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1  mRNA levels occurs during activation of the leukocytic cell line K562.
2 he target cell specificity of LKT for bovine leukocytic cells was retained by the SDS-PAGE LKT, and i
3 dermal keratinocytes, dermal fibroblasts and leukocytic cells.
4 for the extent (diffuse vs focal), score and leukocytic composition in relation to DSA binding streng
5                                   CEACAM1 on leukocytic, endothelial, and epithelial cells functions
6 and peritonitis at ng doses, reducing 40-80% leukocytic exudates.
7 ased shRNA delivery workflow to create human leukocytic HL-60 cell lines that lack up to three glycoT
8             FACS analysis enumerated corneal leukocytic infiltrate and characterized infiltrate pheno
9                                          The leukocytic infiltrate appears to be confined to the adve
10 Immunohistochemistry was used to compare the leukocytic infiltrate between rats and mice.
11  keratitis sequelae, possessed a significant leukocytic infiltrate composed primarily of CD4(+) T cel
12 lung lavage fluid revealed the presence of a leukocytic infiltrate consisting of lymphocytes, neutrop
13 d corneas without HSK exhibited a pronounced leukocytic infiltrate containing a significantly higher
14 in-activated cell sorter analysis of retinal leukocytic infiltrate indicated that there was a reduced
15  HSV infection was accompanied by an intense leukocytic infiltrate into the ophthalmic portion of the
16                       Immunohistology of the leukocytic infiltrate of livers from treated mice reveal
17  all the non-aneurysmal aortas had a similar leukocytic infiltrate that spared the inner media.
18 L6/hi), tumor nodules were removed and their leukocytic infiltrate was analyzed by FACS with Ab marke
19                                          The leukocytic infiltrate was composed of aggregates of B220
20 exposure, as 250 ng ivt LPS produced a brisk leukocytic infiltrate whereas ip injection of 100 mug LP
21                                          The leukocytic infiltrate within the draining lymph node and
22                                  A prominent leukocytic infiltrate within the lamina propria was a co
23 ased on corneal opacity, neovascularization, leukocytic infiltrate, and cytokine/chemokine levels.
24 om untreated mice had no evidence of injury, leukocytic infiltrate, or endothelial cell activation up
25 e accepted renal allografts have a prominent leukocytic infiltrate, suggesting an ongoing, local immu
26 y in the presence of a substantial secondary leukocytic infiltrate, that activated CD4+ T cells, whil
27  degree of fibroplasia, vascularization, and leukocytic infiltrate.
28 ysfunction and the presence of a mononuclear leukocytic infiltrate; however, the presence of a modest
29 cimens harvested after reoperation developed leukocytic infiltrates and displayed diminished in vitro
30                                    Extensive leukocytic infiltrates and necrosis at the decidual-plac
31 ed significantly greater innate and adaptive leukocytic infiltrates compared with uninfected grafts.
32  infection, the Tlr5(-/-) mice had increased leukocytic infiltrates in the alveolar and peribronchial
33  wk of age still contained variably elevated leukocytic infiltrates in their islets when examined at
34  of CCL21 transgenic mice revealed scattered leukocytic infiltrates that consisted primarily of neutr
35                                   Muscularis leukocytic infiltrates were characterized.
36 mage with increased interstitial mononuclear leukocytic infiltrates, and had significantly increased
37 alyses prevent comprehensive descriptions of leukocytic infiltrates, as well as evaluation of the fun
38 -2-deficient MRL-Faslpr mice did not develop leukocytic infiltrates, elevated serum IgG and isotypes
39 munoprivilege that manifests as more intense leukocytic infiltrates, loss of VSMCs, and destruction o
40 rat aortic sprouting assay in the absence of leukocytic infiltrates, we ruled out the possibility tha
41 ot reveal any significant differences in the leukocytic infiltrates.
42  small blood vessels that was accompanied by leukocytic infiltrates.
43 esion molecules, which may lead to increased leukocytic infiltration and tissue damage.
44 PLs was local to the tumor site and elicited leukocytic infiltration and tumor invasion.
45                                              Leukocytic infiltration at different time points was qua
46 ccompanied by a profound decrease in corneal leukocytic infiltration by 44.6% at day 1 (P < 0.003), 7
47 ice demonstrated a striking reduction in the leukocytic infiltration early on in the infection, sugge
48                 The microvascular damage and leukocytic infiltration elicited by TNF were enhanced by
49                      We observed a prominent leukocytic infiltration even around the lowest grade pre
50                                              Leukocytic infiltration in bacterial lipopolysaccharide
51 diesel exhaust (DE) for 1 h induced a marked leukocytic infiltration in the airways of healthy human
52                                  The ensuing leukocytic infiltration in the islets resulted in destru
53 r joint swelling, functional disability, and leukocytic infiltration in the joints than serotype A-in
54                   These results suggest that leukocytic infiltration in this model of inflammatory co
55 d by dysregulated epithelial cell growth and leukocytic infiltration into the lamina propria of the l
56                                              Leukocytic infiltration occurred before manifestation of
57 d-type hypersensitivity (DTH) responses, and leukocytic infiltration of grafts were measured.
58      Immunocytochemical analysis showed that leukocytic infiltration of inner ear tissues coincided w
59 , widespread mucus occlusion of the airways, leukocytic infiltration of the airway tissue and broncho
60                                              Leukocytic infiltration of the pancreatic islets by auto
61 es is the clinical manifestation of aberrant leukocytic infiltration of the pancreatic islets; it is
62  graft arteriosclerosis are characterized by leukocytic infiltration of the vessel wall that spares t
63                 One type of host response is leukocytic infiltration that may develop into chronic in
64 lso in the lung interstitium in the sites of leukocytic infiltration that was not observed in the sal
65 ilarly, in ICAM-1 knockout mice, the corneal leukocytic infiltration was 50.3%, 52.9%, and 36.4%, com
66                                 An increased leukocytic infiltration was also closely associated with
67                                           No leukocytic infiltration was detected at 6 h.
68              Impact on skin erythema, dermal leukocytic infiltration, and concentrations of proinflam
69 ns to reduce early graft lipid peroxidation, leukocytic infiltration, and cytokine production as well
70  capillary thickening, luminal constriction, leukocytic infiltration, mesangial deposits, and changes
71 g thyroid tumors into naive mice resulted in leukocytic infiltration, tumor rejection, and induction
72 ritis) revealed synovial hyperplasia without leukocytic infiltration.
73 ould affect adhesion molecule expression and leukocytic infiltration.
74 ular endothelial activation and interstitial leukocytic infiltration.
75 te intimal hyperplasia and mild perivascular leukocytic infiltration.
76 nges and prominent vascular and perivascular leukocytic infiltration.
77                                              Leukocytic inflammation can be limited by inhibiting sel
78                          However,the role of leukocytic inflammation in promoting or hindering tissue
79 te postoperative ileus is mediated through a leukocytic inflammatory response within the intestinal m
80 ace protects the placenta against a maternal leukocytic influx that reduces fertility.
81 barrier followed by pyrexia and a pronounced leukocytic influx.
82 cient mice are not completely protected from leukocytic invasion.
83 re was no inflammatory response as judged by leukocytic invasion.
84  which may be characteristic of a persistent leukocytic involvement in areas of chronic inflammation
85 found that mature biofilms were resistant to leukocytic killing and did not trigger reactive oxygen s
86                            To identify which leukocytic lineages may be responsible for OPN expressio
87 1-KIR combined therapy resulted in decreased leukocytic organ infiltration, reduction of serum IFN-ga
88                                              Leukocytic phenotype was analyzed by flow cytometry, and
89  We studied beta1,6-branching [determined by leukocytic phytohemagglutinin (LPHA) lectin-histochemist
90                                         This leukocytic reduction results from suppressed infiltratio
91 -tolerance to the functional, molecular, and leukocytic sequelae of intestinal manipulation.
92 entation of human subjects programs specific leukocytic subsets to show enhanced cytokine expression
93  were harvested for quantitative analysis of leukocytic tissue infiltration and thrombus formation.
94 lized Shwartzman reaction and showed greater leukocytic tissue infiltration than their wild-type litt
95  (P<0.005) and 3.3- to 4.5-fold reduction in leukocytic tissue infiltration.
96                                              Leukocytic TLR-5 signalling mediates upregulation of the

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