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1 the opening of gaps before the initiation of leukocyte transmigration.
2 E-cadherin's role as a negative regulator of leukocyte transmigration.
3 at Esm1(KO) mice displayed a 40% decrease in leukocyte transmigration.
4 impairs both docking structure formation and leukocyte transmigration.
5 howed that endothelial RhoG and SGEF control leukocyte transmigration.
6 the involvement of VLDLR in fibrin-dependent leukocyte transmigration.
7 o the effects of shear flow and cytokines on leukocyte transmigration.
8 phology and expression patterns of ICAM-1 in leukocyte transmigration.
9 nvergences are optimally equipped to support leukocyte transmigration.
10 iated with, but TspanC8s remain unstudied in leukocyte transmigration.
11 uld in turn have functional consequences for leukocyte transmigration.
12 ars capable of mediating PECAM-1-independent leukocyte transmigration.
13 iation is required for ICAM-1 clustering and leukocyte transmigration.
14 transmit outside-in signals that facilitate leukocyte transmigration.
15 bling ICAM-1 to form clusters and facilitate leukocyte transmigration.
16 chemokines and other potential regulators of leukocyte transmigration.
17 ession of endothelial adhesion molecules and leukocyte transmigration.
18 on quiescent endothelial cells only mediates leukocyte transmigration.
19 behavior of these adhesion molecules during leukocyte transmigration.
20 othelial cell interactions that occur during leukocyte transmigration.
21 ted in the regulation of tight junctions and leukocyte transmigration.
22 al cells with anti-PECAM-1 antibody inhibits leukocyte transmigration.
23 Gaps rapidly resealed within 5 min after leukocyte transmigration.
24 onoclonal antibodies to JAM failed to reduce leukocyte transmigration.
25 netheless, be a physiological process during leukocyte transmigration.
26 ecules act as counter-receptors in mediating leukocyte transmigration.
27 helial cell migration, and polymorphonuclear leukocyte transmigration.
28 ion of the VE-cadherin complex occurs during leukocyte transmigration.
29 orting a role for astrocytes in facilitating leukocyte transmigration.
30 understanding of the role of EC junctions in leukocyte transmigration.
31 eby controlling endothelial permeability and leukocyte transmigration.
33 ral cell adhesion molecule, implicates it in leukocyte transmigration across the blood-brain barrier.
36 levels of MMP-9 activity, which facilitates leukocyte transmigration across vascular barriers in hep
37 inase-9 (MMP-9) synthesis, which facilitates leukocyte transmigration across vascular barriers in liv
40 ce that VE-cad gap formation is required for leukocyte transmigration and identify p120 as a critical
41 on to show that endothelial Nogo-B regulates leukocyte transmigration and intercellular adhesion mole
45 ting that they contribute to control of host leukocyte transmigration and trafficking during viral in
47 g extracellular domain of endoglin, enhanced leukocyte transmigration, and this increased motility wa
48 in disease was paralleled by a reduction in leukocyte transmigration, as demonstrated by intravital
51 ribution and clustering appear necessary for leukocyte transmigration, but the mechanisms controlling
52 rane molecular scissor that is implicated in leukocyte transmigration by proteolytically cleaving its
53 udy we compared the pulmonary thrombosis and leukocyte transmigration caused by GOX targeting to the
54 contrast to wild-type mice, fibrin-dependent leukocyte transmigration does not occur in such mice.
55 f the animal, and is likely to contribute to leukocyte transmigration events important to intestinal
57 Both dexamethasone and bevacizumab inhibited leukocyte transmigration from angiogenic vessels; howeve
60 al microscopy, there was a 43% inhibition of leukocyte transmigration in mesenteric venules in respon
61 mAb also exhibited a selective reduction in leukocyte transmigration in response to IL-1beta while a
62 AR/nectin molecules mediate virus uptake and leukocyte transmigration in strikingly similar manners.
63 ue from other adhesion molecules involved in leukocyte transmigration in that its adhesiveness appear
66 nd CXCL5 and led to a functional increase in leukocyte transmigration in vivo and CXCR2-dependent neu
67 th the CXCR1/2 antagonist SCH527123 inhibits leukocyte transmigration into lung and subsequently reve
68 scular adhesion molecule-1, which facilitate leukocyte transmigration into the lymphatic vessels.
76 1), a transmembrane glycoprotein involved in leukocyte transmigration, represents a good target for e
77 related endothelial-dependent mechanisms for leukocyte transmigration that involve alterations in lat
78 ause PECAM-1 and JAM have been implicated in leukocyte transmigration, the observed redistribution by
81 hat the cytoplasmic tail of ICAM-1 regulates leukocyte transmigration through MT1-MMP interaction.
83 ich subsequently leads to firm adherence and leukocyte transmigration through the vessel wall into th
84 lood vessels, constitute barriers regulating leukocytes transmigration to the site of inflammation.
85 e operative to finely tune polymorphonuclear leukocytes transmigration to the site of inflammation.
89 nolayers, a commonly used model for studying leukocyte transmigration, were characterized using elect
90 n molecules in periodontitis is regulated by leukocyte transmigration, whereas the neutrophilic antim
91 sion, transporter activity, plasma membrane, leukocyte transmigration, Wnt signaling pathways and ang
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