, and suggest that RBC
250 so purified LFA-1, an integrin that mediates leukocyte adhesion to endothelium.
251 expression in renal vascular endothelium and leukocyte adhesion to endothelium.
252 is study investigated the regulation of firm leukocyte adhesion to human brain endothelium by two dif
253 igation rapidly activated integrin-dependent leukocyte adhesion to immobilized ICAM-1 and fibronectin
254  in multiple leukocyte populations, impaired leukocyte adhesion to inflamed vessels, and accumulation
255                                              Leukocyte adhesion to P-selectin on activated platelets
256 ation of AnxA1(2-50) markedly reduced (>60%) leukocyte adhesion to postcapillary venules in wild type
257 e blockade of NAD(P)H oxidase in turn blocks leukocyte adhesion to retinal vessels during diabetes an
258 tent with these findings, we found increased leukocyte adhesion to sdc-1 knock-out endothelial cells
259 rst description of a mechanism that prevents leukocyte adhesion to the endothelium by a parasite and
260                 Furthermore, IL-35 inhibited leukocyte adhesion to the endothelium in the vessels of
261                                     However, leukocyte adhesion to the fibrin clot can be detrimental
262                                  It promotes leukocyte adhesion to the liver in vivo and drives lymph
263 dose-dependently inhibited TNF-alpha-induced leukocyte adhesion to the murine cremasteric arterioles
264 ed increases in IL-6 mRNA and protein and in leukocyte adhesion to the retinal vessels.
265     ET-1-treated PbN-infected mice displayed leukocyte adhesion to the vascular endothelia and petech
266 ology of diabetic retinopathy is mediated by leukocyte adhesion to the vascular endothelium of the di
267                                Inhibition of leukocyte adhesion to the vascular endothelium represent
268                                              Leukocyte adhesion to the vascular wall is a critical ea
269                                Intriguingly, leukocyte adhesion to the vasculature is significantly i
270 t limits endothelial activation and prevents leukocyte adhesion to the vessel wall.
271 dition of heavy chains to HA cables enhanced leukocyte adhesion to these cables, but it also had seve
272                       Finally, HDL inhibited leukocyte adhesion to TNFalpha-stimulated ECs isolated f
273 to nude mice promotes both SS RBC and murine leukocyte adhesion to vascular endothelium in vivo.
274 s (such as VCAM-1 the ligand for VLA-4), and leukocyte adhesion to vascular endothelium.
275  been implicated in the multistep process of leukocyte adhesion to vascular endothelium.
276                      Human polymorphonuclear leukocytes adhesion to endothelial cells during the earl
277 nd hemorrhagic shock did not further amplify leukocytes adhesion to intestinal venules compared with
278 oxemia and hemorrhagic shock did not amplify leukocytes adhesion to the endothelium further than eith
279     Furthermore, hemorrhagic shock triggered leukocytes adhesion to the venular endothelium to the sa
280                     Hypoxemia also triggered leukocytes adhesion to the venular endothelium.
281 y for rapid activation of integrin-dependent leukocyte adhesion, triggered by toll-like receptor (TLR
282 the essential role for TXNIP in mediating EC-leukocyte adhesion under d-flow, as well as define a nov
283 c IgG and assembled immune complexes amplify leukocyte adhesion under dynamic conditions.
284  core-1/Galbeta1,3GalNAc glycan may regulate leukocyte adhesion under fluid shear.
285 llular adhesion molecule-1 (ICAM-1) mediates leukocyte adhesion under force.
286 ppressed the LPS-induced increase in retinal leukocyte adhesion; vascular leakage; NF-kappaB, HIF-1al
287 r contributed to activation of NF-kappaB and leukocyte adhesion via the EC induction of intercellular
288 glycosylation, sialyl Lewis-X formation, and leukocyte adhesion via the selectins.
289                                              Leukocyte adhesion was assayed by concanavalin A labelin
290                                              Leukocyte adhesion was assessed by labeling adherent leu
291                                 Platelet and leukocyte adhesion was assessed in cortical venules with
292                                 Platelet and leukocyte adhesion was elevated and blood/brain barrier
293                                              Leukocyte adhesion was evaluated by chemotaxis and in vi
294 ferase FUT9 played an important role because leukocyte adhesion was reduced by 50-60% in FUT9-HL-60,
295 tically upregulated at the onset of hypoxia, leukocyte adhesion was unaffected.
296 expression on retinal neovascularization and leukocyte adhesion were abolished in mice lacking TNF-al
297                                   Similarly, leukocyte adhesions were significantly lower, with eleva
298 hyaluronan production or hyaluronan-mediated leukocyte adhesion when treated with the viral mimic pol
299 rolling phase resulting in the transition to leukocyte adhesion, which appear to contribute to chemok
300 tion of CatG specifically abrogated arterial leukocyte adhesion without affecting myeloid cell adhesi

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