ライフサイエンスコーパス: leukocyte migration

1 d the potential role of ALF in regulation of leukocyte migration.

2 investigate the role of Plg in inflammatory leukocyte migration.

3 several innate immune functions that include leukocyte migration.

4 ed in the regulation of barrier function and leukocyte migration.

5 e for DARC expressed on endothelial cells in leukocyte migration.

6 d proteins with roles in axonal guidance and leukocyte migration.

7 nhibits alpha4 integrin signals that support leukocyte migration.

8 chemokines in the key physiologic process of leukocyte migration.

9 nchanged, suggesting that bilirubin inhibits leukocyte migration.

10 hich in turn provide directional signals for leukocyte migration.

11 quired for immune cell-cell interactions and leukocyte migration.

12 hown to regulate neuronal and CXCR4-mediated leukocyte migration.

13 NF in the mouse air pouch reduced SP-induced leukocyte migration.

14 n pathologic conditions may adversely affect leukocyte migration.

15 several aminoacyl-tRNA synthetases to induce leukocyte migration.

16 large family of cytokines that direct normal leukocyte migration.

17 rins, alpha9beta1 plays an important role in leukocyte migration.

18 ar acidification rate, CD11b expression, and leukocyte migration.

19 omologues are known to be potent signals for leukocyte migration.

20 ts arachidonic acid metabolism, and inhibits leukocyte migration.

21 ligand very late antigen-4 (VLA-4), in such leukocyte migration.

22 ignaling role for these molecules in the CNS leukocyte migration.

23 rin expressed on leukocytes, is important in leukocyte migration.

24 te in re-establishing vessel integrity after leukocyte migration.

25 pha had no significant affect on the overall leukocyte migration.

26 rgistic regulation of these receptors during leukocyte migration.

27 sion molecules involved in the regulation of leukocyte migration.

28 forming a physical barrier to intravascular leukocyte migration.

29 mma and promotes Gbetagamma signaling during leukocyte migration.

30 e capable of inducing directed intravascular leukocyte migration.

31 es, and assessment of thioglyccolate-induced leukocyte migration.

32 xhibit differences in thioglyccolate-induced leukocyte migration.

33 ways critically involved in transendothelial leukocyte migration.

34 es reported that CB2R signaling also reduces leukocyte migration.

35 crosis factor-alpha in an air-pouch model of leukocyte migration.

36 o control many cellular processes, including leukocyte migration.

37 ulator, in binding Gbetagamma and inhibiting leukocyte migration.

38 for the efficacy of Gbetagamma signaling and leukocyte migration.

39 Leukocyte migration across endothelial cell borders (par

40 ix and cell death, in addition to regulating leukocyte migration across extracellular matrix barriers

41 Chemokines may play a role in leukocyte migration across the blood-brain barrier (BBB)

42 he pathogenesis of neuroinflammation, namely leukocyte migration across the blood-brain barrier.

43 Leukocyte migration across the endothelial lining is a c

44 The basic route and mechanisms for leukocyte migration across the endothelium remain poorly

45 alpha4 integrins are important mediators of leukocyte migration across vascular endothelium.

46 tibility complex (MHC) protein function; (2) leukocyte migration, activation and cytokine responses;

47 nervous system (CNS)-specific mechanisms of leukocyte migration, activation, and MV clearance.

48 es are small, soluble proteins that regulate leukocyte migration, adhesion, and proliferation.

49 ent zebrafish larvae for in vivo analysis of leukocyte migration after morpholino knockdown of FAN.

50 Leukocyte migration analysis in vivo showed that Acat1(-

51 incipal cell adhesion receptors that mediate leukocyte migration and activation in the immune system.

52 Leukocyte migration and activation is orchestrated by ch

53 In addition to their role as regulators of leukocyte migration and activation, chemokines and their

54 s to the lung, and appropriate regulation of leukocyte migration and adhesion is integral to this pro

55 gulation of CXCL14-a chemokine that controls leukocyte migration and angiogenesis, and whose expressi

56 adhesion molecule 1 (PECAM-1) is involved in leukocyte migration and angiogenesis, which are key comp

57 Chemokines are the principal regulators of leukocyte migration and are essential for initiation and

58 Chemokines and other chemoattractants direct leukocyte migration and are essential for the developmen

59 Chemokines are the principal regulators of leukocyte migration and are essential in the initiation

60 or near genes involved in cellular adhesion, leukocyte migration and atherosclerosis (PECAM1, rs18676

61 e important adhesion molecules necessary for leukocyte migration and cellular interactions.

62 apeutic approach to treat diseases involving leukocyte migration and chemotactic factors.

63 The role of leukocyte migration and chimerism in organ allograft acc

64 P or MMP-14) is a collagenase that is key in leukocyte migration and collagen destruction.

65 ed from eicosapentaenoic acid that regulates leukocyte migration and enhances macrophage phagocytosis

66 of soluble mediators important in regulating leukocyte migration and extravasation, including the CXC

67 How leukocyte migration and function are coordinated is unkn

68 degrading Galpha(i2), Nef directly subverts leukocyte migration and homing.

69 D18 is a key adhesion receptor that mediates leukocyte migration and immune functions.

70 ell (plt) mutation leads to abnormalities in leukocyte migration and immune response.

71 es a basis for their severe abnormalities in leukocyte migration and immune response.

72 NO-np modified leukocyte migration and increased tumor growth factor-be

73 ity of a neuronal guidance cue in regulating leukocyte migration and indicate that there may be a gen

74 n RCD motif have shown promise in modulating leukocyte migration and inflammation presumably by block

75 restricted signaling molecule that regulates leukocyte migration and integrin-mediated adhesion.

76 s and circuitry, including lipid metabolism, leukocyte migration and olfaction.

77 well as angiogenesis, vascular permeability, leukocyte migration and platelet formation in vivo.

78 t of morphine, a known immunosuppressant, on leukocyte migration and recruitment to conditioned media

79 nt inhibition may be effective in preventing leukocyte migration and subsequent local and remote orga

80 nsplantation is directly related to enhanced leukocyte migration and that early islet graft survival

81 first time morphine's inhibitory effects on leukocyte migration and their ability to transmigrate ac

82 rs an avenue for precise characterization of leukocyte migration and therapeutic modulators.

83 alpha(4) integrins play a pivotal role in leukocyte migration and tissue-specific homing.

84 ory pathway based on its ability to modulate leukocyte migration and to inhibit the expression of inf

85 Leukocyte migration and trafficking is dynamically regul

86 f E-selectin receptor/ligand interactions in leukocyte migration and vascular pathology.

87 a beta-arrestin-dependent one that promotes leukocyte migration, and a G-protein/Ca(2+) one that is

88 these QTL, including the glycan degradation, leukocyte migration, and antigen-presenting pathways.

89 otein that costimulates T cells, facilitates leukocyte migration, and inhibits macrophage scavenger f

90 1, a chemokine that regulates cerebellar and leukocyte migration, and its receptor CXCR4 are expresse

91 lar injury, operating after transendothelial leukocyte migration, and presumably binding to alternate

92 meability, endothelial inflammatory markers, leukocyte migration, and susceptibility to LPS-induced d

93 t perceptions concerning the role of PI3K in leukocyte migration are based predominantly around evide

94 with lymphocytes and its in-depth effects on leukocyte migration are poorly understood.

95 Chemokines play a pivotal role in regulating leukocyte migration as well as other biological function

96 glycollate model of peritoneal inflammation, leukocyte migration at 72 hours increased significantly

97 lly related, low m.w. proteins that regulate leukocyte migration both in vitro and in vivo.

98 teins do not only serve as a stop signal for leukocyte migration but also can propagate the extravasa

99 Previously, LEC was shown to induce leukocyte migration but the responsible signaling recept

100 issue hydration, release of collagenase, and leukocyte migration, but their roles in cervical ripenin

101 helial migration, and that polymorphonuclear leukocyte migration can occur without permeability alter

102 This ligand bias correlates with changes in leukocyte migration, consistent with different mechanism

103 In addition to its role in leukocyte migration, CXCL10 inhibits the angiogenic func

104 iated with NK cytotoxicity, Ag presentation, leukocyte migration, cytokine activity, protein kinases,

105 a key role in immune homeostasis regulating leukocyte migration, differentiation, and function.

106 es, shear stress is a contributing factor to leukocyte migration directionality.

107 y contribute to neuropil ECM degradation and leukocyte migration during HAD.

108 on, in embryonic tissue morphogenesis and in leukocyte migration during inflammation.

109 r of the gelatinase family of MMPs, mediates leukocyte migration during inflammation.

110 oduction by endothelial cells and suppresses leukocyte migration during inflammation.

111 spases that regulate cytokine production and leukocyte migration during pathogen infection.

112 tokines studied to date in its regulation of leukocyte migration during primary listeriosis.

113 The effect of strawberry extract and P3G, on leukocyte migration, exudation levels and many inflammat

114 Leukocyte migration from a hemopoietic pool across marro

115 Leukocyte migration from blood into lymphoid and non-lym

116 Leukocyte migration from bloodstream to tissue requires

117 Leukocyte migration from bloodstream to tissue requires

118 ivo lung perfusion (EVLP) to study passenger leukocyte migration from donor lungs into the recipient

119 Leukocyte migration from the bloodstream to a site of in

120 ization of MCP-1 significantly reduced total leukocyte migration (>50% reduction), whereas neutraliza

121 of small, homologous proteins that regulate leukocyte migration, hemopoiesis, and HIV-1 absorption.

122 ing adhesion dynamics, with implications for leukocyte migration, immune responses and potentially pa

123 ial cells and/or to induce polymorphonuclear leukocyte migration in a tissue culture model of mammali

124 own, and impaired both monocyte adhesion and leukocyte migration in a transwell system (p < 0.0001).

125 a causative role for negative regulators of leukocyte migration in chronic inflammation.

126 To better understand the mechanism of leukocyte migration in complex environments, model extra

127 nes CCL2 and CX3CL1 probably are involved in leukocyte migration in diabetic nephropathy.

128 oteinase 9 (MMP-9) is a critical mediator of leukocyte migration in hepatic ischemia/reperfusion (I/R

129 Whether this motility pattern applies for leukocyte migration in inflamed tissue is still unknown.

130 ns plays a central role in the regulation of leukocyte migration in inflammatory responses.

131 te/endothelial adhesion and transendothelial leukocyte migration in inflammatory states.

132 undly inhibited MMP-9 activity and depressed leukocyte migration in livers after I/R injury.

133 Evaluation of fluorescently labeled leukocyte migration in mice revealed that DCs travel via

134 lex, hyper-IL-6, and to effectively modulate leukocyte migration in murine acute peritonitis.

135 in human and its mouse homolog mFPR2 mediate leukocyte migration in response to agonists associated w

136 formylpeptide receptor (FPR), which mediates leukocyte migration in response to bacterial and host-de

137 To study leukocyte migration in response to surface-bound chemoki

138 erum concentration of molecules that control leukocyte migration in serial samples from 34 patients f

139 a metalloproteinase (ADAM) 10 and ADAM17 for leukocyte migration in vitro and in a murine model of ac

140 cell-cell interactions necessary to sustain leukocyte migration in vitro and tissue infiltration in

141 grin CD18 has been shown to be important for leukocyte migration in vitro.

142 d cholinergic agonists significantly blocked leukocyte migration in vivo.

143 fects of PECAM-1 and alphavbeta3 blockade on leukocyte migration in vivo.

144 vivo imaging technique for visualization of leukocyte migration into and out of corneal stroma, we s

145 The chemokine receptor CCR7 drives leukocyte migration into and within lymph nodes (LNs).

146 ta (IL-1beta), to predict chemotactic driven leukocyte migration into and within the artery wall.

147 e studies suggest that autoantigens initiate leukocyte migration into damaged and inflamed tissue tha

148 Chemokine receptors mediate leukocyte migration into inflamed rheumatoid arthritis (

149 ttractant receptors synergistically regulate leukocyte migration into lymphoid tissues and sites of i

150 otein receptor mediates the initial steps of leukocyte migration into secondary lymphoid organs and s

151 n and ICAM-1 expression dramatically reduced leukocyte migration into sites of inflammation beyond wh

152 Leukocyte migration into sites of inflammation involves

153 ammatory response involved polymorphonuclear leukocyte migration into the alveolar space and the accu

154 An analysis of leukocyte migration into the brain revealed that adminis

155 activity with brain capillaries and to block leukocyte migration into the brain was used to identify

156 llowing infection and for the restriction of leukocyte migration into the brain.

157 model of MS, lovastatin treatment inhibited leukocyte migration into the CNS and significantly atten

158 CCL7, in directing monocyte mobilization and leukocyte migration into the CNS is unclear.

159 Selective blockade of leukocyte migration into the gut is a promising strategy

160 f HIFs that myeloid HIFs are dispensable for leukocyte migration into the inflamed eye.

161 0) = 0.75 nM), suggesting that inhibition of leukocyte migration into the knee joint is a likely mech

162 with rheumatoid arthritis, and they promote leukocyte migration into the synovial tissue.

163 ctive absence of Mac-1 impairs transplatelet leukocyte migration into the vessel wall, reducing leuko

164 Leukocyte migration into tissues is characteristic of in

165 t collagen-binding integrins are involved in leukocyte migration into tissues.

166 cytes to vascular endothelium is crucial for leukocyte migration into tissues.

167 ed that poly(I:C) induced T/B cell-dependent leukocyte migration into white pulp regions.

168 conditions, as well as in various diseases, leukocyte migration is a crucial issue for the immune sy

169 Leukocyte migration is critical for the infiltration of

170 Chemokine-directed leukocyte migration is crucial for effective immune and

171 Efficient leukocyte migration is important for an effective host r

172 The control of leukocyte migration is of key interest during conditions

173 Leukocyte migration is the hallmark of inflammation, and

174 The multistep sequence leading to leukocyte migration is thought to be locally regulated a

175 Thus, with regard to leukocyte migration, leukocyte-expressed ADAM10 but not

176 tor agonist can inhibit the normal vectorial leukocyte migration mediated by chemokines.

177 y ligands such as VCAM-1 markedly stimulates leukocyte migration mediated by LFA-1 (integrin alpha(L)

178 involve vessel infiltration by inflammatory leukocytes, migration of medial vascular smooth muscle c

179 onsiveness was not associated with increased leukocyte migration or mucous production in the lung but

180 production, intracellular antioxidation, and leukocyte migration plus genes for proinflammatory cytok

181 (< 40-microns diameter), whereas most of the leukocyte migration (predominantly neutrophils) occurred

182 This blockade in the leukocyte migration process is consistent with the obser

183 e conventional multistep paradigm holds that leukocyte migration represents a cascade of events, init

184 ted with CFTR(172) corrected the exaggerated leukocyte migration seen in these animals.

185 s also displayed defective polymorphonuclear leukocyte migration, suggesting mast cells as one source

186 These studies define a distinct process of leukocyte migration that is initiated by homotypic adhes

187 both remodeling of extracellular matrix and leukocyte migration, their influence on the outcome of i

188 Leukocyte migration through activated venular walls is a

189 cultured cells revealed that sVAP-1 promotes leukocyte migration through catalytic generation of ROS,

190 nd suggested a role for the DDR1a isoform in leukocyte migration through extracellular matrix.

191 f the ImmunoCloak also significantly reduced leukocyte migration through the endothelial cell layer b

192 Leukocyte migration through the endothelial cell wall in

193 The Slit protein guides neuronal and leukocyte migration through the transmembrane receptor R

194 s most likely induce cytokine production and leukocyte migration through TLR7 signaling.

195 The mechanism of leukocyte migration through venular walls in vivo is lar

196 Neutralization of CCL5 and CXCL10 decreases leukocyte migration to areas of infection by 70%.

197 h some, but not all, chemokines and controls leukocyte migration to inflammatory sites.

198 Thus, MCK-1/MCK-2 appears to promote host leukocyte migration to initial sites of infection and ma

199 kocyte activation and may directly influence leukocyte migration to peripheral lymphoid tissues or to

200 G-protein-coupled receptor that facilitates leukocyte migration to regional lymph nodes.

201 uronan substrates and has been implicated in leukocyte migration to sites of inflammation.

202 cross lung grafts, responded to infection by leukocyte migration to small airways and alveoli of the

203 r investigating the mechanisms that regulate leukocyte migration to the joint in systemic models of R

204 dity, survival, clearance of bacteremia, and leukocyte migration to the peritoneal cavity and organs

205 Intravenous 4F-GalNAc infusion reduced leukocyte migration to the peritoneum in a murine model

206 compound was highly effective at inhibiting leukocyte migration toward CypA in vitro as well as in t

207 Leukocyte migration towards injury sites is directed by

208 Chemokines, 8 kDa proteins implicated in leukocyte migration via oligomerization, bind to glycosa

209 the bell-shaped concentration dependence of leukocyte migration was shown to arise from the agonist

210 study the in vivo effects of IP-10 on human leukocyte migration, we then examined the ability of rec

211 a new experimental concept for understanding leukocyte migration within the wounded cornea.

212 recruitment of this membrane to the zones of leukocyte migration, without affecting the constitutive