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1 but under inflammatory conditions acts as a leukocyte adhesion molecule.
2 thelial cells to express at least one of the leukocyte adhesion molecules.
4 (TNF) activates transcription of endothelial leukocyte adhesion molecule-1 (CD62E) in endothelial cel
5 ll adhesion molecule-1 (VCAM-1), endothelial-leukocyte adhesion molecule-1 (E-selectin), and intercel
6 dy 7A9 binds specifically to the endothelial leukocyte adhesion molecule-1 (E-selectin), inhibiting t
7 on of adhesion molecules such as endothelial-leukocyte adhesion molecule-1 (E-selectin), intercellula
9 hesion molecule (ICAM)-1 but not endothelial leukocyte adhesion molecule-1 expression on hepatic endo
11 ar cell adhesion molecule-1, and endothelial leukocyte adhesion molecule-1 in a time- and dose-depend
12 ar cell adhesion molecule-1, and endothelial leukocyte adhesion molecule-1 mRNA were detected by reve
13 appaB and an element, NF-ELAM-1 (endothelial leukocyte adhesion molecule-1), constitutively occupied
14 observed elevated expression of endothelial leukocyte adhesion molecule-1, a human glaucoma marker,
15 alpha (TNFalpha), tissue factor, endothelial leukocyte adhesion molecule-1, and vascular cell adhesio
18 dose of natalizumab, an antibody against the leukocyte adhesion molecule alpha4 integrin, in patients
20 tumor necrosis factor-induced expression of leukocyte adhesion molecules and procoagulant tissue fac
24 proinflammatory cytokines and expression of leukocyte adhesion molecules by endothelial cells in res
26 aB-dependent reporter construct, endothelial leukocyte adhesion molecule CD25 (the 3E10 clone), were
30 this report, we have determined that the EC-leukocyte adhesion molecule E-selectin is a key target f
31 may "superinduce" expression of an important leukocyte adhesion molecule, E-selectin (ELAM-1, CD62E).
33 ulatory role in cytokine induction of the EC-leukocyte adhesion molecules (ELAM) E-selectin and vascu
35 integrin alphaMbeta2 (Mac-1, CD11bCD18) are leukocyte adhesion molecules essential for innate immuni
37 eta2 integrins (CD11/CD18) are heterodimeric leukocyte adhesion molecules expressed on hematopoietic
38 irectly suppressed endothelial activation of leukocyte adhesion molecule expression and inflammation.
40 anges with local expression of cytokines and leukocyte adhesion molecules in mice with disseminated c
42 roke, and to study the potential role of the leukocyte adhesion molecule intercellular adhesion molec
43 ere to investigate the immunostaining of the leukocyte adhesion molecules intercellular adhesion mole
44 s that inhibit the expression of endothelial-leukocyte adhesion molecules intercellular adhesion mole
53 udy, we found that TXA(2) mimetics stimulate leukocyte adhesion molecule (LAM) expression on endothel
54 ncreased endothelial cell (EC) expression of leukocyte adhesion molecules (LAMs), which mediate monoc
57 E-selectin, an endothelial-cell-specific leukocyte adhesion molecule, may also function in angiog
58 whether the CD18 family (beta2-integrin) of leukocyte adhesion molecules mediates initial passage of
59 ulation of the endothelial cell cytokine and leukocyte adhesion molecule mRNAs in response to C. albi
63 t, Rho GTPases affect the expression of some leukocyte adhesion molecules on endothelial cells, such
64 ir ability to regulate the expression of key leukocyte adhesion molecules, on both leukocytes and end
65 ample, sulfation of tyrosine residues in the leukocyte adhesion molecule P-selectin glycoprotein liga
66 ical surface, where JAM-A played a role as a leukocyte adhesion molecule participating in transendoth
67 h AMD, there is increased immunostaining for leukocyte adhesion molecules, particularly in the periph
72 capillaries enlarged into venules expressing leukocyte adhesion molecules, sprouting angiogenesis and
73 esion, and extravasation by up-regulation of leukocyte adhesion molecules such as E-selectin and P-se
75 ription or surface expression of endothelial leukocyte adhesion molecules that are readily induced by
76 e-inducible, NF-kappaB-dependent endothelial-leukocyte adhesion molecules that participate in the leu
77 tigated the role of p75 in TNF-alpha-induced leukocyte adhesion molecules using cultured ECs derived
79 that the phenolic content of RW may modulate leukocyte adhesion molecules, whereas both ethanol and p
80 tress induces expression of endothelial cell leukocyte adhesion molecules, which are centrally import
81 T cells (Tn) are expanded in mice that lack leukocyte adhesion molecules, which have neutrophilia an
82 associated with endothelial upregulation of leukocyte adhesion molecules, which persist even after i
83 lming evidence that blocking the function of leukocyte adhesion molecules would be highly effective i
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